| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| Simone Monika Blattner, Matthias Kretzle. Integrin-linked kinase in renal disease: connecting cell-matrix interaction to the cytoskeleton. Current opinion in nephrology and hypertension. vol 14. issue 4. 2005-12-09. PMID:15931012. |
cellular functions like proliferation, differentiation, migration, morphogenesis and apoptosis are modulated by the extracellular matrix. |
2005-12-09 |
2023-08-12 |
Not clear |
| Chikako Odaka, Shinji Izumiyam. Expression of stromelysin-3 (matrix metalloproteinase-11) in macrophages of murine thymus following thymocyte apoptosis. Cellular immunology. vol 235. issue 1. 2005-12-05. PMID:16165118. |
expression of stromelysin-3 (matrix metalloproteinase-11) in macrophages of murine thymus following thymocyte apoptosis. |
2005-12-05 |
2023-08-12 |
mouse |
| Lesley Ann Smyth, Hugh J M Brad. cMet and Fas receptor interaction inhibits death-inducing signaling complex formation in endothelial cells. Hypertension (Dallas, Tex. : 1979). vol 46. issue 1. 2005-11-29. PMID:15911745. |
endothelial cells undergoing cell matrix detachment, anoikis, are sensitive to fas-mediated apoptosis. |
2005-11-29 |
2023-08-12 |
human |
| Begoña Diosdado, Erica van Oort, Cisca Wijmeng. "Coelionomics": towards understanding the molecular pathology of coeliac disease. Clinical chemistry and laboratory medicine. vol 43. issue 7. 2005-11-22. PMID:16207126. |
four mechanisms, namely apoptosis, oxidative stress, matrix metalloproteinases and dysregulation of proliferation and differentiation, are thought to play a role in the pathophysiology of cd. |
2005-11-22 |
2023-08-12 |
Not clear |
| E Arendt, U Ueberham, R Bittner, R Gebhardt, E Ueberha. Enhanced matrix degradation after withdrawal of TGF-beta1 triggers hepatocytes from apoptosis to proliferation and regeneration. Cell proliferation. vol 38. issue 5. 2005-11-21. PMID:16202037. |
enhanced matrix degradation after withdrawal of tgf-beta1 triggers hepatocytes from apoptosis to proliferation and regeneration. |
2005-11-21 |
2023-08-12 |
mouse |
| S Lísová, J Ehrmann, A Kolek, E Sedláková, Z Kolá. [Immunohistochemical study of the apoptotic and proliferative mechanisms in the intestinal mucosa during coeliac disease]. Ceskoslovenska patologie. vol 41. issue 3. 2005-11-08. PMID:16161453. |
mechanisms leading to morphological changes of the small intestine during coeliac disease are not yet completely recognized, however, two main processes have been suggested recently: remodelling of mucosa by matrix metalloproteinases, and mucosal atrophy by apoptosis. |
2005-11-08 |
2023-08-12 |
Not clear |
| Hossein Ardehali, Brian O'Rourk. Mitochondrial K(ATP) channels in cell survival and death. Journal of molecular and cellular cardiology. vol 39. issue 1. 2005-11-07. PMID:15978901. |
since the discovery of the mitochondrial atp-sensitive potassium channel (mitok(atp)) more than 13 years ago, it has been implicated in the processes of ischemic preconditioning (ipc), apoptosis and mitochondrial matrix swelling. |
2005-11-07 |
2023-08-12 |
Not clear |
| Xuemei Lian, Cong Yan, Yulin Qin, Lana Knox, Tingyu Li, Hong D. Neutral lipids and peroxisome proliferator-activated receptor-{gamma} control pulmonary gene expression and inflammation-triggered pathogenesis in lysosomal acid lipase knockout mice. The American journal of pathology. vol 167. issue 3. 2005-11-07. PMID:16127159. |
among changed genes, matrix metalloproteinase (mmp)-12, apoptosis inhibitor 6 (api-6), erythroblast transformation-specific domain (ets) transcription factor family member spi-c, and oncogene mafb were increased 100-, 70-, 40-, and 10-fold, respectively, in lal-/- lungs versus the wild-type lungs. |
2005-11-07 |
2023-08-12 |
mouse |
| Irfan Rahma. Oxidative stress in pathogenesis of chronic obstructive pulmonary disease: cellular and molecular mechanisms. Cell biochemistry and biophysics. vol 43. issue 1. 2005-11-04. PMID:16043892. |
these include increased sequestration of neutrophils in the pulmonary microvasculature, oxidative inactivation of antiproteases and surfactants, hypersecretion of mucus, membrane lipid peroxidation, mitochondrial respiration, alveolar epithelial injury/permeability, breakdown/remodeling of extracellular matrix, and apoptosis. |
2005-11-04 |
2023-08-12 |
Not clear |
| Clare E Futter, Jonathan G Crowston, Bruce D S Alla. Interaction with collagen IV protects lens epithelial cells from Fas-dependent apoptosis by stimulating the production of soluble survival factors. Investigative ophthalmology & visual science. vol 46. issue 9. 2005-11-02. PMID:16123427. |
to investigate the sensitivity of lens epithelial cells (lecs) to fas-dependent apoptosis and to determine the role of interaction with the extracellular matrix (ecm) in the regulation of fas-dependent apoptosis. |
2005-11-02 |
2023-08-12 |
Not clear |
