| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| R L Hakan, C Hart, C Ey. Specific neurophysiological effects of systemic nicotine on neurons in the nucleus accumbens. Synapse (New York, N.Y.). vol 15. issue 3. 1994-02-09. PMID:7904087. |
thus, the role of dopamine in these nas responses to nicotine remains unclear. |
1994-02-09 |
2023-08-12 |
rat |
| P R Sanberg, D F Emerich, M M el-Etri, M T Shipley, M D Zanol, D W Cahill, A B Norma. Nicotine potentiation of haloperidol-induced catalepsy: striatal mechanisms. Pharmacology, biochemistry, and behavior. vol 46. issue 2. 1994-01-27. PMID:8265684. |
a second experiment examined whether the ability of nicotine to potentiate haloperidol-induced catalepsy was associated with a potentiation of dopamine turnover following haloperidol. |
1994-01-27 |
2023-08-12 |
rat |
| P R Sanberg, D F Emerich, M M el-Etri, M T Shipley, M D Zanol, D W Cahill, A B Norma. Nicotine potentiation of haloperidol-induced catalepsy: striatal mechanisms. Pharmacology, biochemistry, and behavior. vol 46. issue 2. 1994-01-27. PMID:8265684. |
nicotine alone produced a mild increase in dopamine turnover relative to saline treated controls while haloperidol produced a marked increase in dopamine turnover relative to saline- and nicotine-treated controls. |
1994-01-27 |
2023-08-12 |
rat |
| P R Sanberg, D F Emerich, M M el-Etri, M T Shipley, M D Zanol, D W Cahill, A B Norma. Nicotine potentiation of haloperidol-induced catalepsy: striatal mechanisms. Pharmacology, biochemistry, and behavior. vol 46. issue 2. 1994-01-27. PMID:8265684. |
however, the combined administration of haloperidol and nicotine did not further elevate dopamine turnover over that observed following haloperidol alone. |
1994-01-27 |
2023-08-12 |
rat |
| P R Sanberg, D F Emerich, M M el-Etri, M T Shipley, M D Zanol, D W Cahill, A B Norma. Nicotine potentiation of haloperidol-induced catalepsy: striatal mechanisms. Pharmacology, biochemistry, and behavior. vol 46. issue 2. 1994-01-27. PMID:8265684. |
the results indicated that: 1) nicotine could not potentiate haloperidol-induced catalepsy without an intact striatum and 2) the behavioral effect of nicotine and haloperidol cotreatment was not due to any change in dopamine turnover. |
1994-01-27 |
2023-08-12 |
rat |
| M D Schechter, S M Meeha. Dopaminergic mediation of the stimulant generalization of nicotine. Progress in neuro-psychopharmacology & biological psychiatry. vol 17. issue 5. 1994-01-13. PMID:7902994. |
the results are discussed in light of the growing evidence that nicotinic receptors reside upon mesolimbic dopamine neurons and the possibility that the consequent increase in extracellular dopamine may produce the discriminative stimuli, as well as the reinforcing properties, of nicotine. |
1994-01-13 |
2023-08-12 |
rat |
| M Yoshida, H Yokoo, T Tanaka, K Mizoguchi, H Emoto, H Ishii, M Tanak. Facilitatory modulation of mesolimbic dopamine neuronal activity by a mu-opioid agonist and nicotine as examined with in vivo microdialysis. Brain research. vol 624. issue 1-2. 1994-01-13. PMID:8252400. |
facilitatory modulation of mesolimbic dopamine neuronal activity by a mu-opioid agonist and nicotine as examined with in vivo microdialysis. |
1994-01-13 |
2023-08-12 |
rat |
| M Yoshida, H Yokoo, T Tanaka, K Mizoguchi, H Emoto, H Ishii, M Tanak. Facilitatory modulation of mesolimbic dopamine neuronal activity by a mu-opioid agonist and nicotine as examined with in vivo microdialysis. Brain research. vol 624. issue 1-2. 1994-01-13. PMID:8252400. |
administration of either tyr-d-ala-gly-mephe-gly(ol) (dago), a mu-opioid agonist, or nicotine into the vta (a10) caused an increase in both dopamine (da) and its metabolite levels in both the a10 region and da nerve terminals in the nucleus accumbens of rats when assessed with dual probe brain dialysis. |
1994-01-13 |
2023-08-12 |
rat |
| L Monti-Bloch, V Abudara, C Eyzaguirr. Electrical communication between glomus cells of the rat carotid body. Brain research. vol 622. issue 1-2. 1994-01-06. PMID:7902188. |
stimulation by hypoxia (100% n2 or na2s2o4), acidity (lactic acid or 100% co2), dopamine, ach, nicotine and bethanechol depolarized the majority of glomus cells, their input resistance decreased and cells became uncoupled. |
1994-01-06 |
2023-08-12 |
rat |
| D W Schulz, G A Kuchel, R E Zigmon. Decline in response to nicotine in aged rat striatum: correlation with a decrease in a subpopulation of nicotinic receptors. Journal of neurochemistry. vol 61. issue 6. 1993-12-23. PMID:8245973. |
we have utilized the nicotine-mediated stimulation of endogenous dopamine efflux in a striatal slice preparation as a functional index of responsiveness to nicotine in aging. |
