| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| Quan-Zhou Feng, Yu-Sheng Zhao, Eltyeb Abdelwahi. The role of Fas in the progression of ischemic heart failure: prohypertrophy or proapoptosis. Coronary artery disease. vol 19. issue 7. 2009-01-15. PMID:18923250. |
during myocardial ischemia, cardiomyocytes can undergo apoptosis or compensatory hypertrophy. |
2009-01-15 |
2023-08-12 |
Not clear |
| Quan-Zhou Feng, Yu-Sheng Zhao, Eltyeb Abdelwahi. The role of Fas in the progression of ischemic heart failure: prohypertrophy or proapoptosis. Coronary artery disease. vol 19. issue 7. 2009-01-15. PMID:18923250. |
fas expression is upregulated in the myocardial ischemia and is coupled to both apoptosis and hypertrophy of cardiomyocytes. |
2009-01-15 |
2023-08-12 |
Not clear |
| Quan-Zhou Feng, Yu-Sheng Zhao, Eltyeb Abdelwahi. The role of Fas in the progression of ischemic heart failure: prohypertrophy or proapoptosis. Coronary artery disease. vol 19. issue 7. 2009-01-15. PMID:18923250. |
the role of fas in apoptosis induction or cardiomyocyte hypertrophy during ischemic conditions is, however, still unclear. |
2009-01-15 |
2023-08-12 |
Not clear |
| Quan-Zhou Feng, Yu-Sheng Zhao, Eltyeb Abdelwahi. The role of Fas in the progression of ischemic heart failure: prohypertrophy or proapoptosis. Coronary artery disease. vol 19. issue 7. 2009-01-15. PMID:18923250. |
hypertrophic cardiomyocytes easily undergo apoptosis in response to ischemia, after which apoptotic cardiomyocytes are replaced by fibrous tissue. |
2009-01-15 |
2023-08-12 |
Not clear |
| Quan-Zhou Feng, Yu-Sheng Zhao, Eltyeb Abdelwahi. The role of Fas in the progression of ischemic heart failure: prohypertrophy or proapoptosis. Coronary artery disease. vol 19. issue 7. 2009-01-15. PMID:18923250. |
in the late stage of ischemic heart failure, hypertrophy, apoptosis, and fibrosis are thought to accelerate each other and might thus form a vicious circle that eventually results in heart failure. |
2009-01-15 |
2023-08-12 |
Not clear |
| Kerstin Schwarz, Gregor Simonis, Xinjian Yu, Stephan Wiedemann, Ruth H Strasse. Apoptosis at a distance: remote activation of caspase-3 occurs early after myocardial infarction. Molecular and cellular biochemistry. vol 281. issue 1-2. 2009-01-09. PMID:16328956. |
besides hypertrophy, processes of apoptosis may contribute to these remodeling processes. |
2009-01-09 |
2023-08-12 |
Not clear |
| Xiaoping Luo, Nasser Chegin. The expression and potential regulatory function of microRNAs in the pathogenesis of leiomyoma. Seminars in reproductive medicine. vol 26. issue 6. 2009-01-07. PMID:18951332. |
it is clear that ovarian steroids are essential for leiomyoma growth, and local expression of many autocrine/paracrine mediators serving as key regulators of cell-cycle progression, cellular hypertrophy, extracellular matrix accumulation, and apoptosis appear to play central roles in this capacity. |
2009-01-07 |
2023-08-12 |
Not clear |
| Yutaka Matsui, Noritsugu Nakano, Dan Shao, Shumin Gao, Wenting Luo, Chull Hong, Peiyong Zhai, Eric Holle, Xianzhong Yu, Norikazu Yabuta, Wufan Tao, Thomas Wagner, Hiroshi Nojima, Junichi Sadoshim. Lats2 is a negative regulator of myocyte size in the heart. Circulation research. vol 103. issue 11. 2008-12-31. PMID:18927464. |
