| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| Simone A van den Berge, Miriam E van Strien, Elly M Ho. Resident adult neural stem cells in Parkinson's disease--the brain's own repair system? European journal of pharmacology. vol 719. issue 1-3. 2014-07-02. PMID:23872414. |
therefore, restoring the dopamine levels in the striatum of pd patients through stimulating endogenous nscs in the nearby svz to migrate into the striatum and differentiate into dopaminergic neurons might thus be an attractive future therapeutic approach. |
2014-07-02 |
2023-08-12 |
human |
| Ji Yong Lee, Sung Hoon Kim, Ah-Ra Ko, Jin Suk Lee, Ji Hea Yu, Jung Hwa Seo, Byung Pil Cho, Sung-Rae Ch. Therapeutic effects of repetitive transcranial magnetic stimulation in an animal model of Parkinson's disease. Brain research. vol 1537. 2014-06-25. PMID:23998987. |
behavioral assessments such as amphetamine-induced rotational test and treadmill locomotion test were performed, and the dopaminergic (da) neurons of substantia nigra pas compacta (snc) and striatum were histologically examined. |
2014-06-25 |
2023-08-12 |
rat |
| Sachiko Tanaka, Atsuko Ishii, Hirokazu Ohtaki, Seiji Shioda, Takemi Yoshida, Satoshi Numazaw. Activation of microglia induces symptoms of Parkinson's disease in wild-type, but not in IL-1 knockout mice. Journal of neuroinflammation. vol 10. 2014-06-25. PMID:24289537. |
parkinson's disease (pd) is an age-related progressive neurodegenerative disorder caused by selective loss of dopaminergic neurons from the substantia nigra (sn) to the striatum. |
2014-06-25 |
2023-08-12 |
mouse |
| Humsini Viswanath, Asasia Q Carter, Philip R Baldwin, David L Molfese, Ramiro Sala. The medial habenula: still neglected. Frontiers in human neuroscience. vol 7. 2014-06-24. PMID:24478666. |
this structure sends projections to the dopaminergic striatum and receives inputs from the limbic forebrain, making the habenula a unique modulator of cross-talk between these brain regions. |
2014-06-24 |
2023-08-12 |
monkey |
| Ulrich Ettinger, Inga Meyhöfer, Maria Steffens, Michael Wagner, Nikolaos Koutsouleri. Genetics, cognition, and neurobiology of schizotypal personality: a review of the overlap with schizophrenia. Frontiers in psychiatry. vol 5. 2014-06-24. PMID:24600411. |
experimental pharmacological studies have shown that high levels of schizotypy are associated with (i) enhanced dopaminergic response in striatum following administration of amphetamine and (ii) improvement of cognitive performance following administration of antipsychotic compounds. |
2014-06-24 |
2023-08-12 |
Not clear |
| Raimo K R Salokangas, Jukka Huttunen, Jarmo Hietal. Discontinuation of citalopram medication and striatal dopamine activity. International journal of psychiatry in clinical practice. vol 10. issue 4. 2014-06-20. PMID:24941151. |
we suggest that stimulation of serotonergic system in the brain increases dopaminergic activity in the striatum. |
2014-06-20 |
2023-08-13 |
Not clear |
| Andrea de Bartolomeis, Federica Marmo, Elisabetta Filomena Buonaguro, Rodolfo Rossi, Carmine Tomasetti, Felice Iasevol. Imaging brain gene expression profiles by antipsychotics: region-specific action of amisulpride on postsynaptic density transcripts compared to haloperidol. European neuropsychopharmacology : the journal of the European College of Neuropsychopharmacology. vol 23. issue 11. 2014-06-17. PMID:23357084. |
despite the selective dopaminergic profile of both compounds, our results demonstrated a differential modulation of postsynaptic molecules by amisulpride and haloperidol, the former impacting preferentially medial regions of striatum whereas the latter inducing strong gene expression in lateral regions. |
2014-06-17 |
2023-08-12 |
Not clear |
| Xian-Ming Luo, Hai Lin, Wei Wang, Marilyn S Geaney, Lee Law, Shaun Wynyard, Shamim B Shaikh, Henry Waldvogel, Richard L M Faull, Robert B Elliott, Stephen J M Skinner, Jacqueline E Lee, Paul L-J Ta. Recovery of neurological functions in non-human primate model of Parkinson's disease by transplantation of encapsulated neonatal porcine choroid plexus cells. Journal of Parkinson's disease. vol 3. issue 3. 2014-06-17. PMID:24002224. |
parkinson's disease (pd) is a neurodegenerative disease that is primarily characterized by degeneration of dopaminergic (da) neurons in the substantia nigra (sn) and a loss of their fibre projections in the striatum. |
2014-06-17 |
2023-08-12 |
monkey |
| Bianca C Wittmann, Geoffrey C Tan, John E Lisman, Raymond J Dolan, Emrah Düze. Reprint of: DAT genotype modulates striatal processing and long-term memory for items associated with reward and punishment. Neuropsychologia. vol 51. issue 12. 2014-06-15. PMID:24139823. |
these findings suggest that processing of appetitive and aversive motivation in the human striatum involve the dopaminergic system and that dopamine plays a role in memory for both types of motivational information. |
2014-06-15 |
2023-08-12 |
human |
| Claudia M García-Peña, Minkyung Kim, Daniela Frade-Pérez, Daniela Avila-González, Elisa Téllez, Grant S Mastick, Elisa Tamariz, Alfredo Varela-Echavarrí. Ascending midbrain dopaminergic axons require descending GAD65 axon fascicles for normal pathfinding. Frontiers in neuroanatomy. vol 8. 2014-06-13. PMID:24926237. |
the nigrostriatal pathway (nsp) is formed by dopaminergic axons that project from the ventral midbrain to the dorsolateral striatum as part of the medial forebrain bundle. |
