| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| Eric W Olle, Xiaodan Ren, Shannon D McClintock, Roscoe L Warner, Michael P Deogracias, Kent J Johnson, Lisa M Collett. Matrix metalloproteinase-9 is an important factor in hepatic regeneration after partial hepatectomy in mice. Hepatology (Baltimore, Md.). vol 44. issue 3. 2006-09-28. PMID:16941692. |
partial hepatectomy triggers hepatocyte proliferation, hepatic matrix remodeling, and hepatocyte apoptosis, all of which are important processes in the regenerating liver. |
2006-09-28 |
2023-08-12 |
mouse |
| Olivier Morel, Florence Toti, Babé Bakouboula, Lélia Grunebaum, Jean-Marie Freyssine. Procoagulant microparticles: 'criminal partners' in atherothrombosis and deleterious cellular exchanges. Pathophysiology of haemostasis and thrombosis. vol 35. issue 1-2. 2006-09-22. PMID:16855341. |
besides this procoagulant potential, trapped mp could contribute to plaque vulnerability through multiple pathways including angiogenesis, extracellular matrix proteolysis, recruitment of inflammatory cells, smooth muscle cell and endothelial apoptosis. |
2006-09-22 |
2023-08-12 |
human |
| Livia S Machado, Anna Kozak, Adviye Ergul, David C Hess, Cesario V Borlongan, Susan C Faga. Delayed minocycline inhibits ischemia-activated matrix metalloproteinases 2 and 9 after experimental stroke. BMC neuroscience. vol 7. 2006-09-20. PMID:16846501. |
minocycline inhibits inflammation, apoptosis and extracellular matrix degradation. |
2006-09-20 |
2023-08-12 |
rat |
| Hiroyuki Tsutsu. Mitochondrial oxidative stress and heart failure. Internal medicine (Tokyo, Japan). vol 45. issue 13. 2006-09-15. PMID:16880705. |
reactive oxygen species induce myocyte hypertrophy, apoptosis, and interstitial fibrosis by activating matrix metalloproteinases. |
2006-09-15 |
2023-08-12 |
Not clear |
| S Viappiani, M Sariahmetoglu, R Schul. The role of matrix metalloproteinase inhibitors in ischemia-reperfusion injury in the liver. Current pharmaceutical design. vol 12. issue 23. 2006-09-15. PMID:16918422. |
liver ischemia-reperfusion injury is characterized by cell necrosis and apoptosis and by profound modifications in the extracellular matrix (ecm). |
2006-09-15 |
2023-08-12 |
Not clear |
| S Viappiani, M Sariahmetoglu, R Schul. The role of matrix metalloproteinase inhibitors in ischemia-reperfusion injury in the liver. Current pharmaceutical design. vol 12. issue 23. 2006-09-15. PMID:16918422. |
several experimental models of liver ischemia-reperfusion injury have demonstrated protective effects of mmp inhibitors in terms of cell necrosis, apoptosis and rearrangement of the extracellular matrix. |
2006-09-15 |
2023-08-12 |
Not clear |
| Roland D Chapurlat, Pierre D Delma. Drug insight: Bisphosphonates for postmenopausal osteoporosis. Nature clinical practice. Endocrinology & metabolism. vol 2. issue 4. 2006-09-15. PMID:16932286. |
when embedded in bone matrix, bisphosphonates are taken up by osteoclasts engaged in bone resorption, leading--mainly by inhibition of farnesyl diphosphate synthase, a key enzyme of the mevalonate pathway--to osteoclast apoptosis. |
2006-09-15 |
2023-08-12 |
Not clear |
| K Boisvert-Adamo, A E Apli. B-RAF and PI-3 kinase signaling protect melanoma cells from anoikis. Oncogene. vol 25. issue 35. 2006-09-13. PMID:16547495. |
a hallmark feature of cancer is resistance to anoikis, apoptosis induced when cells either lose contact with or encounter an inappropriate extracellular matrix. |
2006-09-13 |
2023-08-12 |
Not clear |
| Jie Shen, Darryl O'Brien, Yi X. Matrix metalloproteinase-2 contributes to tumor necrosis factor alpha induced apoptosis in cultured rat cardiac myocytes. Biochemical and biophysical research communications. vol 347. issue 4. 2006-09-13. PMID:16857167. |
matrix metalloproteinase-2 contributes to tumor necrosis factor alpha induced apoptosis in cultured rat cardiac myocytes. |
2006-09-13 |
2023-08-12 |
human |
| R Frlan, S Gobe. Inhibitors of cathepsin B. Current medicinal chemistry. vol 13. issue 19. 2006-09-11. PMID:16918357. |
it is involved in many physiological processes, such as remodeling of the extracellular matrix (wound healing), apoptosis, and activation of thyroxine and renin. |
2006-09-11 |
2023-08-12 |
Not clear |
| M Mukai, H Endo, T Iwasaki, M Tatsuta, A Togawa, H Nakamura, M Inou. RhoC is essential for TGF-beta1-induced invasive capacity of rat ascites hepatoma cells. Biochemical and biophysical research communications. vol 346. issue 1. 2006-08-28. PMID:16750170. |