| Thomas W Johnson, Yin Xiong Wu, Christian Herdeg, Andreas Baumbach, Andrew C Newby, Karl R Karsch, Martin Oberhof. Stent-based delivery of tissue inhibitor of metalloproteinase-3 adenovirus inhibits neointimal formation in porcine coronary arteries. Arteriosclerosis, thrombosis, and vascular biology. vol 25. issue 4. 2005-10-31. PMID:15681295. |
radtimp-3 decreased smooth muscle cell migration, stabilized the extracellular matrix, and uniquely promoted apoptosis. |
2005-10-31 |
2023-08-12 |
Not clear |
| Ana Cristina García-Ulloa, Oscar Arriet. Tubal occlusion causing infertility due to an excessive inflammatory response in patients with predisposition for keloid formation. Medical hypotheses. vol 65. issue 5. 2005-10-27. PMID:16005574. |
this keloid scarring involves an excess in extracellular matrix production and inhibition of apoptosis, for which a several growth factors and interleukins are needed. |
2005-10-27 |
2023-08-12 |
Not clear |
| M Fini, G Giavaresi, A Carpi, A Nicolini, S Setti, R Giardin. Effects of pulsed electromagnetic fields on articular hyaline cartilage: review of experimental and clinical studies. Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie. vol 59. issue 7. 2005-10-27. PMID:16084055. |
controlling chondrocyte death and apoptosis, function, response to anabolic and catabolic stimuli, matrix synthesis or degradation and inflammation is the most important target of potential chondroprotective treatment, aimed to retard or stabilize the progression of oa. |
2005-10-27 |
2023-08-12 |
Not clear |
| M Yu Kapitonova, Othman Manso. Ultrastructural changes of the articular cartilage in some arthropathies with special reference to chondrocyte cell death. The Malaysian journal of pathology. vol 25. issue 1. 2005-10-27. PMID:16196374. |
to determine in situ using tem the balance of apoptosis and necrosis in the articular cartilage of patients with inflammatory (rheumatoid arthritis and seronegative spondyloarthritis) and degenerative (osteoarthritis) joint diseases and to establish possible correlation between the cell death rate and the matrix vesicles formation. |
2005-10-27 |
2023-08-12 |
Not clear |
| Robert Terkeltaub, Kristen Johnson, Anne Murphy, Soumitra Ghos. Invited review: the mitochondrion in osteoarthritis. Mitochondrion. vol 1. issue 4. 2005-10-20. PMID:16120285. |
these include oxidative stress, inadequacy of chondrocyte biosynthetic and growth responses, up-regulated chondrocyte cytokine-induced inflammation and matrix catabolism, increased chondrocyte apoptosis, and pathologic cartilage matrix calcification. |
2005-10-20 |
2023-08-12 |
Not clear |
| Chithan Kandaswami, Chithan Kanadaswami, Lung-Ta Lee, Ping-Ping H Lee, Jiuan-Jiuan Hwang, Ferng-Chun Ke, Ying-Tung Huang, Ming-Ting Le. The antitumor activities of flavonoids. In vivo (Athens, Greece). vol 19. issue 5. 2005-10-18. PMID:16097445. |
in vitro work has concentrated on the direct and indirect actions of flavonoids on tumor cells, and has found a variety of anticancer effects such as cell growth and kinase activity inhibition, apoptosis induction, suppression of the secretion of matrix metalloproteinases and of tumor invasive behavior. |
2005-10-18 |
2023-08-12 |
human |
| Stephan Kusick, Helga Bertram, Hans Oberleithner, Thomas Ludwi. Nanoscale imaging and quantification of local proteolytic activity. Journal of cellular physiology. vol 204. issue 3. 2005-10-06. PMID:15744770. |
proteolytic cleavage of extracellular matrix (ecm) is a critical feature of tumor cell invasion, and affects cancer cell growth, differentiation, apoptosis, and migration. |
2005-10-06 |
2023-08-12 |
Not clear |
| Irfan Rahma. The role of oxidative stress in the pathogenesis of COPD: implications for therapy. Treatments in respiratory medicine. vol 4. issue 3. 2005-10-06. PMID:15987234. |
these include oxidative inactivation of antiproteases and surfactants, mucus hypersecretion, membrane lipid peroxidation, mitochondrial respiration, alveolar epithelial injury, remodeling of extracellular matrix, and apoptosis. |
2005-10-06 |
2023-08-12 |
Not clear |
| Thomas Effert. Mechanistic perspectives for 1,2,4-trioxanes in anti-cancer therapy. Drug resistance updates : reviews and commentaries in antimicrobial and anticancer chemotherapy. vol 8. issue 1-2. 2005-10-05. PMID:15878303. |
these genes are from classes with different biological function; for example, regulation of proliferation (bub3, cyclins, cdc25a), angiogenesis (vascular endothelial growth factor and its receptor, matrix metalloproteinase-9, angiostatin, thrombospondin-1) or apoptosis (bcl-2, bax). |
2005-10-05 |
2023-08-12 |
human |
| R G Humphrey, S D Smith, L Pang, Y Sadovsky, D M Nelso. Fibrin enhances differentiation, but not apoptosis, and limits hypoxic injury of cultured term human trophoblasts. Placenta. vol 26. issue 6. 2005-10-05. PMID:15950063. |
compared to the two controls, fibrin matrix had no effect on trophoblast apoptosis or total cell death in standard conditions. |
2005-10-05 |
2023-08-12 |
human |