| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| Alzbeta Chorvatova, George Hart, Munir Hussai. Na+/Ca2+ exchange current (I(Na/Ca)) and sarcoplasmic reticulum Ca2+ release in catecholamine-induced cardiac hypertrophy. Cardiovascular research. vol 61. issue 2. 2004-05-20. PMID:14736544. |
we therefore investigated the effects of cardiac hypertrophy, produced by isoprenaline, on i(na/ca) and sarcoplasmic reticulum (sr) function in isolated myocytes. |
2004-05-20 |
2023-08-12 |
Not clear |
| Martin Braun, Gregor Simonis, Katja Birkner, Bernd Pauke, Ruth H Strasse. Regulation of protein kinase C isozyme and calcineurin expression in isoproterenol induced cardiac hypertrophy. Journal of cardiovascular pharmacology. vol 41. issue 6. 2004-04-12. PMID:12775975. |
regulation of protein kinase c isozyme and calcineurin expression in isoproterenol induced cardiac hypertrophy. |
2004-04-12 |
2023-08-12 |
rat |
| Shoko Miura, Isao Ohno, Jun Suzuki, Ko Suzuki, Shinji Okada, Akira Okuyama, Jun Nawata, Jun Ikeda, Kunio Shirat. Inhibition of matrix metalloproteinases prevents cardiac hypertrophy induced by beta-adrenergic stimulation in rats. Journal of cardiovascular pharmacology. vol 42. issue 2. 2004-01-13. PMID:12883319. |
they examined the expression of mmps in cardiac tissues of rats infused with isoproterenol (3 mg/kg per day), the effect of a mmp inhibitor, si-27 (5 mg/rat per day), on cardiac hypertrophy, and the expression of igf-i and igfbp-3. |
2004-01-13 |
2023-08-12 |
rat |
| Shoko Miura, Isao Ohno, Jun Suzuki, Ko Suzuki, Shinji Okada, Akira Okuyama, Jun Nawata, Jun Ikeda, Kunio Shirat. Inhibition of matrix metalloproteinases prevents cardiac hypertrophy induced by beta-adrenergic stimulation in rats. Journal of cardiovascular pharmacology. vol 42. issue 2. 2004-01-13. PMID:12883319. |
these results suggest that the induction of myocyte hypertrophy by isoproterenol is mediated, at least in part, by a modulation of the igf-i axis. |
2004-01-13 |
2023-08-12 |
rat |
| Ahmad Zahabi, Sylvie Picard, Nadia Fortin, Timothy L Reudelhuber, Christian F Descheppe. Expression of constitutively active guanylate cyclase in cardiomyocytes inhibits the hypertrophic effects of isoproterenol and aortic constriction on mouse hearts. The Journal of biological chemistry. vol 278. issue 48. 2004-01-12. PMID:14500707. |
expression of constitutively active guanylate cyclase in cardiomyocytes inhibits the hypertrophic effects of isoproterenol and aortic constriction on mouse hearts. |
2004-01-12 |
2023-08-12 |
mouse |
| Ahmad Zahabi, Sylvie Picard, Nadia Fortin, Timothy L Reudelhuber, Christian F Descheppe. Expression of constitutively active guanylate cyclase in cardiomyocytes inhibits the hypertrophic effects of isoproterenol and aortic constriction on mouse hearts. The Journal of biological chemistry. vol 278. issue 48. 2004-01-12. PMID:14500707. |
however, expression of the transgene attenuated the effects of the pharmacologic hypertrophic agent isoproterenol on cardiac wall thickness and prevented the onset of the fetal gene expression program normally associated with cardiac hypertrophy. |
2004-01-12 |
2023-08-12 |
mouse |
| Rafal M Kedzierski, Paul A Grayburn, Yaz Y Kisanuki, Clay S Williams, Robert E Hammer, James A Richardson, Micheal D Schneider, Masashi Yanagisaw. Cardiomyocyte-specific endothelin A receptor knockout mice have normal cardiac function and an unaltered hypertrophic response to angiotensin II and isoproterenol. Molecular and cellular biology. vol 23. issue 22. 2003-12-10. PMID:14585980. |
cardiomyocyte-specific endothelin a receptor knockout mice have normal cardiac function and an unaltered hypertrophic response to angiotensin ii and isoproterenol. |
2003-12-10 |
