All Relations between hypertrophic and matrix compartment

Publication Sentence Publish Date Extraction Date Species
K Kuroda, O Okamoto, H Shinka. Dermatopontin expression is decreased in hypertrophic scar and systemic sclerosis skin fibroblasts and is regulated by transforming growth factor-beta1, interleukin-4, and matrix collagen. The Journal of investigative dermatology. vol 112. issue 5. 1999-06-18. PMID:10233760. dermatopontin expression is decreased in hypertrophic scar and systemic sclerosis skin fibroblasts and is regulated by transforming growth factor-beta1, interleukin-4, and matrix collagen. 1999-06-18 2023-08-12 Not clear
K Kuroda, O Okamoto, H Shinka. Dermatopontin expression is decreased in hypertrophic scar and systemic sclerosis skin fibroblasts and is regulated by transforming growth factor-beta1, interleukin-4, and matrix collagen. The Journal of investigative dermatology. vol 112. issue 5. 1999-06-18. PMID:10233760. our results suggest that the decreased expression of dermatopontin is associated with the pathogenesis of fibrosis in hypertrophic scar and systemic sclerosis, and that the effect of the cytokines and matrix collagen on dermatopontin may have important implications for skin fibrosis. 1999-06-18 2023-08-12 Not clear
Z H Liu, T J Guan, Z H Chen, L S L. Glucose transporter (GLUT1) allele (XbaI-) associated with nephropathy in non-insulin-dependent diabetes mellitus. Kidney international. vol 55. issue 5. 1999-06-15. PMID:10231446. several pieces of experimental evidence suggest that glucose transporter (glut1) activity is an important modulator for the cell hypertrophy and extracellular matrix formation of glomerular mesangial cells. 1999-06-15 2023-08-12 Not clear
K Tamura, E Chiba, N Yokoyama, Y Sumida, M Yabana, N Tamura, I Takasaki, N Takagi, M Ishii, M Horiuchi, S Umemur. Renin-angiotensin system and fibronectin gene expression in Dahl Iwai salt-sensitive and salt-resistant rats. Journal of hypertension. vol 17. issue 1. 1999-05-26. PMID:10100098. the tissue renin-angiotensin system and extracellular matrix are involved in the cardiovascular hypertrophy and remodeling induced by hypertension. 1999-05-26 2023-08-12 rat
G Li, A H Simpson, J T Triffit. The role of chondrocytes in intramembranous and endochondral ossification during distraction osteogenesis in the rabbit. Calcified tissue international. vol 64. issue 4. 1999-05-06. PMID:10089224. acid phosphatases were found within the cartilage matrix in some of the cartilage/bone transitional regions and type i collagen mrna and type ii collagen protein were found together in some of the marginal hypertrophic chondrocytes. 1999-05-06 2023-08-12 rabbit
C M Ayanoglou, C Lest. Cyclosporin A-induced gingival overgrowth in the rat: a histological, ultrastructural and histomorphometric evaluation. Journal of periodontal research. vol 34. issue 1. 1999-05-03. PMID:10086881. these results indicate that (a) the csa induced modifications are not limited to enlarged gingiva (b) the overgrowth of the gct is the result of a vasodilatation and of an increase in the volume of the extracellular matrix and (c) the increase of the epithelial thickness is mainly the result of a cell hypertrophy in the keratinized epithelia and of a cell hyperplasia in the junctional epithelium. 1999-05-03 2023-08-12 rat
V S Mujumdar, S C Tyag. Temporal regulation of extracellular matrix components in transition from compensatory hypertrophy to decompensatory heart failure. Journal of hypertension. vol 17. issue 2. 1999-04-28. PMID:10067796. temporal regulation of extracellular matrix components in transition from compensatory hypertrophy to decompensatory heart failure. 1999-04-28 2023-08-12 Not clear
H Fujioka, R Thakur, G J Wang, K Mizuno, G Balian, S R Hurwit. Comparison of surgically attached and non-attached repair of the rat Achilles tendon-bone interface. Cellular organization and type X collagen expression. Connective tissue research. vol 37. issue 3-4. 1999-03-23. PMID:9862222. in group i (tendon reattached), at 2 weeks postoperatively, many hypertrophic chondrocytes appeared at the reattachment site adjacent to bone and type x collagen was detected immunologically both in the cells and in the extracellular matrix. 1999-03-23 2023-08-12 rat
D Vranes, M E Cooper, R J Dille. Cellular mechanisms of diabetic vascular hypertrophy. Microvascular research. vol 57. issue 1. 1999-03-22. PMID:9882558. furthermore, an increase in extracellular matrix within the media is an important feature of diabetes associated vascular hypertrophy. 1999-03-22 2023-08-12 rat
G Palmer, J Zhao, J Bonjour, W Hofstetter, J Caverzasi. In vivo expression of transcripts encoding the Glvr-1 phosphate transporter/retrovirus receptor during bone development. Bone. vol 24. issue 1. 1999-03-22. PMID:9916777. in conclusion, the glvr-1 phosphate transporter is selectively expressed in a subset of hypertrophic chondrocytes during endochondral bone formation, in a region where matrix mineralization proceeds. 1999-03-22 2023-08-12 human
