| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| V Bassareo, G Tanda, P Petromilli, C Giua, G Di Chiar. Non-psychostimulant drugs of abuse and anxiogenic drugs activate with differential selectivity dopamine transmission in the nucleus accumbens and in the medial prefrontal cortex of the rat. Psychopharmacology. vol 124. issue 4. 1996-11-25. PMID:8739543. |
systemic administration of low doses of the 5ht3 antagonist ics 205930, previously reported to prevent the increase of extracellular dopamine in the nucleus accumbens elicited by morphine, nicotine, ethanol and haloperidol (carboni et al. |
1996-11-25 |
2023-08-12 |
rat |
| C Ito, K Onodera, E Sakurai, M Sato, T Watanab. Effects of dopamine antagonists on neuronal histamine release in the striatum of rats subjected to acute and chronic treatments with methamphetamine. The Journal of pharmacology and experimental therapeutics. vol 279. issue 1. 1996-11-18. PMID:8859003. |
pretreatments with the dopamine d2 antagonists sulpiride and haloperidol blocked the meth-induced increase of ha release, whereas pretreatment with a dopamine d1 antagonist, sch23390, did not. |
1996-11-18 |
2023-08-12 |
rat |
| R J Boegman, S R Vincen. Involvement of adenosine and glutamate receptors in the induction of c-fos in the striatum by haloperidol. Synapse (New York, N.Y.). vol 22. issue 1. 1996-11-08. PMID:8822480. |
conversely, neuroleptic drugs, such as haloperidol, which block d2-type dopamine receptors, induce c-fos expression in striatal neurons projecting to the globus pallidus. |
1996-11-08 |
2023-08-12 |
Not clear |
| R E See, A M Lynch, M Aravagiri, C B Nemeroff, M J Owen. Chronic haloperidol-induced changes in regional dopamine release and metabolism and neurotensin content in rats. Brain research. vol 704. issue 2. 1996-10-31. PMID:8788915. |
these results suggest a confluence of altered dopamine and neurotensin function in the caudate putamen which may be related to motor side effects of haloperidol, whereas changes in prefrontal dopamine function are not associated with altered neurotensin levels. |
1996-10-31 |
2023-08-12 |
rat |
| R E See, A M Lynch, B A Sor. Subchronic administration of clozapine, but not haloperidol or metoclopramide, decreases dopamine D2 receptor messenger RNA levels in the nucleus accumbens and caudate-putamen in rats. Neuroscience. vol 72. issue 1. 1996-10-24. PMID:8730709. |
subchronic administration of clozapine, but not haloperidol or metoclopramide, decreases dopamine d2 receptor messenger rna levels in the nucleus accumbens and caudate-putamen in rats. |
1996-10-24 |
2023-08-12 |
rat |
| A Bourson, D Wanner, R Wyler, N Petit, C Zwingelstein, A Rudler, A J Sleigh. Pharmacologic evaluation of the discriminative stimulus of metachlorophenylpiperazine. Pharmacology, biochemistry, and behavior. vol 53. issue 1. 1996-10-24. PMID:8848438. |
haloperidol generalises, and amphetamine partially antagonises the mcpp discriminative cue and low doses of apomorphine partially generalises to the mcpp cue, which suggests that a decrease in dopamine neurotransmission may also be involved. |
1996-10-24 |
2023-08-12 |
rat |
| R J Rodgers, N J Johnson, A J Champion, S Mill. Modulation of plus-maze behaviour in mice by the preferential D3-receptor agonist 7-OH-DPAT. Pharmacology, biochemistry, and behavior. vol 54. issue 1. 1996-10-23. PMID:8728542. |
differences in the behavioural profiles of dopamine d2 receptor antagonists (e.g., haloperidol vs. sulpiride) in a animal models of anxiety have prompted speculation concerning the importance of their relative affinities for d2-like receptor populations. |
1996-10-23 |
2023-08-12 |
mouse |
| I Bodis-Wollner, A Anta. On the functional significance of primate retinal dopamine receptors. Journal of neural transmission. Supplementum. vol 45. 1996-10-23. PMID:8748611. |
studies were performed using haloperidol, 1-sulpiride, cy 208-243 and dopamine. |
1996-10-23 |
2023-08-12 |
human |
| A Imperato, M C Obinu, G L Gess. Is in vivo striatal acetylcholine output under a tonic inhibitory control by dopamine? Journal of neural transmission. Supplementum. vol 45. 1996-10-23. PMID:8748614. |
blockade of dopamine d2 receptors with (-)-sulpiride or haloperidol increased acetylcholine release to a maximum of 80% (after 50 and 0.5 mg/kg, respectively). |
1996-10-23 |
2023-08-12 |
rat |
| J T Cheng, T C Lin, F L Hs. Antihypertensive effect of corilagin in the rat. Canadian journal of physiology and pharmacology. vol 73. issue 10. 1996-10-23. PMID:8748933. |
failure of the antagonists for alpha2-adrenoceptors, idazoxan and yohimbine, as well as for dopamine receptors, haloperidol and domperidone, to reverse the antihypertensive actions of corilagin ruled out the participation of these receptors. |
1996-10-23 |
2023-08-12 |
rat |
| L Antkiewicz-Michaluk, B Karolewicz, J Michaluk, J Vetulan. Differences between haloperidol- and pimozide-induced withdrawal syndrome: a role for Ca2+ channels. European journal of pharmacology. vol 294. issue 2-3. 1996-10-23. PMID:8750706. |
after haloperidol treatment we observed an increase in cortical ca2+ channel and limbic dopamine d1 receptor density and an increase in spontaneous motor activity and apomorphine-induced hyperactivity and stereotypy. |