1993-12-23 |
2023-08-12 |
human |
| R Oishi, K Suemaru, K Furuno, Y Gomita, K Saek. Possible explanations for the antagonism by nicotine against reserpine-induced depletion of monoamines in mouse brain. Naunyn-Schmiedeberg's archives of pharmacology. vol 348. issue 2. 1993-12-06. PMID:8232594. |
this effect of nicotine was more marked on dopamine depletion than on noradrenaline or 5-hydroxytryptamine depletion. |
1993-12-06 |
2023-08-12 |
mouse |
| R Oishi, K Suemaru, K Furuno, Y Gomita, K Saek. Possible explanations for the antagonism by nicotine against reserpine-induced depletion of monoamines in mouse brain. Naunyn-Schmiedeberg's archives of pharmacology. vol 348. issue 2. 1993-12-06. PMID:8232594. |
however, when reserpine (0.5 mg/kg) was injected intravenously, pretreatment with nicotine (3 mg/kg, s.c.) inhibited the reserpine-induced dopamine depletion only, and this effect of nicotine was completely blocked by mecamylamine but not by hexamethonium. |
1993-12-06 |
2023-08-12 |
mouse |
| R Oishi, K Suemaru, K Furuno, Y Gomita, K Saek. Possible explanations for the antagonism by nicotine against reserpine-induced depletion of monoamines in mouse brain. Naunyn-Schmiedeberg's archives of pharmacology. vol 348. issue 2. 1993-12-06. PMID:8232594. |
these results suggest that inhibitory effect of nicotine on the intraperitoneal reserpine-induced depletion of brain monoamines is due to an inhibition of absorption of reserpine, and that central nicotinic action is also involved in the antagonism by nicotine of reserpine-induced dopamine depletion. |
1993-12-06 |
2023-08-12 |
mouse |
| B Hiremagalur, B Nankova, J Nitahara, R Zeman, E L Sabba. Nicotine increases expression of tyrosine hydroxylase gene. Involvement of protein kinase A-mediated pathway. The Journal of biological chemistry. vol 268. issue 31. 1993-11-29. PMID:7901211. |
nicotine, a major component of tobacco smoke, stimulates catecholamine secretion and activates catecholamine biosynthetic enzymes such as tyrosine hydroxylase (th) and dopamine beta-hydroxylase (dbh) in adrenal medullary cells. |
1993-11-29 |
2023-08-12 |
rat |
| A Ishikawa, T Miyatak. Effects of smoking in patients with early-onset Parkinson's disease. Journal of the neurological sciences. vol 117. issue 1-2. 1993-11-01. PMID:8410063. |
nicotine is thought to activate the nigrostriatal dopaminergic pathway and increase the release of dopamine in the striatum, and this can explain the effects of smoking in these patients. |
1993-11-01 |
2023-08-12 |
Not clear |
| M J Marks, S R Grady, A C Collin. Downregulation of nicotinic receptor function after chronic nicotine infusion. The Journal of pharmacology and experimental therapeutics. vol 266. issue 3. 1993-10-14. PMID:8371136. |
chronic nicotine infusion resulted in an infusion dose-dependent decrease in [3h]dopamine release from striatum and 86rb+ efflux from midbrain; cortical 86rb+ efflux was not affected by chronic nicotine treatment. |
1993-10-14 |
2023-08-12 |
mouse |
| H Mochizuki, H Miwa, H Imai, Y Muzun. [Hemiballism-hemichorea caused by a putaminal hemorrhage with worsening after smoking--a case report]. Rinsho shinkeigaku = Clinical neurology. vol 33. issue 5. 1993-10-05. PMID:8365067. |
the effect of smoking on his involuntary movements can be understood as a result of increase in the dopamine release from the nigrostriatal terminals induced by nicotine. |
1993-10-05 |
2023-08-12 |
Not clear |
| D Torbati, A K Sherpa, S Lahiri, A Mokashi, K H Albertine, C DiGiuli. Hyperbaric oxygenation alters carotid body ultrastructure and function. Respiration physiology. vol 92. issue 2. 1993-08-06. PMID:8327790. |
to test the hypothesis, we studied the effects of oxygen at 5 atmospheres absolute (ata) on cats (n = 7) carotid body ultrastructure and chemosensory responses to hypoxia, hypercapnia, and to bolus injections of cyanide, nicotine and dopamine. |
1993-08-06 |
2023-08-12 |
Not clear |
| D Torbati, A K Sherpa, S Lahiri, A Mokashi, K H Albertine, C DiGiuli. Hyperbaric oxygenation alters carotid body ultrastructure and function. Respiration physiology. vol 92. issue 2. 1993-08-06. PMID:8327790. |
the responses to hypercapnia or bolus injections of cyanide, nicotine and dopamine were normal. |
1993-08-06 |
2023-08-12 |
Not clear |
| T Watano, K Nakazawa, T Obama, M Mori, K Inoue, K Fujimori, A Takanak. Non-competitive antagonism by hirsuteine of nicotinic receptor-mediated dopamine release from rat pheochromocytoma cells. Japanese journal of pharmacology. vol 61. issue 4. 1993-08-03. PMID:8320880. |
hirsuteine (300 nm-10 microm) inhibited dopamine release evoked by 100 microm nicotine in a concentration-dependent manner. |
1993-08-03 |
2023-08-12 |
rat |