2023-08-12 |
mouse |
| Rafal M Kedzierski, Paul A Grayburn, Yaz Y Kisanuki, Clay S Williams, Robert E Hammer, James A Richardson, Micheal D Schneider, Masashi Yanagisaw. Cardiomyocyte-specific endothelin A receptor knockout mice have normal cardiac function and an unaltered hypertrophic response to angiotensin II and isoproterenol. Molecular and cellular biology. vol 23. issue 22. 2003-12-10. PMID:14585980. |
in addition, these animals exhibit hypertrophic and contractile responses to 10-day infusion of angiotensin ii or isoproterenol similar to those observed in control animals. |
2003-12-10 |
2023-08-12 |
mouse |
| Hyun Kook, John J Lepore, Aaron D Gitler, Min Min Lu, Wendy Wing-Man Yung, Joel Mackay, Rong Zhou, Victor Ferrari, Peter Gruber, Jonathan A Epstei. Cardiac hypertrophy and histone deacetylase-dependent transcriptional repression mediated by the atypical homeodomain protein Hop. The Journal of clinical investigation. vol 112. issue 6. 2003-11-03. PMID:12975471. |
in addition, trichostatin a also attenuates hypertrophy induced by infusion of isoproterenol. |
2003-11-03 |
2023-08-12 |
mouse |
| Remigio L\\xc3\\xb3pez Sol\\xc3\\xads, Margarita Puente D\\xc3\\xadaz, Irene Morales Bozo, Ulrike Kemmerling Weis, Francisco D\\xc3\\xada. Early detection in saliva of polypeptides associated to isoproterenol-induced mouse parotid hypertrophy. Biochimica et biophysica acta. vol 1621. issue 1. 2003-10-16. PMID:12667609. |
chronic administration of isoproterenol (ipr) results in a marked hypertrophy and in the induction of a group of putative proline-rich polypeptides in the mouse parotid glands. |
2003-10-16 |
2023-08-12 |
mouse |
| S Kumar, S Sharma, S S Katoc. Early onset of the maximum protein anabolic effect induced by isoproterenol in chick skeletal and cardiac muscle. Acta physiologica Hungarica. vol 90. issue 1. 2003-09-30. PMID:12666875. |
further, an increase in organ weight and total tissue proteins supported cardiac hypertrophy in chicks as a result of isoproterenol administration. |
2003-09-30 |
2023-08-12 |
chicken |
| L S Pogodina, M V Shornikova, Iu S CheNtso. [Changes in mitochondria and inter-mitochondrial junctions of the rat cardiomyocytes upon adrenergic stimulation by isoproterenol]. Izvestiia Akademii nauk. Seriia biologicheskaia. issue 2. 2003-07-14. PMID:12712574. |
it was found that isoproterenol injections (0.3 mg/kg for eight days) resulted in myocardial hypertrophy, which was more pronounced in the right than in the left ventricle. |
2003-07-14 |
2023-08-12 |
rat |
| Masanori Ozaki, Seinosuke Kawashima, Tomoya Yamashita, Tetsuaki Hirase, Yoshitaka Ohashi, Nobutaka Inoue, Ken-ichi Hirata, Mitsuhiro Yokoyam. Overexpression of endothelial nitric oxide synthase attenuates cardiac hypertrophy induced by chronic isoproterenol infusion. Circulation journal : official journal of the Japanese Circulation Society. vol 66. issue 9. 2003-06-06. PMID:12224825. |
overexpression of endothelial nitric oxide synthase attenuates cardiac hypertrophy induced by chronic isoproterenol infusion. |
2003-06-06 |
2023-08-12 |
mouse |
| Masanori Ozaki, Seinosuke Kawashima, Tomoya Yamashita, Tetsuaki Hirase, Yoshitaka Ohashi, Nobutaka Inoue, Ken-ichi Hirata, Mitsuhiro Yokoyam. Overexpression of endothelial nitric oxide synthase attenuates cardiac hypertrophy induced by chronic isoproterenol infusion. Circulation journal : official journal of the Japanese Circulation Society. vol 66. issue 9. 2003-06-06. PMID:12224825. |
the present study was designed to determine whether overproduction of endothelial no synthase (enos) could inhibit cardiac hypertrophy induced by chronic isoproterenol (iso) infusion (30mg/kg per day) using enos overexpressing (enos-tg) mice and wild-type (wt) mice. |
