All Relations between ache and cholinergic

Publication Sentence Publish Date Extraction Date Species
Z Li, R Yang, Z Che. [Effects of iodine and thyroid hormone deficiency during brain development on activities of cholinergic neurone-related enzymes in central nervous system of rats]. Zhonghua yu fang yi xue za zhi [Chinese journal of preventive medicine]. vol 30. issue 6. 1998-01-20. PMID:9388907. it suggests iodine and thyroid hormone deficiency could affect maturation of chat and disturb transformation and maturation of various molecular types of ache and delay distinctly the development of cholinergic neurone during the critical period of brain development in rats. 1998-01-20 2023-08-12 rat
K Mizukami, A Mishizen, M D Ikonomovic, R Sheffield, D M Armstrong, A Mishizi. Alterations of AMPA-selected glutamate subtype immunoreactivity in the dentate gyrus after perforant pathway lesion. Brain research. vol 768. issue 1-2. 1997-12-18. PMID:9369338. completeness of the lesion was confirmed following examination of nissl-stained tissue sections at all times post-lesion and acetylcholinesterase (ache)-stained sections 14, 30 and 90 days post-lesion, the latter providing evidence of compensatory sprouting of cholinergic fibers in the outer molecular layer of the dentate gyrus. 1997-12-18 2023-08-12 rat
R Beeri, N Le Novère, R Mervis, T Huberman, E Grauer, J P Changeux, H Sore. Enhanced hemicholinium binding and attenuated dendrite branching in cognitively impaired acetylcholinesterase-transgenic mice. Journal of neurochemistry. vol 69. issue 6. 1997-12-12. PMID:9375677. in a search for behavioral, neuroanatomical, and metabolic characteristics of alzheimer's disease that may result from cholinergic malfunction, we used transgenic mice overexpressing acetylcholinesterase (ache) mrna and active enzyme in brain neurons. 1997-12-12 2023-08-12 mouse
G Swiatkiewic. Absence of acetylcholinesterase-positive nerve structure in the superficial part of the pineal gland of adult albino rats. Folia biologica. vol 44. issue 3-4. 1997-12-08. PMID:9342938. if it is assumed that the test for the presence of ache is a form of indirect conclusion concerning the cholinergic nature of given nerve structures, then in the case of the superficial innervation of the pineal gland, the outcome of the test is negative. 1997-12-08 2023-08-12 rat
M Koga, H Miyata, N Tsuno, K Nakayama, S Ushijima, Y Tanaka, Y Hiraga, N Kobayash. [Relationship between cholinergic symptoms caused by distigmine and the activities of serum AChE and BChE]. Nihon shinkei seishin yakurigaku zasshi = Japanese journal of psychopharmacology. vol 17. issue 3. 1997-11-19. PMID:9278940. [relationship between cholinergic symptoms caused by distigmine and the activities of serum ache and bche]. 1997-11-19 2023-08-12 Not clear
M Koga, H Miyata, N Tsuno, K Nakayama, S Ushijima, Y Tanaka, Y Hiraga, N Kobayash. [Relationship between cholinergic symptoms caused by distigmine and the activities of serum AChE and BChE]. Nihon shinkei seishin yakurigaku zasshi = Japanese journal of psychopharmacology. vol 17. issue 3. 1997-11-19. PMID:9278940. we evaluated the possibility of using serum acetylcholinesterase (ache) and butyrylcholinesterase (bche) as specific clinical markers for the adverse cholinergic effects of distigmine. 1997-11-19 2023-08-12 Not clear
M Koga, H Miyata, N Tsuno, K Nakayama, S Ushijima, Y Tanaka, Y Hiraga, N Kobayash. [Relationship between cholinergic symptoms caused by distigmine and the activities of serum AChE and BChE]. Nihon shinkei seishin yakurigaku zasshi = Japanese journal of psychopharmacology. vol 17. issue 3. 1997-11-19. PMID:9278940. of the twelve patients treated with distigmine for dysuria caused by psychotropic drugs six patients presented both adverse cholinergic effects and decreased levels of serum ache and bche. 1997-11-19 2023-08-12 Not clear
M Koga, H Miyata, N Tsuno, K Nakayama, S Ushijima, Y Tanaka, Y Hiraga, N Kobayash. [Relationship between cholinergic symptoms caused by distigmine and the activities of serum AChE and BChE]. Nihon shinkei seishin yakurigaku zasshi = Japanese journal of psychopharmacology. vol 17. issue 3. 1997-11-19. PMID:9278940. this study suggests that the values of serum ache and bche may be useful markers for the manifestation of adverse cholinergic effects caused by distigmine. 1997-11-19 2023-08-12 Not clear
A Alvarez, C Opazo, R Alarcón, J Garrido, N C Inestros. Acetylcholinesterase promotes the aggregation of amyloid-beta-peptide fragments by forming a complex with the growing fibrils. Journal of molecular biology. vol 272. issue 3. 1997-10-28. PMID:9325095. in order to identify the structural domains of the amyloid-beta-peptide (abeta) involved in the aggregation induced by ache, we have studied the effect of this cholinergic enzyme on abeta peptide fragments of different sizes. 1997-10-28 2023-08-12 Not clear
