All Relations between mesencephalon and dopamine

Publication Sentence Publish Date Extraction Date Species
P P Rompré, S M Boye, J Moisa. Activation of neurotensin receptors in the prefrontal cortex stimulates midbrain dopamine cell firing. European journal of pharmacology. vol 341. issue 2-3. 1998-05-21. PMID:9543236. activation of neurotensin receptors in the prefrontal cortex stimulates midbrain dopamine cell firing. 1998-05-21 2023-08-12 rat
P P Rompré, S M Boye, J Moisa. Activation of neurotensin receptors in the prefrontal cortex stimulates midbrain dopamine cell firing. European journal of pharmacology. vol 341. issue 2-3. 1998-05-21. PMID:9543236. the effects of medial prefrontal cortex microinjections of 3 nmol/0.5 microl of neurotensin-(1-13), the inactive fragment neurotensin-(1-8), or vehicle on the firing rate of midbrain dopamine neurons were studied in anesthetized rats. 1998-05-21 2023-08-12 rat
P P Rompré, S M Boye, J Moisa. Activation of neurotensin receptors in the prefrontal cortex stimulates midbrain dopamine cell firing. European journal of pharmacology. vol 341. issue 2-3. 1998-05-21. PMID:9543236. these results suggest that activation of neurotensin receptors in the medial prefrontal cortex can modulate neural activity of a subpopulation of midbrain dopamine neurons. 1998-05-21 2023-08-12 rat
B A Bennett, C K Hollingsworth, R S Martin, J J Har. Methamphetamine-induced alterations in dopamine transporter function. Brain research. vol 782. issue 1-2. 1998-05-14. PMID:9519266. this study was designed to examine the effects of acute and chronic meth exposure on uptake and release of [3h]dopamine (da) in cultured midbrain dopamine neurons to determine if persistent neuronal adaptations ensue. 1998-05-14 2023-08-12 rat
A J Levinson, S Garside, P I Rosebush, M F Mazure. Haloperidol induces persistent down-regulation of tyrosine hydroxylase immunoreactivity in substantia nigra but not ventral tegmental area in the rat. Neuroscience. vol 84. issue 1. 1998-05-08. PMID:9522374. these results indicate that haloperidol can produce selective changes in midbrain dopamine neurons that persist long after discontinuation of the drug. 1998-05-08 2023-08-12 rat
O Saucedo-Cardenas, J D Quintana-Hau, W D Le, M P Smidt, J J Cox, F De Mayo, J P Burbach, O M Conneel. Nurr1 is essential for the induction of the dopaminergic phenotype and the survival of ventral mesencephalic late dopaminergic precursor neurons. Proceedings of the National Academy of Sciences of the United States of America. vol 95. issue 7. 1998-05-01. PMID:9520484. further, as development progresses, these mid-brain dopamine precursor cells degenerate in the absence of nurr1, resulting in loss of ptx-3 expression and a concomitant increase in apoptosis of ventral midbrain neurons in newborn null mutant mice. 1998-05-01 2023-08-12 mouse
M Melis, G Mereu, V Lilliu, M Quartu, M Diana, G L Gess. Haloperidol does not produce dopamine cell depolarization-block in paralyzed, unanesthetized rats. Brain research. vol 783. issue 1. 1998-04-30. PMID:9479061. a widely accepted theory postulates that chronic treatment with neuroleptics causes, in rats, the depolarization block of the majority of midbrain dopamine (da) neurons. 1998-04-30 2023-08-12 rat
M A Wigmore, M G Lace. Metabotropic glutamate receptors depress glutamate-mediated synaptic input to rat midbrain dopamine neurones in vitro. British journal of pharmacology. vol 123. issue 4. 1998-04-16. PMID:9517386. metabotropic glutamate receptors depress glutamate-mediated synaptic input to rat midbrain dopamine neurones in vitro. 1998-04-16 2023-08-12 rat
M A Wigmore, M G Lace. Metabotropic glutamate receptors depress glutamate-mediated synaptic input to rat midbrain dopamine neurones in vitro. British journal of pharmacology. vol 123. issue 4. 1998-04-16. PMID:9517386. ), evoked by electrical stimulation rostral to the recording site, were examined by intracellular microelectrode recording from dopamine neurones in parasagittal slices of rat ventral midbrain. 1998-04-16 2023-08-12 rat
M A Wigmore, M G Lace. Metabotropic glutamate receptors depress glutamate-mediated synaptic input to rat midbrain dopamine neurones in vitro. British journal of pharmacology. vol 123. issue 4. 1998-04-16. PMID:9517386. these results provide evidence for the presence of presynaptic inhibitory metabotropic glutamate autoreceptors from the mglu receptor groups ii and iii on descending glutamatergic inputs to midbrain dopamine neurones. 1998-04-16 2023-08-12 rat
T E Koeltzow, M Xu, D C Cooper, X T Hu, S Tonegawa, M E Wolf, F J Whit. Alterations in dopamine release but not dopamine autoreceptor function in dopamine D3 receptor mutant mice. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 18. issue 6. 1998-04-13. PMID:9482807. dopamine (da) autoreceptors expressed along the somatodendritic extent of midbrain da neurons modulate impulse activity, whereas those expressed at da nerve terminals regulate both da synthesis and release. 1998-04-13 2023-08-12 mouse