2003-06-06 |
2023-08-12 |
mouse |
| Julian C Braz, Orlando F Bueno, Qiangrong Liang, Benjamin J Wilkins, Yan-Shan Dai, Stephanie Parsons, Joseph Braunwart, Betty J Glascock, Raisa Klevitsky, Thomas F Kimball, Timothy E Hewett, Jeffery D Molkenti. Targeted inhibition of p38 MAPK promotes hypertrophic cardiomyopathy through upregulation of calcineurin-NFAT signaling. The Journal of clinical investigation. vol 111. issue 10. 2003-06-04. PMID:12750397. |
moreover, dominant-negative p38alpha, mkk3, and mkk6 transgenic mice each showed enhanced cardiac hypertrophy following aortic banding, ang ii infusion, isoproterenol infusion, or phenylephrine infusion for 14 days. |
2003-06-04 |
2023-08-12 |
mouse |
| Keita Odashiro, Shin-ichi Hiramatsu, Nobuhito Yanagi, Takeshi Arita, Toru Maruyama, Yoshikazu Kaji, Mine Harad. Arrhythmogenic and inotropic effects of interferon investigated in perfused and in vivo rat hearts: influences of cardiac hypertrophy and isoproterenol. Circulation journal : official journal of the Japanese Circulation Society. vol 66. issue 12. 2003-05-08. PMID:12499625. |
arrhythmogenic and inotropic effects of interferon investigated in perfused and in vivo rat hearts: influences of cardiac hypertrophy and isoproterenol. |
2003-05-08 |
2023-08-12 |
human |
| Danelle Badenhorst, Demetri Veliotes, Muzi Maseko, Oupa J Tsotetsi, Richard Brooksbank, Alvin Naidoo, Angela J Woodiwiss, Gavin R Norto. Beta-adrenergic activation initiates chamber dilatation in concentric hypertrophy. Hypertension (Dallas, Tex. : 1979). vol 41. issue 3. 2003-04-11. PMID:12623950. |
we evaluated the effect of 5 months of isoprenaline (0.02 mg x kg(-1) x d(-1)) on hemodynamics, lv wall thickness, cavity size, and interstitial characteristics in spontaneously hypertensive rats (shr) with compensated lv hypertrophy. |
2003-04-11 |
2023-08-12 |
rat |
| Andrzej M Janczewski, Toshiaki Kadokami, Bonnie Lemster, Carole S Frye, Charles F McTiernan, Arthur M Feldma. Morphological and functional changes in cardiac myocytes isolated from mice overexpressing TNF-alpha. American journal of physiology. Heart and circulatory physiology. vol 284. issue 3. 2003-03-11. PMID:12578819. |
tg-f mice showed lv hypertrophy without dilatation and only a small reduction of basal fractional shortening (fs) and response to isoproterenol (iso). |
2003-03-11 |
2023-08-12 |
mouse |
| Mar\\xc3\\xada Paz Ocaranza, Guillermo D\\xc3\\xadaz-Araya, Mario Chiong, David Mu\\xc3\\xb1oz, Juan Pablo Riveros, Roberto Ebensperger, Sebasti\\xc3\\xa1n Sabat, Pablo Irarr\\xc3\\xa1zaval, Jorge E Jalil, Sergio Lavander. Isoproterenol and angiotensin I-converting enzyme in lung, left ventricle, and plasma during myocardial hypertrophy and fibrosis. Journal of cardiovascular pharmacology. vol 40. issue 2. 2002-12-24. PMID:12131554. |
isoproterenol and angiotensin i-converting enzyme in lung, left ventricle, and plasma during myocardial hypertrophy and fibrosis. |
2002-12-24 |
2023-08-12 |
rat |
| Mar\\xc3\\xada Paz Ocaranza, Guillermo D\\xc3\\xadaz-Araya, Mario Chiong, David Mu\\xc3\\xb1oz, Juan Pablo Riveros, Roberto Ebensperger, Sebasti\\xc3\\xa1n Sabat, Pablo Irarr\\xc3\\xa1zaval, Jorge E Jalil, Sergio Lavander. Isoproterenol and angiotensin I-converting enzyme in lung, left ventricle, and plasma during myocardial hypertrophy and fibrosis. Journal of cardiovascular pharmacology. vol 40. issue 2. 2002-12-24. PMID:12131554. |
this study investigated whether long-term administration of isoproterenol (iso) induces differential expression of angiotensin-converting enzyme (ace) in lung, plasma, and left ventricle (lv) during development of left ventricular hypertrophy (lvh) and myocardial fibrosis. |
2002-12-24 |
2023-08-12 |
rat |