| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| Rossella Russo, Elisa Siviglia, Micaela Gliozzi, Diana Amantea, Annamaria Paoletti, Laura Berliocchi, G Bagetta, M Tiziana Corasanit. Evidence implicating matrix metalloproteinases in the mechanism underlying accumulation of IL-1beta and neuronal apoptosis in the neocortex of HIV/gp120-exposed rats. International review of neurobiology. vol 82. 2007-09-05. PMID:17678975. |
evidence implicating matrix metalloproteinases in the mechanism underlying accumulation of il-1beta and neuronal apoptosis in the neocortex of hiv/gp120-exposed rats. |
2007-09-05 |
2023-08-12 |
rat |
| Lihua Wang, Yuzhu Tang, Deborah C Rubin, Marc S Levi. Chronically administered retinoic acid has trophic effects in the rat small intestine and promotes adaptation in a resection model of short bowel syndrome. American journal of physiology. Gastrointestinal and liver physiology. vol 292. issue 6. 2007-08-30. PMID:17307727. |
at 2 wk postoperation, changes in gut morphology, crypt cell proliferation and apoptosis, enterocyte migration, the extracellular matrix, and gene expression were assessed. |
2007-08-30 |
2023-08-12 |
rat |
| Marjo de Graauw, Sylvia Le Dévédec, Ine Tijdens, Mirjam B Smeets, André M Deelder, Bob van de Wate. Proteomic analysis of alternative protein tyrosine phosphorylation in 1,2-dichlorovinyl-cysteine-induced cytotoxicity in primary cultured rat renal proximal tubular cells. The Journal of pharmacology and experimental therapeutics. vol 322. issue 1. 2007-08-30. PMID:17442843. |
treatment of primary cultured rat proximal tubular epithelial cells with the model nephrotoxicant s-(1,2-dichlorovinyl)-l-cysteine (dcvc) resulted in early reorganization of f-actin stress fibers and formation of lamellipodia, which was followed by cell detachment from the matrix and apoptosis. |
2007-08-30 |
2023-08-12 |
rat |
| Cornelia Amăline. [Morphogenesis and differentiation of the female genital tract. Genetic determinism and epithelium-stromal interactions]. Revista medico-chirurgicala a Societatii de Medici si Naturalisti din Iasi. vol 111. issue 1. 2007-08-30. PMID:17595868. |
tgf-beta, whose expression is modulated by ovarian steroids, regulates cell growth, differentiation, apoptosis, inflammatory and immune responses, extracellular matrix deposition, adhesion molecules, proteases, and protease inhibitor expression. |
2007-08-30 |
2023-08-12 |
Not clear |
| b' T Pelik\\xc3\\xa1nov\\xc3\\xa. [Pathogenesis of diabetic retinopathy]. Vnitrni lekarstvi. vol 53. issue 5. 2007-08-30. PMID:17642432.' |
these activated cells change the production pattern of a number of mediators such as growth factors, vasoactive agents, coagulation factors and adhesion molecules resulting in increased blood flow, increased capillary permeability, proliferation of extracellular matrix and thickening of basal membranes, altered cell turnover (apoptosis, proliferation, hypertrophy), procoagulant and proaggregant patterns, and finally in angiogenesis and tissue remodelling. |
2007-08-30 |
2023-08-12 |
Not clear |
| Mira Lim, Michael H Goldstein, Sonal Tuli, Gregory S Schult. Growth factor, cytokine and protease interactions during corneal wound healing. The ocular surface. vol 1. issue 2. 2007-08-28. PMID:17075633. |
simultaneously, keratocytes adjacent to the injured area undergo apoptosis under the fas/fas ligand system, while more distant keratocytes transform into activated fibroblasts and migrate into the wound, where they begin synthesizing new extracellular matrix components that form the scar tissue under the dominant influence of the tgfb/ ctgf system. |
2007-08-28 |
2023-08-12 |
