All Relations between hypertrophic and matrix compartment

Publication Sentence Publish Date Extraction Date Species
A D Hodgkinson, T Page, B A Millward, A G Demain. A novel polymorphism in the 5' flanking region of the glucose transporter (GLUT1) gene is strongly associated with diabetic nephropathy in patients with Type 1 diabetes mellitus. Journal of diabetes and its complications. vol 19. issue 2. 2005-06-30. PMID:15745834. glucose transporter 1 (glut1) activity has been implicated in renal hypertrophy and extracellular matrix formation in mesangial cells. 2005-06-30 2023-08-12 Not clear
Young-Sook Kim, Zhong-Gao Xu, Marpadga A Reddy, Shu-Lian Li, Linda Lanting, Kumar Sharma, Sharon G Adler, Rama Nataraja. Novel interactions between TGF-{beta}1 actions and the 12/15-lipoxygenase pathway in mesangial cells. Journal of the American Society of Nephrology : JASN. vol 16. issue 2. 2005-06-28. PMID:15615821. diabetic nephropathy (dn) is characterized by mesangial cell (mc) hypertrophy and progressive accumulation of glomerular extracellular matrix (ecm). 2005-06-28 2023-08-12 mouse
Kristen A Johnson, Robert A Terkeltau. External GTP-bound transglutaminase 2 is a molecular switch for chondrocyte hypertrophic differentiation and calcification. The Journal of biological chemistry. vol 280. issue 15. 2005-06-07. PMID:15691824. chondrocyte maturation to hypertrophy, associated with up-regulated transglutaminase 2 (tg2) expression, mediates not only physiologic growth plate mineralization but also pathologic matrix calcification and dys-regulated matrix repair in osteoarthritic articular cartilage. 2005-06-07 2023-08-12 mouse
Kristen A Johnson, Robert A Terkeltau. External GTP-bound transglutaminase 2 is a molecular switch for chondrocyte hypertrophic differentiation and calcification. The Journal of biological chemistry. vol 280. issue 15. 2005-06-07. PMID:15691824. following transfection of tg2 site-directed mutants into chondrocytic cells, we observed that wild type tg2, and tg catalytic site and fibronectin-binding mutants promoted type x collagen expression and matrix calcification consistent with chondrocyte hypertrophic differentiation. 2005-06-07 2023-08-12 mouse
P Pacaud, V Sauzeau, G Loiran. Rho proteins and vascular diseases. Archives des maladies du coeur et des vaisseaux. vol 98. issue 3. 2005-05-26. PMID:15816329. typically, arterial diseases are associated with functional and structural wall alterations including modified contractile properties, smooth muscle cell hypertrophy and proliferation, endothelial dysfunction, excessive extracellular matrix accumulation and inflammation. 2005-05-26 2023-08-12 Not clear
Yan Wang, Frank Middleton, Jason A Horton, Lee Reichel, Cornelia E Farnum, Timothy A Damro. Microarray analysis of proliferative and hypertrophic growth plate zones identifies differentiation markers and signal pathways. Bone. vol 35. issue 6. 2005-05-19. PMID:15589209. longitudinal bone growth results from coordination of proliferation and hypertrophy of chondrocytes, calcification of the matrix, vascular invasion, and completion of endochondral bone formation in the growth plate. 2005-05-19 2023-08-12 rat
Anke Schnapper, Wilfried Meye. Osteopontin distribution in the canine skeleton during growth and structural maturation. Cells, tissues, organs. vol 178. issue 3. 2005-05-19. PMID:15655333. opn was expressed in hypertrophic chondrocytes of epiphyseal cartilage and in chondrocytes of the deep zone of mature articular cartilage, where extracellular matrix was also stained. 2005-05-19 2023-08-12 Not clear
Judith Bellemare, Charles J Roberge, Danielle Bergeron, Carlos A Lopez-Vallé, Michel Roy, Véronique J Mouli. Epidermis promotes dermal fibrosis: role in the pathogenesis of hypertrophic scars. The Journal of pathology. vol 206. issue 1. 2005-05-17. PMID:15772942. hypertrophic scarring is a pathological process characterized by fibroblastic hyperproliferation and by excessive deposition of extracellular matrix components. 2005-05-17 2023-08-12 Not clear
Björn Samnegård, Stefan H Jacobson, Georg Jaremko, Bo-Lennart Johansson, Karin Ekberg, Britta Isaksson, Linda Eriksson, John Wahren, Mats Sjöquis. C-peptide prevents glomerular hypertrophy and mesangial matrix expansion in diabetic rats. Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association. vol 20. issue 3. 2005-05-16. PMID:15665028. c-peptide prevents glomerular hypertrophy and mesangial matrix expansion in diabetic rats. 2005-05-16 2023-08-12 rat
Norio Amizuka, Junko Shimomura, Takeyasu Maeda, Hidehiro Ozaw. [Mineralization and vascular invasion during endochondral bone formation]. Clinical calcium. vol 13. issue 4. 2005-05-11. PMID:15775105. the intercolumnar cartilage matrix of the hypertrophic zone is subjected to mineralization whereas the transverse partitions of the cartilage column are poorly mineralized. 2005-05-11 2023-08-12 Not clear
