All Relations between hypertrophic and matrix compartment

Publication Sentence Publish Date Extraction Date Species
K J Noonan, E B Hunziker, J Nessler, J A Buckwalte. Changes in cell, matrix compartment, and fibrillar collagen volumes between growth-plate zones. Journal of orthopaedic research : official publication of the Orthopaedic Research Society. vol 16. issue 4. 1998-10-06. PMID:9747793. growth rate was inversely related to the cell numerical density in the upper proliferative and lower hypertrophic zones and was directly related to interterritorial matrix volume per cell in the upper proliferative zone and to pericellular/territorial matrix volume per cell in the lower hypertrophic zone. 1998-10-06 2023-08-12 Not clear
K J Noonan, E B Hunziker, J Nessler, J A Buckwalte. Changes in cell, matrix compartment, and fibrillar collagen volumes between growth-plate zones. Journal of orthopaedic research : official publication of the Orthopaedic Research Society. vol 16. issue 4. 1998-10-06. PMID:9747793. these results show that cell enlargement contributes more to longitudinal bone growth than does increased matrix volume, that increased pericellular/territorial matrix volume makes a greater contribution to growth than does increased interterritorial matrix volume, and that the total amount of fibrillar collagen per cell increases between the upper proliferative and lower hypertrophic zones. 1998-10-06 2023-08-12 Not clear
C Farquharson, J L Berry, E B Mawer, E Seawright, C C Whitehea. Ascorbic acid-induced chondrocyte terminal differentiation: the role of the extracellular matrix and 1,25-dihydroxyvitamin D. European journal of cell biology. vol 76. issue 2. 1998-10-05. PMID:9696350. ascorbic acid-induced chondrocyte terminal differentiation: the role of the extracellular matrix and 1,25-dihydroxyvitamin d. chondrocyte terminal differentiation is associated with cellular hypertrophy increased activity of plasma membrane alkaline phosphatase and the synthesis of collagen type x. 1998-10-05 2023-08-12 Not clear
S M Ali, N J Laping, T A Fredrickson, L C Contino, B a Olson, K Anderson, D P Brook. Angiotensin-converting enzyme inhibition attenuates proteinuria and renal TGF-beta 1 mRNA expression in rats with chronic renal disease. Pharmacology. vol 57. issue 1. 1998-10-01. PMID:9670209. evidence suggests that transforming growth factor beta 1 (tgf-beta 1), a multifunctional cytokine, induces renal extracellular matrix production and glomerular hypertrophy. 1998-10-01 2023-08-12 rat
M Nurminskaya, C Magee, D Nurminsky, T F Linsenmaye. Plasma transglutaminase in hypertrophic chondrocytes: expression and cell-specific intracellular activation produce cell death and externalization. The Journal of cell biology. vol 142. issue 4. 1998-09-25. PMID:9722623. the enzymatic analyses also show that appreciable transglutaminase activity in the hypertrophic zone becomes externalized into the extracellular matrix. 1998-09-25 2023-08-12 human
M Nurminskaya, C Magee, D Nurminsky, T F Linsenmaye. Plasma transglutaminase in hypertrophic chondrocytes: expression and cell-specific intracellular activation produce cell death and externalization. The Journal of cell biology. vol 142. issue 4. 1998-09-25. PMID:9722623. this produces autocatalytic cell death, externalization of the enzyme, and presumably cross-linking of components within the hypertrophic matrix. 1998-09-25 2023-08-12 human
M Nurminskaya, C Magee, D Nurminsky, T F Linsenmaye. Plasma transglutaminase in hypertrophic chondrocytes: expression and cell-specific intracellular activation produce cell death and externalization. The Journal of cell biology. vol 142. issue 4. 1998-09-25. PMID:9722623. these changes may in turn regulate the removal and/or calcification of this hypertrophic matrix, which are its ultimate fates. 1998-09-25 2023-08-12 human
S Lehoux, A Tedgu. Signal transduction of mechanical stresses in the vascular wall. Hypertension (Dallas, Tex. : 1979). vol 32. issue 2. 1998-09-09. PMID:9719064. the processes involved in vascular remodeling include cellular hypertrophy and hyperplasia, as well as enhanced protein synthesis or extracellular matrix protein reorganization. 1998-09-09 2023-08-12 Not clear
Y Sasano, I Takahashi, I Mizoguchi, M Kagayama, H Takita, Y Kubok. Type X collagen is not localized in hypertrophic or calcified cartilage in the developing rat trachea. Anatomy and embryology. vol 197. issue 5. 1998-08-07. PMID:9623674. our previous studies have shown that rat tracheal chondrocytes become larger and hypertrophic, and that the cartilage matrix calcifies during development. 1998-08-07 2023-08-12 rat
R L Broverman, K H Nguyen, A da Silveira, L L Brinkley, S P Macauley, T Zeng, H Yamamoto, R W Tarnuzzer, G S Schultz, M Kerr, M G Humphreys-Behe. Changes in the expression of extracellular matrix (ECM) and matrix metalloproteinases (MMP) of proliferating rat parotid acinar cells. Journal of dental research. vol 77. issue 7. 1998-07-30. PMID:9663435. daily administration of the broad-spectrum matrix metalloproteinase inhibitor galardin for 3 days in combination with isoproterenol resulted in enhanced gland hypertrophy compared with that produced by isoproterenol alone. 1998-07-30 2023-08-12 rat
