| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| N Bonnefoy-Berard, L Genestier, X Preville, J P Revillar. TNF alpha and CD95-L contribute to apoptosis of activated lymphocytes triggered by ATGs. Transplantation proceedings. vol 31. issue 1-2. 1999-04-15. PMID:10083328. |
tnf alpha and cd95-l contribute to apoptosis of activated lymphocytes triggered by atgs. |
1999-04-15 |
2023-08-12 |
Not clear |
| M Iacobelli, F Rohwer, P Shanahan, J A Quiroz, K L McGuir. IL-2-mediated cell cycle progression and inhibition of apoptosis does not require NF-kappa B or activating protein-1 activation in primary human T cells. Journal of immunology (Baltimore, Md. : 1950). vol 162. issue 6. 1999-04-13. PMID:10092783. |
the current study clearly demonstrates that il-2 does not induce i kappa b alpha degradation or nf-kappa b activation in primary human t cells that respond to il-2 by entering the cell cycle and avoiding apoptosis. |
1999-04-13 |
2023-08-12 |
human |
| B Murphy, C C Magee, S I Alexander, A M Waaga, H W Snoeck, J P Vella, C B Carpenter, M H Sayeg. Inhibition of allorecognition by a human class II MHC-derived peptide through the induction of apoptosis. The Journal of clinical investigation. vol 103. issue 6. 1999-04-02. PMID:10079107. |
these data demonstrate that synthetic peptides derived from highly conserved regions of the class ii mhc alpha chain can alter cd4(+) t-lymphocyte alloimmune responses in vitro, and this effect is mediated by the induction of apoptosis in activated t cells. |
1999-04-02 |
2023-08-12 |
mouse |
| J J Lemasters, T Qian, S P Elmore, L C Trost, Y Nishimura, B Herman, C A Bradham, D A Brenner, A L Niemine. Confocal microscopy of the mitochondrial permeability transition in necrotic cell killing, apoptosis and autophagy. BioFactors (Oxford, England). vol 8. issue 3-4. 1999-03-24. PMID:9914830. |
the mpt is a causative event in many types of necrotic and apoptotic cell death, including oxidative stress, ischemia/reperfusion injury, ca2+ ionophore toxicity and tumor necrosis factor alpha (tnf alpha) induced apoptosis, and may contribute to reye's-related drug toxicity. |
1999-03-24 |
2023-08-12 |
Not clear |
| P Nørgaard, B Law, H Joseph, D L Page, Y Shyr, D Mays, J A Pietenpol, N E Kohl, A Oliff, R J Coffey, H S Poulsen, H L Mose. Treatment with farnesyl-protein transferase inhibitor induces regression of mammary tumors in transforming growth factor (TGF) alpha and TGF alpha/neu transgenic mice by inhibition of mitogenic activity and induction of apoptosis. Clinical cancer research : an official journal of the American Association for Cancer Research. vol 5. issue 1. 1999-03-24. PMID:9918200. |
treatment with farnesyl-protein transferase inhibitor induces regression of mammary tumors in transforming growth factor (tgf) alpha and tgf alpha/neu transgenic mice by inhibition of mitogenic activity and induction of apoptosis. |
1999-03-24 |
2023-08-12 |
mouse |
| E Mortola, M Okuda, K Ohno, T Watari, H Tsujimoto, A Hasegaw. Inhibition of apoptosis and virus replication in feline immunodeficiency virus-infected cells by N-acetylcysteine and ascorbic acid. The Journal of veterinary medical science. vol 60. issue 11. 1999-03-18. PMID:9853298. |
the treatment with nac or aa induced a significant inhibition of viral replication and apoptosis in fel-039 cells and tumor necrosis factor alpha (tnf-alpha)-treated crfk cells infected with fiv. |
1999-03-18 |
2023-08-12 |
Not clear |
