All Relations between hypertrophic and matrix compartment

Publication Sentence Publish Date Extraction Date Species
Maria Mirotsou, Victor J Dzau, Richard E Pratt, Ellen O Weinber. Physiological genomics of cardiac disease: quantitative relationships between gene expression and left ventricular hypertrophy. Physiological genomics. vol 27. issue 1. 2006-11-02. PMID:16835353. of genes with significant correlations to numerically continuous measurements of hypertrophy, we found enrichment for genes encoding extracellular matrix, growth-related and secreted proteins in the directly correlated subset, and for genes encoding mitochondria and metabolic/fatty acid oxidation proteins in the inversely correlated subset. 2006-11-02 2023-08-12 mouse
Yung-Tsung Chiu, Ching-Chang Cheng, Nai-Nu Lin, Yi-Wen Hung, Ying-Tsung Chen, Shih-Lan Hsu, Ching-Shiang Chi, Yun-Ching F. High-dose norepinephrine induces disruption of myocardial extracellular matrix and left ventricular dilatation and dysfunction in a novel feline model. Journal of the Chinese Medical Association : JCMA. vol 69. issue 8. 2006-10-26. PMID:16970269. intravenous norepinephrine (ne) at a dose of 1-6 microg/kg/minute can induce increased extracellular matrix (ecm) and hypertrophic cardiomyopathy. 2006-10-26 2023-08-12 Not clear
Sada Boodoo, Ernst W Spannhake, Jonathan D Powell, Maureen R Horto. Differential regulation of hyaluronan-induced IL-8 and IP-10 in airway epithelial cells. American journal of physiology. Lung cellular and molecular physiology. vol 291. issue 3. 2006-10-05. PMID:16581825. at the cellular level, the hallmarks of airway inflammation are mucus gland hypertrophy with excess mucus production, accumulation of inflammatory mediators, inflammation in the airway walls and lumen, and breakdown and turnover of the extracellular matrix. 2006-10-05 2023-08-12 Not clear
Hiroyuki Tsutsu. Mitochondrial oxidative stress and heart failure. Internal medicine (Tokyo, Japan). vol 45. issue 13. 2006-09-15. PMID:16880705. reactive oxygen species induce myocyte hypertrophy, apoptosis, and interstitial fibrosis by activating matrix metalloproteinases. 2006-09-15 2023-08-12 Not clear
Siham Madani, Sabrina De Girolamo, Diana Marcela Muñoz, Ren-Ke Li, Gary Sweene. Direct effects of leptin on size and extracellular matrix components of human pediatric ventricular myocytes. Cardiovascular research. vol 69. issue 3. 2006-08-18. PMID:16376323. both extracellular matrix remodeling and left ventricular hypertrophy are well-defined components of remodeling in heart failure, and here we further investigate the role of leptin, the obese gene product, on these parameters. 2006-08-18 2023-08-12 human
Susanne Rutschow, Jun Li, Heinz-Peter Schultheiss, Matthias Pauschinge. Myocardial proteases and matrix remodeling in inflammatory heart disease. Cardiovascular research. vol 69. issue 3. 2006-08-18. PMID:16417902. the myocardial inflammatory reaction not only affects myocardial hypertrophy and apoptosis, but it has a major influence on the regulation of extracellular matrix turnover. 2006-08-18 2023-08-12 Not clear
Ernestina Schipan. Hypoxia and HIF-1alpha in chondrogenesis. Annals of the New York Academy of Sciences. vol 1068. 2006-08-08. PMID:16831906. in endochondral bone development chondrocytes undergo proliferation, hypertrophic differentiation, mineralization of the surrounding matrix, death, blood vessel invasion, and finally replacement of cartilage with bone. 2006-08-08 2023-08-12 Not clear
Ernestina Schipan. Hypoxia and HIF-1 alpha in chondrogenesis. Seminars in cell & developmental biology. vol 16. issue 4-5. 2006-07-14. PMID:16144691. in endochondral bone development chondrocytes undergo well-ordered and controlled phases of proliferation, hypertrophic differentiation, mineralization of the surrounding matrix, death, blood vessel invasion, and finally replacement of cartilage with bone. 2006-07-14 2023-08-12 Not clear
Xiangsheng Feng, Jiaju Tan, Yingen Pan, Qiuhe Wu, Shubin Ruan, Rui Shen, Xiaodong Chen, Yongjun D. Control of hypertrophic scar from inception by using xenogenic (porcine) acellular dermal matrix (ADM) to cover deep second degree burn. Burns : journal of the International Society for Burn Injuries. vol 32. issue 3. 2006-06-29. PMID:16487662. control of hypertrophic scar from inception by using xenogenic (porcine) acellular dermal matrix (adm) to cover deep second degree burn. 2006-06-29 2023-08-12 Not clear
C W Park, Y Zhang, X Zhang, J Wu, L Chen, D R Cha, D Su, M-T Hwang, X Fan, L Davis, G Striker, F Zheng, M Breyer, Y Gua. PPARalpha agonist fenofibrate improves diabetic nephropathy in db/db mice. Kidney international. vol 69. issue 9. 2006-06-21. PMID:16672921. this was accompanied by dramatically reduced glomerular hypertrophy and mesangial matrix expansion. 2006-06-21 2023-08-12 mouse
