| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| Sukumar Chattopadhyay, Sraboni Bhaumik, Madhumita Purkayastha, Srabanti Basu, Aditi Nag Chaudhuri, Shyamal Das Gupt. Apoptosis and necrosis in developing brain cells due to arsenic toxicity and protection with antioxidants. Toxicology letters. vol 136. issue 1. 2002-12-02. PMID:12368058. |
the morphological analyses showed growth of viable cells, neural networking vis-à-vis apoptosis on exposure to arsenic for 24 h and necrosis and loss of ground matrix on arsenic exposure for 18 days. |
2002-12-02 |
2023-08-12 |
human |
| Andrew E Aplin, Brian P Hogan, Jeannie Tomeu, R L Julian. Cell adhesion differentially regulates the nucleocytoplasmic distribution of active MAP kinases. Journal of cell science. vol 115. issue Pt 13. 2002-11-26. PMID:12077368. |
cells decide whether to undergo processes, such as proliferation, differentiation and apoptosis, based upon the cues they receive from both circulating factors and integrin-mediated adhesion to the extracellular matrix. |
2002-11-26 |
2023-08-12 |
Not clear |
| Won-Jing Wang, Jean-Cheng Kuo, Chung-Chen Yao, Ruey-Hwa Che. DAP-kinase induces apoptosis by suppressing integrin activity and disrupting matrix survival signals. The Journal of cell biology. vol 159. issue 1. 2002-11-19. PMID:12370243. |
dap-kinase induces apoptosis by suppressing integrin activity and disrupting matrix survival signals. |
2002-11-19 |
2023-08-12 |
Not clear |
| O P Zhirnov, A L Ksenofontov, S G Kuzmina, H D Klen. Interaction of influenza A virus M1 matrix protein with caspases. Biochemistry. Biokhimiia. vol 67. issue 5. 2002-11-15. PMID:12059772. |
in this investigation, an ability of influenza a virus m1 matrix protein to bind intracellular caspases, the key enzymes of cell apoptosis, has been examined. |
2002-11-15 |
2023-08-12 |
Not clear |
| T G Frey, C W Renken, G A Perkin. Insight into mitochondrial structure and function from electron tomography. Biochimica et biophysica acta. vol 1555. issue 1-3. 2002-10-18. PMID:12206915. |
results of structural studies of mitochondria during apoptosis demonstrate that cytochrome c is released without detectable disruption of the outer membrane or extensive swelling of the mitochondrial matrix, suggesting the formation of an outer membrane pore large enough to allow passage of holo-cytochrome c. the possible compartmentation of inner membrane function between the ibm and the cristae membrane is also discussed. |
2002-10-18 |
2023-08-12 |
Not clear |
| George M Yousef, Eleftherios P Diamandi. Expanded human tissue kallikrein family--a novel panel of cancer biomarkers. Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine. vol 23. issue 3. 2002-10-16. PMID:12218299. |
preliminary reports indicate a possible role of kallikreins in controlling vital processes, like apoptosis, angiogenesis and tumor metastasis by cleavage of critical substrates such as growth factors, hormones or extracellular matrix. |
2002-10-16 |
2023-08-12 |
human |
| Steven D Webb, Jonathan A Sherratt, Reginald G Fis. Cells behaving badly: a theoretical model for the Fas/FasL system in tumour immunology. Mathematical biosciences. vol 179. issue 2. 2002-10-10. PMID:12208611. |
the model predicts that (a) the production of soluble forms of fas and fasl will lead to the down-regulation of the immune response; (b) matrix metalloproteinase (mmp) inactivation should lead to increased membrane fasl and result in a higher rate of fas-mediated apoptosis for lymphocytes than for tumour cells. |
2002-10-10 |
2023-08-12 |
Not clear |
| Jean-Yves Jouzeau, Sandrine Pacquelet, Christelle Boileau, Emmanuelle Nedelec, Nathalie Presle, Patrick Netter, Bernard Terlai. Nitric oxide (NO) and cartilage metabolism: NO effects are modulated by superoxide in response to IL-1. Biorheology. vol 39. issue 1-2. 2002-10-04. PMID:12082283. |
in vitro evidence includes the decreased synthesis of extracellular matrix components, the abnormal cell renewal, the decreased production of il-1 receptor antagonist, the induction of apoptosis and the enhanced sensitivity of chondrocytes to oxidative stress. |
2002-10-04 |
2023-08-12 |
mouse |
| J Barry Wright, Kan Lam, Andre G Buret, Merle E Olson, Robert E Burrel. Early healing events in a porcine model of contaminated wounds: effects of nanocrystalline silver on matrix metalloproteinases, cell apoptosis, and healing. Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair Society. vol 10. issue 3. 2002-09-27. PMID:12100375. |
early healing events in a porcine model of contaminated wounds: effects of nanocrystalline silver on matrix metalloproteinases, cell apoptosis, and healing. |
2002-09-27 |
2023-08-12 |
Not clear |
| J Barry Wright, Kan Lam, Andre G Buret, Merle E Olson, Robert E Burrel. Early healing events in a porcine model of contaminated wounds: effects of nanocrystalline silver on matrix metalloproteinases, cell apoptosis, and healing. Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair Society. vol 10. issue 3. 2002-09-27. PMID:12100375. |
these benefits are associated with reduced local matrix metalloproteinase levels and enhanced cellular apoptosis. |
2002-09-27 |
