| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| Mahendra Bishnoi, Kanwaljit Chopra, Shrinivas K Kulkarn. Involvement of adenosinergic receptor system in an animal model of tardive dyskinesia and associated behavioural, biochemical and neurochemical changes. European journal of pharmacology. vol 552. issue 1-3. 2007-01-04. PMID:17064683. |
on chronic administration of haloperidol there was a decrease in dopamine and norepinephrine turnover which was dose-dependently reversed by treatment with adenosine or caffeine. |
2007-01-04 |
2023-08-12 |
rat |
| A Fredriksson, T Arche. Subchronic administration of haloperidol influences the functional deficits of postnatal iron administration in mice. Neurotoxicity research. vol 10. issue 2. 2007-01-03. PMID:17062374. |
in the context of these and other observations, it is suggested that subchronic administration of haloperidol interacting with postnatal iron induces different expressions of dopamine neuron comorbidity underlying movement disorder. |
2007-01-03 |
2023-08-12 |
mouse |
| Huma Saeedi, Gary Remington, Bruce K Christense. Impact of haloperidol, a dopamine D2 antagonist, on cognition and mood. Schizophrenia research. vol 85. issue 1-3. 2006-12-28. PMID:16679001. |
impact of haloperidol, a dopamine d2 antagonist, on cognition and mood. |
2006-12-28 |
2023-08-12 |
human |
| Dineshree V Naiker, Stanley V Catts, Vibeke S Catts, Kuldip S Bedi, Lesley J Bryan-Lluk. Dose determination of haloperidol, risperidone and olanzapine using an in vivo dopamine D2-receptor occupancy method in the rat. European journal of pharmacology. vol 540. issue 1-3. 2006-12-11. PMID:16730699. |
dose determination of haloperidol, risperidone and olanzapine using an in vivo dopamine d2-receptor occupancy method in the rat. |
2006-12-11 |
2023-08-12 |
rat |
| Dineshree V Naiker, Stanley V Catts, Vibeke S Catts, Kuldip S Bedi, Lesley J Bryan-Lluk. Dose determination of haloperidol, risperidone and olanzapine using an in vivo dopamine D2-receptor occupancy method in the rat. European journal of pharmacology. vol 540. issue 1-3. 2006-12-11. PMID:16730699. |
the purpose of the present study was to determine antipsychotic doses that achieve 80% striatal dopamine d2-receptor occupancy for haloperidol, risperidone and olanzapine in rats. |
2006-12-11 |
2023-08-12 |
rat |
| Dineshree V Naiker, Stanley V Catts, Vibeke S Catts, Kuldip S Bedi, Lesley J Bryan-Lluk. Dose determination of haloperidol, risperidone and olanzapine using an in vivo dopamine D2-receptor occupancy method in the rat. European journal of pharmacology. vol 540. issue 1-3. 2006-12-11. PMID:16730699. |
the doses required to achieve dopamine d2-receptor occupancy of 80% in 11- and 24-week old rats were: haloperidol 0.25 mg/kg/day, risperidone 5 mg/kg/day and olanzapine 10 mg/kg/day. |
2006-12-11 |
2023-08-12 |
rat |
| Ella H Sklan, Amit Berson, Klara R Birikh, Amos Gutnick, Or Shahar, Shai Shoham, Hermona Sore. Acetylcholinesterase modulates stress-induced motor responses through catalytic and noncatalytic properties. Biological psychiatry. vol 60. issue 7. 2006-11-07. PMID:16904653. |
more specifically, we induced stress-associated hypofunction of dopaminergic, mainly d2 dopamine receptor-mediated neurotransmission by haloperidol and explored stress induced hyperlocomotion and catalepsy, an extreme form of immobility, induced in mice with ache deficiencies. |
2006-11-07 |
2023-08-12 |
mouse |
| C R Maxwell, Y Liang, M P Kelly, S J Kanes, T Abel, S J Siege. Mice expressing constitutively active Gsalpha exhibit stimulus encoding deficits similar to those observed in schizophrenia patients. Neuroscience. vol 141. issue 3. 2006-10-18. PMID:16750890. |
n40 deficits in transgenic animals were ameliorated with low dose haloperidol and reversed with higher dose, suggesting dopamine d2 receptor-linked gi activity contributes to the impairment. |
2006-10-18 |
2023-08-12 |
mouse |
| Jeffrey B Eells, Jaime A Misler, Vera M Nikode. Reduced tyrosine hydroxylase and GTP cyclohydrolase mRNA expression, tyrosine hydroxylase activity, and associated neurochemical alterations in Nurr1-null heterozygous mice. Brain research bulletin. vol 70. issue 2. 2006-10-10. PMID:16782508. |
furthermore, dopamine levels in the striatum of +/- mice were significantly reduced after inhibition of dopamine synthesis or after haloperidol treatment but not under basal conditions while dopamine levels in the nucleus accumbens were reduced under basal conditions. |
2006-10-10 |
2023-08-12 |
mouse |
| Ellen R A de Bruijn, Bernard G C Sabbe, Wouter Hulstijn, Gé S F Ruigt, Robbert J Verke. Effects of antipsychotic and antidepressant drugs on action monitoring in healthy volunteers. Brain research. vol 1105. issue 1. 2006-09-29. PMID:16499887. |
the attenuated erns after the dopamine antagonist haloperidol are in line with the presumed role of dopamine in action monitoring. |
