All Relations between dopaminergic and dopamine

Publication Sentence Publish Date Extraction Date Species
J D Jentsch, R H Roth, J R Taylo. Role for dopamine in the behavioral functions of the prefrontal corticostriatal system: implications for mental disorders and psychotropic drug action. Progress in brain research. vol 126. 2001-01-18. PMID:11105661. moreover, the afferent control of dopamine neurons from brainstem and forebrain sites need to be fully explored in order to begin to understand what mechanisms are involved in regulating the dopaminergic response to stimuli with incentive value. 2001-01-18 2023-08-12 human
E A Nofzinger, A Fasiczka, S Berman, M E Thas. Bupropion SR reduces periodic limb movements associated with arousals from sleep in depressed patients with periodic limb movement disorder. The Journal of clinical psychiatry. vol 61. issue 11. 2001-01-11. PMID:11105739. given the role of dopamine in plmd and the effects of bupropion sustained-release (sr) on central dopaminergic function, we hypothesized that bupropion sr would not be associated with antidepressant-induced plmd. 2001-01-11 2023-08-12 Not clear
S J Glatt, C A Bolaños, G H Trksak, D Jackso. Effects of prenatal cocaine exposure on dopamine system development: a meta-analysis. Neurotoxicology and teratology. vol 22. issue 5. 2001-01-11. PMID:11106856. several studies have investigated the effects of prenatal cocaine (pcoc) exposure on the nigrostriatal dopaminergic system in animal models of maternal drug abuse, yet independent examinations of striatal dopamine (da) receptors and tissue da levels have produced equivocal results. 2001-01-11 2023-08-12 Not clear
S A Treseder, L A Smith, P Jenne. Endogenous dopaminergic tone and dopamine agonist action. Movement disorders : official journal of the Movement Disorder Society. vol 15. issue 5. 2001-01-09. PMID:11009183. endogenous dopaminergic tone and dopamine agonist action. 2001-01-09 2023-08-12 Not clear
A Heller, A Freeney, S Hessefort, M Villereal, L Wo. Cellular dopamine is increased following exposure to a factor derived from immortalized striatal neurons [corrected]. Neuroscience letters. vol 295. issue 1-2. 2001-01-09. PMID:11078922. a factor obtained from an immortalized hybrid monoclonal cell line (x61) of striatal origin is capable of increasing the dopamine content of hybrid, monoclonal cells expressing a dopaminergic phenotype (mn9d) and of aggregate cultures containing primary dopaminergic neurons. 2001-01-09 2023-08-12 Not clear
A Heller, A Freeney, S Hessefort, M Villereal, L Wo. Cellular dopamine is increased following exposure to a factor derived from immortalized striatal neurons [corrected]. Neuroscience letters. vol 295. issue 1-2. 2001-01-09. PMID:11078922. given that parkinsonian symptoms are only apparent following degeneration of a substantial portion of the mesencephalic dopaminergic cell population, activity capable of increasing dopamine content of the surviving cells may represent an interesting candidate therapeutic agent. 2001-01-09 2023-08-12 Not clear
J Lavalaye, J Booij, L Reneman, J B Habraken, E A van Roye. Effect of age and gender on dopamine transporter imaging with [123I]FP-CIT SPET in healthy volunteers. European journal of nuclear medicine. vol 27. issue 7. 2001-01-08. PMID:10952500. dopamine transporter imaging is a valuable tool to investigate the integrity of the dopaminergic neurons. 2001-01-08 2023-08-12 human
K K Jai. An assessment of iloperidone for the treatment of schizophrenia. Expert opinion on investigational drugs. vol 9. issue 12. 2001-01-08. PMID:11093363. iloperidone, with a balance of activity at the dopaminergic and serotonergic receptors, has obvious advantages over clozapine and olanzapine, both of which have a similar receptor profile as they favour serotonergic over dopamine receptors. 2001-01-08 2023-08-12 mouse
S F Taylor, R A Koeppe, R Tandon, J K Zubieta, K A Fre. In vivo measurement of the vesicular monoamine transporter in schizophrenia. Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology. vol 23. issue 6. 2001-01-04. PMID:11063922. given evidence for excessive striatal dopamine activity in schizophrenia, we sought to test the hypothesis that dopaminergic innervation in the striatum is abnormally elevated, and a secondary hypothesis that age-related loss is accelerated. 2001-01-04 2023-08-12 human
G G Nomikos, B Schilström, B E Hildebrand, G Panagis, J Grenhoff, T H Svensso. Role of alpha7 nicotinic receptors in nicotine dependence and implications for psychiatric illness. Behavioural brain research. vol 113. issue 1-2. 2001-01-03. PMID:10942036. based on the data reviewed in the present study, it is suggested that nicotine by stimulating presynaptic alpha7 nicotinic receptors within the vta, that are probably localized on glutamatergic afferents from the medial prefrontal cortex, produces sequentially an increase in glutamate concentrations, stimulation of nmda receptors found on dopamine (da)-containing neurons in the vta, enhanced firing activity of vta-da neurons, augmented da release in the nerve terminal regions, and enhanced c-fos expression in the dopaminergic projection areas through activation of d1-da receptors. 2001-01-03 2023-08-12 Not clear
