| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| Kiyoshi Egami, Silaja Yitta, Suhail Kasim, J Chris Lewers, Rosalinda C Roberts, Mohamed Lehar, H A Jinna. Basal ganglia dopamine loss due to defect in purine recycling. Neurobiology of disease. vol 26. issue 2. 2007-06-28. PMID:17374562. |
basal ganglia dopamine loss due to defect in purine recycling. |
2007-06-28 |
2023-08-12 |
mouse |
| H Wang, J Katz, P Dagostino, J-J Soghomonia. Unilateral 6-hydroxydopamine lesion of dopamine neurons and subchronic L-DOPA administration in the adult rat alters the expression of the vesicular GABA transporter in different subsets of striatal neurons and in the substantia nigra, pars reticulata. Neuroscience. vol 145. issue 2. 2007-06-26. PMID:17218060. |
the loss of dopamine neurons combined or not with the subsequent administration of l-dopa in patients with parkinson's disease or in experimental models of the disease results in altered gabaergic signaling throughout the basal ganglia, including the striatum and the substantia nigra, pars reticulata. |
2007-06-26 |
2023-08-12 |
rat |
| Takao Hashimot. [Functional models of movement disorders of basal ganglia origin and effects of functional neurosurgery]. Rinsho shinkeigaku = Clinical neurology. vol 47. issue 1. 2007-06-19. PMID:17491332. |
dopamine depletion in parkinson's disease increases basal ganglia output, then decreases thalamocortical output, leading to bradykinesia. |
2007-06-19 |
2023-08-12 |
monkey |
| R Franco, C Lluis, E I Canela, J Mallol, L Agnati, V Casadó, F Ciruela, S Ferré, K Fux. Receptor-receptor interactions involving adenosine A1 or dopamine D1 receptors and accessory proteins. Journal of neural transmission (Vienna, Austria : 1996). vol 114. issue 1. 2007-06-12. PMID:17024327. |
adenosine and dopamine receptors in the basal ganglia have been fundamental to demonstrate the existence of receptor heteromers and the functional consequences of such molecular interactions. |
2007-06-12 |
2023-08-12 |
Not clear |
| Flora Mela, Matteo Marti, Andrzej Dekundy, Wojciech Danysz, Michele Morari, M Angela Cenc. Antagonism of metabotropic glutamate receptor type 5 attenuates l-DOPA-induced dyskinesia and its molecular and neurochemical correlates in a rat model of Parkinson's disease. Journal of neurochemistry. vol 101. issue 2. 2007-06-01. PMID:17359492. |
in this study, we addressed the role of mglur5 in l-dopa-induced dyskinesia, a movement disorder that is due to abnormal activation of both dopamine and glutamate receptors in the basal ganglia. |
2007-06-01 |
2023-08-12 |
rat |
| A N Talalaenko, G K Krivobok, N P Bulgakova, D V Pankrat'e. [Functional significance of monoamine- and aminoacidergic mechanisms of dorsal pallidum in anxiety of different aversive genesis]. Zhurnal vysshei nervnoi deiatelnosti imeni I P Pavlova. vol 57. issue 1. 2007-05-30. PMID:17432321. |
dopamine and gaba injected locally into this formation of basal ganglia do not affect the mechanisms of voluntary movement, but counteract the conditions of anxiety in both models of behavior. |
2007-05-30 |
2023-08-12 |
rat |
| Adam J Schwarz, Alessandro Gozzi, Torsten Reese, Angelo Bifon. Functional connectivity in the pharmacologically activated brain: resolving networks of correlated responses to d-amphetamine. Magnetic resonance in medicine. vol 57. issue 4. 2007-05-24. PMID:17390353. |
a cluster analysis resolved three distinct networks of brain regions that exhibited closely coupled responses: one corresponding to primary dopamine projections from the midbrain to the striatum, a second consisting predominantly of forebrain cortical and basal ganglia regions that share a widespread correlation pattern resembling the univariate group response, and a third including structures in the periventricular dopamine system. |
2007-05-24 |
2023-08-12 |
rat |
| J-A Arias-Montaño, B Floran, L Floran, J Aceves, J M Youn. Dopamine D(1) receptor facilitation of depolarization-induced release of gamma-amino-butyric acid in rat striatum is mediated by the cAMP/PKA pathway and involves P/Q-type calcium channels. Synapse (New York, N.Y.). vol 61. issue 5. 2007-05-04. PMID:17318879. |
transmission in the "direct" pathway through the basal ganglia, which has an important role in the control of motor movement, is markedly facilitated by the concurrent activation of dopamine d(1) receptors. |
2007-05-04 |
2023-08-12 |
rat |
| Ayelet Sapir, Julie B Kaplan, Biyu J He, Maurizio Corbett. Anatomical correlates of directional hypokinesia in patients with hemispatial neglect. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 27. issue 15. 2007-05-03. PMID:17428982. |
the localization of neglect's motor deficits to the basal ganglia establishes interesting homologies with animal data; it also suggests that a relative depletion of dopamine in the nigrostriatal pathway on the same side of the lesion may be an important pathophysiological mechanism potentially amenable to intervention. |
2007-05-03 |
2023-08-12 |
Not clear |
| Jing-Yu Chang, Li-Hong Shi, Fei Luo, Donald J Woodwar. Neural responses in multiple basal ganglia regions following unilateral dopamine depletion in behaving rats performing a treadmill locomotion task. Experimental brain research. vol 172. issue 2. 2007-04-26. PMID:16369786. |
neural responses in multiple basal ganglia regions following unilateral dopamine depletion in behaving rats performing a treadmill locomotion task. |
2007-04-26 |
2023-08-12 |
rat |
