| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| S A Renshaw, S J Timmons, V Eaton, L R Usher, M Akil, C D Bingle, M K Whyt. Inflammatory neutrophils retain susceptibility to apoptosis mediated via the Fas death receptor. Journal of leukocyte biology. vol 67. issue 5. 2000-06-01. PMID:10811006. |
similarly, granulocyte-macrophage colony-stimulating factor reduced apoptosis but did not abolish signaling via fas. |
2000-06-01 |
2023-08-12 |
Not clear |
| M Younes, J Lechago, A Ertan, D Finnie, A Youne. Decreased expression of Fas (CD95/APO1) associated with goblet cell metaplasia in Barrett's esophagus. Human pathology. vol 31. issue 4. 2000-06-01. PMID:10821489. |
fas ligand (fasl) has been shown to induce apoptosis in cells expressing its receptor fas. |
2000-06-01 |
2023-08-12 |
Not clear |
| Y Yoshika. The interaction of intestinal epithelial cells and intraepithelial lymphocytes in host defense. Immunologic research. vol 20. issue 3. 2000-05-31. PMID:10741862. |
we show here that cd4+ cd8alphaalpha+ i-iel belongs to th1 type t cells capable of responding to self-mhc class i on i-ec and that a significant fraction of i-iel expressed fas ligand (fas-l) and induced apoptosis in the i-ec via fas-dependent pathway. |
2000-05-31 |
2023-08-12 |
mouse |
| D P Nelson, E Setser, D G Hall, S M Schwartz, T Hewitt, R Klevitsky, H Osinska, D Bellgrau, R C Duke, J Robbin. Proinflammatory consequences of transgenic fas ligand expression in the heart. The Journal of clinical investigation. vol 105. issue 9. 2000-05-25. PMID:10791994. |
fasl expression in the heart does result in mild leukocyte infiltration, but despite coexpression of fas and fasl in tg hearts, neither myocardial tissue apoptosis nor necrosis accompanies the leukocyte infiltration. |
2000-05-25 |
2023-08-12 |
mouse |
| K J Hamann, J E Vieira, A J Halayko, D Dorscheid, S R White, S M Forsythe, B Camoretti-Mercado, K F Rabe, J Solwa. Fas cross-linking induces apoptosis in human airway smooth muscle cells. American journal of physiology. Lung cellular and molecular physiology. vol 278. issue 3. 2000-05-24. PMID:10710535. |
fas cross-linking induces apoptosis in human airway smooth muscle cells. |
2000-05-24 |
2023-08-12 |
human |
| K J Hamann, J E Vieira, A J Halayko, D Dorscheid, S R White, S M Forsythe, B Camoretti-Mercado, K F Rabe, J Solwa. Fas cross-linking induces apoptosis in human airway smooth muscle cells. American journal of physiology. Lung cellular and molecular physiology. vol 278. issue 3. 2000-05-24. PMID:10710535. |
we therefore tested the hypotheses that 1) human airway smooth muscle (hasm) expresses fas, 2) fas cross-linking induces apoptosis in these cells, and 3) tumor necrosis factor (tnf)-alpha potentiates fas-mediated airway myocyte killing. |
2000-05-24 |
2023-08-12 |
human |
| K J Hamann, J E Vieira, A J Halayko, D Dorscheid, S R White, S M Forsythe, B Camoretti-Mercado, K F Rabe, J Solwa. Fas cross-linking induces apoptosis in human airway smooth muscle cells. American journal of physiology. Lung cellular and molecular physiology. vol 278. issue 3. 2000-05-24. PMID:10710535. |
fas cross-linking with ch-11 antibody induced apoptosis in cultured hasm cells, and this effect was reduced by long-term serum deprivation and synergistically potentiated by concomitant tnf-alpha exposure. |
2000-05-24 |
2023-08-12 |
human |
| K J Hamann, J E Vieira, A J Halayko, D Dorscheid, S R White, S M Forsythe, B Camoretti-Mercado, K F Rabe, J Solwa. Fas cross-linking induces apoptosis in human airway smooth muscle cells. American journal of physiology. Lung cellular and molecular physiology. vol 278. issue 3. 2000-05-24. PMID:10710535. |
tnf-alpha did not induce substantial apoptosis in the absence of fas cross-linking. |
2000-05-24 |
2023-08-12 |
human |
| K J Hamann, J E Vieira, A J Halayko, D Dorscheid, S R White, S M Forsythe, B Camoretti-Mercado, K F Rabe, J Solwa. Fas cross-linking induces apoptosis in human airway smooth muscle cells. American journal of physiology. Lung cellular and molecular physiology. vol 278. issue 3. 2000-05-24. PMID:10710535. |
these data represent the first demonstration that fas is expressed on hasm and suggest a mechanism by which fas-mediated apoptosis could act to oppose excess smooth muscle accumulation during airway remodeling in asthma. |
2000-05-24 |
2023-08-12 |
human |
| B Badie, J Schartner, J Vorpahl, K Presto. Interferon-gamma induces apoptosis and augments the expression of Fas and Fas ligand by microglia in vitro. Experimental neurology. vol 162. issue 2. 2000-05-23. PMID:10739635. |
