| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| W E Stehben. The significance of programmed cell death or apoptosis and matrix vesicles in atherogenesis. Cellular and molecular biology (Noisy-le-Grand, France). vol 46. issue 1. 2000-04-13. PMID:10726976. |
the significance of programmed cell death or apoptosis and matrix vesicles in atherogenesis. |
2000-04-13 |
2023-08-12 |
Not clear |
| W E Stehben. The significance of programmed cell death or apoptosis and matrix vesicles in atherogenesis. Cellular and molecular biology (Noisy-le-Grand, France). vol 46. issue 1. 2000-04-13. PMID:10726976. |
evaluation of apoptosis must take into account the concomitant changes in the whole vessel wall and its matrix. |
2000-04-13 |
2023-08-12 |
Not clear |
| T Di Matola, F Mueller, G Fenzi, G Rossi, M Bifulco, L A Marzano, M Vital. Serum withdrawal-induced apoptosis in thyroid cells is caused by loss of fibronectin-integrin interaction. The Journal of clinical endocrinology and metabolism. vol 85. issue 3. 2000-03-30. PMID:10720060. |
in some cell types, including a fetal thyroid cell line, denial of adhesion to extracellular matrix induces a type of apoptosis called anoikis. |
2000-03-30 |
2023-08-12 |
human |
| T Di Matola, F Mueller, G Fenzi, G Rossi, M Bifulco, L A Marzano, M Vital. Serum withdrawal-induced apoptosis in thyroid cells is caused by loss of fibronectin-integrin interaction. The Journal of clinical endocrinology and metabolism. vol 85. issue 3. 2000-03-30. PMID:10720060. |
because serum can stimulate cells to produce some components of the extracellular matrix, it was of interest to determine the role of the matrix in the apoptosis induced by serum withdrawal in normal human thyroid cells in primary culture. |
2000-03-30 |
2023-08-12 |
human |
| S Kook, S R Shim, J I Kim, J H Ahnn, Y K Jung, S G Paik, W K Son. Degradation of focal adhesion proteins during nocodazole-induced apoptosis in rat-1 cells. Cell biochemistry and function. vol 18. issue 1. 2000-03-29. PMID:10686577. |
during nocodazole-induced apoptosis, normally flat cells became rounded in shape and detached from the extracellular matrix. |
2000-03-29 |
2023-08-12 |
Not clear |
| G Feldmann, D Haouzi, A Moreau, A M Durand-Schneider, A Bringuier, A Berson, A Mansouri, D Fau, D Pessayr. Opening of the mitochondrial permeability transition pore causes matrix expansion and outer membrane rupture in Fas-mediated hepatic apoptosis in mice. Hepatology (Baltimore, Md.). vol 31. issue 3. 2000-03-15. PMID:10706558. |
opening of the mitochondrial permeability transition pore causes matrix expansion and outer membrane rupture in fas-mediated hepatic apoptosis in mice. |
2000-03-15 |
2023-08-12 |
mouse |
| W Ahmad, A A Panteleyev, A M Christian. The molecular basis of congenital atrichia in humans and mice: mutations in the hairless gene. The journal of investigative dermatology. Symposium proceedings. vol 4. issue 3. 2000-03-14. PMID:10674375. |
in hairless mice, the hair matrix cells appear to undergo a premature and massive apoptosis, together with a concomitant decline in bcl-2 expression, a loss of ncam positivity, and a disconnection with the overlying epithelial sheath essential for the movement of the dermal papilla. |
2000-03-14 |
2023-08-12 |
mouse |
| Y G Liang, A G Jorgensen, C G Kaestel, A K Wiencke, G M Lui, M H la Cour, C H Röpke, M H Nisse. Bcl-2, Bax, and c-Fos expression correlates to RPE cell apoptosis induced by UV-light and daunorubicin. Current eye research. vol 20. issue 1. 2000-03-08. PMID:10611712. |
the aim of this study was to determine the role of bcl-2, bcl-x l, bax, and c-fos in regulation of apoptosis, induced by ultraviolet-light a (uv-a) and daunorubicin (dnr), in retinal pigment epithelium (rpe) cells grown on bovine extracellular matrix (ecm)-coated or uncoated plastic dishes. |
2000-03-08 |
2023-08-12 |
cattle |
| X Cai, Y L Shen, Q Zhu, P M Jia, Y Yu, L Zhou, Y Huang, J W Zhang, S M Xiong, S J Chen, Z Y Wang, Z Chen, G Q Che. Arsenic trioxide-induced apoptosis and differentiation are associated respectively with mitochondrial transmembrane potential collapse and retinoic acid signaling pathways in acute promyelocytic leukemia. Leukemia. vol 14. issue 2. 2000-03-02. PMID:10673743. |
1.0 microm as2o3-induced apoptosis was associated with condensation of the mitochondrial matrix, disruption of mitochondrial transmembrane potentials (deltapsim) and activation of caspase-3 in acute promyelocytic leukemia (apl) cells regardless of their sensitivity to all-trans retinoic acid (atra). |
2000-03-02 |
2023-08-12 |
Not clear |
| S M Frisc. Evidence for a function of death-receptor-related, death-domain-containing proteins in anoikis. Current biology : CB. vol 9. issue 18. 2000-02-14. PMID:10508612. |
normal epithelial cells undergo apoptosis if integrinmediated matrix contacts are lost, in a process termed 'anoikis'. |