2006-09-29 |
2023-08-12 |
human |
| Tim Karl, Liesl Duffy, Elizabeth O'brien, Izuru Matsumoto, Irina Dedov. Behavioural effects of chronic haloperidol and risperidone treatment in rats. Behavioural brain research. vol 171. issue 2. 2006-09-25. PMID:16697060. |
the therapeutic properties of typical antipsychotic drugs (apds) such as haloperidol in schizophrenia treatment are mainly associated with their ability to block dopamine d2 receptors. |
2006-09-25 |
2023-08-12 |
rat |
| S J Chrapusta, M F Ega. Poor evidence for depolarization block but uncoupling of nigral from striatal dopamine metabolism after chronic haloperidol treatment in the rat. Journal of neural transmission (Vienna, Austria : 1996). vol 113. issue 5. 2006-09-19. PMID:16082510. |
poor evidence for depolarization block but uncoupling of nigral from striatal dopamine metabolism after chronic haloperidol treatment in the rat. |
2006-09-19 |
2023-08-12 |
rat |
| S J Chrapusta, M F Ega. Poor evidence for depolarization block but uncoupling of nigral from striatal dopamine metabolism after chronic haloperidol treatment in the rat. Journal of neural transmission (Vienna, Austria : 1996). vol 113. issue 5. 2006-09-19. PMID:16082510. |
chronic haloperidol treatment induces depolarization block in midbrain dopamine neuronal systems. |
2006-09-19 |
2023-08-12 |
rat |
| S J Chrapusta, M F Ega. Poor evidence for depolarization block but uncoupling of nigral from striatal dopamine metabolism after chronic haloperidol treatment in the rat. Journal of neural transmission (Vienna, Austria : 1996). vol 113. issue 5. 2006-09-19. PMID:16082510. |
haloperidol pretreatment significantly modified haloperidol challenge effect on regional dopamine metabolite contents. |
2006-09-19 |
2023-08-12 |
rat |
| S J Chrapusta, M F Ega. Poor evidence for depolarization block but uncoupling of nigral from striatal dopamine metabolism after chronic haloperidol treatment in the rat. Journal of neural transmission (Vienna, Austria : 1996). vol 113. issue 5. 2006-09-19. PMID:16082510. |
these results suggest that chronic haloperidol treatment uncouples somatodendritic dopamine turnover and release from those in the axon terminals of nigrostriatal dopamine neurons. |
2006-09-19 |
2023-08-12 |
rat |
| Michael J Burke, Jana Lincol. Aripiprazole and haloperidol: a clinically relevant interaction with a dopamine antagonist and partial agonist. Annals of clinical psychiatry : official journal of the American Academy of Clinical Psychiatrists. vol 18. issue 2. 2006-08-24. PMID:16754419. |
aripiprazole and haloperidol: a clinically relevant interaction with a dopamine antagonist and partial agonist. |
2006-08-24 |
2023-08-12 |
Not clear |
| Anders Fredriksson, Trevor Arche. Subchronic administration of haloperidol influences the functional deficits of postnatal iron administration in mice. Neurotoxicity research. vol 9. issue 4. 2006-08-23. PMID:16782590. |
in the context of these and other observations, it is suggested that subchronic administration of haloperidol interacting with postnatal iron induces different expressions of dopamine neuron comorbidity underlying movement disorder. |
2006-08-23 |
2023-08-12 |
mouse |
| M A Cheĭdo, G V Idov. [Differential contribution of dopamine D1 and D2 receptors in the mu-opioidergic immunomodulation]. Rossiiskii fiziologicheskii zhurnal imeni I.M. Sechenova. vol 92. issue 5. 2006-08-18. PMID:16869283. |
haloperidol (2 mg/kg), a selective antagonist of the postsynaptic dopamine (da) receptors, prevented immunostimulating effect of dago. |
2006-08-18 |
2023-08-12 |
mouse |
| G V Idova, M A Cheĭdo, E N Zhukov. [Effect of the activation and blockade of D2 dopamine receptors on the immune response in mice with different types of social behavior]. Rossiiskii fiziologicheskii zhurnal imeni I.M. Sechenova. vol 92. issue 5. 2006-08-18. PMID:16869284. |
at the same time, the blockade of d2 dopamine receptors with haloperidol suppressed immunogenesis in aggressive and control mice, whereas the immune reactions in submissive mice were unchanged. |
2006-08-18 |
2023-08-12 |
mouse |
| Seth Y Ablordeppey, Margaret Lyles-Eggleston, Barbara Bricker, Wang Zhang, Xue Zhu, Carl Goodman, Bryan L Rot. Evaluation of the eutomer of 4-{3-(4-chlorophenyl)-3-hydroxypyrrolidin-1-yl}-1-(4-fluorophenyl)butan-1-one, {(+)-SYA 09}, a pyrrolidine analog of haloperidol. Bioorganic & medicinal chemistry letters. vol 16. issue 12. 2006-08-16. PMID:16621538. |
enantiomeric separation of the racemic 4-{3-(4-chlorophenyl)-3-hydroxypyrrolidin-1-yl}-1-(4-fluorophenyl)butan-1-one, a pyrrolidine analog of haloperidol, {(+/-)-sya 09}, and subsequent binding studies revealed that most of the binding affinity at dopamine and serotonin receptors resides in the (+)-isomer {(+)-sya 09} or the eutomer. |
2006-08-16 |
2023-08-12 |
rat |