| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| L C Gerstenfeld, F D Shapir. Expression of bone-specific genes by hypertrophic chondrocytes: implication of the complex functions of the hypertrophic chondrocyte during endochondral bone development. Journal of cellular biochemistry. vol 62. issue 1. 1996-12-11. PMID:8836870. |
while it has been widely demonstrated that the majority of hypertrophic chondrocytes undergo apoptosis during terminal stages of the developmental sequence, their response to specific exogenous regulatory signals and their expression of bone-specific proteins give rise to questions about whether all growth chondrocytes have the same developmental fates and have identical functions. |
1996-12-11 |
2023-08-12 |
Not clear |
| F B Key, D McClean, J C Mather. Tissue hypertrophy and epithelial proliferation rate in the gut of rats fed on bread and haricot beans (Phaseolus vulgaris). The British journal of nutrition. vol 76. issue 2. 1996-10-17. PMID:8813901. |
in those instances where enhanced scfa supply is associated with increased epr, the increase may be (1) from a hypoproliferative state towards normal, (2) a transient phenomenon accompanying tissue hypertrophy or (3) a homeostatic response to increased cell loss by cell sloughing or apoptosis. |
1996-10-17 |
2023-08-12 |
rat |
| N Kyprianou, H Tu, S C Jacob. Apoptotic versus proliferative activities in human benign prostatic hyperplasia. Human pathology. vol 27. issue 7. 1996-09-05. PMID:8698310. |
balancing the apoptotic versus the proliferative activities revealed a substantial net decrease (fourfold) in the total number of cells dying via apoptosis in both the glandular and basal epithelial cell compartments of the hypertrophic prostate (bph) when compared with the normal gland. |
1996-09-05 |
2023-08-12 |
human |
| E Teiger, V D Than, L Richard, C Wisnewsky, B S Tea, L Gaboury, J Tremblay, K Schwartz, P Hame. Apoptosis in pressure overload-induced heart hypertrophy in the rat. The Journal of clinical investigation. vol 97. issue 12. 1996-08-12. PMID:8675703. |
apoptosis in pressure overload-induced heart hypertrophy in the rat. |
1996-08-12 |
2023-08-12 |
rat |
| E Teiger, V D Than, L Richard, C Wisnewsky, B S Tea, L Gaboury, J Tremblay, K Schwartz, P Hame. Apoptosis in pressure overload-induced heart hypertrophy in the rat. The Journal of clinical investigation. vol 97. issue 12. 1996-08-12. PMID:8675703. |
these data suggest that cardiac hypertrophy is initiated by a wave of apoptosis of cardiomyocytes. |
1996-08-12 |
2023-08-12 |
rat |
| M Hatori, K J Klatte, C C Teixeira, I M Shapir. End labeling studies of fragmented DNA in the avian growth plate: evidence of apoptosis in terminally differentiated chondrocytes. Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research. vol 10. issue 12. 1996-06-07. PMID:8619377. |
further evidence in support of apoptosis was provided by electron microscopic observation of cells in the hypertrophic region of the growth plate. |
1996-06-07 |
2023-08-12 |
human |
| M Hatori, K J Klatte, C C Teixeira, I M Shapir. End labeling studies of fragmented DNA in the avian growth plate: evidence of apoptosis in terminally differentiated chondrocytes. Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research. vol 10. issue 12. 1996-06-07. PMID:8619377. |
finally, we used flow cytometry to analyze chondrocytes isolated from the proliferating and hypertrophic regions of the growth plate for apoptosis. |
1996-06-07 |
2023-08-12 |
human |
| A M Kat. Cell death in the failing heart: role of an unnatural growth response to overload. Clinical cardiology. vol 18. issue 9 Suppl 4. 1996-01-04. PMID:7489619. |
instead, overload initiates an unnatural growth response that appears to shorten cardiac myocyte survival, possibly because the same growth factors that mediate the hypertrophic response of the adult heart can also induce programmed cell death (apoptosis). |
1996-01-04 |
2023-08-12 |
Not clear |
| H I Roach, J Erenpreisa, T Aigne. Osteogenic differentiation of hypertrophic chondrocytes involves asymmetric cell divisions and apoptosis. The Journal of cell biology. vol 131. issue 2. 1995-12-01. PMID:7593173. |
osteogenic differentiation of hypertrophic chondrocytes involves asymmetric cell divisions and apoptosis. |
1995-12-01 |
2023-08-12 |
chicken |
| D M Chisholm, M M Adi, I M Ervine, G R Ogde. Cell deletion by apoptosis during regression of rat parotid sialadenosis. Virchows Archiv : an international journal of pathology. vol 427. issue 2. 1995-11-27. PMID:7582249. |
following withdrawal of the drug, quantitative histological techniques were used to investigate the balance between hypertrophy, hyperplasia and apoptosis. |
1995-11-27 |
2023-08-12 |
rat |
| T N Jame. Normal and abnormal consequences of apoptosis in the human heart: from postnatal morphogenesis to paroxysmal arrhythmias. Transactions of the American Clinical and Climatological Association. vol 105. 1994-12-21. PMID:7974966. |
these features make apoptosis a valuable component of morphogenesis, mediation of hormonal and immunological responses, and the homeostatic balance between hypertrophy and atrophy or involution. |
1994-12-21 |
2023-08-12 |
human |
| T N Jame. Normal and abnormal consequences of apoptosis in the human heart. From postnatal morphogenesis to paroxysmal arrhythmias. Circulation. vol 90. issue 1. 1994-08-11. PMID:8026044. |
these features make apoptosis a valuable component of morphogenesis, mediation of hormonal and immunologic responses, and the homeostatic balance between hypertrophy and atrophy or involution. |
1994-08-11 |
2023-08-12 |
human |
| D Lewinson, M Silberman. Chondroclasts and endothelial cells collaborate in the process of cartilage resorption. The Anatomical record. vol 233. issue 4. 1992-08-13. PMID:1626710. |
in this ultrastructural study we demonstrate that the terminal hypertrophic chondrocytes undergo apoptosis and disintegration while simultaneously chondroclasts dissolve gaps in the calcified cartilage that engulfs them. |
1992-08-13 |
2023-08-11 |
rat |
| C W Kische. The microvessels in hypertrophic scars, keloids and related lesions: a review. Journal of submicroscopic cytology and pathology. vol 24. issue 2. 1992-07-15. PMID:1600518. |
differential degeneration, or apoptosis, of the fibroblasts, pericytes and microvessels occurs from granulation tissue through hypertrophic scarring. |
1992-07-15 |
2023-08-11 |
Not clear |
| H G Verhage, P A Mavrogianis, M L Boice, W Li, A T Fazleaba. Oviductal epithelium of the baboon: hormonal control and the immuno-gold localization of oviduct-specific glycoproteins. The American journal of anatomy. vol 187. issue 1. 1990-02-12. PMID:2296912. |
within the ampulla, estradiol induced hypertrophy, hyperplasia, ciliogenesis, and secretory activity, whereas adding progesterone to the treatment regimen (+/- estradiol) resulted in atrophy, deciliation, apoptosis, and loss of the secretory activity. |
1990-02-12 |
2023-08-11 |
Not clear |