2000-02-14 |
2023-08-12 |
Not clear |
| M Rytömaa, L M Martins, J Downwar. Involvement of FADD and caspase-8 signalling in detachment-induced apoptosis. Current biology : CB. vol 9. issue 18. 2000-02-14. PMID:10508619. |
detachment of most untransformed adherent cells from the extracellular matrix promotes apoptosis, in a process termed anoikis [1] [2]. |
2000-02-14 |
2023-08-12 |
human |
| T Furui, R LaPushin, M Mao, H Khan, S R Watt, M A Watt, Y Lu, X Fang, S Tsutsui, Z H Siddik, R C Bast, G B Mill. Overexpression of edg-2/vzg-1 induces apoptosis and anoikis in ovarian cancer cells in a lysophosphatidic acid-independent manner. Clinical cancer research : an official journal of the American Association for Cancer Research. vol 5. issue 12. 2000-02-14. PMID:10632375. |
when cells were separated from the extracellular matrix, they underwent a specialized form of apoptosis called anoikis, which is particularly important in survival of cells in the circulation during metastasis. |
2000-02-14 |
2023-08-12 |
Not clear |
| O Verborgt, G J Gibson, M B Schaffle. Loss of osteocyte integrity in association with microdamage and bone remodeling after fatigue in vivo. Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research. vol 15. issue 1. 2000-02-11. PMID:10646115. |
the strong associations between microdamage, osteocyte apoptosis, and subsequent bone remodeling support the hypothesis that osteocyte apoptosis provides a key part of the activation or signaling mechanisms by which osteoclasts target bone for removal after fatigue-induced matrix injury. |
2000-02-11 |
2023-08-12 |
rat |
| M Sabatini, G Rolland, S Léonce, M Thomas, C Lesur, V Pérez, G de Nanteuil, J Bonne. Effects of ceramide on apoptosis, proteoglycan degradation, and matrix metalloproteinase expression in rabbit articular cartilage. Biochemical and biophysical research communications. vol 267. issue 1. 2000-02-08. PMID:10623638. |
effects of ceramide on apoptosis, proteoglycan degradation, and matrix metalloproteinase expression in rabbit articular cartilage. |
2000-02-08 |
2023-08-12 |
rabbit |
| M Sabatini, G Rolland, S Léonce, M Thomas, C Lesur, V Pérez, G de Nanteuil, J Bonne. Effects of ceramide on apoptosis, proteoglycan degradation, and matrix metalloproteinase expression in rabbit articular cartilage. Biochemical and biophysical research communications. vol 267. issue 1. 2000-02-08. PMID:10623638. |
ceramide also caused chondrocyte apoptosis at doses ranging from 10(-5) to 10(-4) m. this study supports the hypothesis that ceramide might play a mediatory role in both matrix degradation and apoptosis in processes of cartilage loss such as those observed in osteoarthritis. |
2000-02-08 |
2023-08-12 |
rabbit |
| M Y Park, R H Lee, S H Lee, J S Jun. Apoptosis induced by inhibition of contact with extracellular matrix in mouse collecting duct cells. Nephron. vol 83. issue 4. 2000-02-04. PMID:10575296. |
apoptosis induced by inhibition of contact with extracellular matrix in mouse collecting duct cells. |
2000-02-04 |
2023-08-12 |
mouse |
| M Y Park, R H Lee, S H Lee, J S Jun. Apoptosis induced by inhibition of contact with extracellular matrix in mouse collecting duct cells. Nephron. vol 83. issue 4. 2000-02-04. PMID:10575296. |
detachment from the matrix epithelial cells induces programmed cell death, and this cell detachment induced apoptosis has been referred to as 'anoikis'. |
2000-02-04 |
2023-08-12 |
mouse |
| M Y Park, R H Lee, S H Lee, J S Jun. Apoptosis induced by inhibition of contact with extracellular matrix in mouse collecting duct cells. Nephron. vol 83. issue 4. 2000-02-04. PMID:10575296. |
this study was undertaken to determine whether apoptosis is induced by inhibition of contact with extracellular matrix (ecm) in collecting duct cells and to investigate the signaling mechanisms of the process. |
2000-02-04 |
2023-08-12 |
mouse |
| Y Yamada, S Endo, H Nakae, Y Kamei, S Taniguchi, H Ishikura, T Tanaka, K Taki, K Inad. Nuclear matrix protein levels in burn patients with multiple organ dysfunction syndrome. Burns : journal of the International Society for Burn Injuries. vol 25. issue 8. 2000-02-04. PMID:10630850. |
in this study, we investigated multiple organ dysfunction syndrome (mods) and nuclear matrix protein (nmp), which is an indicator for apoptosis, in burn patients. |
2000-02-04 |
2023-08-12 |
human |
| K N Cowan, P L Jones, M Rabinovitc. Elastase and matrix metalloproteinase inhibitors induce regression, and tenascin-C antisense prevents progression, of vascular disease. The Journal of clinical investigation. vol 105. issue 1. 2000-01-28. PMID:10619858. |
our data suggest that proteinase inhibition is a novel strategy to induce regression of vascular disease because this overcomes the pluripotentiality of smc-matrix survival interactions and induces coordinated apoptosis and resorption of matrix. |
2000-01-28 |
2023-08-12 |
rat |