mammalian sterile 20-like kinase (mst)1 plays an important role in mediating apoptosis and inhibiting hypertrophy in the heart. |
2008-12-31 |
2023-08-12 |
mouse |
| Yutaka Matsui, Noritsugu Nakano, Dan Shao, Shumin Gao, Wenting Luo, Chull Hong, Peiyong Zhai, Eric Holle, Xianzhong Yu, Norikazu Yabuta, Wufan Tao, Thomas Wagner, Hiroshi Nojima, Junichi Sadoshim. Lats2 is a negative regulator of myocyte size in the heart. Circulation research. vol 103. issue 11. 2008-12-31. PMID:18927464. |
dn-lats2 significantly attenuated induction of apoptosis and inhibition of hypertrophy by mst1, indicating that lats2 mediates the function of mst1 in cardiac myocytes. |
2008-12-31 |
2023-08-12 |
mouse |
| Yutaka Matsui, Noritsugu Nakano, Dan Shao, Shumin Gao, Wenting Luo, Chull Hong, Peiyong Zhai, Eric Holle, Xianzhong Yu, Norikazu Yabuta, Wufan Tao, Thomas Wagner, Hiroshi Nojima, Junichi Sadoshim. Lats2 is a negative regulator of myocyte size in the heart. Circulation research. vol 103. issue 11. 2008-12-31. PMID:18927464. |
overexpression of dn-lats2 significantly enhanced cardiac hypertrophy and inhibited cardiac myocyte apoptosis induced by transverse aortic constriction. |
2008-12-31 |
2023-08-12 |
mouse |
| Dirk Westermann, Alexander Riad, Olga Lettau, Anton Roks, Konstantinos Savvatis, Peter Moritz Becher, Felicitas Escher, A H Jan Danser, Heinz-Peter Schultheiss, Carsten Tschöp. Renin inhibition improves cardiac function and remodeling after myocardial infarction independent of blood pressure. Hypertension (Dallas, Tex. : 1979). vol 52. issue 6. 2008-12-31. PMID:18955663. |
this was associated with the amelioration of cardiac remodelling, hypertrophy, and apoptosis. |
2008-12-31 |
2023-08-12 |
mouse |
| Nadia Hedhli, Paulo Lizano, Chull Hong, Luke F Fritzky, Sunil K Dhar, Huasheng Liu, Yimin Tian, Shumin Gao, Kiran Madura, Stephen F Vatner, Christophe Depr. Proteasome inhibition decreases cardiac remodeling after initiation of pressure overload. American journal of physiology. Heart and circulatory physiology. vol 295. issue 4. 2008-12-05. PMID:18676687. |
therefore, we propose that proteasome inhibition after the onset of pressure overload rescues ventricular remodeling by stabilizing cardiac function, suppressing further progression of hypertrophy, repressing collagen accumulation, and reducing myocyte apoptosis. |
2008-12-05 |
2023-08-12 |
mouse |
| David Bialostozky, Gerardo Rodríguez-Diez, Cecilia Zazuet. [Apoptosis detection in cardiovascular diseases through nuclear cardiology SPECT images]. Archivos de cardiologia de Mexico. vol 78. issue 2. 2008-12-04. PMID:18754413. |
apoptosis activation in cardiomyocytes is a common problem in a large variety of cardiomyopathies; it has been suggested that it is an important contributor ventricular hypertrophy and to the increase of the infarct size in patients with cardiac failure and cardiovascular disease. |
2008-12-04 |
2023-08-12 |
Not clear |
| Shinichirou Miura, Kazunori Hanaoka, Shin Togash. Skeletogenesis in Xenopus tropicalis: characteristic bone development in an anuran amphibian. Bone. vol 43. issue 5. 2008-12-02. PMID:18692165. |
chondrocytes in the cartilaginous anlagen undergo processes of maturation such as hypertrophy, calcification and apoptosis. |
2008-12-02 |
2023-08-12 |
xenopus_laevis |