transforming growth factor-beta1 (tgf-beta1) is a multifunctional growth factor that plays a role in cell proliferation, differentiation, extracellular matrix production, apoptosis, and cell motility. |
2006-08-28 |
2023-08-12 |
rat |
| Denise M T Yu, Xin M Wang, Geoffrey W McCaughan, Mark D Gorrel. Extraenzymatic functions of the dipeptidyl peptidase IV-related proteins DP8 and DP9 in cell adhesion, migration and apoptosis. The FEBS journal. vol 273. issue 11. 2006-08-23. PMID:16704418. |
we have previously shown that the in vitro roles of dipeptidyl peptidase iv and fibroblast activation protein in cell-extracellular matrix interactions and apoptosis are similarly independent of catalytic activity. |
2006-08-23 |
2023-08-12 |
Not clear |
| En-Hui Yao, Yi Yu, Noboru Fukud. Oxidative stress on progenitor and stem cells in cardiovascular diseases. Current pharmaceutical biotechnology. vol 7. issue 2. 2006-08-22. PMID:16724944. |
these effects include cell growth, apoptosis, migration, inflammatory gene expression and matrix regulation. |
2006-08-22 |
2023-08-12 |
Not clear |
| Susanne Rutschow, Jun Li, Heinz-Peter Schultheiss, Matthias Pauschinge. Myocardial proteases and matrix remodeling in inflammatory heart disease. Cardiovascular research. vol 69. issue 3. 2006-08-18. PMID:16417902. |
the myocardial inflammatory reaction not only affects myocardial hypertrophy and apoptosis, but it has a major influence on the regulation of extracellular matrix turnover. |
2006-08-18 |
2023-08-12 |
Not clear |
| Virna L Sales, George C Engelmayr, Bret A Mettler, John A Johnson, Michael S Sacks, John E Maye. Transforming growth factor-beta1 modulates extracellular matrix production, proliferation, and apoptosis of endothelial progenitor cells in tissue-engineering scaffolds. Circulation. vol 114. issue 1 Suppl. 2006-08-18. PMID:16820571. |
transforming growth factor-beta1 modulates extracellular matrix production, proliferation, and apoptosis of endothelial progenitor cells in tissue-engineering scaffolds. |
2006-08-18 |
2023-08-12 |
Not clear |
| Allen Cheng, Tom C Nguyen, Marcin Malinowski, Frank Langer, David Liang, George T Daughters, Neil B Ingels, D Craig Mille. Passive ventricular constraint prevents transmural shear strain progression in left ventricle remodeling. Circulation. vol 114. issue 1 Suppl. 2006-08-18. PMID:16820650. |
altered wall strain results in cytokine and reactive oxygen species production, which, in turn, stimulates apoptosis and extracellular matrix disruption and could be an important trigger for adverse global lv dilatation and remodeling. |
2006-08-18 |
2023-08-12 |
Not clear |
| Jaron Lockett, Shuping Yin, Xiaohua Li, Yonghong Meng, Shijie Shen. Tumor suppressive maspin and epithelial homeostasis. Journal of cellular biochemistry. vol 97. issue 4. 2006-08-14. PMID:16329135. |
it is further noted that extracellular maspin is sufficient to block tumor induced extracellular matrix degradation, tumor cell motility and invasion, whereas intracellular maspin is responsible for the increased cellular sensitivity to apoptosis. |
2006-08-14 |
2023-08-12 |
Not clear |
| Danyu Sun, Keith R McCra. Endothelial-cell apoptosis induced by cleaved high-molecular-weight kininogen (HKa) is matrix dependent and requires the generation of reactive oxygen species. Blood. vol 107. issue 12. 2006-08-14. PMID:16418331. |
endothelial-cell apoptosis induced by cleaved high-molecular-weight kininogen (hka) is matrix dependent and requires the generation of reactive oxygen species. |
2006-08-14 |
2023-08-12 |
Not clear |
| Danyu Sun, Keith R McCra. Endothelial-cell apoptosis induced by cleaved high-molecular-weight kininogen (HKa) is matrix dependent and requires the generation of reactive oxygen species. Blood. vol 107. issue 12. 2006-08-14. PMID:16418331. |
the ability of hka to induce endothelial-cell apoptosis is dependent on the generation of intracellular reactive oxygen species and associated with depletion of glutathione and peroxidation of endothelial-cell lipids, effects that occur only in cells cultured on matrix proteins permissive for hka-induced apoptosis. |
2006-08-14 |
2023-08-12 |
Not clear |
| Liezhen Fu, Akihiro Tomita, Hua Wang, Daniel R Buchholz, Yun-Bo Sh. Transcriptional regulation of the Xenopus laevis Stromelysin-3 gene by thyroid hormone is mediated by a DNA element in the first intron. The Journal of biological chemistry. vol 281. issue 25. 2006-08-11. PMID:16606608. |
the matrix metalloproteinase (mmp) stromelysin-3 (st3) (mmp11) was first isolated as a breast cancer-associated gene and is expressed in diverse human carcinomas and various developmental processes involving apoptosis. |
2006-08-11 |
2023-08-12 |
human |