All Relations between cocaine and nucleus accumbens

Publication Sentence Publish Date Extraction Date Species
K Grasing, S Ghos. Selegiline prevents long-term changes in dopamine efflux and stress immobility during the second and third weeks of abstinence following opiate withdrawal. Neuropharmacology. vol 37. issue 8. 1999-02-26. PMID:9833630. brain slices obtained from the nucleus accumbens of opiate-withdrawn animals immediately following swim stress testing displayed diminished efflux of tritiated dopamine after two in vitro exposures to cocaine or amphetamine. 1999-02-26 2023-08-12 human
A Y Deutch, M Bubser, C D Youn. Psychostimulant-induced Fos protein expression in the thalamic paraventricular nucleus. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 18. issue 24. 1999-02-25. PMID:9852603. we examined the effects of acute amphetamine and cocaine administration on expression of fos protein in the thalamic paraventricular nucleus (pvt), which provides glutamatergic inputs to the nucleus accumbens and also receives dopaminergic afferents. 1999-02-25 2023-08-12 Not clear
D J Henry, X T Hu, F J Whit. Adaptations in the mesoaccumbens dopamine system resulting from repeated administration of dopamine D1 and D2 receptor-selective agonists: relevance to cocaine sensitization. Psychopharmacology. vol 140. issue 2. 1999-02-25. PMID:9860115. among the adaptations implicated in cocaine sensitization are transient subsensitivity of impulse-regulating da d2 autoreceptors on ventral tegmental area (vta) da neurons leading to hyperactivity of the mesoaccumbens da pathway, and persistently enhanced da d1 receptor responses of nucleus accumbens (nac) neurons. 1999-02-25 2023-08-12 rat
N Thiriet, N Humblot, C Burgun, D Aunis, J Zwille. Cocaine and fluoxetine induce the expression of the hVH-5 gene encoding a MAP kinase phosphatase. Brain research. Molecular brain research. vol 62. issue 2. 1999-02-18. PMID:9813294. injection of cocaine, amphetamine and caffeine induced hvh-5 mrna expression within 40 min in the nucleus accumbens (nac), caudate putamen, frontal cortex and hippocampus, with a maximal effect in the nac. 1999-02-18 2023-08-12 rat
A S Keys, G P Mark, N Emre, C K Meshu. Reduced glutamate immunolabeling in the nucleus accumbens following extended withdrawal from self-administered cocaine. Synapse (New York, N.Y.). vol 30. issue 4. 1999-01-22. PMID:9826231. reduced glutamate immunolabeling in the nucleus accumbens following extended withdrawal from self-administered cocaine. 1999-01-22 2023-08-12 rat
A S Keys, G P Mark, N Emre, C K Meshu. Reduced glutamate immunolabeling in the nucleus accumbens following extended withdrawal from self-administered cocaine. Synapse (New York, N.Y.). vol 30. issue 4. 1999-01-22. PMID:9826231. alterations in the density of gaba and glutamate immunolabeling within nerve terminals in the shell region of the nucleus accumbens were assessed in rats withdrawn from intravenous cocaine exposure. 1999-01-22 2023-08-12 rat
A S Keys, G P Mark, N Emre, C K Meshu. Reduced glutamate immunolabeling in the nucleus accumbens following extended withdrawal from self-administered cocaine. Synapse (New York, N.Y.). vol 30. issue 4. 1999-01-22. PMID:9826231. these results demonstrate that glutamate density is suppressed in the shell region of the nucleus accumbens following withdrawal from 2 weeks of cocaine exposure. 1999-01-22 2023-08-12 rat
J D Stekete. Injection of SCH 23390 into the ventral tegmental area blocks the development of neurochemical but not behavioral sensitization to cocaine. Behavioural pharmacology. vol 9. issue 1. 1998-12-29. PMID:9832950. motor activity and extracellular dopamine concentrations in the nucleus accumbens were monitored on the day the animals received the first of their four daily treatments and/or on the day animals received their cocaine challenge injection. 1998-12-29 2023-08-12 Not clear
D J David, N R Zahniser, B J Hoffer, G A Gerhard. In vivo electrochemical studies of dopamine clearance in subregions of rat nucleus accumbens: differential properties of the core and shell. Experimental neurology. vol 153. issue 2. 1998-11-25. PMID:9784287. in addition, these data indicate that dats are more sensitive to the effects of psychomotor stimulants, such as cocaine, in the shell of the nucleus accumbens. 1998-11-25 2023-08-12 rat
J L Neisewander, R A Fuchs, L E O'Dell, T V Khroya. Effects of SCH-23390 on dopamine D1 receptor occupancy and locomotion produced by intraaccumbens cocaine infusion. Synapse (New York, N.Y.). vol 30. issue 2. 1998-11-16. PMID:9723789. in experiment 2, rats received coinfusion of sch-23390 (0-1 microg/side) and cocaine (0 or 100 microg/side) into the nucleus accumbens (nac). 1998-11-16 2023-08-12 rat
