| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| Fanghao Lu, Jun Xing, Xinying Zhang, Shiyun Dong, Yajun Zhao, Lina Wang, Hulun Li, Fan Yang, Changqing Xu, Weihua Zhan. Exogenous hydrogen sulfide prevents cardiomyocyte apoptosis from cardiac hypertrophy induced by isoproterenol. Molecular and cellular biochemistry. vol 381. issue 1-2. 2014-02-12. PMID:23660955. |
oxidative stress is a crucial factor inducing cardiomyocyte apoptosis due to cardiac hypertrophy. |
2014-02-12 |
2023-08-12 |
Not clear |
| Fanghao Lu, Jun Xing, Xinying Zhang, Shiyun Dong, Yajun Zhao, Lina Wang, Hulun Li, Fan Yang, Changqing Xu, Weihua Zhan. Exogenous hydrogen sulfide prevents cardiomyocyte apoptosis from cardiac hypertrophy induced by isoproterenol. Molecular and cellular biochemistry. vol 381. issue 1-2. 2014-02-12. PMID:23660955. |
hence, we aimed to elucidate whether h2s prevents cardiomyocyte apoptosis due to cardiac hypertrophy via its antioxidant function. |
2014-02-12 |
2023-08-12 |
Not clear |
| Christian Hill, Alona Würfel, Jacqueline Heger, Bettina Meyering, Klaus-Dieter Schlüter, Martin Weber, Peter Ferdinandy, Ami Aronheim, Rainer Schulz, Gerhild Eule. Inhibition of AP-1 signaling by JDP2 overexpression protects cardiomyocytes against hypertrophy and apoptosis induction. Cardiovascular research. vol 99. issue 1. 2014-01-31. PMID:23612584. |
inhibition of ap-1 signaling by jdp2 overexpression protects cardiomyocytes against hypertrophy and apoptosis induction. |
2014-01-31 |
2023-08-12 |
mouse |
| Christian Hill, Alona Würfel, Jacqueline Heger, Bettina Meyering, Klaus-Dieter Schlüter, Martin Weber, Peter Ferdinandy, Ami Aronheim, Rainer Schulz, Gerhild Eule. Inhibition of AP-1 signaling by JDP2 overexpression protects cardiomyocytes against hypertrophy and apoptosis induction. Cardiovascular research. vol 99. issue 1. 2014-01-31. PMID:23612584. |
in order to test if ap-1 inhibition may limit processes contributing to cardiac remodelling, ventricular cardiomyocytes of mice with cardiac overexpression of the ap-1 inhibitor jdp2 were analysed under stimulation of hypertrophy, apoptosis, or contractile function. |
2014-01-31 |
2023-08-12 |
mouse |
| Mark K Friedberg, Mi-Young Cho, Jing Li, Renato S Assad, Mei Sun, Sagar Rohailla, Osami Honjo, Christian Apitz, Andrew N Redingto. Adverse biventricular remodeling in isolated right ventricular hypertension is mediated by increased transforming growth factor-β1 signaling and is abrogated by angiotensin receptor blockade. American journal of respiratory cell and molecular biology. vol 49. issue 6. 2014-01-27. PMID:23841477. |
five weeks after surgery, the animals were killed for assessments of biventricular hypertrophy, fibrosis, apoptosis, and the components of their signaling pathways. |
2014-01-27 |
2023-08-12 |
Not clear |
| Mark K Friedberg, Mi-Young Cho, Jing Li, Renato S Assad, Mei Sun, Sagar Rohailla, Osami Honjo, Christian Apitz, Andrew N Redingto. Adverse biventricular remodeling in isolated right ventricular hypertension is mediated by increased transforming growth factor-β1 signaling and is abrogated by angiotensin receptor blockade. American journal of respiratory cell and molecular biology. vol 49. issue 6. 2014-01-27. PMID:23841477. |
pab animals developed biventricular hypertrophy, fibrosis, and apoptosis, versus sham rabbits, in which these conditions were decreased with losartan. |
2014-01-27 |
2023-08-12 |
Not clear |