| S Dar, S Mundle, T Andric, H Qawi, V Shetty, S Reza, B Y Mativi, K Allampallam, A Ali, P Venugopal, S Gezer, L Broady-Robinson, J Cartlidge, M Showel, S Hussaini, D Ragasa, I Ali, A Chaudhry, S Waggoner, L Lisak, R W Huang, A Raz. Biological characteristics of myelodysplastic syndrome patients who demonstrated high versus no intramedullary apoptosis. European journal of haematology. vol 62. issue 2. 1999-03-16. PMID:10052711. |
patients with high apoptosis levels tended to have a high li (p=0.013), more macrophages in their bm biopsies (p=0.006) and higher tumor necrosis factor alpha (tnf-alpha) levels (not significant) compared to patients with no apoptosis. |
1999-03-16 |
2023-08-12 |
Not clear |
| J L Kissil, O Cohen, T Raveh, A Kimch. Structure-function analysis of an evolutionary conserved protein, DAP3, which mediates TNF-alpha- and Fas-induced cell death. The EMBO journal. vol 18. issue 2. 1999-03-15. PMID:9889192. |
expression of anti-sense dap3 rna and of the dominant interfering form of dap3 both protected cells from apoptosis induced by activation of fas and tumor necrosis factor alpha (tnf-alpha) receptors. |
1999-03-15 |
2023-08-12 |
human |
| W El-Assaad, M El-Sabban, C Awaraji, N Abboushi, G S Dbaib. Distinct sites of action of Bcl-2 and Bcl-xL in the ceramide pathway of apoptosis. The Biochemical journal. vol 336 ( Pt 3). 1999-02-25. PMID:9841888. |
we studied the inhibition of tumour necrosis factor alpha (tnfalpha)- and camptothecin-induced apoptosis by bcl-2 and bcl-xl as they relate to the ceramide pathway. |
1999-02-25 |
2023-08-12 |
Not clear |
| N H James, J H Gill, R Brindle, N J Woodyatt, N Macdonald, M Rolfe, S C Hasmall, J D Tugwood, P R Holden, R A Robert. Peroxisome proliferator-activated receptor (PPAR) alpha-regulated growth responses and their importance to hepatocarcinogenesis. Toxicology letters. vol 102-103. 1999-02-25. PMID:10022238. |
more recently, we have demonstrated that pps can suppress apoptosis induced by more diverse stimuli such as dna damage or ligation of fas, a receptor related to the tumour necrosis factor alpha (tnf alpha) family of cell surface receptors. |
1999-02-25 |
2023-08-12 |
human |
| N H James, J H Gill, R Brindle, N J Woodyatt, N Macdonald, M Rolfe, S C Hasmall, J D Tugwood, P R Holden, R A Robert. Peroxisome proliferator-activated receptor (PPAR) alpha-regulated growth responses and their importance to hepatocarcinogenesis. Toxicology letters. vol 102-103. 1999-02-25. PMID:10022238. |
we investigated whether activation of ppar alpha mediates the suppression of rat hepatocyte apoptosis induced by pps. |
1999-02-25 |
2023-08-12 |
human |
| N H James, J H Gill, R Brindle, N J Woodyatt, N Macdonald, M Rolfe, S C Hasmall, J D Tugwood, P R Holden, R A Robert. Peroxisome proliferator-activated receptor (PPAR) alpha-regulated growth responses and their importance to hepatocarcinogenesis. Toxicology letters. vol 102-103. 1999-02-25. PMID:10022238. |
when introduced into primary rat liver cell cultures by transient transfection, hppar alpha-6/29 prevented the suppression of hepatocyte apoptosis by the pp nafenopin, but not that seen in response to phenobarbitone (pb), a non-genotoxic carcinogen whose action does not involve ppar alpha. |
1999-02-25 |
2023-08-12 |
human |
| R Homburg, A Amsterda. Polysystic ovary syndrome--loss of the apoptotic mechanism in the ovarian follicles? Journal of endocrinological investigation. vol 21. issue 9. 1999-02-23. PMID:9856409. |
egf/tgf alpha) in pcos, considered to be survival or antiapoptotic factors, led to the hypothesis of their involvement in the blocking of apoptosis and atresia leading to an accumulation of multiple small antral follicles. |
1999-02-23 |
2023-08-12 |
Not clear |