R Usha, D Muralikrishnan, B Thomas, S Ghosh, C Mandal, K P Mohanakuma. Region-specific attenuation of a trypsin-like protease in substantia nigra following dopaminergic neurotoxicity by 1-methyl-4-phenyl-1,2, 3,6-tetrahydropyridine. Brain research. vol 882. issue 1-2. 2000-12-28. PMID:11056198. the absence of typical dna 'ladder' when there was severe striatal dopamine depletion suggests that in vivo mptp-mediated dopaminergic neurotoxicity may not involve apoptotic cell death, and explains why in mice mptp-induced dopamine depletion is transient. 2000-12-28 2023-08-12 mouse
H Khaldy, G Escames, J León, F Vives, J D Luna, D Acuña-Castroviej. Comparative effects of melatonin, L-deprenyl, Trolox and ascorbate in the suppression of hydroxyl radical formation during dopamine autoxidation in vitro. Journal of pineal research. vol 29. issue 2. 2000-12-22. PMID:10981823. dopamine (da) autoxidation produces reactive oxygen species implicated in the loss of dopaminergic neurons in the nigrostriatal pathway. 2000-12-22 2023-08-12 Not clear
F Gonon, J B Burie, M Jaber, M Benoit-Marand, B Dumartin, B Bloc. Geometry and kinetics of dopaminergic transmission in the rat striatum and in mice lacking the dopamine transporter. Progress in brain research. vol 125. 2000-12-22. PMID:11098665. geometry and kinetics of dopaminergic transmission in the rat striatum and in mice lacking the dopamine transporter. 2000-12-22 2023-08-12 mouse
A Friedma. [Treatment of Parkinson's disease--from theory to practice]. Neurologia i neurochirurgia polska. vol 32 Suppl 6. 2000-12-22. PMID:11107568. however, not all symptoms can be explained by this damage, moreover, substitution therapy /with preparations containing dopamine precursor--levodopa and agonists of the dopaminergic receptor /not only fails to remove all disease symptoms but can even produce adverse effects. 2000-12-22 2023-08-12 Not clear
G D Stanwood, R P Artymyshyn, M P Kung, H F Kung, I Lucki, P McGonigl. Quantitative autoradiographic mapping of rat brain dopamine D3 binding with [(125)I]7-OH-PIPAT: evidence for the presence of D3 receptors on dopaminergic and nondopaminergic cell bodies and terminals. The Journal of pharmacology and experimental therapeutics. vol 295. issue 3. 2000-12-20. PMID:11082459. quantitative autoradiographic mapping of rat brain dopamine d3 binding with [(125)i]7-oh-pipat: evidence for the presence of d3 receptors on dopaminergic and nondopaminergic cell bodies and terminals. 2000-12-20 2023-08-12 rat
H Inou. Effects of naltrexone on the accumulation of L-3, 4-dihydroxyphenylalanine and 5-hydroxy-L-tryptophan and on the firing rate induced by acute ethanol administration. European journal of pharmacology. vol 406. issue 3. 2000-12-11. PMID:11040344. in order to characterize the effects of naltrexone, a mu-opioid receptor antagonist, on acute ethanol-induced functional modification of dopaminergic neurons in the nigrastriatal and mesolimbic dopamine systems, the accumulation of l-3, 4-dihydroxyphenylalanine (l-dopa) in the cerebral cortex, dorsal striatum and nucleus accumbens and of 5-hydroxy-l-tryptophan (5-htp) in the hippocampus was measured in normal rats using the mu-hydroxybenzylhydrazine dihydrochloride (nsd-1015) enzymatic inhibition method. 2000-12-11 2023-08-12 rat
H Inou. Effects of naltrexone on the accumulation of L-3, 4-dihydroxyphenylalanine and 5-hydroxy-L-tryptophan and on the firing rate induced by acute ethanol administration. European journal of pharmacology. vol 406. issue 3. 2000-12-11. PMID:11040344. naltrexone resulted in a decrease in the dopaminergic neuronal firing rates activated by ethanol and eventually in a reduction of the dopamine synthesis induced by ethanol in the dorsal striatum and nucleus accumbens, but not in the cerebral cortex. 2000-12-11 2023-08-12 rat
J E Demaria, G M Nagy, M E Freema. Immunoneutralization of prolactin prevents stimulatory feedback of prolactin on hypothalamic neuroendocrine dopaminergic neurons. Endocrine. vol 12. issue 3. 2000-12-07. PMID:10963056. sites of neuroendocrine dopaminergic nerve terminals, the median eminence (me), and intermediate and neural lobes of the pituitary gland were excised and stored for determination of dopamine (da) and 3,4-dihydroxyphenyl acetic acid (dopac) concentrations by high-performance liquid chromatography electrochemical detection (ec). 2000-12-07 2023-08-12 rat
X Gao, Y Zhang, G W. Effects of dopaminergic agents on carrageenan hyperalgesia in rats. European journal of pharmacology. vol 406. issue 1. 2000-12-07. PMID:11011033. the present study explored the role of central dopaminergic transmission in a model of carrageenan-induced inflammatory pain by examining the effects of selective agonists and antagonists of dopamine receptors. 2000-12-07 2023-08-12 rat
G Bringmann, D Feineis, R Brückner, M Blank, K Peters, E M Peters, H Reichmann, B Janetzky, C Grote, H W Clement, W Weseman. Bromal-derived tetrahydro-beta-carbolines as neurotoxic agents: chemistry, impairment of the dopamine metabolism, and inhibitory effects on mitochondrial respiration. Bioorganic & medicinal chemistry. vol 8. issue 6. 2000-12-06. PMID:10896123. the mammalian alkaloids tryptoline (1) and eleagnine (2) as well as the highly halogenated (x = f, cl, br) tetrahydro-beta-carbolines (thbetacs) 3-5, structurally similar to the dopaminergic neurotoxin 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (mptp, 6), were found to have a common feature of inducing a severe impairment of the nigrostriatal dopamine metabolism and inhibiting complex i of the mitochondrial respiratory chain highly selectively. 2000-12-06 2023-08-12 rat