| Jing-Yu Chang, Li-Hong Shi, Fei Luo, Donald J Woodwar. Neural responses in multiple basal ganglia regions following unilateral dopamine depletion in behaving rats performing a treadmill locomotion task. Experimental brain research. vol 172. issue 2. 2007-04-26. PMID:16369786. |
to investigate basal ganglia (bg) neural responses to dopamine (da) depletion, multiple channel, single unit recording was carried out in freely moving rats performing a treadmill locomotion task. |
2007-04-26 |
2023-08-12 |
rat |
| Thomas Müller, Hermann Rus. Levodopa, motor fluctuations and dyskinesia in Parkinson's disease. Expert opinion on pharmacotherapy. vol 7. issue 13. 2007-04-19. PMID:16925499. |
parkinson's disease (pd) is one of the most frequent, chronic, progressive degenerative disorders of the cns, characterised by altered neurotransmission of dopamine in the basal ganglia. |
2007-04-19 |
2023-08-12 |
Not clear |
| Maria Zalesska-Krecicka, Monika Morawska-Kochman, Ewa Wierzbicka, Aleksandra Skrzypek, Tomasz Krecick. [Spasmodic dysphonia in course of Meige's syndrome--case report]. Otolaryngologia polska = The Polish otolaryngology. vol 60. issue 5. 2007-04-05. PMID:17263254. |
the disorders of basal ganglia function and neurotransmitters' imbalance (dopamine and acetylocholine) can be with reason of pronouncement of symptoms presumably. |
2007-04-05 |
2023-08-12 |
Not clear |
| David Weisman, Ian McKeit. Dementia with Lewy bodies. Seminars in neurology. vol 27. issue 1. 2007-04-03. PMID:17226740. |
suggestive features include rem sleep behavior disorder, severe neuroleptic sensitivity, and low dopamine transporter uptake in the basal ganglia on functional neuroimaging. |
2007-04-03 |
2023-08-12 |
Not clear |
| J R Walters, D Hu, C A Itoga, L C Parr-Brownlie, D A Bergstro. Phase relationships support a role for coordinated activity in the indirect pathway in organizing slow oscillations in basal ganglia output after loss of dopamine. Neuroscience. vol 144. issue 2. 2007-03-28. PMID:17112675. |
phase relationships support a role for coordinated activity in the indirect pathway in organizing slow oscillations in basal ganglia output after loss of dopamine. |
2007-03-28 |
2023-08-12 |
rat |
| J R Walters, D Hu, C A Itoga, L C Parr-Brownlie, D A Bergstro. Phase relationships support a role for coordinated activity in the indirect pathway in organizing slow oscillations in basal ganglia output after loss of dopamine. Neuroscience. vol 144. issue 2. 2007-03-28. PMID:17112675. |
the goal of the present study was to determine the phase relationships of the slow oscillatory activity that emerges in basal ganglia nuclei in anesthetized rats after dopamine cell lesion in order to gain insight into the passage of this oscillatory activity through the basal ganglia network. |
2007-03-28 |
2023-08-12 |
rat |
| J R Walters, D Hu, C A Itoga, L C Parr-Brownlie, D A Bergstro. Phase relationships support a role for coordinated activity in the indirect pathway in organizing slow oscillations in basal ganglia output after loss of dopamine. Neuroscience. vol 144. issue 2. 2007-03-28. PMID:17112675. |
while these observations do not rule out additional mechanisms contributing to the emergence of slow oscillations in the basal ganglia after dopamine cell lesion in the anesthetized preparation, they are compatible with 1) increased oscillatory activity in the gp facilitated by an effect of dopamine loss on striatal 'filtering' of slow components of oscillatory cortical input, 2) increased oscillatory activity in stn spike trains supported by convergent antiphase inhibitory and excitatory oscillatory input from gp and cortex, respectively, and 3) increased oscillatory activity in snpr spike trains organized by convergent antiphase inhibitory and excitatory oscillatory input from gp and stn, respectively. |
2007-03-28 |
2023-08-12 |
rat |
| Przemysław Nowak, Grazyna Szczerbak, Joanna Dabrowska, Aleksandra Bortel, Izabela Biedka, Richard M Kostrzew. [Molecular mechanisms of levodopa action in animal models of Parkinson's disease]. Neurologia i neurochirurgia polska. vol 40. issue 6. 2007-03-27. PMID:17199178. |
one of the most important hallmarks of parkinson's disease is the loss of neuronal cell bodies containing neuromelanin in the substantia nigra zona compacta, and subsequently, loss of dopamine terminals in basal ganglia nuclei of the brain. |
2007-03-27 |
2023-08-12 |
Not clear |
| Héctor R Martínez, Leonel Cantú-Martínez, Humberto C González, Laura de León Flores, Héctor J Villarreal, Javier Onofre-Castill. Epilepsy, parkinsonism, and neuroleptic malignant syndrome in a child. Journal of child neurology. vol 21. issue 12. 2007-02-23. PMID:17156702. |
the basal ganglia and dopamine are involved in the pathophysiology of parkinsonism and neuroleptic malignant syndrome and have been recognized in seizure propagation and seizure threshold. |
2007-02-23 |
2023-08-12 |
Not clear |
| Mark D Bevan, Jeremy F Atherton, Jérôme Baufreto. Cellular principles underlying normal and pathological activity in the subthalamic nucleus. Current opinion in neurobiology. vol 16. issue 6. 2007-02-01. PMID:17084618. |
recent findings suggest that dopamine denervation of the striatum and extrastriatal basal ganglia profoundly alters the transmission and integration of glutamatergic cortical and gabaergic pallidal inputs to subthalamic neurons, leading to pathological activity that resonates throughout the basal ganglia and wider motor system. |
2007-02-01 |
2023-08-12 |
Not clear |