| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| B Moghaddam, B S Bunne. Differential effect of cocaine on extracellular dopamine levels in rat medial prefrontal cortex and nucleus accumbens: comparison to amphetamine. Synapse (New York, N.Y.). vol 4. issue 2. 1989-10-19. PMID:2781466. |
differential effect of cocaine on extracellular dopamine levels in rat medial prefrontal cortex and nucleus accumbens: comparison to amphetamine. |
1989-10-19 |
2023-08-11 |
rat |
| B Moghaddam, B S Bunne. Differential effect of cocaine on extracellular dopamine levels in rat medial prefrontal cortex and nucleus accumbens: comparison to amphetamine. Synapse (New York, N.Y.). vol 4. issue 2. 1989-10-19. PMID:2781466. |
the technique of in vivo microdialysis was used to measure the extracellular levels of dopamine in the nucleus accumbens septi and medial prefrontal cortex of chloral-hydrate-anaesthetized rats following systemic administration of cocaine and amphetamine. |
1989-10-19 |
2023-08-11 |
rat |
| B Moghaddam, B S Bunne. Differential effect of cocaine on extracellular dopamine levels in rat medial prefrontal cortex and nucleus accumbens: comparison to amphetamine. Synapse (New York, N.Y.). vol 4. issue 2. 1989-10-19. PMID:2781466. |
intravenous injection of cocaine increased the extracellular levels of dopamine in the medial prefrontal cortex and the nucleus accumbens septi in a dose-dependent manner. |
1989-10-19 |
2023-08-11 |
rat |
| B Moghaddam, B S Bunne. Differential effect of cocaine on extracellular dopamine levels in rat medial prefrontal cortex and nucleus accumbens: comparison to amphetamine. Synapse (New York, N.Y.). vol 4. issue 2. 1989-10-19. PMID:2781466. |
in comparison to cocaine, amphetamine increased the extracellular levels of dopamine in the nucleus accumbens and medial prefrontal cortex to the same degree. |
1989-10-19 |
2023-08-11 |
rat |
| B Moghaddam, B S Bunne. Differential effect of cocaine on extracellular dopamine levels in rat medial prefrontal cortex and nucleus accumbens: comparison to amphetamine. Synapse (New York, N.Y.). vol 4. issue 2. 1989-10-19. PMID:2781466. |
based on the relatively small increase of extracellular dopamine levels in medial prefrontal cortex by cocaine, it is postulated that dopaminergic innervation of other structures besides medial prefrontal cortex may be involved in maintenance of cocaine self-administration. |
1989-10-19 |
2023-08-11 |
rat |
| S D Martin, V K Yeragani, R Lodhi, M P Gallowa. Clinical ratings and plasma HVA during cocaine abstinence. Biological psychiatry. vol 26. issue 4. 1989-09-27. PMID:2765598. |
the data provide preliminary evidence to suggest that changes in cocaine craving during abstinence are positively correlated with changes in dopamine turnover. |
1989-09-27 |
2023-08-11 |
Not clear |
| K Akimoto, T Hamamura, S Otsuk. Subchronic cocaine treatment enhances cocaine-induced dopamine efflux, studied by in vivo intracerebral dialysis. Brain research. vol 490. issue 2. 1989-09-25. PMID:2765867. |
subchronic cocaine treatment enhances cocaine-induced dopamine efflux, studied by in vivo intracerebral dialysis. |
1989-09-25 |
2023-08-11 |
rat |
| K Akimoto, T Hamamura, S Otsuk. Subchronic cocaine treatment enhances cocaine-induced dopamine efflux, studied by in vivo intracerebral dialysis. Brain research. vol 490. issue 2. 1989-09-25. PMID:2765867. |
2: 7 days after withdrawal from the drug, we measured extracellular dopamine (da), 3,4-dihydroxyphenylacetic acid (dopac) and homovanillic acid (hva) after the challenge administration of cocaine using an in vivo intracerebral dialysis technique. |
1989-09-25 |
2023-08-11 |
rat |
| R Cavanagh, J S Clifford, W L Gregor. The use of bromocriptine for the treatment of attention deficit disorder in two chemically dependent patients. Journal of psychoactive drugs. vol 21. issue 2. 1989-09-20. PMID:2668485. |
the dually diagnosed patient with attention deficit disorder, residual type (add-rt) may be especially prone to cocaine abuse, because add includes dopamine deficiencies and cocaine is a dopamine agonist. |
1989-09-20 |
2023-08-11 |
Not clear |
| R Cavanagh, J S Clifford, W L Gregor. The use of bromocriptine for the treatment of attention deficit disorder in two chemically dependent patients. Journal of psychoactive drugs. vol 21. issue 2. 1989-09-20. PMID:2668485. |
