All Relations between dopaminergic and dopamine

Publication Sentence Publish Date Extraction Date Species
Xiomara A Perez, Neeraja Parameswaran, Luping Z Huang, Kathryn T O'Leary, Maryka Qui. Pre-synaptic dopaminergic compensation after moderate nigrostriatal damage in non-human primates. Journal of neurochemistry. vol 105. issue 5. 2008-06-12. PMID:18248617. to address this issue, we used cyclic voltammetry to directly measure endogenous dopamine release from striatal slices of control monkeys and animals with a moderate or severe mptp-induced dopaminergic lesion. 2008-06-12 2023-08-12 monkey
Jelena Mijatovic, Outi Patrikainen, Leonid Yavich, Mikko Airavaara, Liisa Ahtee, Mart Saarma, T Petteri Pieppone. Characterization of the striatal dopaminergic neurotransmission in MEN2B mice with elevated cerebral tissue dopamine. Journal of neurochemistry. vol 105. issue 5. 2008-06-12. PMID:18248620. characterization of the striatal dopaminergic neurotransmission in men2b mice with elevated cerebral tissue dopamine. 2008-06-12 2023-08-12 mouse
Jun Takahash. [Neuromodulation for Parkinson's disease]. Rinsho shinkeigaku = Clinical neurology. vol 48. issue 4. 2008-06-10. PMID:18453154. parkinson's disease (pd) is a neurodegenerative disorder characterized by a progressive loss of midbrain dopaminergic (da) neurons and a subsequent reduction in striatal dopamine. 2008-06-10 2023-08-12 human
Ruth F Villarán, M Tomás-Camardiel, Rocío M de Pablos, Marti Santiago, Antonio J Herrera, Ana Navarro, Alberto Machado, Josefina Can. Endogenous dopamine enhances the neurotoxicity of 3-nitropropionic acid in the striatum through the increase of mitochondrial respiratory inhibition and free radicals production. Neurotoxicology. vol 29. issue 2. 2008-06-05. PMID:18093658. all these results suggest that endogenous dopamine within the dopaminergic terminals of the striatum enhances the mitochondrial production of radical oxygen species along with the respiratory inhibition produced by 3-np and thus increases the toxicity produced by 3-np in the striatum. 2008-06-05 2023-08-12 Not clear
Yasuhiko Izumi, Noriyuki Yamamoto, Toshiaki Kume, Hiroshi Katsuki, Hideyuki Sawada, Akinori Akaik. Regulation of intracellular dopamine levels by dopaminergic drugs: involvement of vesicular monoamine transporter. European journal of pharmacology. vol 582. issue 1-3. 2008-06-05. PMID:18261722. regulation of intracellular dopamine levels by dopaminergic drugs: involvement of vesicular monoamine transporter. 2008-06-05 2023-08-12 Not clear
Yasuhiko Izumi, Noriyuki Yamamoto, Toshiaki Kume, Hiroshi Katsuki, Hideyuki Sawada, Akinori Akaik. Regulation of intracellular dopamine levels by dopaminergic drugs: involvement of vesicular monoamine transporter. European journal of pharmacology. vol 582. issue 1-3. 2008-06-05. PMID:18261722. endogenous dopamine could serve as a susceptibility factor for dopaminergic neuronal death. 2008-06-05 2023-08-12 Not clear
Yasuhiko Izumi, Noriyuki Yamamoto, Toshiaki Kume, Hiroshi Katsuki, Hideyuki Sawada, Akinori Akaik. Regulation of intracellular dopamine levels by dopaminergic drugs: involvement of vesicular monoamine transporter. European journal of pharmacology. vol 582. issue 1-3. 2008-06-05. PMID:18261722. the dopaminergic drugs which gave rise to reduction in dopamine content also interfered with vesicular dopamine transport. 2008-06-05 2023-08-12 Not clear
Yasuhiko Izumi, Noriyuki Yamamoto, Toshiaki Kume, Hiroshi Katsuki, Hideyuki Sawada, Akinori Akaik. Regulation of intracellular dopamine levels by dopaminergic drugs: involvement of vesicular monoamine transporter. European journal of pharmacology. vol 582. issue 1-3. 2008-06-05. PMID:18261722. these results suggest that dopaminergic drugs can reduce intracellular dopamine via inhibition of vesicular monoamine uptake. 2008-06-05 2023-08-12 Not clear
Nicolas Mallet, Alek Pogosyan, Andrew Sharott, Jozsef Csicsvari, J Paul Bolam, Peter Brown, Peter J Magil. Disrupted dopamine transmission and the emergence of exaggerated beta oscillations in subthalamic nucleus and cerebral cortex. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 28. issue 18. 2008-06-05. PMID:18448656. abnormal beta oscillations and motor symptoms are concomitantly and acutely suppressed by dopaminergic therapies, suggesting that these inappropriate rhythms might also emerge acutely from disrupted dopamine transmission. 2008-06-05 2023-08-12 Not clear
Patrick B Woo. Role of central dopamine in pain and analgesia. Expert review of neurotherapeutics. vol 8. issue 5. 2008-06-04. PMID:18457535. several novel classes of medication with analgesic properties have bearing on dopaminergic activity as evident in the capacity of dopamine antagonists to attenuate their analgesic capacity. 2008-06-04 2023-08-12 Not clear
Gérard Lambert, Laurent Karila, William Lowenstei. [Neuroimaging and cocaine: mapping dependence?]. Presse medicale (Paris, France : 1983). vol 37. issue 4 Pt 2. 2008-06-03. PMID:18164583. this blockage is expressed by depletion of dopaminergic d2 postsynaptic receptors and low levels of dopamine release. 2008-06-03 2023-08-12 Not clear
