| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| R Venezian, B J Shenker, S Datar, P S Lebo. Modulation of chondrocyte proliferation by ascorbic acid and BMP-2. Journal of cellular physiology. vol 174. issue 3. 1998-02-13. PMID:9462695. |
these results imply that in chondrocytes the transition to hypertrophy is associated with a decreased number of proliferating cells and a relatively high level of apoptosis. |
1998-02-13 |
2023-08-12 |
chicken |
| D R Meldrum, R Shenkar, B C Sheridan, B S Cain, E Abraham, A H Harke. Hemorrhage activates myocardial NFkappaB and increases TNF-alpha in the heart. Journal of molecular and cellular cardiology. vol 29. issue 10. 1998-02-03. PMID:9344778. |
locally (v systemically)-produced tnfalpha likely contributes to myocardial dysfunction via direct suppression of myocardial contractile function, the induction of myocardial apoptosis, and the genesis of cardiac hypertrophy. |
1998-02-03 |
2023-08-12 |
rat |
| W S Colucc. Molecular and cellular mechanisms of myocardial failure. The American journal of cardiology. vol 80. issue 11A. 1998-01-20. PMID:9412539. |
these alterations include hypertrophy and cellular apoptosis of myocytes, changes in the molecular phenotype of the myocardium with reinduction of a fetal gene program, and alterations in the quantity and composition of the extracellular matrix. |
1998-01-20 |
2023-08-12 |
Not clear |
| J Díez, A Panizo, M Hernández, F Vega, I Sola, M A Fortuño, J Pard. Cardiomyocyte apoptosis and cardiac angiotensin-converting enzyme in spontaneously hypertensive rats. Hypertension (Dallas, Tex. : 1979). vol 30. issue 5. 1997-12-10. PMID:9369251. |
increased apoptosis has been reported in the heart of rats with spontaneous hypertension and cardiac hypertrophy. |
1997-12-10 |
2023-08-12 |
rat |
| J Díez, A Panizo, M Hernández, F Vega, I Sola, M A Fortuño, J Pard. Cardiomyocyte apoptosis and cardiac angiotensin-converting enzyme in spontaneously hypertensive rats. Hypertension (Dallas, Tex. : 1979). vol 30. issue 5. 1997-12-10. PMID:9369251. |
this study was designed to investigate the relationship between apoptosis and hypertrophy in cardiomyocytes from the left ventricle of spontaneously hypertensive rats (shr). |
1997-12-10 |
2023-08-12 |
rat |
| J Díez, A Panizo, M Hernández, F Vega, I Sola, M A Fortuño, J Pard. Cardiomyocyte apoptosis and cardiac angiotensin-converting enzyme in spontaneously hypertensive rats. Hypertension (Dallas, Tex. : 1979). vol 30. issue 5. 1997-12-10. PMID:9369251. |
these results suggest that the timing and mechanisms responsible for apoptosis and hypertrophy of cardiomyocytes are different in shr. |
1997-12-10 |
2023-08-12 |
rat |
| J Díez, A Panizo, M Hernández, F Vega, I Sola, M A Fortuño, J Pard. Cardiomyocyte apoptosis and cardiac angiotensin-converting enzyme in spontaneously hypertensive rats. Hypertension (Dallas, Tex. : 1979). vol 30. issue 5. 1997-12-10. PMID:9369251. |
whereas hypertrophy seems to be an earlier alteration that develops in parallel with hypertension, apoptosis develops later in association with overactivity of the local ace. |
1997-12-10 |
2023-08-12 |
rat |
| R Jaffe, M Y Flugelman, D A Halon, B S Lewi. Ventricular remodeling: from bedside to molecule. Advances in experimental medicine and biology. vol 430. 1997-11-19. PMID:9330735. |
in many instances, however, progressive ventricular dilatation and heart failure occur as a result of maladaptive hypertrophy (abnormal myosin-actin production), programmed cell death (apoptosis) and/or changes in the interstitial vasculature and collagen composition. |
1997-11-19 |
2023-08-12 |
Not clear |
| C A Praul, C V Gay, R M Leac. Chondrocytes of the tibial dyschondroplastic lesion are apoptotic. The International journal of developmental biology. vol 41. issue 4. 1997-10-09. PMID:9303350. |
genomic dna isolated from cells of severe td lesions show the nucleosomal laddering indicative of apoptosis, while dna isolated from proliferative and hypertrophic cells does not. |
1997-10-09 |
2023-08-12 |
Not clear |
| Y Ishii, S Tsutsui, K Doi, S Itagak. Hair follicles of young Wistar strain hairless rats: a histological study. Journal of anatomy. vol 191 ( Pt 1). 1997-09-18. PMID:9279663. |
in late anagen phase, the follicles in which the apoptosis did not occur showed enlarged hair roots with hypertrophy of the inner root sheath. |
1997-09-18 |
2023-08-12 |
rat |
| S Mesiano, R B Jaff. Developmental and functional biology of the primate fetal adrenal cortex. Endocrine reviews. vol 18. issue 3. 1997-08-06. PMID:9183569. |