Z Q Xu, J Y Lew, K Harada, K Aman, M Goldstein, A Deutch, J W Haycock, T Hökfel. Immunohistochemical studies on phosphorylation of tyrosine hydroxylase in central catecholamine neurons using site- and phosphorylation state-specific antibodies. Neuroscience. vol 82. issue 3. 1998-04-09. PMID:9483531. in the perfused brains ths40p-like immunoreactivity could be visualized in some dopamine neurons in the ventral mesencephalon, especially the a10 area, and in noradrenergic locus coeruleus neurons, whereas ths19p-like immunoreactivity was found in all catecholamine groups studied, similar to the results obtained with the pan-tyrosine hydroxylase antiserum and the tyrosine hydroxylase-monoclonal antibody. 1998-04-09 2023-08-12 mouse
J G Kohlert, R K Rowe, R L Meise. Intromissive stimulation from the male increases extracellular dopamine release from fluoro-gold-identified neurons within the midbrain of female hamsters. Hormones and behavior. vol 32. issue 3. 1998-03-26. PMID:9454665. intromissive stimulation from the male increases extracellular dopamine release from fluoro-gold-identified neurons within the midbrain of female hamsters. 1998-03-26 2023-08-12 Not clear
J G Kohlert, R K Rowe, R L Meise. Intromissive stimulation from the male increases extracellular dopamine release from fluoro-gold-identified neurons within the midbrain of female hamsters. Hormones and behavior. vol 32. issue 3. 1998-03-26. PMID:9454665. probe placements in the rostral accumbens, caudal accumbens, or rostral bed nucleus of the stria terminalis were not distinguishable based on analyses of basal dopamine levels, volume of fluoro-gold injection sites, or fluoro-gold labeling of midbrain, tyrosine hydroxylase-stained neurons. 1998-03-26 2023-08-12 Not clear
J G Kohlert, R K Rowe, R L Meise. Intromissive stimulation from the male increases extracellular dopamine release from fluoro-gold-identified neurons within the midbrain of female hamsters. Hormones and behavior. vol 32. issue 3. 1998-03-26. PMID:9454665. the number of midbrain neurons containing fluoro-gold was positively related to basal dopamine levels, indicating that the amount of dopamine recovered from the nucleus accumbens in microdialysis studies is a function of the number of neurons contributing to the terminal field in the region of the probe. 1998-03-26 2023-08-12 Not clear
K Chergui, P Svenningsson, G G Nomikos, F Gonon, B B Fredholm, T H Svennso. Increased expression of NGFI-A mRNA in the rat striatum following burst stimulation of the medial forebrain bundle. The European journal of neuroscience. vol 9. issue 11. 1998-03-20. PMID:9464931. these results provide additional support for the physiological importance of burst firing activity in midbrain dopamine neurons for the activation of their target cells. 1998-03-20 2023-08-12 rat
Y Fang, A Janowsky, O K Rønneklei. Cocaine exposure in fetal rhesus monkey: consequences for dopamine D1- and D2-like receptor binding densities. Brain research. Developmental brain research. vol 104. issue 1-2. 1998-03-20. PMID:9466719. these findings suggest that d1- and d2-like receptors are present in dopamine target neurons, whereas d2-like autoreceptors can not be detected in day-70 fetal monkey midbrain. 1998-03-20 2023-08-12 monkey
R Masalha, Y Herishanu, A Alfahel-Kakunda, W F Silverma. Selective dopamine neurotoxicity by an industrial chemical: an environmental cause of Parkinson's disease? Brain research. vol 774. issue 1-2. 1998-03-11. PMID:9452222. one of these agents, a light stabilizer employed universally in the production of polyolifins used in plastics, depleted primary mesencephalic cultures of dopamine neurons, and produced a dopamine-specific lesion of the substantia nigra pars compacta when injected stereotactically into the ventral midbrain of adult rats. 1998-03-11 2023-08-12 rat
P Pierret, N Quenneville, S Vandaele, R Abbaszadeh, C Lanctôt, P Crine, G Douce. Trophic and tropic effects of striatal astrocytes on cografted mesencephalic dopamine neurons and their axons. Journal of neuroscience research. vol 51. issue 1. 1998-03-10. PMID:9452306. astrocytes from the ventral mesencephalon and from the striatum respectively promote the dendritic and axonal arborization of dopamine (da) neurons in vitro. 1998-03-10 2023-08-12 rat
M Dziedzicka-Wasylewsk. The effect of imipramine on the amount of mRNA coding for rat dopamine D2 autoreceptors. European journal of pharmacology. vol 337. issue 2-3. 1998-03-03. PMID:9430428. the amount of mrna coding for dopamine d2 autoreceptors in the rat mesencephalon was analyzed following acute and repeated treatment with imipramine, the most widely used antidepressant drug. 1998-03-03 2023-08-12 rat