All Relations between basal ganglia and dopamine

Publication Sentence Publish Date Extraction Date Species
J R Walters, D Hu, C A Itoga, L C Parr-Brownlie, D A Bergstro. Phase relationships support a role for coordinated activity in the indirect pathway in organizing slow oscillations in basal ganglia output after loss of dopamine. Neuroscience. vol 144. issue 2. 2007-03-28. PMID:17112675. phase relationships support a role for coordinated activity in the indirect pathway in organizing slow oscillations in basal ganglia output after loss of dopamine. 2007-03-28 2023-08-12 rat
J R Walters, D Hu, C A Itoga, L C Parr-Brownlie, D A Bergstro. Phase relationships support a role for coordinated activity in the indirect pathway in organizing slow oscillations in basal ganglia output after loss of dopamine. Neuroscience. vol 144. issue 2. 2007-03-28. PMID:17112675. the goal of the present study was to determine the phase relationships of the slow oscillatory activity that emerges in basal ganglia nuclei in anesthetized rats after dopamine cell lesion in order to gain insight into the passage of this oscillatory activity through the basal ganglia network. 2007-03-28 2023-08-12 rat
J R Walters, D Hu, C A Itoga, L C Parr-Brownlie, D A Bergstro. Phase relationships support a role for coordinated activity in the indirect pathway in organizing slow oscillations in basal ganglia output after loss of dopamine. Neuroscience. vol 144. issue 2. 2007-03-28. PMID:17112675. while these observations do not rule out additional mechanisms contributing to the emergence of slow oscillations in the basal ganglia after dopamine cell lesion in the anesthetized preparation, they are compatible with 1) increased oscillatory activity in the gp facilitated by an effect of dopamine loss on striatal 'filtering' of slow components of oscillatory cortical input, 2) increased oscillatory activity in stn spike trains supported by convergent antiphase inhibitory and excitatory oscillatory input from gp and cortex, respectively, and 3) increased oscillatory activity in snpr spike trains organized by convergent antiphase inhibitory and excitatory oscillatory input from gp and stn, respectively. 2007-03-28 2023-08-12 rat
Przemysław Nowak, Grazyna Szczerbak, Joanna Dabrowska, Aleksandra Bortel, Izabela Biedka, Richard M Kostrzew. [Molecular mechanisms of levodopa action in animal models of Parkinson's disease]. Neurologia i neurochirurgia polska. vol 40. issue 6. 2007-03-27. PMID:17199178. one of the most important hallmarks of parkinson's disease is the loss of neuronal cell bodies containing neuromelanin in the substantia nigra zona compacta, and subsequently, loss of dopamine terminals in basal ganglia nuclei of the brain. 2007-03-27 2023-08-12 Not clear
Héctor R Martínez, Leonel Cantú-Martínez, Humberto C González, Laura de León Flores, Héctor J Villarreal, Javier Onofre-Castill. Epilepsy, parkinsonism, and neuroleptic malignant syndrome in a child. Journal of child neurology. vol 21. issue 12. 2007-02-23. PMID:17156702. the basal ganglia and dopamine are involved in the pathophysiology of parkinsonism and neuroleptic malignant syndrome and have been recognized in seizure propagation and seizure threshold. 2007-02-23 2023-08-12 Not clear
Mark D Bevan, Jeremy F Atherton, Jérôme Baufreto. Cellular principles underlying normal and pathological activity in the subthalamic nucleus. Current opinion in neurobiology. vol 16. issue 6. 2007-02-01. PMID:17084618. recent findings suggest that dopamine denervation of the striatum and extrastriatal basal ganglia profoundly alters the transmission and integration of glutamatergic cortical and gabaergic pallidal inputs to subthalamic neurons, leading to pathological activity that resonates throughout the basal ganglia and wider motor system. 2007-02-01 2023-08-12 Not clear
Philip Win. How best to consider the structure and function of the pedunculopontine tegmental nucleus: evidence from animal studies. Journal of the neurological sciences. vol 248. issue 1-2. 2007-01-23. PMID:16765383. the former contains dopamine neurons that project widely within the basal ganglia while the latter is in receipt of corticostriatal output. 2007-01-23 2023-08-12 Not clear
George W Hubert, Michael J Kuha. Colocalization of CART peptide with prodynorphin and dopamine D1 receptors in the rat nucleus accumbens. Neuropeptides. vol 40. issue 6. 2007-01-18. PMID:17064765. because cart has been shown to colocalize with substance p, a marker of the basal ganglia direct pathway, we now test for colocalization with other markers of the direct pathway to determine if cart colocalizes with dynorphin and dopamine d1 receptors. 2007-01-18 2023-08-12 rat
Dorothée Domenger, Rainer K W Schwartin. The serial reaction time task in the rat: effects of D1 and D2 dopamine-receptor antagonists. Behavioural brain research. vol 175. issue 2. 2007-01-11. PMID:17011054. work with such tasks has shown that sequential behaviour seems to be mediated by specific brain systems, including the basal ganglia and the neurotransmitter dopamine. 2007-01-11 2023-08-12 human
