| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| Andréa M A Costa, Beatriz Tuchweber, Thierry Lamireau, Ibrahim M Yousef, Charles Balabaud, Jean Rosenbaum, Alexis Desmoulièr. Role of apoptosis in the remodeling of cholestatic liver injury following release of the mechanical stress. Virchows Archiv : an international journal of pathology. vol 442. issue 4. 2003-06-17. PMID:12715172. |
after anastomosis or decompression, apoptosis of both fibroblastic and bile ductular cells increased greatly and was accompanied by ultrastructural features of extracellular matrix degradation. |
2003-06-17 |
2023-08-12 |
rat |
| David J Hunter, Tim D Specto. The role of bone metabolism in osteoarthritis. Current rheumatology reports. vol 5. issue 1. 2003-06-10. PMID:12590880. |
the increased turnover of collagen, in particular, may affect the biomechanical competence of bone and is driven by a number of processes, including osteoblast phenotypic expression, chondrocyte apoptosis, matrix metalloproteinases, and growth factors. |
2003-06-10 |
2023-08-12 |
Not clear |
| Suresh C Tyagi, Brian D Hoi. Metalloproteinase in myocardial adaptation and maladaptation. Journal of cardiovascular pharmacology and therapeutics. vol 7. issue 4. 2003-06-04. PMID:12490970. |
ventricular remodeling is a compensatory response that comprises the processes of apoptosis, muscle cell hypertrophy, and rearrangement of the extracellular matrix fibers connecting the muscles. |
2003-06-04 |
2023-08-12 |
Not clear |
| Yuko Ibuki, Rensuke Got. The antiapoptotic effect of low-dose UVB irradiation in NIH3T3 cells involves caspase inhibitions. Photochemistry and photobiology. vol 77. issue 3. 2003-05-27. PMID:12685655. |
however, we have previously found that low doses of uvb irradiation inhibited apoptosis induced by both serum starvation and lack of extracellular matrix, involving a significant inhibition of caspase-3/7 activation. |
2003-05-27 |
2023-08-12 |
Not clear |
| Thierry Vincent, Laurence Molina, Lucile Espert, Nadir Mecht. Hyaluronan, a major non-protein glycosaminoglycan component of the extracellular matrix in human bone marrow, mediates dexamethasone resistance in multiple myeloma. British journal of haematology. vol 121. issue 2. 2003-05-27. PMID:12694247. |
in this study, hyaluronan (ha), a major non-protein glycosaminoglycan component of the extracellular matrix in mammalian bone marrow, acted as a survival factor against dexamethasone (dex)-induced apoptosis in mm cell lines. |
2003-05-27 |
2023-08-12 |
human |
| Tomohiko Fukuda, Lida Guo, Xiaohua Shi, Chuanyue W. CH-ILKBP regulates cell survival by facilitating the membrane translocation of protein kinase B/Akt. The Journal of cell biology. vol 160. issue 7. 2003-05-23. PMID:12654898. |
cells that are depleted of ch-ilkbp undergo extensive apoptosis despite the presence of cell-extracellular matrix contacts and soluble growth factors. |
2003-05-23 |
2023-08-12 |
Not clear |
| F Grabellus, C Schmid, B Levkau, J Stypmann, H H Scheld, H A Bab. [Myocardial alterations with mechanical left ventricular assist devices]. Der Pathologe. vol 24. issue 2. 2003-05-23. PMID:12673495. |
various molecular pathways of the human myocardium associated with apoptosis, response to stress, or matrix changes are known to be altered under conditions of heart failure, and some have been shown to be reversibly regulated during left ventricular mechanical support, suggesting that the descriptive term "reverse remodeling" is, at least in parts, a reversible mechanism. |
2003-05-23 |
2023-08-12 |
human |
| Stephanie Groos, Enrico Reale, Günter Hünefeld, Liliana Lucian. Changes in epithelial cell turnover and extracellular matrix in human small intestine after TPN. The Journal of surgical research. vol 109. issue 2. 2003-05-22. PMID:12643847. |
the present study aims at examining (1) whether there are differences in proliferation and apoptosis of epithelial cells between enterally and parenterally nourished patients and (2) whether the distribution pattern of extracellular matrix (ecm) proteins known to influence cell turnover along the the crypt-villus axis is changed after tpn. |
2003-05-22 |
2023-08-12 |
human |
| Yuhong He, Jing Chen, Jianguo Ren, Guochun Wang, Guoping Ca. Type I collagen inhibits hydroxyl radical-induced apoptosis. Journal of biochemistry. vol 132. issue 3. 2003-05-16. PMID:12204105. |
the extracellular matrix (ecm) plays an important role in cell differentiation and apoptosis. |
2003-05-16 |
2023-08-12 |
Not clear |
| Mari T Kaartinen, Sherif El-Maadawy, Niina H Räsänen, Marc D McKe. Tissue transglutaminase and its substrates in bone. Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research. vol 17. issue 12. 2003-05-16. PMID:12469910. |
tissue transglutaminase (ttg) is an intra- and extracellular, protein-cross-linking enzyme that has been implicated in apoptosis, matrix stabilization, and cell attachment in a variety of tissues. |