it is hypothesized that bromocriptine, a dopamine agonist, will reduce a patient's craving for cocaine and simultaneously address the patient's add-rt symptoms. |
1989-09-20 |
2023-08-11 |
Not clear |
| W L Woolverton, R M Viru. The effects of a D1 and a D2 dopamine antagonist on behavior maintained by cocaine or food. Pharmacology, biochemistry, and behavior. vol 32. issue 3. 1989-08-01. PMID:2662223. |
the effects of a d1 and a d2 dopamine antagonist on behavior maintained by cocaine or food. |
1989-08-01 |
2023-08-11 |
monkey |
| W L Woolverton, R M Viru. The effects of a D1 and a D2 dopamine antagonist on behavior maintained by cocaine or food. Pharmacology, biochemistry, and behavior. vol 32. issue 3. 1989-08-01. PMID:2662223. |
the purpose of the present experiment was to determine whether a d1 or a d2 dopamine antagonist could alter responding maintained by cocaine at doses that did not affect responding maintained by food. |
1989-08-01 |
2023-08-11 |
monkey |
| W L Woolverton, R M Viru. The effects of a D1 and a D2 dopamine antagonist on behavior maintained by cocaine or food. Pharmacology, biochemistry, and behavior. vol 32. issue 3. 1989-08-01. PMID:2662223. |
thus both a d1 and a d2 dopamine antagonist decreased behavior maintained by cocaine but only at doses that also decreased behavior maintained by another reinforcer, food. |
1989-08-01 |
2023-08-11 |
monkey |
| R Lew, D E Grigoriadis, J Sharkey, M J Kuha. Dopamine transporter: solubilization from dog caudate nucleus. Synapse (New York, N.Y.). vol 3. issue 4. 1989-07-31. PMID:2740994. |
in competition studies, soluble 3h-gbr 12935 binding was inhibited by mazindol, gbr 12909, nomifensine, dimethocaine, dopamine, (-) cocaine, and (+) cocaine in a manner typical of binding to the dopamine transporter. |
1989-07-31 |
2023-08-11 |
rat |
| J Peris, N R Zahnise. Persistent augmented dopamine release after acute cocaine requires dopamine receptor activation. Pharmacology, biochemistry, and behavior. vol 32. issue 1. 1989-07-17. PMID:2660163. |
persistent augmented dopamine release after acute cocaine requires dopamine receptor activation. |
1989-07-17 |
2023-08-11 |
rat |
| J Peris, N R Zahnise. Persistent augmented dopamine release after acute cocaine requires dopamine receptor activation. Pharmacology, biochemistry, and behavior. vol 32. issue 1. 1989-07-17. PMID:2660163. |
pretreatment of rats with a single injection of cocaine produces a persistent augmentation of amphetamine-induced [3h]dopamine [( 3h]da) release measured using the in vitro striatal slice preparation. |
1989-07-17 |
2023-08-11 |
rat |
| S J Cooper, I N Rusk, D J Barbe. Sucrose sham-feeding in the rat after administration of the selective dopamine D2 receptor agonist N-0437, d-amphetamine or cocaine. Pharmacology, biochemistry, and behavior. vol 32. issue 2. 1989-07-12. PMID:2567016. |
sucrose sham-feeding in the rat after administration of the selective dopamine d2 receptor agonist n-0437, d-amphetamine or cocaine. |
1989-07-12 |
2023-08-11 |
rat |
| S J Cooper, I N Rusk, D J Barbe. Sucrose sham-feeding in the rat after administration of the selective dopamine D2 receptor agonist N-0437, d-amphetamine or cocaine. Pharmacology, biochemistry, and behavior. vol 32. issue 2. 1989-07-12. PMID:2567016. |
drugs which act as agonists at dopamine receptors, or which increase dopamine release (e.g., d-amphetamine, cocaine) are known to reduce food intake. |
1989-07-12 |
2023-08-11 |
rat |
| S J Cooper, I N Rusk, D J Barbe. Sucrose sham-feeding in the rat after administration of the selective dopamine D2 receptor agonist N-0437, d-amphetamine or cocaine. Pharmacology, biochemistry, and behavior. vol 32. issue 2. 1989-07-12. PMID:2567016. |
the present experiments investigated, for the first time, the effects of a highly selective dopamine d2 receptor agonist, n-0437 (0.3-3.0 mg/kg, ip), on 5% sucrose sham-feeding in gastric fistulated rats, and compared these results with those of d-amphetamine (0.1-3.0 mg/kg, ip) and cocaine (3.0-10.0 mg/kg, ip). |
1989-07-12 |
2023-08-11 |
rat |
| L L Cregle. Adverse health consequences of cocaine abuse. Journal of the National Medical Association. vol 81. issue 1. 1989-07-10. PMID:2657079. |
the dopamine depletion hypothesis may explain why cocaine is repeatedly administered; cocaine produces a transient increase in synaptic dopamine. |
1989-07-10 |
2023-08-11 |
Not clear |