| Charles H Rundle, Xiaoguang Wang, Matilda H-C Sheng, Jon E Wergedal, K-H William Lau, Subburaman Moha. Bax deficiency in mice increases cartilage production during fracture repair through a mechanism involving increased chondrocyte proliferation without changes in apoptosis. Bone. vol 43. issue 5. 2008-12-02. PMID:18708175. |
in conclusion, this study demonstrates for the first time that bax has an important role in the early stage of fracture healing, and that the increased callus size and cartilage area in bax ko fractures was due to increased chondrocyte proliferation and not to reduced apoptosis or increased chondrocyte hypertrophy. |
2008-12-02 |
2023-08-12 |
mouse |
| Marco Cassano, Stefano Biressi, Amanda Finan, Laura Benedetti, Claudia Omes, Renata Boratto, Frank Martin, Marcello Allegretti, Vania Broccoli, Gabriella Cusella De Angelis, Paolo M Comoglio, Cristina Basilico, Yvan Torrente, Paolo Michieli, Giulio Cossu, Maurilio Sampaoles. Magic-factor 1, a partial agonist of Met, induces muscle hypertrophy by protecting myogenic progenitors from apoptosis. PloS one. vol 3. issue 9. 2008-11-25. PMID:18795097. |
magic-factor 1, a partial agonist of met, induces muscle hypertrophy by protecting myogenic progenitors from apoptosis. |
2008-11-25 |
2023-08-12 |
Not clear |
| Zhiguo Wang, Xioabin Luo, Yanjie Lu, Baofeng Yan. miRNAs at the heart of the matter. Journal of molecular medicine (Berlin, Germany). vol 86. issue 7. 2008-11-24. PMID:18415070. |
here, we summarize and analyze the currently available data from our own laboratory and other groups, providing a comprehensive overview of mirna function in the heart, including a brief introduction of mirna biology, expression profile of mirnas in cardiac tissue, role of mirnas in cardiac hypertrophy and heart failure, the arrhythmogenic potential of mirnas, the involvement of mirnas in vascular angiogenesis, and regulation of cardiomyocyte apoptosis by mirnas. |
2008-11-24 |
2023-08-12 |
human |
| Rui Sheng, Zhen-lun Gu, Mei-lin Xie, Wen-xuan Zhou, Ci-yi Gu. EGCG inhibits cardiomyocyte apoptosis in pressure overload-induced cardiac hypertrophy and protects cardiomyocytes from oxidative stress in rats. Acta pharmacologica Sinica. vol 28. issue 2. 2008-11-18. PMID:17241521. |
egcg inhibits cardiomyocyte apoptosis in pressure overload-induced cardiac hypertrophy and protects cardiomyocytes from oxidative stress in rats. |
2008-11-18 |
2023-08-12 |
rat |
| Marianna Evans, Kevin Morine, Cyelee Kulkarni, Elisabeth R Barto. Expression profiling reveals heightened apoptosis and supports fiber size economy in the murine muscles of mastication. Physiological genomics. vol 35. issue 1. 2008-11-18. PMID:18593863. |
specifically there are small diameter fibers that have an attenuated ability to hypertrophy, but an increased propensity to undergo apoptosis. |
2008-11-18 |
2023-08-12 |
mouse |
| Ulrich H Frey, Wolfgang Lieb, Jeanette Erdmann, Danai Savidou, Gerd Heusch, Kirsten Leineweber, Heinz Jakob, Hans-Werner Hense, Hannelore Löwel, Norbert H Brockmeyer, Heribert Schunkert, Winfried Siffer. Characterization of the GNAQ promoter and association of increased Gq expression with cardiac hypertrophy in humans. European heart journal. vol 29. issue 7. 2008-11-13. PMID:18326504. |
transgenic mice with cardiac overexpression of gq develop cardiac hypertrophy, apoptosis, and heart failure. |
2008-11-13 |
2023-08-12 |
mouse |