C Erb, J Klein, A Köppen, K Löffelholz, H Jeltsch, J C Casse. Modulation of hippocampal acetylcholine release after fimbria-fornix lesions and septal transplantation in rats. Neuroscience letters. vol 231. issue 1. 1997-10-23. PMID:9280154. the extent of cholinergic (re)innervation was determined by acetylcholinesterase (ache) staining and densitometry. 1997-10-23 2023-08-12 rat
X Cousin, C Bo. [Acetylcholinesterase from snake venoms]. Comptes rendus des seances de la Societe de biologie et de ses filiales. vol 191. issue 3. 1997-10-02. PMID:9295965. acetylcholinesterase (ache) plays a key role in cholinergic transmission. 1997-10-02 2023-08-12 Not clear
X Cousin, C Bo. [Acetylcholinesterase from snake venoms]. Comptes rendus des seances de la Societe de biologie et de ses filiales. vol 191. issue 3. 1997-10-02. PMID:9295965. the presence of ache in tissues devoid of cholinergic function is also well known and raises the question of its role. 1997-10-02 2023-08-12 Not clear
G Sberna, J Sáez-Valero, K Beyreuther, C L Masters, D H Smal. The amyloid beta-protein of Alzheimer's disease increases acetylcholinesterase expression by increasing intracellular calcium in embryonal carcinoma P19 cells. Journal of neurochemistry. vol 69. issue 3. 1997-09-25. PMID:9282941. one of the characteristic changes that occurs in alzheimer's disease is the loss of acetylcholinesterase (ache) from both cholinergic and noncholinergic neurons of the brain. 1997-09-25 2023-08-12 Not clear
W Yao, D A Godfre. Densitometric evaluation of markers for cholinergic transmission in rat superior olivary complex. Neuroscience letters. vol 229. issue 1. 1997-09-12. PMID:9224792. cholinergic transmission in the rat superior olivary complex (soc) was assessed by densitometric measurements of labeling with several markers: acetylcholinesterase (ache) histochemistry, choline acetyltransferase (chat) immunohistochemistry, and muscarinic acetylcholine receptor (m35) immunohistochemistry and n-methylscopolamine (nms) binding autoradiography, as well as its subtype 2 (m2) immunohistochemistry. 1997-09-12 2023-08-12 rat
W Yao, D A Godfre. Densitometric evaluation of markers for cholinergic transmission in rat superior olivary complex. Neuroscience letters. vol 229. issue 1. 1997-09-12. PMID:9224792. the markers which may occur in cholinergic neurons (chat, m2 receptor and ache) showed dense labeling in the ventral nucleus of the trapezoid body (vntb), in line with other evidence that it contains cholinergic neuron somata. 1997-09-12 2023-08-12 rat
T Naumann, T Deller, R Bender, M Frotsche. 192 IgG-saporin-induced loss of cholinergic neurons in the septum abolishes cholinergic sprouting after unilateral entorhinal lesion in the rat. The European journal of neuroscience. vol 9. issue 6. 1997-09-03. PMID:9215714. this cholinergic sprouting has been demonstrated by acetylcholinesterase (ache) histochemistry, a method said selectively to label cholinergic septohippocampal fibres in the hippocampus. 1997-09-03 2023-08-12 rat
T Naumann, T Deller, R Bender, M Frotsche. 192 IgG-saporin-induced loss of cholinergic neurons in the septum abolishes cholinergic sprouting after unilateral entorhinal lesion in the rat. The European journal of neuroscience. vol 9. issue 6. 1997-09-03. PMID:9215714. however, a recent report has questioned this concept, suggesting that ache may not be an adequate marker to monitor cholinergic sprouting and that other, non-cholinergic axons sprouting after entorhinal cortex lesion cause the dense ache-positive band in the denervated outer molecular layer. 1997-09-03 2023-08-12 rat
T Naumann, T Deller, R Bender, M Frotsche. 192 IgG-saporin-induced loss of cholinergic neurons in the septum abolishes cholinergic sprouting after unilateral entorhinal lesion in the rat. The European journal of neuroscience. vol 9. issue 6. 1997-09-03. PMID:9215714. in order to determine the contribution of cholinergic septohippocampal fibres to the dense ache band appearing after entorhinal cortex lesion, the neurotoxin 192 igg-saporin, known to destroy cholinergic neurons in the basal forebrain selectively, was used. 1997-09-03 2023-08-12 rat
T Naumann, T Deller, R Bender, M Frotsche. 192 IgG-saporin-induced loss of cholinergic neurons in the septum abolishes cholinergic sprouting after unilateral entorhinal lesion in the rat. The European journal of neuroscience. vol 9. issue 6. 1997-09-03. PMID:9215714. the cholinergic sprouting response in the molecular layer, as visualized with ache histochemistry, was abolished in all animals treated with immunotoxin. 1997-09-03 2023-08-12 rat
T Naumann, T Deller, R Bender, M Frotsche. 192 IgG-saporin-induced loss of cholinergic neurons in the septum abolishes cholinergic sprouting after unilateral entorhinal lesion in the rat. The European journal of neuroscience. vol 9. issue 6. 1997-09-03. PMID:9215714. these data indicate that the dense ache band forming after entorhinal cortex lesion represents the sprouting of cholinergic septohippocampal fibres. 1997-09-03 2023-08-12 rat