| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| K C Wollert, J Heineke, J Westermann, M Lüdde, B Fiedler, W Zierhut, D Laurent, M K Bauer, K Schulze-Osthoff, H Drexle. The cardiac Fas (APO-1/CD95) Receptor/Fas ligand system : relation to diastolic wall stress in volume-overload hypertrophy in vivo and activation of the transcription factor AP-1 in cardiac myocytes. Circulation. vol 101. issue 10. 2000-03-30. PMID:10715265. |
cross-linking of fas by fas ligand (fasl), a tumor necrosis factor-alpha-related cytokine, promotes apoptosis and/or transcription factor activation in a highly cell-type-specific manner. |
2000-03-30 |
2023-08-12 |
Not clear |
| T Okamot. The protective effect of glycyrrhizin on anti-Fas antibody-induced hepatitis in mice. European journal of pharmacology. vol 387. issue 2. 2000-03-28. PMID:10650164. |
fas ligand, which is a type ii membrane protein, is a major inducer of apoptosis. |
2000-03-28 |
2023-08-12 |
mouse |
| M J Kaplan, D Ray, R R Mo, R L Yung, B C Richardso. TRAIL (Apo2 ligand) and TWEAK (Apo3 ligand) mediate CD4+ T cell killing of antigen-presenting macrophages. Journal of immunology (Baltimore, Md. : 1950). vol 164. issue 6. 2000-03-28. PMID:10706675. |
however, d10, an iak-restricted cloned th2 line, will similarly induce apoptosis in ag-presenting macrophages, and d10 cells lack fas ligand. |
2000-03-28 |
2023-08-12 |
human |
| S Kim, K A Kim, D Y Hwang, T H Lee, N Kayagaki, H Yagita, M S Le. Inhibition of autoimmune diabetes by Fas ligand: the paradox is solved. Journal of immunology (Baltimore, Md. : 1950). vol 164. issue 6. 2000-03-28. PMID:10706679. |
we addressed why diabetes cannot be transferred to nod-lpr mice despite the nonessential role of fas in beta cell apoptosis. |
2000-03-28 |
2023-08-12 |
mouse |
| V E Watson, L L Hill, L B Owen-Schaub, D W Davis, D J McConkey, C Jagannath, R L Hunter, J K Acto. Apoptosis in mycobacterium tuberculosis infection in mice exhibiting varied immunopathology. The Journal of pathology. vol 190. issue 2. 2000-03-27. PMID:10657021. |
the contribution of fas and fas ligand-mediated apoptosis was assessed via bioluminescent reverse transcription-polymerase chain reaction (rt-pcr), immunohistochemical staining, and tunel assessment of dna fragmentation. |
2000-03-27 |
2023-08-12 |
mouse |
| K Kuwano, N Hagimoto, T Tanaka, M Kawasaki, R Kunitake, H Miyazaki, Y Kaneko, T Matsuba, T Maeyama, N Har. Expression of apoptosis-regulatory genes in epithelial cells in pulmonary fibrosis in mice. The Journal of pathology. vol 190. issue 2. 2000-03-27. PMID:10657022. |
up-regulation of fas and fas ligand and excessive apoptosis of bronchiolar and alveolar epithelial cells were identified in bleomycin-induced pulmonary fibrosis in mice. |
2000-03-27 |
2023-08-12 |
mouse |
| D R Balkundi, N Hanna, M Hileb, J Dougherty, S Sharm. Labor-associated changes in Fas ligand expression and function in human placenta. Pediatric research. vol 47. issue 3. 2000-03-24. PMID:10709727. |
fas ligand (fasl)-dependent apoptosis has been implicated in the control of tissue-damaging inflammatory responses in several immune privileged sites. |
2000-03-24 |
2023-08-12 |
human |
| M Kornmann, T Ishiwata, J Kleeff, H G Beger, M Kor. Fas and Fas-ligand expression in human pancreatic cancer. Annals of surgery. vol 231. issue 3. 2000-03-24. PMID:10714630. |
to investigate fas and fasl expression in pancreatic tissues and cultured pancreatic cancer cell lines, and to assess the ability of anti-fas antibodies to induce apoptosis. |
2000-03-24 |
2023-08-12 |
human |
| S Kawaguchi, T Mineta, M Ichinose, J Masuoka, T Shiraishi, K Tabuch. Induction of apoptosis in glioma cells by recombinant human Fas ligand. Neurosurgery. vol 46. issue 2. 2000-03-23. PMID:10690733. |
induction of apoptosis in glioma cells by recombinant human fas ligand. |
2000-03-23 |
2023-08-12 |
human |
| S Kawaguchi, T Mineta, M Ichinose, J Masuoka, T Shiraishi, K Tabuch. Induction of apoptosis in glioma cells by recombinant human Fas ligand. Neurosurgery. vol 46. issue 2. 2000-03-23. PMID:10690733. |
fas ligand (fasl) belongs to the tumor necrosis factor family and has the ability to induce apoptosis in susceptible target cells by binding to its receptor, fas. |
2000-03-23 |
2023-08-12 |
human |
| U T Brunk, I Svensso. Oxidative stress, growth factor starvation and Fas activation may all cause apoptosis through lysosomal leak. Redox report : communications in free radical research. vol 4. issue 1-2. 2000-03-23. PMID:10714269. |