T H Lee, K R Gee, E H Ellinwood, F J Seidle. Altered cocaine potency in the nucleus accumbens following 7-day withdrawal from intermittent but not continuous treatment: voltammetric assessment of dopamine uptake in the rat. Psychopharmacology. vol 137. issue 3. 1998-10-28. PMID:9683008. altered cocaine potency in the nucleus accumbens following 7-day withdrawal from intermittent but not continuous treatment: voltammetric assessment of dopamine uptake in the rat. 1998-10-28 2023-08-12 rat
A V Azaryan, B J Clock, J G Rosenberger, B M Co. Transient upregulation of mu opioid receptor mRNA levels in nucleus accumbens during chronic cocaine administration. Canadian journal of physiology and pharmacology. vol 76. issue 3. 1998-10-22. PMID:9673791. transient upregulation of mu opioid receptor mrna levels in nucleus accumbens during chronic cocaine administration. 1998-10-22 2023-08-12 rat
A V Azaryan, B J Clock, J G Rosenberger, B M Co. Transient upregulation of mu opioid receptor mRNA levels in nucleus accumbens during chronic cocaine administration. Canadian journal of physiology and pharmacology. vol 76. issue 3. 1998-10-22. PMID:9673791. chronic continuous cocaine administration for 3 days has been shown to upregulate the level of mu opioid receptor (mor) mrna in the nucleus accumbens (n. 1998-10-22 2023-08-12 rat
P Di Ciano, C D Blaha, A G Phillip. The relation between dopamine oxidation currents in the nucleus accumbens and conditioned increases in motor activity in rats following repeated administration of d-amphetamine or cocaine. The European journal of neuroscience. vol 10. issue 3. 1998-10-22. PMID:9753179. the relation between dopamine oxidation currents in the nucleus accumbens and conditioned increases in motor activity in rats following repeated administration of d-amphetamine or cocaine. 1998-10-22 2023-08-12 rat
P Di Ciano, C D Blaha, A G Phillip. The relation between dopamine oxidation currents in the nucleus accumbens and conditioned increases in motor activity in rats following repeated administration of d-amphetamine or cocaine. The European journal of neuroscience. vol 10. issue 3. 1998-10-22. PMID:9753179. chronoamperometric recording techniques were used to monitor extracellular dopamine efflux in the nucleus accumbens associated with unconditioned and conditioned increases in motor activity in rats, following the intravenous administration of either d-amphetamine (0.63 mg/kg) or cocaine (3 mg/kg), or the presentation of a conditioned stimulus paired repeatedly with one of these psychostimulants. 1998-10-22 2023-08-12 rat
L L Peoples, A J Uzwiak, F X Guyette, M O Wes. Tonic inhibition of single nucleus accumbens neurons in the rat: a predominant but not exclusive firing pattern induced by cocaine self-administration sessions. Neuroscience. vol 86. issue 1. 1998-10-20. PMID:9692739. tonic inhibition of single nucleus accumbens neurons in the rat: a predominant but not exclusive firing pattern induced by cocaine self-administration sessions. 1998-10-20 2023-08-12 rat
L L Peoples, A J Uzwiak, F X Guyette, M O Wes. Tonic inhibition of single nucleus accumbens neurons in the rat: a predominant but not exclusive firing pattern induced by cocaine self-administration sessions. Neuroscience. vol 86. issue 1. 1998-10-20. PMID:9692739. inhibition of nucleus accumbens neurons is hypothesized to be a mechanism that contributes to the reinforcing (addictive) effects of cocaine and other drugs. 1998-10-20 2023-08-12 rat
L L Peoples, A J Uzwiak, F X Guyette, M O Wes. Tonic inhibition of single nucleus accumbens neurons in the rat: a predominant but not exclusive firing pattern induced by cocaine self-administration sessions. Neuroscience. vol 86. issue 1. 1998-10-20. PMID:9692739. to test this hypothesis, the activity of single nucleus accumbens neurons of rats was recorded extracellularly during cocaine self-administration sessions. 1998-10-20 2023-08-12 rat
O Manzoni, D Pujalte, J Williams, J Bockaer. Decreased presynaptic sensitivity to adenosine after cocaine withdrawal. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 18. issue 19. 1998-10-09. PMID:9742166. the nucleus accumbens (nac) is a site mediating the rewarding properties of drugs of abuse, such as cocaine, amphetamine, opiates, nicotine, and alcohol (wise and bozarth, 1987; koob, 1992; samson andharris, 1992; woolverton and johnson, 1992; self and nestler, 1995; pontieri et al., 1996). 1998-10-09 2023-08-12 Not clear
L L Peoples, F Gee, R Bibi, M O Wes. Phasic firing time locked to cocaine self-infusion and locomotion: dissociable firing patterns of single nucleus accumbens neurons in the rat. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 18. issue 18. 1998-10-02. PMID:9736676. phasic firing time locked to cocaine self-infusion and locomotion: dissociable firing patterns of single nucleus accumbens neurons in the rat. 1998-10-02 2023-08-12 rat