interferon-gamma induces apoptosis and augments the expression of fas and fas ligand by microglia in vitro. |
2000-05-23 |
2023-08-12 |
Not clear |
| B Badie, J Schartner, J Vorpahl, K Presto. Interferon-gamma induces apoptosis and augments the expression of Fas and Fas ligand by microglia in vitro. Experimental neurology. vol 162. issue 2. 2000-05-23. PMID:10739635. |
these findings suggest that in addition to its role as a microglia activator, ifn-gamma may also induce apoptosis of microglia, possibly through simultaneous upregulation of fas and fasl. |
2000-05-23 |
2023-08-12 |
Not clear |
| B Badie, J Schartner, J Vorpahl, K Presto. Interferon-gamma induces apoptosis and augments the expression of Fas and Fas ligand by microglia in vitro. Experimental neurology. vol 162. issue 2. 2000-05-23. PMID:10739635. |
interferon-gamma modulation of the fas pathway and apoptosis in microglia may be important in the pathogenesis of inflammatory cns disease processes. |
2000-05-23 |
2023-08-12 |
Not clear |
| J Nilsen, G Mor, F Naftoli. Estrogen-regulated developmental neuronal apoptosis is determined by estrogen receptor subtype and the Fas/Fas ligand system. Journal of neurobiology. vol 43. issue 1. 2000-05-23. PMID:10756067. |
moreover, we show that estrogen-induced apoptosis through er-beta requires the expression of fas- and fas ligand (fasl) proteins, since the absence of fasl in neurons prevents this effect. |
2000-05-23 |
2023-08-12 |
rat |
| S C Ko, V L Johnson, S C Cho. Functional characterization of Jurkat T cells rescued from CD95/Fas-induced apoptosis through the inhibition of caspases. Biochemical and biophysical research communications. vol 270. issue 3. 2000-05-22. PMID:10772942. |
after 72 h, the z-vad-fmk-rescued cells proliferate normally and responded to activation induced cell death after phytohaemaglutinin treatment, and readily undergo apoptosis when restimulated with agonistic fas antibodies. |
2000-05-22 |
2023-08-12 |
human |
| C D Hsu, S F Hong, H Harirah, R Bahado-Singh, L c L. Amniotic fluid soluble fas levels in intra-amniotic infection. Obstetrics and gynecology. vol 95. issue 5. 2000-05-22. PMID:10775726. |
membrane fas can induce apoptosis in sensitive cells. |
2000-05-22 |
2023-08-12 |
Not clear |
| L A Norian, K M Latinis, S L Eliason, K Lyson, C Yang, T Ratliff, G A Koretzk. The regulation of CD95 (Fas) ligand expression in primary T cells: induction of promoter activation in CD95LP-Luc transgenic mice. Journal of immunology (Baltimore, Md. : 1950). vol 164. issue 9. 2000-05-22. PMID:10779747. |
the interaction between cd95 (fas) and cd95l (fas ligand) initiates apoptosis in a variety of cell types. |
2000-05-22 |
2023-08-12 |
mouse |
| W Xia, R Voellmy, N L Specto. Sensitization of tumor cells to fas killing through overexpression of heat-shock transcription factor 1. Journal of cellular physiology. vol 183. issue 3. 2000-05-22. PMID:10797318. |
the effects of fas on heat-shock-gene transcription do not appear to be a consequence of cell death as they (1) precede apoptotic changes and (2) are not abrogated by yvad-cmk, an inhibitor of fas apoptosis that acts by blocking downstream effector proteases. |
2000-05-22 |
2023-08-12 |
Not clear |
| W Xia, R Voellmy, N L Specto. Sensitization of tumor cells to fas killing through overexpression of heat-shock transcription factor 1. Journal of cellular physiology. vol 183. issue 3. 2000-05-22. PMID:10797318. |
despite expressing fas, hela cells are relatively insensitive to fas-mediated killing, indicating that fas expression alone, although necessary, is not sufficient for apoptosis. |
2000-05-22 |
2023-08-12 |
Not clear |
| D J Brick, R D Burke, A A Minkley, C Upto. Ectromelia virus virulence factor p28 acts upstream of caspase-3 in response to UV light-induced apoptosis. The Journal of general virology. vol 81. issue Pt 4. 2000-05-18. PMID:10725436. |
here, we report that both evp28 and overexpression of sfv n1r in poxvirus-infected hela cells protect specifically from uv light-induced apoptosis, but not from apoptosis induced by fas or tnf. |
2000-05-18 |
2023-08-12 |
Not clear |
| D J Brick, R D Burke, A A Minkley, C Upto. Ectromelia virus virulence factor p28 acts upstream of caspase-3 in response to UV light-induced apoptosis. The Journal of general virology. vol 81. issue Pt 4. 2000-05-18. PMID:10725436. |
further, we report that both vv and ev protect from apoptosis induced by uv, fas and tnf. |
2000-05-18 |
2023-08-12 |
Not clear |