D Nie, Y Ishikawa, Y Guo, L N Wu, B R Genge, R E Wuthier, G R Saue. Inhibition of terminal differentiation and matrix calcification in cultured avian growth plate chondrocytes by Rous sarcoma virus transformation. Journal of cellular biochemistry. vol 69. issue 4. 1998-07-23. PMID:9620171. endochondral bone formation involves the progression of epiphyseal growth plate chondrocytes through a sequence of developmental stages which include proliferation, differentiation, hypertrophy, and matrix calcification. 1998-07-23 2023-08-12 Not clear
R S Ross, C Pham, S Y Shai, J I Goldhaber, C Fenczik, C C Glembotski, M H Ginsberg, J C Loftu. Beta1 integrins participate in the hypertrophic response of rat ventricular myocytes. Circulation research. vol 82. issue 11. 1998-07-02. PMID:9633916. the hypertrophic response of this model required interaction with extracellular matrix proteins. 1998-07-02 2023-08-12 rat
S R Gudi, A A Lee, C B Clark, J A Frango. Equibiaxial strain and strain rate stimulate early activation of G proteins in cardiac fibroblasts. The American journal of physiology. vol 274. issue 5. 1998-07-01. PMID:9612231. cardiac fibroblasts are responsible for the production of the extracellular matrix of the heart, with alterations of fibroblast function implicated in myocardial infarction and cardiac hypertrophy. 1998-07-01 2023-08-12 rat
E E Mancilla, F De Luca, J A Uyeda, F S Czerwiec, J Baro. Effects of fibroblast growth factor-2 on longitudinal bone growth. Endocrinology. vol 139. issue 6. 1998-06-24. PMID:9607800. we next assessed each major component of longitudinal growth: proliferation, cellular hypertrophy, and cartilage matrix synthesis. 1998-06-24 2023-08-12 rat
E E Mancilla, F De Luca, J A Uyeda, F S Czerwiec, J Baro. Effects of fibroblast growth factor-2 on longitudinal bone growth. Endocrinology. vol 139. issue 6. 1998-06-24. PMID:9607800. we conclude that fgf-2 inhibits longitudinal bone growth by three mechanisms: decreased growth plate chondrocyte proliferation, decreased cellular hypertrophy, and, at high concentrations, decreased cartilage matrix production. 1998-06-24 2023-08-12 rat
M Yoshiyama, K Takeuchi, S Kim, A Hanatani, T Omura, I Toda, K Akioka, M Teragaki, H Iwao, J Yoshikaw. Effect of manidipine hydrochloride, a calcium antagonist, on isoproterenol-induced left ventricular hypertrophy. Japanese circulation journal. vol 62. issue 1. 1998-06-16. PMID:9559417. we examined the effect of a calcium antagonist, manidipine hydrochloride, on cardiac hypertrophy and the expression of the atrial natriuretic peptide (anp), transforming growth factor beta 1 (tgf-beta 1), and extracellular matrix protein genes in rats with isoproterenol-induced cardiac hypertrophy. 1998-06-16 2023-08-12 rat
M Yoshiyama, K Takeuchi, S Kim, A Hanatani, T Omura, I Toda, K Akioka, M Teragaki, H Iwao, J Yoshikaw. Effect of manidipine hydrochloride, a calcium antagonist, on isoproterenol-induced left ventricular hypertrophy. Japanese circulation journal. vol 62. issue 1. 1998-06-16. PMID:9559417. thus, manidipine hydrochloride prevented cardiac hypertrophy and changes in the expression of genes for anp and interstitial components of extracellular matrix induced by isoproterenol. 1998-06-16 2023-08-12 rat
T F Linsenmayer, F Long, M Nurminskaya, Q Chen, T M Schmi. Type X collagen and other up-regulated components of the avian hypertrophic cartilage program. Progress in nucleic acid research and molecular biology. vol 60. 1998-06-15. PMID:9594572. we provide evidence that the molecule has unique diffusion and regulatory properties that combine to alter the hypertrophic cartilage matrix. 1998-06-15 2023-08-12 Not clear
B O Oyajobi, A Frazer, A P Hollander, R M Graveley, C Xu, A Houghton, P V Hatton, R G Russell, B M Stringe. Expression of type X collagen and matrix calcification in three-dimensional cultures of immortalized temperature-sensitive chondrocytes derived from adult human articular cartilage. Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research. vol 13. issue 3. 1998-06-10. PMID:9525344. in addition, in the presence of ascorbate but in the absence of a phosphate donor or inorganic phosphate supplement, the cells also begin to express a hypertrophic phenotype characterized by type x collagen synthesis and extensive mineralization of the extracellular matrix in late stage cultures. 1998-06-10 2023-08-12 human
K Ohshiro, P Pur. Pathogenesis of infantile hypertrophic pyloric stenosis: recent progress. Pediatric surgery international. vol 13. issue 4. 1998-06-10. PMID:9553181. reviews of the recent progress in the pathogenesis of ihps show: (1) there is increasing evidence to suggest that smooth-muscle cells in ihps are not properly innervated; (2) because non-adrenergic, non-cholinergic nerves are mediators of smooth-muscle relaxation, it is likely that the absence of these nerves in pyloric muscle is the cause of excessively contracted hypertrophic circular pyloric muscle; (3) there are abnormal amounts of extracellular matrix proteins in hypertrophic pyloric muscle. 1998-06-10 2023-08-12 Not clear