Andrew R Kompa, Walter G Thomas, Fiona See, Alex Tzanidis, Ross D Hannan, Henry Kru. Cardiovascular role of urotensin II: effect of chronic infusion in the rat. Peptides. vol 25. issue 10. 2005-05-06. PMID:15476946. direct effects of uii on the myocardium, include myocyte hypertrophy, extracellular matrix deposition and contractility. 2005-05-06 2023-08-12 rat
Gunnar Sjöberg, Anna Kostareva, Thomas Sejerse. [Hereditary cardiomyopathies: a review. Mutation of structural proteins a common cause of hereditary cardiomyopathy]. Lakartidningen. vol 102. issue 11. 2005-04-20. PMID:15835520. in general the genes causing hypertrophic cardiomyopathies code for proteins involved in the contractile apparatus, the sarcomere, and the genes causing dilated cardiomyopathy code for proteins that anchor the sarcomere to the cell membrane and extracellular matrix. 2005-04-20 2023-08-12 Not clear
Francisco Forriol, Frederic Shapir. Bone development: interaction of molecular components and biophysical forces. Clinical orthopaedics and related research. issue 432. 2005-04-06. PMID:15738800. most endochondral growth in length occurs at the physes by the coordinated actions of cell proliferation, matrix synthesis, and chondrocyte hypertrophy. 2005-04-06 2023-08-12 Not clear
Blanche Schroen, Stephane Heymans, Umesh Sharma, W Matthijs Blankesteijn, Saraswati Pokharel, Jack P M Cleutjens, J Gordon Porter, Chris T A Evelo, Rudy Duisters, Rick E W van Leeuwen, Ben J A Janssen, Jacques J M Debets, Jos F M Smits, Mat J A P Daemen, Harry J G M Crijns, Paul Bornstein, Yigal M Pint. Thrombospondin-2 is essential for myocardial matrix integrity: increased expression identifies failure-prone cardiac hypertrophy. Circulation research. vol 95. issue 5. 2005-04-01. PMID:15284191. thrombospondin-2 is essential for myocardial matrix integrity: increased expression identifies failure-prone cardiac hypertrophy. 2005-04-01 2023-08-12 mouse
Xinping Zhang, Navid Ziran, J Jeffery Goater, Edward M Schwarz, J Edward Puzas, Randy N Rosier, Michael Zuscik, Hicham Drissi, Regis J O'Keef. Primary murine limb bud mesenchymal cells in long-term culture complete chondrocyte differentiation: TGF-beta delays hypertrophy and PGE2 inhibits terminal differentiation. Bone. vol 34. issue 5. 2005-03-29. PMID:15121012. here, we show that stage e11.5 embryonic murine limb bud mesenchymal stem cells in micromass cell culture progress through the stages of chondrogenesis, chondrocyte hypertrophy, terminal differentiation, and matrix calcification. 2005-03-29 2023-08-12 mouse
Xinping Zhang, Navid Ziran, J Jeffery Goater, Edward M Schwarz, J Edward Puzas, Randy N Rosier, Michael Zuscik, Hicham Drissi, Regis J O'Keef. Primary murine limb bud mesenchymal cells in long-term culture complete chondrocyte differentiation: TGF-beta delays hypertrophy and PGE2 inhibits terminal differentiation. Bone. vol 34. issue 5. 2005-03-29. PMID:15121012. pge2 had minimal effects on chondrocyte hypertrophy but delayed terminal differentiation and matrix calcification. 2005-03-29 2023-08-12 mouse
Hiroyuki Tsutsu. Novel pathophysiological insight and treatment strategies for heart failure--lessons from mice and patients--. Circulation journal : official journal of the Japanese Circulation Society. vol 68. issue 12. 2005-03-29. PMID:15564690. oxidative stress causes myocyte hypertrophy, apoptosis, and interstitial fibrosis by activating matrix metalloproteinases, all of which result in myocardial remodeling and failure. 2005-03-29 2023-08-12 mouse
Myra H Wyckoff, Chirine El-Turk, Abbot Laptook, Charles Timmons, Francis H Gannon, Xiafang Zhang, Steven Mumm, Michael P Whyt. Neonatal lethal osteochondrodysplasia with low serum levels of alkaline phosphatase and osteocalcin. The Journal of clinical endocrinology and metabolism. vol 90. issue 2. 2005-03-24. PMID:15562030. histopathology at autopsy excluded impaired mineralization of skeletal matrix, but endochondral bone formation appeared disorganized with growth plate clustering of chondrocytes in hypertrophic zones and in zones of provisional calcification. 2005-03-24 2023-08-12 Not clear
C Alexakis, C Strup, P Mestries, D Mathé, J P Caruelle, D Barritault, P Ker. Regulation of the collagen phenotype expression of gamma-irradiated vascular smooth muscle cells by heparan mimetics (RGTA). Journal of biomedical materials research. Part A. vol 70. issue 4. 2005-03-22. PMID:15307164. restenosis is characterized by vascular smooth muscle cell (vsmc) proliferation and accumulation of collagen iii in a hypertrophic and disorganized extracellular matrix. 2005-03-22 2023-08-12 Not clear
Kathleen M Daumer, A Cevik Tufan, Rocky S Tua. Long-term in vitro analysis of limb cartilage development: involvement of Wnt signaling. Journal of cellular biochemistry. vol 93. issue 3. 2005-03-18. PMID:15372624. endochondral skeletal development involves the condensation of mesenchymal cells, their differentiation into chondrocytes, followed by chondrocyte maturation, hypertrophy, and matrix mineralization, and replacement by osteoblasts. 2005-03-18 2023-08-12 chicken