2003-05-16 |
2023-08-12 |
rat |
| Sung-Yu Hong, Hyeryung Lee, Weon-Kyoo You, Kwang-Hoe Chung, Doo-Sik Kim, Kiwon Son. The snake venom disintegrin salmosin induces apoptosis by disassembly of focal adhesions in bovine capillary endothelial cells. Biochemical and biophysical research communications. vol 302. issue 3. 2003-05-13. PMID:12615062. |
these observations strongly suggest that salmosin disorganizes focal contacts to detach cells by competing with the extracellular matrix (ecm) for direct binding to integrin alpha(v)beta(3) on the cell surface, eventually leading to apoptosis. |
2003-05-13 |
2023-08-12 |
cattle |
| L V Kremneva, O V Abaturov. [Molecular and cellular mechanisms of myocardial remodeling in heart failure]. Klinicheskaia meditsina. vol 81. issue 2. 2003-05-07. PMID:12685226. |
the apoptosis of smooth muscle cells, the translocation, structural and functional impairments of remaining cardiomyocytes, ventricular dilatation, and the altered extracellular matrix underlie myocardial remodeling in chronic heart failure (chf). |
2003-05-07 |
2023-08-12 |
Not clear |
| Sarah A Kopecky, Douglas S Lyle. Contrasting effects of matrix protein on apoptosis in HeLa and BHK cells infected with vesicular stomatitis virus are due to inhibition of host gene expression. Journal of virology. vol 77. issue 8. 2003-05-05. PMID:12663772. |
contrasting effects of matrix protein on apoptosis in hela and bhk cells infected with vesicular stomatitis virus are due to inhibition of host gene expression. |
2003-05-05 |
2023-08-12 |
Not clear |
| Sarah A Kopecky, Douglas S Lyle. Contrasting effects of matrix protein on apoptosis in HeLa and BHK cells infected with vesicular stomatitis virus are due to inhibition of host gene expression. Journal of virology. vol 77. issue 8. 2003-05-05. PMID:12663772. |
recently, it has been shown that two vsv products are involved in the induction of apoptosis, the matrix (m) protein, and another viral product that has yet to be identified (s. a. kopecky et. |
2003-05-05 |
2023-08-12 |
Not clear |
| b' Z Humlov\\xc3\\xa. Protooncogene bcl-2 in process of apoptosis. Review article. Sbornik lekarsky. vol 103. issue 4. 2003-04-29. PMID:12688154.' |
changes in the flux of ions and water across the inner mitochondrial membrane characterize the early phase of apoptosis, during which an increase in matrix volume may precede a collapse of mitochondrial membrane potential (delta psi m). |
2003-04-29 |
2023-08-12 |
Not clear |
| Victoria M L Herrera, Tamara Didishvili, Lyle V Lopez, Nelson Ruiz-Opaz. Differential regulation of functional gene clusters in overt coronary artery disease in a transgenic atherosclerosis-hypertensive rat model. Molecular medicine (Cambridge, Mass.). vol 8. issue 7. 2003-04-16. PMID:12393934. |
typically studied as distinct pathologies, emerging pathogenic paradigms implicate multiple processes beyond thrombosis and ischemic cell injury respectively, with significant pathway overlap involving inflammation, apoptosis, matrix degradation, and oxidative stress. |
2003-04-16 |
2023-08-12 |
human |
| Ding Yu, Zihui Wang, Liyuan Zhu, E C Che. Nuclear matrix associated protein PML: an arsenic trioxide apoptosis therapeutic target protein in HepG2 cells. Chinese medical journal. vol 116. issue 1. 2003-04-15. PMID:12667397. |
nuclear matrix associated protein pml: an arsenic trioxide apoptosis therapeutic target protein in hepg2 cells. |
2003-04-15 |
2023-08-12 |
Not clear |
| Ding Yu, Zihui Wang, Liyuan Zhu, E C Che. Nuclear matrix associated protein PML: an arsenic trioxide apoptosis therapeutic target protein in HepG2 cells. Chinese medical journal. vol 116. issue 1. 2003-04-15. PMID:12667397. |
to investigate arsenic trioxide (as(2)o(3))-induced apoptosis and the effects on cell nuclear matrix related protein promyelocytic leukaemia (pml). |
2003-04-15 |
2023-08-12 |
Not clear |
| Feng Jiang, Zhou Wan. Identification of androgen-responsive genes in the rat ventral prostate by complementary deoxyribonucleic acid subtraction and microarray. Endocrinology. vol 144. issue 4. 2003-04-10. PMID:12639908. |
the identified genes can be classified into several functional groups, including metabolism, protein chaperoning and trafficking, protein synthesis, secretions, cell cycle and apoptosis, structural and extracellular matrix proteins, and novel proteins. |
2003-04-10 |
2023-08-12 |
rat |
| Inder S Anan. Ventricular remodeling without cellular contractile dysfunction. Journal of cardiac failure. vol 8. issue 6 Suppl. 2003-04-08. PMID:12555152. |
important among these are loss of myocytes from necrosis or apoptosis, changes in the extracellular matrix (ecm), myocyte contractile dysfunction, and the purely geometric or mechanical effects of structural alterations in the lv. |
2003-04-08 |
2023-08-12 |
Not clear |