Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
P W Kalivas, P Duff. Similar effects of daily cocaine and stress on mesocorticolimbic dopamine neurotransmission in the rat. Biological psychiatry. vol 25. issue 7. 1989-06-23. PMID:2541803. |
similar effects of daily cocaine and stress on mesocorticolimbic dopamine neurotransmission in the rat. |
1989-06-23 |
2023-08-11 |
rat |
P W Kalivas, P Duff. Similar effects of daily cocaine and stress on mesocorticolimbic dopamine neurotransmission in the rat. Biological psychiatry. vol 25. issue 7. 1989-06-23. PMID:2541803. |
considering that both cocaine and stress enhance dopamine neurotransmission in the central nervous system, it was of interest to determine the effects of daily cocaine and stress on the capacity of acute stress to alter dopamine neurotransmission. |
1989-06-23 |
2023-08-11 |
rat |
P W Kalivas, P Duff. Similar effects of daily cocaine and stress on mesocorticolimbic dopamine neurotransmission in the rat. Biological psychiatry. vol 25. issue 7. 1989-06-23. PMID:2541803. |
it is proposed that pretreatment with cocaine or stress alters the response of the mesocorticolimbic dopamine neurons to subsequent stress, so that axonal dopamine neurotransmission is enhanced in the terminal fields and somatodendritic dopamine neurotransmission is diminished. |
1989-06-23 |
2023-08-11 |
rat |
P W Kalivas, P Duff. Similar effects of daily cocaine and stress on mesocorticolimbic dopamine neurotransmission in the rat. Biological psychiatry. vol 25. issue 7. 1989-06-23. PMID:2541803. |
furthermore, the long-lasting influence of daily cocaine and stress on mesocorticolimbic dopamine responsiveness to subsequent stressful experiences may be relevant in the etiology of psychostimulant-induced psychosis. |
1989-06-23 |
2023-08-11 |
rat |
G R Hanson, P Smiley, M Johnson, A Letter, L Bush, J W Gib. Response by the neurotensin systems of the basal ganglia to cocaine treatment. European journal of pharmacology. vol 160. issue 1. 1989-06-20. PMID:2714362. |
the response of the striatal nt systems to cocaine appeared to be mediated principally by dopamine d-1 receptors, while both d-1 and d-2 receptors contributed to the response by the nigral nt projections. |
1989-06-20 |
2023-08-11 |
Not clear |
G R Hanson, P Smiley, M Johnson, A Letter, L Bush, J W Gib. Response by the neurotensin systems of the basal ganglia to cocaine treatment. European journal of pharmacology. vol 160. issue 1. 1989-06-20. PMID:2714362. |
specific dopamine, but not serotonin, uptake blockers caused increases in striatal and nigral ntli concentrations similar to that seen with cocaine treatments, suggesting that interference with the dopamine uptake carrier complex by cocaine was responsible for its actions on extrapyramidal nt systems. |
1989-06-20 |
2023-08-11 |
Not clear |
J M Witkin, S R Goldberg, J L Kat. Lethal effects of cocaine are reduced by the dopamine-1 receptor antagonist SCH 23390 but not by haloperidol. Life sciences. vol 44. issue 18. 1989-06-14. PMID:2523996. |
these results suggest that dopamine receptors may play a role in the lethal effects of cocaine and that the d1 dopamine receptor subtype appears to be more relevant to lethality than the d2 subtype. |
1989-06-14 |
2023-08-11 |
rat |
L C Nicolaysen, J B Justic. Effects of cocaine on release and uptake of dopamine in vivo: differentiation by mathematical modeling. Pharmacology, biochemistry, and behavior. vol 31. issue 2. 1989-06-06. PMID:3072568. |
effects of cocaine on release and uptake of dopamine in vivo: differentiation by mathematical modeling. |
1989-06-06 |
2023-08-11 |
Not clear |
L C Nicolaysen, J B Justic. Effects of cocaine on release and uptake of dopamine in vivo: differentiation by mathematical modeling. Pharmacology, biochemistry, and behavior. vol 31. issue 2. 1989-06-06. PMID:3072568. |
although considerable effort has been invested trying to distinguish between the effects of cocaine on dopamine (da) uptake and release in both in vitro and in vivo experiments, disagreement over the specific actions of cocaine remains. |
1989-06-06 |
2023-08-11 |
Not clear |
U Filibeck, S Cabib, C Castellano, S Puglisi-Allegr. Chronic cocaine enhances defensive behaviour in the laboratory mouse: involvement of D2 dopamine receptors. Psychopharmacology. vol 96. issue 4. 1989-06-02. PMID:2907661. |
chronic cocaine enhances defensive behaviour in the laboratory mouse: involvement of d2 dopamine receptors. |
1989-06-02 |
