| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| Paul Usechak, Anna Gates, Cynthia Rl Webste. Activation of focal adhesion kinase and JNK contributes to the extracellular matrix and cAMP-GEF mediated survival from bile acid induced apoptosis in rat hepatocytes. Journal of hepatology. vol 49. issue 2. 2009-01-09. PMID:18550202. |
activation of focal adhesion kinase and jnk contributes to the extracellular matrix and camp-gef mediated survival from bile acid induced apoptosis in rat hepatocytes. |
2009-01-09 |
2023-08-12 |
rat |
| Paul Usechak, Anna Gates, Cynthia Rl Webste. Activation of focal adhesion kinase and JNK contributes to the extracellular matrix and cAMP-GEF mediated survival from bile acid induced apoptosis in rat hepatocytes. Journal of hepatology. vol 49. issue 2. 2009-01-09. PMID:18550202. |
adherence to an extracellular matrix (ecm) rescues hepatocytes from apoptosis, but how hepatocytes adhered to different ecm and respond to apoptotic and cytoprotective stimuli is unknown. |
2009-01-09 |
2023-08-12 |
rat |
| Erdener Oze. Telomerase activity of chorionic trophoblasts in preterm premature rupture of membranes. Pediatric and developmental pathology : the official journal of the Society for Pediatric Pathology and the Paediatric Pathology Society. vol 11. issue 5. 2009-01-07. PMID:18260693. |
to evaluate other suggested mechanisms, such as apoptosis and extracellular matrix degradation, immunohistochemistry for matrix metalloproteinase (mmp)-9, tissue inhibitor of metalloproteinase (timp)-3, and m30 cytodeath antibodies were also performed. |
2009-01-07 |
2023-08-12 |
Not clear |
| Erdener Oze. Telomerase activity of chorionic trophoblasts in preterm premature rupture of membranes. Pediatric and developmental pathology : the official journal of the Society for Pediatric Pathology and the Paediatric Pathology Society. vol 11. issue 5. 2009-01-07. PMID:18260693. |
although study of the pprom has focused on activation of matrix metalloproteinases and apoptosis that result in the consequence of a programmed membrane weakening process, decreased telomerase activity of chorionic trophoblasts is likely to have a contributing role in the pathogenesis of pprom as well. |
2009-01-07 |
2023-08-12 |
Not clear |
| Laney M Weber, Kirsten N Hayda, Kristi S Anset. Cell-matrix interactions improve beta-cell survival and insulin secretion in three-dimensional culture. Tissue engineering. Part A. vol 14. issue 12. 2009-01-07. PMID:18724831. |
correspondingly, apoptosis in encapsulated beta-cells was less in the presence of each matrix protein, suggesting the ability of individual matrix interactions to prevent matrix signaling-related apoptosis (anoikis). |
2009-01-07 |
2023-08-12 |
Not clear |
| Xiaoping Luo, Nasser Chegin. The expression and potential regulatory function of microRNAs in the pathogenesis of leiomyoma. Seminars in reproductive medicine. vol 26. issue 6. 2009-01-07. PMID:18951332. |
it is clear that ovarian steroids are essential for leiomyoma growth, and local expression of many autocrine/paracrine mediators serving as key regulators of cell-cycle progression, cellular hypertrophy, extracellular matrix accumulation, and apoptosis appear to play central roles in this capacity. |
2009-01-07 |
2023-08-12 |
Not clear |
| Tiina Skoog, Outi Elomaa, Sanna M Pasonen-Seppänen, Sofi Forsberg, Katja Ahokas, Leila Jeskanen, Jenita Pärssinen, Inkeri Tiala, Ola Rollman, Jouko Lohi, Ulpu Saarialho-Ker. Matrix metalloproteinase-21 expression is associated with keratinocyte differentiation and upregulated by retinoic acid in HaCaT cells. The Journal of investigative dermatology. vol 129. issue 1. 2009-01-06. PMID:18633436. |