2023-08-12 |
Not clear |
| Anne-Marie Preaux, Marie-Pia D'ortho, Marie-Pierre Bralet, Yannick Laperche, Philippe Mavie. Apoptosis of human hepatic myofibroblasts promotes activation of matrix metalloproteinase-2. Hepatology (Baltimore, Md.). vol 36. issue 3. 2002-09-27. PMID:12198653. |
apoptosis of human hepatic myofibroblasts promotes activation of matrix metalloproteinase-2. |
2002-09-27 |
2023-08-12 |
human |
| Anne-Marie Preaux, Marie-Pia D'ortho, Marie-Pierre Bralet, Yannick Laperche, Philippe Mavie. Apoptosis of human hepatic myofibroblasts promotes activation of matrix metalloproteinase-2. Hepatology (Baltimore, Md.). vol 36. issue 3. 2002-09-27. PMID:12198653. |
fibrosis resolution is characterized by apoptosis of hepatic myofibroblasts and degradation of extracellular matrix components. |
2002-09-27 |
2023-08-12 |
human |
| Justus Gille, Eva-M Ehlers, Mathias Okroi, Martin Russlies, Peter Behren. Apoptotic chondrocyte death in cell-matrix biocomposites used in autologous chondrocyte transplantation. Annals of anatomy = Anatomischer Anzeiger : official organ of the Anatomische Gesellschaft. vol 184. issue 4. 2002-09-24. PMID:12201041. |
it is known that matrix provides a trophic signal for the cells and an altered matrix may influence the availability of factors that regulate apoptosis. |
2002-09-24 |
2023-08-12 |
Not clear |
| Johanna Laukkanen, Seppo Ylä-Herttual. Genes involved in atherosclerosis. Experimental nephrology. vol 10. issue 2. 2002-09-17. PMID:11937762. |
the purpose of this article is to focus on the arterial wall and to review lipoprotein receptors, growth factors, cytokines, chemokines, matrix metalloproteinases, adhesion molecules, and apoptosis genes and their involvement in atherogenesis. |
2002-09-17 |
2023-08-12 |
Not clear |
| X Luan, T G H Diekwisc. CP27 affects viability, proliferation, attachment and gene expression in embryonic fibroblasts. Cell proliferation. vol 35. issue 4. 2002-09-13. PMID:12153613. |
following cell culture, cell viability, apoptosis, cell proliferation, cell shape, cellular attachment and fibronectin matrix production were assayed using mtt colourimetric assay, brdu staining, morphometry, immunostaining and western blot analysis. |
2002-09-13 |
2023-08-12 |
rabbit |
| Maria P Morgan, Geraldine M McCarth. Signaling mechanisms involved in crystal-induced tissue damage. Current opinion in rheumatology. vol 14. issue 3. 2002-09-12. PMID:11981329. |
some of the downstream effects known to date include increased mitogenesis, up-regulation of members of the matrix metalloproteinase family, down-regulation of tissue inhibitor of metalloproteinase-1 and -2 in fibroblasts, induction of neutrophil chemotactic chemokines such as interleukin-8, activation and degranulation of neutrophils, and inhibition of neutrophil apoptosis. |
2002-09-12 |
2023-08-12 |
Not clear |
| S Yamasaki, T Nakashima, A Kawakami, T Miyashita, H Ida, K Migita, K Nakata, K Eguch. Functional changes in rheumatoid fibroblast-like synovial cells through activation of peroxisome proliferator-activated receptor gamma-mediated signalling pathway. Clinical and experimental immunology. vol 129. issue 2. 2002-09-11. PMID:12165097. |
incubation of fls with a synthetic ppargamma ligand, troglitazone, inhibited endogenous production of tnf-alpha, il-6 and il-8, as well as matrix metalloprotease-3 (mmp-3), without inducing apoptosis of the cells. |
2002-09-11 |
2023-08-12 |
Not clear |
| William A Ricke, George W Smith, Eric W McIntush, Michael F Smit. Analysis of luteal tissue inhibitor of metalloproteinase-1, -2, and -3 during prostaglandin F(2alpha)-induced luteolysis. Biology of reproduction. vol 66. issue 5. 2002-09-10. PMID:11967202. |
increased matrix metalloproteinase (mmp) expression and activities help to mediate tissue involution through increasing extracellular matrix remodeling and promoting dedifferentiation and, ultimately, apoptosis. |
2002-09-10 |
2023-08-12 |
Not clear |
| F Luchetti, S Burattini, P Ferri, S Papa, E Falcier. Actin involvement in apoptotic chromatin changes of hemopoietic cells undergoing hyperthermia. Apoptosis : an international journal on programmed cell death. vol 7. issue 2. 2002-08-30. PMID:11865198. |
we suggest that, similarly to the role played by nuclear matrix in interphase and mitotic nucleus, actin could be directly involved in chromatin rearrangement occurring in apoptosis. |
2002-08-30 |
2023-08-12 |
Not clear |
| Marc J Curtis, Thomas J Wolper. The oat mitochondrial permeability transition and its implication in victorin binding and induced cell death. The Plant journal : for cell and molecular biology. vol 29. issue 3. 2002-08-16. PMID:11844107. |
previous results suggest that a mpt may facilitate victorin's access to the mitochondrial matrix and binding to the p-protein: (i) victorin-induced cell death displays features similar to apoptosis; (ii) in vivo, victorin binds to the mitochondrial p-protein only in toxin-sensitive genotypes whereas victorin binds equally well to p-protein isolated from toxin-sensitive and insensitive oats; (iii) isolated, untreated mitochondria are impermeable to victorin. |
2002-08-16 |
2023-08-12 |
rat |