Wayne A Cass, Richard Grondin, Anders H Andersen, Zhiming Zhang, Peter A Hardy, Lindsay K Hussey-Andersen, William S Rayens, Greg A Gerhardt, Don M Gas. Iron accumulation in the striatum predicts aging-related decline in motor function in rhesus monkeys. Neurobiology of aging. vol 28. issue 2. 2007-01-09. PMID:16442671. these perturbations include alterations in dopamine (da) release, regulation and transport in the striatum and substantia nigra, striatal atrophy and elevated iron levels in the basal ganglia. 2007-01-09 2023-08-12 monkey
Pershia Samadi, Paul J Bédard, Claude Rouillar. Opioids and motor complications in Parkinson's disease. Trends in pharmacological sciences. vol 27. issue 10. 2006-12-13. PMID:16908075. in animal models of parkinson's disease, dopamine denervation and repeated l-dopa administration are associated with an enhancement of opioid transmission in the basal ganglia. 2006-12-13 2023-08-12 Not clear
George V Rebe. Behavioral electrophysiology of psychostimulants. Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology. vol 31. issue 11. 2006-12-12. PMID:16855534. the motor-activating effects of amphetamine and other psychostimulants such as cocaine depend on an increase in dopamine (da) transmission in the striatum, a key component of the basal ganglia and the forebrain motive circuit. 2006-12-12 2023-08-12 Not clear
Brian S McKay, Brooke Goodman, Torsten Falk, Scott J Sherma. Retinal pigment epithelial cell transplantation could provide trophic support in Parkinson's disease: results from an in vitro model system. Experimental neurology. vol 201. issue 1. 2006-11-14. PMID:16764861. transplantation of retinal pigment epithelial (rpe) cells in the basal ganglia could provide a novel cell-based therapy for parkinson's disease by providing a constant source of dopamine replacement via the melanin synthetic pathway enzyme tyrosinase. 2006-11-14 2023-08-12 human
Brian S McKay, Brooke Goodman, Torsten Falk, Scott J Sherma. Retinal pigment epithelial cell transplantation could provide trophic support in Parkinson's disease: results from an in vitro model system. Experimental neurology. vol 201. issue 1. 2006-11-14. PMID:16764861. these results indicate that transplantation rpe cells could potentially provide a dual benefit in parkinson's disease producing both dopamine and neurotrophic support of the basal ganglia. 2006-11-14 2023-08-12 human
David J Scott, Mary M Heitzeg, Robert A Koeppe, Christian S Stohler, Jon-Kar Zubiet. Variations in the human pain stress experience mediated by ventral and dorsal basal ganglia dopamine activity. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 26. issue 42. 2006-11-14. PMID:17050717. variations in the human pain stress experience mediated by ventral and dorsal basal ganglia dopamine activity. 2006-11-14 2023-08-12 human
David J Scott, Mary M Heitzeg, Robert A Koeppe, Christian S Stohler, Jon-Kar Zubiet. Variations in the human pain stress experience mediated by ventral and dorsal basal ganglia dopamine activity. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 26. issue 42. 2006-11-14. PMID:17050717. basal ganglia dopamine (da) neurotransmission may then respond to environmental events depending on their saliency, orienting the subsequent responses of the organism to both positive and negative stimuli. 2006-11-14 2023-08-12 human
David J Scott, Mary M Heitzeg, Robert A Koeppe, Christian S Stohler, Jon-Kar Zubiet. Variations in the human pain stress experience mediated by ventral and dorsal basal ganglia dopamine activity. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 26. issue 42. 2006-11-14. PMID:17050717. these data demonstrate that basal ganglia dopamine d2 receptor-mediated neurotransmission is involved in responses to pain and that it contributes to individual variations in the pain experience at the levels of physical and emotional elements, albeit with different neuroanatomical substrates. 2006-11-14 2023-08-12 human
Naila Ismayilova, Alexei Verkhratsky, Michael J Dascomb. Changes in mGlu5 receptor expression in the basal ganglia of reserpinised rats. European journal of pharmacology. vol 545. issue 2-3. 2006-11-13. PMID:16890937. dopamine depletion in parkinson's disease results in a series of pathophysiological changes in the basal ganglia circuitry. 2006-11-13 2023-08-12 rat
A N Talalaenko, G K Krivobok, D V Pankrat'ev, N V Goncharenk. Neurochemical mechanisms of the dorsal pallidum in the antiaversive effects of anxiolytics in various models of anxiety. Neuroscience and behavioral physiology. vol 36. issue 7. 2006-10-31. PMID:16841156. dopamine, apomorphine, gaba, chlordiazepoxide, phenibut, and indoter injected locally into this formation of the basal ganglia had no effect on the mechanisms of voluntary movement but counteracted anxiety states in both behavioral models. 2006-10-31 2023-08-12 rat
Ubaldo Bonuccelli, Paolo Del Dott. New pharmacologic horizons in the treatment of Parkinson disease. Neurology. vol 67. issue 7 Suppl 2. 2006-10-30. PMID:17030738. the nonphysiologic pulsatile stimulation of striatal dopamine receptors, produced by the currently available dopaminergic drugs, may trigger a dysregulation of many neurotransmitter systems within the basal ganglia, mainly localized on medium spiny striatal neurons. 2006-10-30 2023-08-12 Not clear