F Spencer, L Chi, M X Zh. Biochemical characterization of benomyl inhibition on endometrial growth during decidualization in rats. Advances in experimental medicine and biology. vol 444. 1999-03-10. PMID:10026946. measured endometrial parameters were the wet weight, protein for hypertrophy, dna indicative of hyperplasia; enzymatic biomarkers- isocitrate dehydrogenase (icdh) and the matrix metalloproteinases (mmps); and serum progesterone which hormonally maintains decidual growth. 1999-03-10 2023-08-12 rat
I Koffi, P Lacolley, M Kirchengaast, J P Pomiès, S Laurent, A Beneto. Prevention of arterial structural alterations with verapamil and trandolapril and consequences for mechanical properties in spontaneously hypertensive rats. European journal of pharmacology. vol 361. issue 1. 1999-03-04. PMID:9851541. we compared the chronic effects in spontaneously hypertensive rats (shr) of low doses of an angiotensin converting enzyme inhibitor, trandolapril, a ca2+ channel antagonist, verapamil, and their combination (trandolapril-verapamil), on arterial mechanical properties, arterial wall hypertrophy and extracellular matrix proteins. 1999-03-04 2023-08-12 rat
R K Dubey, D G Gillespie, E K Jackso. Adenosine inhibits collagen and total protein synthesis in vascular smooth muscle cells. Hypertension (Dallas, Tex. : 1979). vol 33. issue 1 Pt 2. 1999-02-26. PMID:9931103. thus, exogenous a2b receptor agonists and drugs that modulate endogenous adenosine levels may protect against vasoocclusive disorders by attenuating extracellular matrix synthesis by and cellular hypertrophy of vascular smcs. 1999-02-26 2023-08-12 Not clear
T Szuwart, H Kierdorf, U Kierdorf, G Cleme. Ultrastructural aspects of cartilage formation, mineralization, and degeneration during primary antler growth in fallow deer (Dama dama). Annals of anatomy = Anatomischer Anzeiger : official organ of the Anatomische Gesellschaft. vol 180. issue 6. 1999-02-25. PMID:9862029. concomitant with cell hypertrophy, formation of a lacunar and a perilacunar extracellular matrix was observed, the latter containing numerous collagenous fibers. 1999-02-25 2023-08-12 Not clear
T Szuwart, H Kierdorf, U Kierdorf, G Cleme. Ultrastructural aspects of cartilage formation, mineralization, and degeneration during primary antler growth in fallow deer (Dama dama). Annals of anatomy = Anatomischer Anzeiger : official organ of the Anatomische Gesellschaft. vol 180. issue 6. 1999-02-25. PMID:9862029. the hypertrophic chondrocytes of the mineralized cartilage then degenerated, a process that was also occasionally observed in more distally located cells surrounded by still unmineralized matrix. 1999-02-25 2023-08-12 Not clear
H Watanabe, Y Yamad. Mice lacking link protein develop dwarfism and craniofacial abnormalities. Nature genetics. vol 21. issue 2. 1999-02-25. PMID:9988279. during endochondral bone formation, hypertrophic chondrocytes die and the cartilage is replaced with bone matrix. 1999-02-25 2023-08-12 mouse
H Watanabe, Y Yamad. Mice lacking link protein develop dwarfism and craniofacial abnormalities. Nature genetics. vol 21. issue 2. 1999-02-25. PMID:9988279. these results indicate that lp is important for the formation of proteoglycan aggregates and normal organization of hypertrophic chondrocytes, and suggest that cartilage matrix has a role in chondrocyte differentiation and maturation. 1999-02-25 2023-08-12 mouse
J Mollenhauer, M T Mok, K B King, M Gupta, S Chubinskaya, H Koepp, A A Col. Expression of anchorin CII (cartilage annexin V) in human young, normal adult, and osteoarthritic cartilage. The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society. vol 47. issue 2. 1999-02-12. PMID:9889256. anx v epitopes are also located in the pericellular matrix of hypertrophic cartilage. 1999-02-12 2023-08-12 human
M Turcani, H Rup. Development of pressure overload induced cardiac hypertrophy is unaffected by long-term treatment with losartan. Molecular and cellular biochemistry. vol 188. issue 1-2. 1999-02-05. PMID:9823028. left ventricular hypertrophy with adequate wall thickness, preserved adult phenotype and extracellular matrix may be useful in the prevention of heart failure. 1999-02-05 2023-08-12 rat
M Riminucci, J N Bradbeer, A Corsi, C Gentili, F Descalzi, R Cancedda, P Bianc. Vis-à-vis cells and the priming of bone formation. Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research. vol 13. issue 12. 1999-02-03. PMID:9844103. we further showed that in an in vitro avian model of chondrocyte differentiation in vitro to osteoblast-like cells, early hypertrophic chondrocytes replated as adherent cells turned on the expression of high levels of bsp in conjunction with the switch to collagen type i synthesis and matrix mineralization. 1999-02-03 2023-08-12 Not clear