Not clear |
| Net Das Evcimen, George L Kin. The role of protein kinase C activation and the vascular complications of diabetes. Pharmacological research. vol 55. issue 6. 2007-08-24. PMID:17574431. |
increased pkc activation have been associated with changes in blood flow, basement membrane thickening, extracellular matrix expansion, increases in vascular permeability, abnormal angiogenesis, excessive apoptosis and changes in enzymatic activity alterations such as na(+)-k(+)-atpase, cpla(2), pi3kinase and map kinase. |
2007-08-24 |
2023-08-12 |
Not clear |
| Tetyana L Vasylyeva, Robert J Ferr. Novel roles of the IGF-IGFBP axis in etiopathophysiology of diabetic nephropathy. Diabetes research and clinical practice. vol 76. issue 2. 2007-08-20. PMID:17011663. |
high ambient glucose level transforms intracellular pathways, promoting stable phenotypic changes in the glomerulus such as mesangial cell hypertrophy, podocyte apoptosis, and matrix expansion. |
2007-08-20 |
2023-08-12 |
Not clear |
| Gordon L Warren, Mukesh Summan, Xin Gao, Rebecca Chapman, Tracy Hulderman, Petia P Simeonov. Mechanisms of skeletal muscle injury and repair revealed by gene expression studies in mouse models. The Journal of physiology. vol 582. issue Pt 2. 2007-08-20. PMID:17478534. |
only ci activated a set of genes associated with the repair of impaired proteins and structures including genes related to apoptosis, whereas fi uniquely activated gene sets involved in extensive inflammatory responses, tissue remodelling, angiogenesis and myofibre/extracellular matrix synthesis. |
2007-08-20 |
2023-08-12 |
mouse |
| Antonio Di Sabatino, Sylvia L F Pender, Claire L Jackson, Joanna D Prothero, John N Gordon, Lucia Picariello, Laura Rovedatti, Guillermo Docena, Giovanni Monteleone, David S Rampton, Francesco Tonelli, Gino R Corazza, Thomas T MacDonal. Functional modulation of Crohn's disease myofibroblasts by anti-tumor necrosis factor antibodies. Gastroenterology. vol 133. issue 1. 2007-08-16. PMID:17631138. |
we investigated the action of infliximab on apoptosis, the production of matrix metalloproteinases (mmps) and tissue inhibitor of metalloproteinases (timp)-1, and migration of crohn's disease (cd) myofibroblasts. |
2007-08-16 |
2023-08-12 |
Not clear |
| Yi Yang, Fabio M V Rossi, Edward E Putnin. Ex vivo expansion of rat bone marrow mesenchymal stromal cells on microcarrier beads in spin culture. Biomaterials. vol 28. issue 20. 2007-08-13. PMID:17433434. |
we hypothesized that disruption of cell-extracellular matrix contact either during cell expansion or immediately prior to cell transplantation may impair cell viability and facilitate apoptosis. |
2007-08-13 |
2023-08-12 |
rat |
| Pål Aukrust, Arne Yndestad, Jan Kristian Damås, Thor Ueland, Erik Øie, Lars Gullesta. Potential mechanisms of benefit with thalidomide in chronic heart failure. American journal of cardiovascular drugs : drugs, devices, and other interventions. vol 7. issue 2. 2007-08-07. PMID:17503883. |
persistent inflammation, involving increased levels of inflammatory cytokines, seems to play a pathogenic role in chronic hf by influencing heart contractility, inducing matrix degradation and fibrosis, and promoting apoptosis, contributing to myocardial remodeling. |
2007-08-07 |
2023-08-12 |
human |
| Fiona J Matthews, Stuart D Cook, Mohammed A Majid, Andrew D Dick, Valerie A Smit. Changes in the balance of the tissue inhibitor of matrix metalloproteinases (TIMPs)-1 and -3 may promote keratocyte apoptosis in keratoconus. Experimental eye research. vol 84. issue 6. 2007-08-06. PMID:17449031. |