in the skin, expression of several matrix metalloproteinases (mmps) occurs in response to tissue injury, tumorigenesis, angiogenesis, apoptosis, and inflammation. |
2009-01-06 |
2023-08-12 |
Not clear |
| Zhi-Hua Chen, Hong Pyo Kim, Stefan W Ryter, Augustine M K Cho. Identifying targets for COPD treatment through gene expression analyses. International journal of chronic obstructive pulmonary disease. vol 3. issue 3. 2009-01-06. PMID:18990963. |
such studies have revealed a number of differentially-regulated genes associated with copd progression, which include genes involved in the regulation of inflammation, extracellular matrix, cytokines, chemokines, apoptosis, and stress responses. |
2009-01-06 |
2023-08-12 |
Not clear |
| Ju-mei Zhao, Yan-jun Wen, Qiu Li, Yong-sheng Wang, Hong-bo Wu, Jian-rong Xu, Xian-cheng Chen, Yang Wu, Ling-yu Fan, Han-shuo Yang, Tao Liu, Zhen-yu Ding, Xiao-bo Du, Peng Diao, Jiong Li, Hong-bing Wu, Bing Kan, Song Lei, Hong-xin Deng, Yong-qiu Mao, Xia Zhao, Yu-quan We. A promising cancer gene therapy agent based on the matrix protein of vesicular stomatitis virus. FASEB journal : official publication of the Federation of American Societies for Experimental Biology. vol 22. issue 12. 2008-12-31. PMID:18716030. |
the matrix (m) protein of vesicular stomatitis virus (vsv) plays a key role in inducing cell apoptosis during infection. |
2008-12-31 |
2023-08-12 |
mouse |
| Adnan Al-Ayoubi, Adel Tarcsafalvi, Hui Zheng, Wayne Sakati, Scott T Eble. ERK activation and nuclear signaling induced by the loss of cell/matrix adhesion stimulates anchorage-independent growth of ovarian cancer cells. Journal of cellular biochemistry. vol 105. issue 3. 2008-12-31. PMID:18726893. |
for normal epithelial cells and fibroblasts, cell adhesion to the extracellular matrix is required to prevent apoptosis and for proper activation and nuclear signaling of the erk map kinase. |
2008-12-31 |
2023-08-12 |
Not clear |
| Craig D Simpson, Kika Anyiwe, Aaron D Schimme. Anoikis resistance and tumor metastasis. Cancer letters. vol 272. issue 2. 2008-12-29. PMID:18579285. |
as a barrier to metastases, cells normally undergo apoptosis after they lose contact with their extra cellular matrix or their neighbouring cells. |
2008-12-29 |
2023-08-12 |
Not clear |
| Keren Tzukert, Raphael Gorodestky, Idit Avrahami, Lina Krasny, Nilly Shimony, Gregory Elkin, Dirk M Nettelbeck, Yosef S Havi. A novel dynamic matrix detachment model reveals a shift from apoptosis to necrosis in melanoma cells. Cancer letters. vol 272. issue 2. 2008-12-29. PMID:18723276. |
a novel dynamic matrix detachment model reveals a shift from apoptosis to necrosis in melanoma cells. |
2008-12-29 |
2023-08-12 |
Not clear |
| Bente Halvorsen, Kari Otterdal, Tuva B Dahl, Mona Skjelland, Lars Gullestad, Erik Øie, Pål Aukrus. Atherosclerotic plaque stability--what determines the fate of a plaque? Progress in cardiovascular diseases. vol 51. issue 3. 2008-12-24. PMID:19026853. |
thus, inflammatory cytokines may attenuate interstitial collagen synthesis, increase matrix degradation, and promote apoptosis in several atheroma-associated cell types, and all these cellular events may enhance plaque vulnerability. |
2008-12-24 |
2023-08-12 |
Not clear |
| Dingsheng Lin, Jie Feng, Weishan Che. Bcl-2 and caspase-8 related anoikis resistance in human osteosarcoma MG-63 cells. Cell biology international. vol 32. issue 10. 2008-12-22. PMID:18675369. |
detachment of adherent cells from extracellular matrix results in apoptosis, a process termed "anoikis". |
2008-12-22 |
2023-08-12 |
human |