| Xianrong Zhang, Ji Zhu, Yumei Li, Tiao Lin, Valerie A Siclari, Abhishek Chandra, Elena M Candela, Eiki Koyama, Motomi Enomoto-Iwamoto, Ling Qi. Epidermal growth factor receptor (EGFR) signaling regulates epiphyseal cartilage development through β-catenin-dependent and -independent pathways. The Journal of biological chemistry. vol 288. issue 45. 2014-01-20. PMID:24047892. |
this delay was accompanied by normal chondrocyte hypertrophy but decreased mineralization and apoptosis of hypertrophic chondrocytes and reduced osteoclast number at the border of marrow space. |
2014-01-20 |
2023-08-12 |
mouse |
| Kevin A Lawson, Colin J Teteak, Junhui Zou, Jacques Hacquebord, Andrew Ghatan, Anna Zielinska-Kwiatkowska, Russell J Fernandes, Howard A Chansky, Liu Yan. Mesenchyme-specific knockout of ESET histone methyltransferase causes ectopic hypertrophy and terminal differentiation of articular chondrocytes. The Journal of biological chemistry. vol 288. issue 45. 2014-01-20. PMID:24056368. |
at 1 month, ectopic hypertrophy, proliferation, and apoptosis of articular chondrocytes were seen in the articular cartilage of eset-null animals. |
2014-01-20 |
2023-08-12 |
mouse |
| U Bahrudin, N Ikeda, S B Utami, N Maharani, K Morikawa, P Li, M A Sobirin, A Hasegawa, S Sakata, R Endo, S Rifqi, Y Shirayoshi, K Yamamoto, H Ninomiya, I Hisatom. Simultaneous treatment with azelnidipine and olmesartan inhibits apoptosis of Hl-1 cardiac myocytes expressing E334k cMyBPC. Drug research. vol 63. issue 10. 2014-01-16. PMID:23740383. |
apoptosis appears to play an important role in the pathogenesis of hypertrophic cardiomyopathy (hcm). |
2014-01-16 |
2023-08-12 |
Not clear |
| Shanshan Zhou, Yucheng Liu, Kendell Prater, Yang Zheng, Lu Ca. Roles of microRNAs in pressure overload- and ischemia-related myocardial remodeling. Life sciences. vol 93. issue 23. 2014-01-13. PMID:24021888. |
in clinic, pressure overload and ischemia are the two most common reasons to induce cardiac remodeling and hf, which includes but is not limited to cardiac hypertrophy, fibrosis, and cardiomyocyte apoptosis. |
2014-01-13 |
2023-08-12 |
Not clear |
| Tong Tang, N Chin Lai, Adam T Wright, Mei Hua Gao, Paul Lee, Tracy Guo, Ruoying Tang, Andrew D McCulloch, H Kirk Hammon. Adenylyl cyclase 6 deletion increases mortality during sustained β-adrenergic receptor stimulation. Journal of molecular and cellular cardiology. vol 60. 2013-12-31. PMID:23587598. |
there were no group differences in left ventricular hypertrophy, apoptosis, and fibrosis. |
2013-12-31 |
2023-08-12 |
mouse |
| Wenliang Zhu, Yilei Zhao, Yingqi Xu, Yong Sun, Zhe Wang, Wei Yuan, Zhimin D. Dissection of protein interactomics highlights microRNA synergy. PloS one. vol 8. issue 5. 2013-12-30. PMID:23691029. |
the synergistic effect of mir-21 and mir-1 were functionally validated for their significant influences on myocardial apoptosis, cardiac hypertrophy and fibrosis. |
2013-12-30 |
2023-08-12 |
human |
| Fang-Hao Lu, Song-Bin Fu, Xiaoning Leng, Xinying Zhang, Shiyun Dong, Ya-Jun Zhao, Huan Ren, Hulun Li, Xin Zhong, Chang-Qing Xu, Wei-Hua Zhan. Role of the calcium-sensing receptor in cardiomyocyte apoptosis via the sarcoplasmic reticulum and mitochondrial death pathway in cardiac hypertrophy and heart failure. Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology. vol 31. issue 4-5. 2013-12-17. PMID:23711498. |
role of the calcium-sensing receptor in cardiomyocyte apoptosis via the sarcoplasmic reticulum and mitochondrial death pathway in cardiac hypertrophy and heart failure. |
2013-12-17 |
2023-08-12 |
Not clear |