1996-10-23 |
2023-08-12 |
rat |
| K Ossowsk. The subsensitivity of striatal glutamate receptors induced by chronic haloperidol in rats. European journal of pharmacology. vol 294. issue 2-3. 1996-10-23. PMID:8750734. |
the present results seem to suggest that, apart from supersensitivity to dopamine, chronic treatment with haloperidol also induces subsensitivity of striatal nmda and non-nmda receptors. |
1996-10-23 |
2023-08-12 |
rat |
| J P Doucet, Y Nakabeppu, P J Bedard, B T Hope, E J Nestler, B J Jasmin, J S Chen, M J Iadarola, M St-Jean, N Wigle, P Blanchet, R Grondin, G S Robertso. Chronic alterations in dopaminergic neurotransmission produce a persistent elevation of deltaFosB-like protein(s) in both the rodent and primate striatum. The European journal of neuroscience. vol 8. issue 2. 1996-10-22. PMID:8714707. |
chronic haloperidol administration also selectively elevated expression of these fos-related antigens, suggesting that their induction after dopaminergic denervation is mediated by reduced activation of d2-like dopamine receptors. |
1996-10-22 |
2023-08-12 |
rat |
| A J Chesi, K J Feasey-Truger, C Alzheimer, G ten Bruggencat. Dopamine autoreceptor sensitivity is unchanged in rat nucleus accumbens after chronic haloperidol treatment: an in vivo and in vitro voltammetric study. The European journal of neuroscience. vol 7. issue 12. 1996-10-22. PMID:8845950. |
dopamine autoreceptor sensitivity is unchanged in rat nucleus accumbens after chronic haloperidol treatment: an in vivo and in vitro voltammetric study. |
1996-10-22 |
2023-08-12 |
rat |
| A J Chesi, K J Feasey-Truger, C Alzheimer, G ten Bruggencat. Dopamine autoreceptor sensitivity is unchanged in rat nucleus accumbens after chronic haloperidol treatment: an in vivo and in vitro voltammetric study. The European journal of neuroscience. vol 7. issue 12. 1996-10-22. PMID:8845950. |
fast cyclic voltammetry was used to assess the effects of chronic oral haloperidol treatment (0.7 mg/kg/day for 21 days) on the sensitivity of dopamine autoreceptors in the rat nucleus accumbens both in vivo and in vitro. |
1996-10-22 |
2023-08-12 |
rat |
| A J Chesi, K J Feasey-Truger, C Alzheimer, G ten Bruggencat. Dopamine autoreceptor sensitivity is unchanged in rat nucleus accumbens after chronic haloperidol treatment: an in vivo and in vitro voltammetric study. The European journal of neuroscience. vol 7. issue 12. 1996-10-22. PMID:8845950. |
evoked dopamine overflow was significantly reduced after chronic haloperidol treatment, but the sensitivity of dopamine overflow to sulpiride, an antagonist at release-inhibiting dopamine autoreceptors, and quinpirole, an agonist at these receptors, was unchanged. |
1996-10-22 |
2023-08-12 |
rat |
| A J Chesi, K J Feasey-Truger, C Alzheimer, G ten Bruggencat. Dopamine autoreceptor sensitivity is unchanged in rat nucleus accumbens after chronic haloperidol treatment: an in vivo and in vitro voltammetric study. The European journal of neuroscience. vol 7. issue 12. 1996-10-22. PMID:8845950. |
the finding that the reduced dopamine overflow in the nucleus accumbens observed after chronic treatment with a classical neuroleptic is not due to dopamine autoreceptor supersensitivity may therefore be the first functional evidence for unchanged autoreceptor activity in the nucleus accumbens, supporting biochemical findings of a lack of d3 autoreceptor up-regulation after chronic haloperidol treatment. |
1996-10-22 |
2023-08-12 |
rat |
| I Ushijima, Y Mizuki, M Yamad. Alteration of cataleptic responses induced by dopamine receptor antagonists after chronic cocaine administration in mice. European journal of pharmacology. vol 285. issue 1. 1996-10-21. PMID:8846811. |
the influence of chronic treatment of mice with cocaine, an indirect dopamine receptor agonist, on the cataleptic effects of r-(+)-chloro-2,3,4,5-tetrahydro-3-methyl-5-phenyl-1h-3-benzazepin- 7ol hydrochloride (sch23390), a dopamine d1 receptor antagonist, or haloperidol, mainly a dopamine d2 receptor antagonist, was investigated. |
1996-10-21 |
2023-08-12 |
mouse |
| H L Andersen, I C Kilpatric. Prevention by (+/-)-8-hydroxy-2-(di-n-propylamino)tetralin of both catalepsy and the rises in rat striatal dopamine metabolism caused by haloperidol. British journal of pharmacology. vol 118. issue 2. 1996-10-16. PMID:8735647. |
prevention by (+/-)-8-hydroxy-2-(di-n-propylamino)tetralin of both catalepsy and the rises in rat striatal dopamine metabolism caused by haloperidol. |
1996-10-16 |
2023-08-12 |
rat |
| H L Andersen, I C Kilpatric. Prevention by (+/-)-8-hydroxy-2-(di-n-propylamino)tetralin of both catalepsy and the rises in rat striatal dopamine metabolism caused by haloperidol. British journal of pharmacology. vol 118. issue 2. 1996-10-16. PMID:8735647. |
these results suggest that part of the mechanism by which 8-oh-dpat prevents haloperidol-induced catalepsy is reflected by a reversal of the compensatory increase in meso-striatal and/or meso-cortical dopamine neuronal activity that normally accompanies postsynaptic dopamine receptor blockade with haloperidol. |
1996-10-16 |
2023-08-12 |
rat |