Panayotis K Thanos, Michael Michaelides, Helene Benveniste, Gene Jack Wang, Nora D Volko. The effects of cocaine on regional brain glucose metabolism is attenuated in dopamine transporter knockout mice. Synapse (New York, N.Y.). vol 62. issue 5. 2008-06-03. PMID:18286542. to separate the dopaminergic from the non-dopaminergic effects of cocaine on brain function we compared the regional brain metabolic responses to cocaine between dopamine transporter deficient (dat(-/-)) mice with that of their dat(+/+) littermates. 2008-06-03 2023-08-12 mouse
Lane J Wallace, Laura E Connel. Mechanisms by which amphetamine redistributes dopamine out of vesicles: a computational study. Synapse (New York, N.Y.). vol 62. issue 5. 2008-06-03. PMID:18297690. computer simulations of dopamine (da) and amphetamine interactions associated with dopaminergic storage vesicles were developed in order to better explain how amphetamine causes redistribution of da out of the vesicles. 2008-06-03 2023-08-12 Not clear
Nicola Simola, Sandro Fenu, Pier G Baraldi, Mojgan Aghazadeh Tabrizi, Micaela Morell. Blockade of globus pallidus adenosine A(2A) receptors displays antiparkinsonian activity in 6-hydroxydopamine-lesioned rats treated with D(1) or D(2) dopamine receptor agonists. Synapse (New York, N.Y.). vol 62. issue 5. 2008-06-03. PMID:18297692. we have recently demonstrated how antagonism of adenosine a(2a) receptors within the globus pallidus (gp) ipsilateral to dopaminergic denervation potentiates contralateral rotational behavior induced by the dopamine precursor l-dopa in 6-hydroxydopamine-lesioned hemiparkinsonian rats. 2008-06-03 2023-08-12 rat
G Z Ziiazetdinova, A Ia Sapronova, V Ia Kiiasova, A K Nanaev, V S Kudrin, N Martina, I Tyllet, M V Ugriumo. [Compensatory reaction during degeneration of arcuate nucleus dopaminergic neurons in rats]. Zhurnal evoliutsionnoi biokhimii i fiziologii. vol 44. issue 1. 2008-06-03. PMID:18411516. the study has been carried out to verify the authors' hypothesis that degeneration of dopaminergic (da-ergic) neurons of the hypothalamic tuberoinfundibular system and concomitant development of hyperprolactinemia are accompanied by involvement of compensatory synthesis of dopamine (da) by non-dopaminergic neurons expressing single complementary enzymes of synthesis of this neurotransmitter. 2008-06-03 2023-08-12 rat
Roser Nadal Aleman. [Novelty-seeking: its relationship with vulnerability to addiction and stress]. Adicciones. vol 20. issue 1. 2008-05-30. PMID:18299782. with regard to dopaminergic systems, microdialysis studies show that cocaine has a greater neurochemical effect (more increase in dopamine levels) in hr than in lr animals. 2008-05-30 2023-08-12 human
Antonio J Herrera, Rocío M de Pablos, Eloisa Carreño-Müller, Ruth F Villarán, José L Venero, Mayka Tomás-Camardiel, Josefina Cano, Alberto Machad. The intrastriatal injection of thrombin in rat induced a retrograde apoptotic degeneration of nigral dopaminergic neurons through synaptic elimination. Journal of neurochemistry. vol 105. issue 3. 2008-05-22. PMID:18179476. when we studied the effects on substantia nigra, we found the disappearance of dopaminergic neurons, shown by loss of th immunoreactivity, loss of expression of th and dopamine transporter mrnas, and disappearance of fluorogold-labelled nigral neurons. 2008-05-22 2023-08-12 rat
Ayumi Fukakusa, Hiroyuki Mizoguchi, Hiroyuki Koike, Toshitaka Nabeshima, Kazuhiro Takuma, Kiyofumi Yamad. Tissue plasminogen activator is not involved in methamphetamine-induced neurotoxicity. Journal of pharmacological sciences. vol 106. issue 2. 2008-05-22. PMID:18277051. to investigate the role of tissue plasminogen activator (tpa) in methamphetamine (meth)-induced dopaminergic neurotoxicity, we compared the changes in tyrosine hydroxylase (th) and dopamine transporter (dat) levels in the striatum after repetitive treatment of meth at 4 mg/kg among wild-type, tpa-deficient (tpa-/-), and protease activated receptor-1-deficient (par-1-/-) mice. 2008-05-22 2023-08-12 mouse
Diego A Pizzagalli, A Eden Evins, Erika Cowman Schetter, Michael J Frank, Petra E Pajtas, Diane L Santesso, Melissa Culhan. Single dose of a dopamine agonist impairs reinforcement learning in humans: behavioral evidence from a laboratory-based measure of reward responsiveness. Psychopharmacology. vol 196. issue 2. 2008-05-19. PMID:17909750. the dopaminergic system, particularly d2-like dopamine receptors, has been strongly implicated in reward processing. 2008-05-19 2023-08-12 Not clear
Gregory T Collins, Diane M Calinski, Amy Hauck Newman, Peter Grundt, James H Wood. Food restriction alters N'-propyl-4,5,6,7-tetrahydrobenzothiazole-2,6-diamine dihydrochloride (pramipexole)-induced yawning, hypothermia, and locomotor activity in rats: evidence for sensitization of dopamine D2 receptor-mediated effects. The Journal of pharmacology and experimental therapeutics. vol 325. issue 2. 2008-05-19. PMID:18305018. food restriction has also been shown to alter a variety of behavioral and pharmacological responses to dopaminergic agonists, including an increased sensitivity to the locomotor stimulatory effects of direct- and indirect-dopamine agonists, elevated extracellular dopamine levels in responses to psychostimulants, as well as suppression of agonist-induced yawning. 2008-05-19 2023-08-12 rat