2023-08-11 |
mouse |
L Estañ, I Martinez-Mir, E Rubio, F J Morales-Oliva. Relaxant effect of dopamine on the isolated rat uterus. Naunyn-Schmiedeberg's archives of pharmacology. vol 338. issue 5. 1989-06-02. PMID:3072485. |
similarly, cocaine (3 x 10(-6) m) failed to modify the relaxant effect of dopamine. |
1989-06-02 |
2023-08-11 |
rat |
E Carboni, A Imperato, L Perezzani, G Di Chiar. Amphetamine, cocaine, phencyclidine and nomifensine increase extracellular dopamine concentrations preferentially in the nucleus accumbens of freely moving rats. Neuroscience. vol 28. issue 3. 1989-05-31. PMID:2710338. |
amphetamine, cocaine, phencyclidine and nomifensine increase extracellular dopamine concentrations preferentially in the nucleus accumbens of freely moving rats. |
1989-05-31 |
2023-08-11 |
rat |
E Carboni, A Imperato, L Perezzani, G Di Chiar. Amphetamine, cocaine, phencyclidine and nomifensine increase extracellular dopamine concentrations preferentially in the nucleus accumbens of freely moving rats. Neuroscience. vol 28. issue 3. 1989-05-31. PMID:2710338. |
the effect of systemically administered amphetamine, cocaine, phencyclidine and nomifensine on the extracellular concentrations of dopamine in freely moving rats was estimated by microdialysis in the nucleus accumbens and in the dorsal caudate. |
1989-05-31 |
2023-08-11 |
rat |
E Carboni, A Imperato, L Perezzani, G Di Chiar. Amphetamine, cocaine, phencyclidine and nomifensine increase extracellular dopamine concentrations preferentially in the nucleus accumbens of freely moving rats. Neuroscience. vol 28. issue 3. 1989-05-31. PMID:2710338. |
low doses of cocaine (1.0 mg/kg s.c.) stimulated dopamine output only in the nucleus accumbens. |
1989-05-31 |
2023-08-11 |
rat |
E Carboni, A Imperato, L Perezzani, G Di Chiar. Amphetamine, cocaine, phencyclidine and nomifensine increase extracellular dopamine concentrations preferentially in the nucleus accumbens of freely moving rats. Neuroscience. vol 28. issue 3. 1989-05-31. PMID:2710338. |
thus, in contrast with amphetamine, cocaine, phencyclidine and nomifensine increase synaptic dopamine concentrations in vivo by a mechanism which depends on intact activity of dopaminergic neurons and by an exocytotic process. |
1989-05-31 |
2023-08-11 |
rat |
D Branco, W Osswal. Spontaneous and ouabain-induced efflux of catecholamines and dihydroxyphenylglycol in two canine blood vessels. Fundamental & clinical pharmacology. vol 2. issue 6. 1989-05-16. PMID:3240915. |
the ratio dopamine:noradrenaline was decreased by cocaine and by ouabain. |
1989-05-16 |
2023-08-11 |
Not clear |
J B Kamien, W L Woolverto. A pharmacological analysis of the discriminative stimulus properties of d-amphetamine in rhesus monkeys. The Journal of pharmacology and experimental therapeutics. vol 248. issue 3. 1989-05-12. PMID:2649658. |
amph (0.08-2.6 mumol/kg), cocaine (0.06-1.0 mumol/kg; n = 4), the dopamine (da) uptake inhibitor bupropion (0.25-2.0 mumol/kg; n = 2) and the norepinephrine (ne) uptake inhibitor nisoxetine (1.0-16 mumol/kg; n = 4) produced dose-related increases in the percentage of responses that occurred on the amph-appropriate lever during test sessions in all monkeys tested. |
1989-05-12 |
2023-08-11 |
monkey |
M C Ritz, M J Kuha. Relationship between self-administration of amphetamine and monoamine receptors in brain: comparison with cocaine. The Journal of pharmacology and experimental therapeutics. vol 248. issue 3. 1989-05-12. PMID:2703961. |
we have shown previously that the reinforcing effects of cocaine can be correlated with drug binding to the mazindol and gbr 12935 binding sites on the dopamine transporter. |
1989-05-12 |
2023-08-11 |
Not clear |
D W Teller, P Deveny. Bromocriptine in cocaine withdrawal--does it work? The International journal of the addictions. vol 23. issue 11. 1989-05-05. PMID:3069753. |
heavy cocaine use has been reported to lead to dopamine depletion in the brain, which in turn may be responsible for strong cocaine craving after withdrawal. |
1989-05-05 |
2023-08-11 |
Not clear |
D W Teller, P Deveny. Bromocriptine in cocaine withdrawal--does it work? The International journal of the addictions. vol 23. issue 11. 1989-05-05. PMID:3069753. |
in an uncontrolled trial of 25 heavy cocaine users, measurements of pre- and post-bromocriptine serum prolactin levels-as indicators of inhibitory dopaminergic control-did not suggest dopamine depletion. |
1989-05-05 |
2023-08-11 |
Not clear |