Shintaro Kinugawa, Hiroyuki Tsutsu. [Oxidative stress and heart failure]. Nihon rinsho. Japanese journal of clinical medicine. vol 64. issue 5. 2006-06-15. PMID:16689364. the excessive reactive oxygen species (ros), produced by the electron leak in mitochondria of failing myocardium, play an important role in development and progression of heart failure (hf) and cardiac remodeling, which is associated with induction of myocyte hypertrophy and apoptosis and activation of matrix metalloproteinases. 2006-06-15 2023-08-12 Not clear
T Pantsulai. Role of TGF-beta in pathogenesis of diabetic nephropathy. Georgian medical news. issue 131. 2006-06-12. PMID:16575121. diabetic nephropathy, a major long-term complication of diabetes mellitus is characterized by hypertrophy of the glomerular and tubuloepithelial structures and thickening of glomerular and tubular basement membranes and progressive accumulation of extracellular matrix proteins (fibronectin, collagens and laminin) in the mesangium and the interstitium. 2006-06-12 2023-08-12 Not clear
T Pantsulai. Role of TGF-beta in pathogenesis of diabetic nephropathy. Georgian medical news. issue 131. 2006-06-12. PMID:16575121. of these, the cytokine transforming growth factor (tgf-beta) has emerged as having a key role in the development of renal hypertrophy and accumulation of extracellular matrix in diabetes. 2006-06-12 2023-08-12 Not clear
Joji Ishikawa, Kazuomi Kario, Yoshio Matsui, Seiichi Shibasaki, Masato Morinari, Ruri Kaneda, Satoshi Hoshide, Kazuo Eguchi, Yukihiro Hojo, Kazuyuki Shimad. Collagen metabolism in extracellular matrix may be involved in arterial stiffness in older hypertensive patients with left ventricular hypertrophy. Hypertension research : official journal of the Japanese Society of Hypertension. vol 28. issue 12. 2006-06-05. PMID:16671339. collagen metabolism in extracellular matrix may be involved in arterial stiffness in older hypertensive patients with left ventricular hypertrophy. 2006-06-05 2023-08-12 Not clear
Hsiu-Wen Chan, Nicola J Smith, Ross D Hannan, Walter G Thoma. Tackling the EGFR in pathological tissue remodelling. Pulmonary pharmacology & therapeutics. vol 19. issue 1. 2006-05-11. PMID:15979363. tissue remodelling is an adaptive physiological event initiated by physical and/or hormonal stimuli and characterised by extracellular matrix modifications, inflammation, cellular hypertrophy, proliferation and/or apoptosis. 2006-05-11 2023-08-12 Not clear
John V Bonacci, Alastair G Stewar. Regulation of human airway mesenchymal cell proliferation by glucocorticoids and beta2-adrenoceptor agonists. Pulmonary pharmacology & therapeutics. vol 19. issue 1. 2006-05-11. PMID:16286235. one of the most consistently observed changes in asthmatic airways is an increased volume of airway smooth muscle (asm), that has been explained by proliferation, hypertrophy, extracellular matrix deposition within the smooth muscle bundles, and more recently, the migration of mesenchymal precursor cells to the airways. 2006-05-11 2023-08-12 human
V J Karthikeyan, Gregory Y H Li. Matrix metalloproteinases and hypertension: a link between left ventricular hypertrophy and diastolic dysfunction? The Tohoku journal of experimental medicine. vol 208. issue 2. 2006-05-10. PMID:16434829. matrix metalloproteinases and hypertension: a link between left ventricular hypertrophy and diastolic dysfunction? 2006-05-10 2023-08-12 Not clear
Mustafa Saglam, Osman Karakaya, Ali Metin Esen, Irfan Barutcu, Serkan Dogan, Yusuf Karavelioglu, Hekim Karapinar, Taylan Akgun, Ozlem Esen, Nihal Ozdemir, Sembol Turkmen, Cihangir Kayma. Contribution of plasma matrix metalloproteinases to development of left ventricular hypertrophy and diastolic dysfunction in hypertensive subjects. The Tohoku journal of experimental medicine. vol 208. issue 2. 2006-05-10. PMID:16434834. contribution of plasma matrix metalloproteinases to development of left ventricular hypertrophy and diastolic dysfunction in hypertensive subjects. 2006-05-10 2023-08-12 human
Mustafa Saglam, Osman Karakaya, Ali Metin Esen, Irfan Barutcu, Serkan Dogan, Yusuf Karavelioglu, Hekim Karapinar, Taylan Akgun, Ozlem Esen, Nihal Ozdemir, Sembol Turkmen, Cihangir Kayma. Contribution of plasma matrix metalloproteinases to development of left ventricular hypertrophy and diastolic dysfunction in hypertensive subjects. The Tohoku journal of experimental medicine. vol 208. issue 2. 2006-05-10. PMID:16434834. matrix metalloproteinases (mmps) are involved in the regulation of the extracellular matrix (ecm) of the myocardium and thus the pathogenesis of vascular and cardiac hypertrophy. 2006-05-10 2023-08-12 human
Mustafa Saglam, Osman Karakaya, Ali Metin Esen, Irfan Barutcu, Serkan Dogan, Yusuf Karavelioglu, Hekim Karapinar, Taylan Akgun, Ozlem Esen, Nihal Ozdemir, Sembol Turkmen, Cihangir Kayma. Contribution of plasma matrix metalloproteinases to development of left ventricular hypertrophy and diastolic dysfunction in hypertensive subjects. The Tohoku journal of experimental medicine. vol 208. issue 2. 2006-05-10. PMID:16434834. in this study, we investigated contribution of plasma matrix metalloproteinases to development of left ventricular hypertrophy (lvh) and diastolic dysfunction in hypertensive subjects. 2006-05-10 2023-08-12 human