| Wan-Gang Guo, Zhi-Bin Yu, Man-Jiang Xi. [Protein kinase Cdelta is possibly involved in the transition from hypertrophy to apoptosis of myocardiocytes]. Sheng li xue bao : [Acta physiologica Sinica]. vol 58. issue 3. 2013-12-12. PMID:16786112. |
[protein kinase cdelta is possibly involved in the transition from hypertrophy to apoptosis of myocardiocytes]. |
2013-12-12 |
2023-08-12 |
Not clear |
| Wan-Gang Guo, Zhi-Bin Yu, Man-Jiang Xi. [Protein kinase Cdelta is possibly involved in the transition from hypertrophy to apoptosis of myocardiocytes]. Sheng li xue bao : [Acta physiologica Sinica]. vol 58. issue 3. 2013-12-12. PMID:16786112. |
in the present study, experiments were designed to explore the susceptibility of hypertrophic myocardiocytes to apoptotic stimuli and the role of protein kinase cdelta (pkcdelta) during the transition from hypertrophy to apoptosis. |
2013-12-12 |
2023-08-12 |
Not clear |
| Wan-Gang Guo, Zhi-Bin Yu, Man-Jiang Xi. [Protein kinase Cdelta is possibly involved in the transition from hypertrophy to apoptosis of myocardiocytes]. Sheng li xue bao : [Acta physiologica Sinica]. vol 58. issue 3. 2013-12-12. PMID:16786112. |
these results suggest that inhibition of pkcdelta can reduce ang ii-induced apoptosis of hypertrophic cardiomyocytes and that pkcdelta is possibly involved in the apoptotic process of hypertrophic cardiomyocytes. |
2013-12-12 |
2023-08-12 |
Not clear |
| Gustavo Blanco, Darren P Wallac. Novel role of ouabain as a cystogenic factor in autosomal dominant polycystic kidney disease. American journal of physiology. Renal physiology. vol 305. issue 6. 2013-12-11. PMID:23761677. |
however, the new and unexpected role of the na-k-atpase as a signal transducer revealed a novel facet for ouabain in the regulation of a myriad of cell functions, including cell proliferation, hypertrophy, apoptosis, mobility, and metabolism. |
2013-12-11 |
2023-08-12 |
Not clear |
| Ivonne Loeffler, Christiane Rüster, Sybille Franke, Marita Liebisch, Gunter Wol. Erythropoietin ameliorates podocyte injury in advanced diabetic nephropathy in the db/db mouse. American journal of physiology. Renal physiology. vol 305. issue 6. 2013-12-11. PMID:23825071. |
the pathophysiology of age-challenged podocytes, such as hypertrophy, apoptosis, and reduced cell migration, is closely related to the induction of the cell cycle inhibitor p27(kip1) and to the inhibition of neuropilin 1 (nrp1). |
2013-12-11 |
2023-08-12 |
mouse |
| Bing Feng, Wei Liu, Jing Xu, Zuo-Yun He, Hui-Biao Yan. [Relationship between apoptosis and alteration of the energetic metabolism pathways of hypertrophic cardiomyocytes induced by hypoxia-reoxygenation]. Sheng li xue bao : [Acta physiologica Sinica]. vol 57. issue 5. 2013-12-05. PMID:16220203. |
[relationship between apoptosis and alteration of the energetic metabolism pathways of hypertrophic cardiomyocytes induced by hypoxia-reoxygenation]. |
2013-12-05 |
2023-08-12 |
Not clear |
| Bing Feng, Wei Liu, Jing Xu, Zuo-Yun He, Hui-Biao Yan. [Relationship between apoptosis and alteration of the energetic metabolism pathways of hypertrophic cardiomyocytes induced by hypoxia-reoxygenation]. Sheng li xue bao : [Acta physiologica Sinica]. vol 57. issue 5. 2013-12-05. PMID:16220203. |
the apoptosis of cardiomyocytes plays a pivotal role in the pathogenesis of cardiac failure transformed from cardiac hypertrophy, so that suppression of cardiomyocytes apoptosis is an effective pharmacotherapeutic target to prevent cardiac failure. |
2013-12-05 |
2023-08-12 |
Not clear |