| R Wisdom, R S Johnson, C Moor. c-Jun regulates cell cycle progression and apoptosis by distinct mechanisms. The EMBO journal. vol 18. issue 1. 1999-02-18. PMID:9878062. |
in addition, c-jun protects cells from uv-induced cell death and cooperates with nf-kappab to prevent apoptosis induced by tumor necrosis factor alpha (tnfalpha). |
1999-02-18 |
2023-08-12 |
Not clear |
| M Gupta, A George, R Sen, S Rath, J M Durdik, V Ba. Presence of pentoxifylline during T cell priming increases clonal frequencies in secondary proliferative responses and inhibits apoptosis. Journal of immunology (Baltimore, Md. : 1950). vol 162. issue 2. 1999-02-08. PMID:9916687. |
the presence of pox during priming alters the outcome of t cell activation, resulting in a lower frequency of cells expressing il-2r alpha (cd25) and a decrease in their subsequent apoptosis, and this antiapoptotic effect is consistent with the enhanced commitment of t cells to secondary responsiveness by pox. |
1999-02-08 |
2023-08-12 |
human |
| H J Deeg, V Lesniko. MHC class II and negative regulators of hematopoiesis. Archivum immunologiae et therapiae experimentalis. vol 46. issue 5. 1999-02-03. PMID:9832067. |
signals generated via hla-dr result in upregulation of tumor necrosis factor alpha (tnf-alpha) and nitric oxide as well as an enhanced expression of fas and its natural ligand (fas-l), all molecules associated with programmed cell death (apoptosis). |
1999-02-03 |
2023-08-12 |
Not clear |
| D M Monack, J Mecsas, D Bouley, S Falko. Yersinia-induced apoptosis in vivo aids in the establishment of a systemic infection of mice. The Journal of experimental medicine. vol 188. issue 11. 1999-01-20. PMID:9841926. |
we conclude here that yopj plays a role in the establishment of a systemic infection by inducing apoptosis and that this is consistent with the ability to suppress the production of the proinflammatory cytokine tumor necrosis factor alpha. |
1999-01-20 |
2023-08-12 |
mouse |
| A J Seely, D E Swartz, B Giannias, N V Christo. Reduction in neutrophil cell surface expression of tumor necrosis factor receptors but not Fas after transmigration: implications for the regulation of neutrophil apoptosis. Archives of surgery (Chicago, Ill. : 1960). vol 133. issue 12. 1999-01-20. PMID:9865647. |
to test the hypothesis that loss of polymorphonuclear neutrophil tumor necrosis factor alpha (tnf-alpha) receptors during transmigration renders the exudate neutrophil refractory to tnf-alpha-mediated stimulation of apoptosis; and to investigate the surface expression of fas on both circulating and exudate neutrophils. |
1999-01-20 |
2023-08-12 |
Not clear |
| T Stokłosa, C Wójcik, J Gołab, A Giermasz, S Wil. Inhibition of proteasome, apoptosis and sensitization to tumour necrosis factor alpha: do they always go together? British journal of cancer. vol 79. issue 2. 1999-01-19. PMID:9888486. |
inhibition of proteasome, apoptosis and sensitization to tumour necrosis factor alpha: do they always go together? |
1999-01-19 |
2023-08-12 |
Not clear |
| E Sueoka, N Sueoka, Y Kai, S Okabe, M Suganuma, K Kanematsu, T Yamamoto, H Fujik. Anticancer activity of morphine and its synthetic derivative, KT-90, mediated through apoptosis and inhibition of NF-kappaB activation. Biochemical and biophysical research communications. vol 252. issue 3. 1999-01-14. PMID:9837747. |
as for mechanisms of action, kt-90 and morphine induced apoptosis, and inhibited tumor necrosis factor alpha (tnf-alpha) gene expression induced by tumor promoters, okadaic acid and 12-o-tetradecanoylphorbol-13-acetate, associated with reduction of nf-kappab dna binding activity. |
1999-01-14 |
2023-08-12 |
human |