2014-06-13 |
2023-08-13 |
mouse |
| Claudia M García-Peña, Minkyung Kim, Daniela Frade-Pérez, Daniela Avila-González, Elisa Téllez, Grant S Mastick, Elisa Tamariz, Alfredo Varela-Echavarrí. Ascending midbrain dopaminergic axons require descending GAD65 axon fascicles for normal pathfinding. Frontiers in neuroanatomy. vol 8. 2014-06-13. PMID:24926237. |
we observed in mouse and rat embryos that midbrain dopaminergic axons ascend in close apposition to descending gad65-positive axon bundles throughout their trajectory to the striatum. |
2014-06-13 |
2023-08-13 |
mouse |
| Claudia M García-Peña, Minkyung Kim, Daniela Frade-Pérez, Daniela Avila-González, Elisa Téllez, Grant S Mastick, Elisa Tamariz, Alfredo Varela-Echavarrí. Ascending midbrain dopaminergic axons require descending GAD65 axon fascicles for normal pathfinding. Frontiers in neuroanatomy. vol 8. 2014-06-13. PMID:24926237. |
in these embryos we observed dopaminergic misprojection into the hypothalamic region and abnormal projection in the striatum. |
2014-06-13 |
2023-08-13 |
mouse |
| C Aldinio, G Valenti, G E Savoini, G Kirschner, L F Agnati, G Toffan. Monosialoganglioside internal ester stimulates the dopaminergic reinnervation of the striatum after unilateral hemitransection in rat. International journal of developmental neuroscience : the official journal of the International Society for Developmental Neuroscience. vol 2. issue 3. 2014-06-02. PMID:24874041. |
monosialoganglioside internal ester stimulates the dopaminergic reinnervation of the striatum after unilateral hemitransection in rat. |
2014-06-02 |
2023-08-13 |
rat |
| C Aldinio, G Valenti, G E Savoini, G Kirschner, L F Agnati, G Toffan. Monosialoganglioside internal ester stimulates the dopaminergic reinnervation of the striatum after unilateral hemitransection in rat. International journal of developmental neuroscience : the official journal of the International Society for Developmental Neuroscience. vol 2. issue 3. 2014-06-02. PMID:24874041. |
the effects of the administration of gm1 monosialoganglioside internal ester, agf2, on the dopaminergic reinnervation of the striatum in rats with unilateral hemitransection has been studied. |
2014-06-02 |
2023-08-13 |
rat |
| R N Kalaria, A K Princ. Effects of thyroid deficiency on the development of cholinergic, GABA, dopaminergic and glutamate neuron markers and DNA concentrations in the rat corpus striatum. International journal of developmental neuroscience : the official journal of the International Society for Developmental Neuroscience. vol 3. issue 6. 2014-06-02. PMID:24875111. |
effects of thyroid deficiency on the development of cholinergic, gaba, dopaminergic and glutamate neuron markers and dna concentrations in the rat corpus striatum. |
2014-06-02 |
2023-08-13 |
rat |
| R N Kalaria, A K Princ. Effects of thyroid deficiency on the development of cholinergic, GABA, dopaminergic and glutamate neuron markers and DNA concentrations in the rat corpus striatum. International journal of developmental neuroscience : the official journal of the International Society for Developmental Neuroscience. vol 3. issue 6. 2014-06-02. PMID:24875111. |
the effects of propylthiouracil (ptu)-induced thyroid deficiency on the postnatal development of synaptic markers for cholinergic, gaba, dopaminergic and glutamate neurons in the rat corpus striatum were investigated. |
2014-06-02 |
2023-08-13 |
rat |
| Busisiwe C Maseko, Nina Patzke, Kjell Fuxe, Paul R Mange. Architectural organization of the african elephant diencephalon and brainstem. Brain, behavior and evolution. vol 82. issue 2. 2014-05-29. PMID:24021932. |
each island may send topologically organized projections to the striatum forming a dopaminergic innervation mosaic that may relate to trunk movements. |
2014-05-29 |
2023-08-12 |
Not clear |
| Kazuto Kobayashi, Ryoji Fukabori, Kayo Nishizaw. Neural circuit mechanism for learning dependent on dopamine transmission: roles of striatal direct and indirect pathways in sensory discrimination. Advances in pharmacology (San Diego, Calif.). vol 68. 2014-05-29. PMID:24054143. |
the striatum receives excitatory inputs from many cortical areas and the thalamic nuclei and dopaminergic inputs from the ventral midbrain and projects to the output nuclei through direct and indirect pathways. |
2014-05-29 |
2023-08-12 |
mouse |
| David Arkadir, Hagai Bergman, Stanley Fah. Redundant dopaminergic activity may enable compensatory axonal sprouting in Parkinson disease. Neurology. vol 82. issue 12. 2014-05-27. PMID:24663231. |
collateral axonal sprouting of dopaminergic terminals into the denervated striatum is the most studied compensatory mechanism in animal (almost exclusively rodent) models of parkinson disease and is correlated with behavioral recovery after partial lesions. |
2014-05-27 |
2023-08-12 |
human |
| David Arkadir, Hagai Bergman, Stanley Fah. Redundant dopaminergic activity may enable compensatory axonal sprouting in Parkinson disease. Neurology. vol 82. issue 12. 2014-05-27. PMID:24663231. |
we suggest here that the unique physiologic property of the dopaminergic innervation of the striatum, namely redundancy of information encoding, is crucial to the efficacy of compensatory axonal sprouting in the presence of aberrant anatomical connections. |
2014-05-27 |
2023-08-12 |
human |