| J-C Pasquier, M Dore. [Fetal membranes: embryological development, structure and the physiopathology of the preterm premature rupture of membranes]. Journal de gynecologie, obstetrique et biologie de la reproduction. vol 37. issue 6. 2008-12-19. PMID:18424017. |
two main mechanisms are involved in the degradation process: apoptosis in the cellular compartment and matrix metalloproteinase (mmp) in the extracellular matrix. |
2008-12-19 |
2023-08-12 |
Not clear |
| Xiao-Bo Du, Jin-Yi Lang, Jian-Rong Xu, You Lu, Yan-Jun Wen, Ju-Mei Zhao, Peng Diao, Zhi-Ping Yuan, Bin Yao, Ling-Yu Fan, Guo-Qing Wang, Li Liu, Zhen-Yu Ding, Yong-Sheng Wang, Tao Li, Rui Wang, Yun-Qiu Mao, Bin Kan, Hong-Bin Wu, Hong-Xia Li, Han-Suo Yang, Hong-Bo Wu, Yu-Quan Wei, Xia Zha. Vesicular stomatitis virus matrix protein gene enhances the antitumor effects of radiation via induction of apoptosis. Apoptosis : an international journal on programmed cell death. vol 13. issue 10. 2008-12-16. PMID:18726188. |
vesicular stomatitis virus matrix protein gene enhances the antitumor effects of radiation via induction of apoptosis. |
2008-12-16 |
2023-08-12 |
mouse |
| Xiao-Bo Du, Jin-Yi Lang, Jian-Rong Xu, You Lu, Yan-Jun Wen, Ju-Mei Zhao, Peng Diao, Zhi-Ping Yuan, Bin Yao, Ling-Yu Fan, Guo-Qing Wang, Li Liu, Zhen-Yu Ding, Yong-Sheng Wang, Tao Li, Rui Wang, Yun-Qiu Mao, Bin Kan, Hong-Bin Wu, Hong-Xia Li, Han-Suo Yang, Hong-Bo Wu, Yu-Quan Wei, Xia Zha. Vesicular stomatitis virus matrix protein gene enhances the antitumor effects of radiation via induction of apoptosis. Apoptosis : an international journal on programmed cell death. vol 13. issue 10. 2008-12-16. PMID:18726188. |
vesicular stomatitis virus (vsv) matrix (m) protein can directly induce apoptosis by inhibiting host gene expression when it is expressed in the absence of other viral components. |
2008-12-16 |
2023-08-12 |
mouse |
| Melinda E Lull, Willard M Freeman, Kent E Vrana, Deborah C Mas. Correlating human and animal studies of cocaine abuse and gene expression. Annals of the New York Academy of Sciences. vol 1141. 2008-12-12. PMID:18991951. |
in total, human and animal studies of cocaine abuse have uncovered gene expression changes in the brain related to a number of molecular functions, including the extracellular matrix, synaptic communication and neuroplasticity, receptors, ion channels and transporters, oligodendrocytes and myelin, apoptosis and cell death, mitochondrial function, signal transduction, and transcription factors. |
2008-12-12 |
2023-08-12 |
human |
| Mamoru Satoh, Yoshitaka Minami, Yuji Takahashi, Motoyuki Nakamur. Immune modulation: role of the inflammatory cytokine cascade in the failing human heart. Current heart failure reports. vol 5. issue 2. 2008-12-10. PMID:18765076. |
indeed, sustained increases in cytokines, including tnf-alpha and its receptor, lead to monocyte phenotype transition, myocytic apoptosis, and activation of matrix metalloproteinase. |
2008-12-10 |
2023-08-12 |
human |
| Preetha Anand, Ajaikumar B Kunnumakkara, Kuzhuvelil B Harikumar, Kwang Seok Ahn, Vladimir Badmaev, Bharat B Aggarwa. Modification of cysteine residue in p65 subunit of nuclear factor-kappaB (NF-kappaB) by picroliv suppresses NF-kappaB-regulated gene products and potentiates apoptosis. Cancer research. vol 68. issue 21. 2008-12-08. PMID:18974130. |
nf-kappab inhibition by picroliv leads to suppression of nf-kappab-regulated proteins, including those linked with cell survival (inhibitor of apoptosis protein 1, bcl-2, bcl-xl, survivin, and tnf receptor-associated factor 2), proliferation (cyclin d1 and cyclooxygenase-2), angiogenesis (vascular endothelial growth factor), and invasion (intercellular adhesion molecule-1 and matrix metalloproteinase-9). |
2008-12-08 |
2023-08-12 |
Not clear |