more recently, it has become clear that fetal adrenal cortical growth involves cellular hypertrophy, hyperplasia, apoptosis, and migration and is best described by the migration theory, i.e. |
1997-08-06 |
2023-08-12 |
human |
| L G Gomella, B W Godwi. This month in investigative urology. Apoptosis and benign prostatic hypertrophy. The Journal of urology. vol 158. issue 1. 1997-07-14. PMID:9186312. |
apoptosis and benign prostatic hypertrophy. |
1997-07-14 |
2023-08-12 |
Not clear |
| G Gibson, D L Lin, M Roqu. Apoptosis of terminally differentiated chondrocytes in culture. Experimental cell research. vol 233. issue 2. 1997-07-10. PMID:9194499. |
we conclude that commitment of chondrocytes to terminal differentiation is associated with a commitment to apoptosis and apoptosis of hypertrophic chondrocytes in growth cartilage does not require initiation by external signals. |
1997-07-10 |
2023-08-12 |
chicken |
| C E Sarraf, M Horgan, R J Edwards, M R Aliso. Reversal of phenobarbital-induced hyperplasia and hypertrophy in the livers of lpr mice. International journal of experimental pathology. vol 78. issue 1. 1997-06-13. PMID:9166105. |
phenobarbital treatment of experimental animals induces cytochrome p450 enzymes, and thus acts as a growth stimulus to the liver with both hyperplasia and hypertrophy; cessation results in reversion of liver to normal size with apoptosis playing a role. |
1997-06-13 |
2023-08-12 |
mouse |
| P Zhang, N J Liégeois, C Wong, M Finegold, H Hou, J C Thompson, A Silverman, J W Harper, R A DePinho, S J Elledg. Altered cell differentiation and proliferation in mice lacking p57KIP2 indicates a role in Beckwith-Wiedemann syndrome. Nature. vol 387. issue 6629. 1997-05-28. PMID:9144284. |
mice lacking the imprinted cdk inhibitor p57(kip2) have altered cell proliferation and differentiation, leading to abdominal muscle defects; cleft palate; endochondral bone ossification defects with incomplete differentiation of hypertrophic chondrocytes; renal medullary dysplasia; adrenal cortical hyperplasia and cytomegaly; and lens cell hyperproliferation and apoptosis. |
1997-05-28 |
2023-08-12 |
mouse |
| T Ino, K Nishimoto, M Okubo, K Akimoto, K Yabuta, S Kawai, R Okada, N Sueyosh. Apoptosis as a possible cause of wall thinning in end-stage hypertrophic cardiomyopathy. The American journal of cardiology. vol 79. issue 8. 1997-05-08. PMID:9114784. |
apoptosis as a possible cause of wall thinning in end-stage hypertrophic cardiomyopathy. |
1997-05-08 |
2023-08-12 |
Not clear |
| A Desmoulière, C Badid, M L Bochaton-Piallat, G Gabbian. Apoptosis during wound healing, fibrocontractive diseases and vascular wall injury. The international journal of biochemistry & cell biology. vol 29. issue 1. 1997-04-25. PMID:9076938. |
during excessive scarring (hypertrophic scar or fibrosis), it is conceivable that the process of apoptosis cannot take place. |
1997-04-25 |
2023-08-12 |
human |
| M W Chen, R M Levin, R Buttya. Peptide growth factors in normal and hypertrophied bladder. World journal of urology. vol 13. issue 6. 1997-04-24. PMID:9116753. |
the extensive tissue remodeling associated with each of these changes requires different bladder components to undergo cellular proliferation, cellular hypertrophy, and even programmed cell death (apoptosis). |
1997-04-24 |
2023-08-12 |
Not clear |
| P Hamet, P Moreau, T V Dam, S N Orlov, B S Tea, D de Blois, J Trembla. The time window of apoptosis: a new component in the therapeutic strategy for cardiovascular remodeling. Journal of hypertension. Supplement : official journal of the International Society of Hypertension. vol 14. issue 5. 1997-04-24. PMID:9120687. |
apoptosis and experimental hypertrophy: apoptosis (programmed cell death) is a physiological counterpart of cell replication with shared as well as specific pathways. |
1997-04-24 |
2023-08-12 |
mouse |
| D deBlois, B S Tea, V D Than, J Tremblay, P Hame. Smooth muscle apoptosis during vascular regression in spontaneously hypertensive rats. Hypertension (Dallas, Tex. : 1979). vol 29. issue 1 Pt 2. 1997-03-13. PMID:9039125. |
as an initial in vivo exploration, we now examined smooth muscle cell apoptosis regulation during the regression of vascular hypertrophy in the thoracic aorta media of spontaneously hypertensive rats receiving the antihypertensive drug enalapril (30 mg.kg-1.d-1), losartan (30 mg.kg-1.d-1), nifedipine (35 mg.kg-1.d-1), hydralazine (40 mg.kg-1.d-1), propranolol (50 mg.kg-1.d-1), or hydrochlorothiazide (75 mg.kg-1.d-1) for 1 to 4 weeks starting at 10 to 11 weeks of age. |
1997-03-13 |
2023-08-12 |
rat |