changes in the balance of the tissue inhibitor of matrix metalloproteinases (timps)-1 and -3 may promote keratocyte apoptosis in keratoconus. |
2007-08-06 |
2023-08-12 |
Not clear |
| Fiona J Matthews, Stuart D Cook, Mohammed A Majid, Andrew D Dick, Valerie A Smit. Changes in the balance of the tissue inhibitor of matrix metalloproteinases (TIMPs)-1 and -3 may promote keratocyte apoptosis in keratoconus. Experimental eye research. vol 84. issue 6. 2007-08-06. PMID:17449031. |
this could result from corneal stromal cell apoptosis or be induced or perpetuated by the activation of matrix degrading enzymes, particularly members of the matrix metalloproteinase (mmp) family. |
2007-08-06 |
2023-08-12 |
Not clear |
| M Coolman, M de Maat, W L Van Heerde, L Felida, S Schoormans, E A P Steegers, R M Bertina, C J M de Groo. Matrix metalloproteinase-9 gene -1562C/T polymorphism mitigates preeclampsia. Placenta. vol 28. issue 7. 2007-08-03. PMID:17137622. |
prerequisites for successful placentation include trophoblast invasion, degradation and remodelling of the uterine decidual extracellular matrix, and apoptosis without thrombosis. |
2007-08-03 |
2023-08-12 |
Not clear |
| Marko Jović, Naava Naslavsky, Debora Rapaport, Mia Horowitz, Steve Capla. EHD1 regulates beta1 integrin endosomal transport: effects on focal adhesions, cell spreading and migration. Journal of cell science. vol 120. issue Pt 5. 2007-07-31. PMID:17284518. |
beta1 integrins bind to the extracellular matrix and stimulate signaling pathways leading to crucial cellular functions, including proliferation, apoptosis, cell spreading and migration. |
2007-07-31 |
2023-08-12 |
mouse |
| Betty Schwartz, Oded Shoseyov, Vladislava O Melnikova, Marya McCarty, Michael Leslie, Levava Roiz, Patricia Smirnoff, Guo-Fu Hu, Dina Lev, Menashe Bar-El. ACTIBIND, a T2 RNase, competes with angiogenin and inhibits human melanoma growth, angiogenesis, and metastasis. Cancer research. vol 67. issue 11. 2007-07-26. PMID:17545605. |
thus, there was decreased expression and activity of the matrix metalloproteinase 2 in melanoma and vascular endothelial cells, decreased vascularization, and increased tumor cell apoptosis in vivo. |
2007-07-26 |
2023-08-12 |
human |
| Feng M Ho, Shing H Liu, Wan W Lin, Chiau S Lia. Opposite effects of high glucose on MMP-2 and TIMP-2 in human endothelial cells. Journal of cellular biochemistry. vol 101. issue 2. 2007-07-24. PMID:17203468. |
although high glucose can induce matrix metalloproteinases (mmps), its inhibitors and cell apoptosis, little is known about the roles of mmps in regulating cell apoptosis in response to high glucose. |
2007-07-24 |
2023-08-12 |
human |
| Christoph Daniel, Kerstin Amann, Bernd Hohenstein, Paul Bornstein, Christian Hug. Thrombospondin 2 functions as an endogenous regulator of angiogenesis and inflammation in experimental glomerulonephritis in mice. Journal of the American Society of Nephrology : JASN. vol 18. issue 3. 2007-07-24. PMID:17287428. |
tsp2-null mice were analyzed for kidney function, renal cortical matrix expansion, influx of inflammatory cells, proliferation, and apoptosis, as well as for capillary rarefaction after induction of anti-gbm disease. |
2007-07-24 |
2023-08-12 |
mouse |
| Joachim Weil, Heribert Schunker. [Pathophysiology of chronic heart failure]. Clinical research in cardiology : official journal of the German Cardiac Society. vol 95 Suppl 4. 2007-07-17. PMID:16598604. |
these changes in gene expression are complex and involve contractile proteins, ion channels, ca(++) handling, apoptosis, cell metabolism, the extracellular matrix, signal transduction pathways and growth factors. |
2007-07-17 |
2023-08-12 |
Not clear |