oxidative stress, growth factor starvation and fas activation may all cause apoptosis through lysosomal leak. |
2000-03-23 |
2023-08-12 |
human |
| C Gajate, R I Fonteriz, C Cabaner, G Alvarez-Noves, Y Alvarez-Rodriguez, M Modolell, F Mollined. Intracellular triggering of Fas, independently of FasL, as a new mechanism of antitumor ether lipid-induced apoptosis. International journal of cancer. vol 85. issue 5. 2000-03-22. PMID:10699948. |
intracellular triggering of fas, independently of fasl, as a new mechanism of antitumor ether lipid-induced apoptosis. |
2000-03-22 |
2023-08-12 |
human |
| C Gajate, R I Fonteriz, C Cabaner, G Alvarez-Noves, Y Alvarez-Rodriguez, M Modolell, F Mollined. Intracellular triggering of Fas, independently of FasL, as a new mechanism of antitumor ether lipid-induced apoptosis. International journal of cancer. vol 85. issue 5. 2000-03-22. PMID:10699948. |
in human lymphoid leukemic cells, et-18-och(3) does not promote fas or fasl expression and et-18-och(3)-induced apoptosis is not inhibited by pre-incubation with an anti-fas blocking antibody that abrogates cell killing mediated by fas/fasl interactions. |
2000-03-22 |
2023-08-12 |
human |
| C Gajate, R I Fonteriz, C Cabaner, G Alvarez-Noves, Y Alvarez-Rodriguez, M Modolell, F Mollined. Intracellular triggering of Fas, independently of FasL, as a new mechanism of antitumor ether lipid-induced apoptosis. International journal of cancer. vol 85. issue 5. 2000-03-22. PMID:10699948. |
confocal laser scanning microscopy shows that et-18-och(3) induces fas clustering and capping during triggering of et-18-och(3)-induced apoptosis. |
2000-03-22 |
2023-08-12 |
human |
| C Gajate, R I Fonteriz, C Cabaner, G Alvarez-Noves, Y Alvarez-Rodriguez, M Modolell, F Mollined. Intracellular triggering of Fas, independently of FasL, as a new mechanism of antitumor ether lipid-induced apoptosis. International journal of cancer. vol 85. issue 5. 2000-03-22. PMID:10699948. |
microinjection-induced apoptosis and fas clustering are specific for the molecular structure of et-18-och(3). |
2000-03-22 |
2023-08-12 |
human |
| C Gajate, R I Fonteriz, C Cabaner, G Alvarez-Noves, Y Alvarez-Rodriguez, M Modolell, F Mollined. Intracellular triggering of Fas, independently of FasL, as a new mechanism of antitumor ether lipid-induced apoptosis. International journal of cancer. vol 85. issue 5. 2000-03-22. PMID:10699948. |
our data indicate that et-18-och(3) induces apoptosis via fas after the ether lipid is inside the cell, and this fas activation is independent of the interaction of fas with its natural ligand fasl. |
2000-03-22 |
2023-08-12 |
human |
| A Hamzaoui, K Hamzaoui, H Salah, A Chabbo. Lymphocytes apoptosis in patients with acute exacerbation of asthma. Mediators of inflammation. vol 8. issue 4-5. 2000-03-21. PMID:10704078. |
ten patients with asthma were investigated during acute exacerbation for the quantification of apoptosis, for bcl-2 and fas expression, in induced sputum lymphocytes. |
2000-03-21 |
2023-08-12 |
Not clear |
| A Hamzaoui, K Hamzaoui, H Salah, A Chabbo. Lymphocytes apoptosis in patients with acute exacerbation of asthma. Mediators of inflammation. vol 8. issue 4-5. 2000-03-21. PMID:10704078. |
distinct mechanisms of apoptosis were found in patients with asthma and patients with copd, characterized by different levels of bcl-2 and fas expression. |
2000-03-21 |
2023-08-12 |
Not clear |
| S Vassiliadis, V Dragiotis, E Protopapadakis, I Athanassakis, P Mitlianga, K Konidaris, G K Papadopoulo. The destructive action of IL-1alpha and IL-1beta in IDDM is a multistage process: evidence and confirmation by apoptotic studies, induction of intermediates and electron microscopy. Mediators of inflammation. vol 8. issue 2. 2000-03-21. PMID:10704145. |
in addition, the interplay between no and fas is shown, while scanning electron microscopy (sem) confirms apoptosis by revealing the degree and type of cellular damage which, in the case of il-1alpha, can be reversed by an inhibitor to no synthesis. |
2000-03-21 |
2023-08-12 |
rat |
| T Reimer, C Herrnring, D Koczan, D Richter, B Gerber, D Kabelitz, K Friese, H J Thiese. FasL:Fas ratio--a prognostic factor in breast carcinomas. Cancer research. vol 60. issue 4. 2000-03-20. PMID:10706087. |
programmed cell death (apoptosis) is primarily mediated by fas ligand (fasl; cd95l) and the fas receptor (fas; cd95). |
2000-03-20 |
2023-08-12 |
human |