All Relations between hypertrophic and matrix compartment

Publication Sentence Publish Date Extraction Date Species
Bing-Ying Wan, Dong-Wei Cao, Yi-Gang Wan, Dai Chen, Wei Wu, Qi-Jun Fang, Si-Yi Liu, Yue Tu, Yu Wang, Zi-Yue Wa. [Effects and mechanisms of total flavones of Abelmoschus manihot in attenuating diabetic tubulopathy by targeting endoplasmic reticulum stress-induced cell apoptosis]. Zhongguo Zhong yao za zhi = Zhongguo zhongyao zazhi = China journal of Chinese materia medica. vol 48. issue 10. 2023-06-07. PMID:37282927. the results indicated that hypertrophy of renal tubular epithelial cells, renal tubular hyperplasia and occlusion, as well as interstitial extracellular matrix and collagen deposition occurred in the dt model rats. 2023-06-07 2023-08-14 rat
Bingyang Dai, Yuwei Zhu, Xu Li, Zuru Liang, Shunxiang Xu, Shian Zhang, Zhe Zhang, Shanshan Bai, Wenxue Tong, Mingde Cao, Ye Li, Xiaobo Zhu, Wei Liu, Yuantao Zhang, Liang Chang, Patrick Shu-Hang Yung, Kevin Ki-Wai Ho, Jiankun Xu, To Ngai, Ling Qi. Blockage of Osteopontin-Integrin β3 Signaling in Infrapatellar Fat Pad Attenuates Osteoarthritis in Mice. Advanced science (Weinheim, Baden-Wurttemberg, Germany). 2023-05-23. PMID:37218542. it is further demonstrated that ipfp-derived opn participates in oa progression, including activated matrix metallopeptidase 9 in chondrocyte hypertrophy and integrin β3 in ipfp fibrosis. 2023-05-23 2023-08-14 mouse
Debao Guan, Xin Zhuan, Xiaoyu Luo, Hao Ga. An Updated Lagrangian Constrained Mixture Model of Pathological Cardiac Growth and Remodelling. Acta biomaterialia. 2023-05-18. PMID:37201740. progressive left ventricular (lv) growth and remodelling (g&r) is often induced by volume and pressure overload, characterized by structural and functional adaptation through myocyte hypertrophy and extracellular matrix remodelling, which are dynamically regulated by biomechanical factors, inflammation, neurohormonal pathways, etc. 2023-05-18 2023-08-14 human
Giuseppe Filiberto Serraino, Federica Jiritano, Davide Costa, Nicola Ielapi, Desirèe Napolitano, Pasquale Mastroroberto, Umberto Marcello Bracale, Michele Andreucci, Raffaele Serr. Metalloproteinases and Hypertrophic Cardiomyopathy: A Systematic Review. Biomolecules. vol 13. issue 4. 2023-05-16. PMID:37189412. hypertrophic cardiomyopathy (hcm) is a genetic condition determined by an altered collagen turnover of the extracellular matrix. 2023-05-16 2023-08-14 human
Nikolaos Chrysakis, Andrew Xanthopoulos, Dimitrios Magouliotis, Randall C Starling, Stavros G Drakos, Filippos Triposkiadis, John Skoularigi. Myocardial Recovery. Diagnostics (Basel, Switzerland). vol 13. issue 8. 2023-05-16. PMID:37189604. the changes that take place in cardiac hypertrophy, extracellular matrix, cell populations and their structural elements, β-receptors, energetics, and several biological processes, are reviewed. 2023-05-16 2023-08-14 Not clear
X Cheng, L Huan. The Mechanism of the Anti-Cardiac Hypertrophy Effect of Glycyrrhizic Acid Is Related to Reducing STIM1-Dependent Store-Operated Calcium Entry. Bulletin of experimental biology and medicine. 2023-05-10. PMID:37162628. in rats with modeled cardiac hypertrophy, the effect of ga on expression of type 1 matrix interaction molecules was determined in horizontal tissues and cultured cardiomyocytes of the left ventricle. 2023-05-10 2023-08-14 rat
Jiayin Ye, Cheng Gao, Yong Liang, Zongliu Hou, Yufang Shi, Ying Wan. Characteristic and fate determination of adipose precursors during adipose tissue remodeling. Cell regeneration (London, England). vol 12. issue 1. 2023-05-03. PMID:37138165. changes in adipocytes, hypertrophy or hyperplasia, contribute to the remodeling of obese adipose tissues, accompanied by abundant immune cell accumulation, decreased angiogenesis, and aberrant extracellular matrix deposition. 2023-05-03 2023-08-14 Not clear
Rosa Lillo, Francesca Graziani, Francesco Franceschi, Giulia Iannaccone, Massimo Massetti, Iacopo Olivotto, Filippo Crea, Giovanna Liuzz. Inflammation across the spectrum of hypertrophic cardiac phenotypes. Heart failure reviews. 2023-04-28. PMID:37115472. indeed, inflammation can trigger molecular pathways which contribute to cardiomyocyte hypertrophy and dysfunction, extracellular matrix accumulation, and microvascular dysfunction. 2023-04-28 2023-08-14 Not clear
Jianfei Li, Feiju M. Polo-Like Kinase 2 Is Identified in Hypertrophy, Extracellular Matrix Accumulation, and Oxidative Stress of Mesangial Cells in Diabetic Nephropathy through p38-MAPK Signaling. Annals of clinical and laboratory science. vol 53. issue 2. 2023-04-24. PMID:37094847. polo-like kinase 2 is identified in hypertrophy, extracellular matrix accumulation, and oxidative stress of mesangial cells in diabetic nephropathy through p38-mapk signaling. 2023-04-24 2023-08-14 Not clear
Maria Uscategui Calderon, Brittany A Gonzalez, Katherine E Yutze. Cardiomyocyte-fibroblast crosstalk in the postnatal heart. Frontiers in cell and developmental biology. vol 11. 2023-04-20. PMID:37077417. moreover, postnatal cardiomyocytes undergo binucleation and cell cycle arrest with induction of hypertrophic growth, while cardiac fibroblasts proliferate and produce extracellular matrix (ecm) that transitions from components that support cellular maturation to production of the mature fibrous skeleton of the heart. 2023-04-20 2023-08-14 Not clear
L Chen, H Yan, B Zhou, Y F Xu, J L. [New research advances in hypertrophic scar formation, prevention and treatment]. Zhonghua yu fang yi xue za zhi [Chinese journal of preventive medicine]. vol 57. issue 4. 2023-04-09. PMID:37032171. excessive fb proliferation which produces the excessive collagen deposition, including increased extracellular matrix synthesis or insufficient decomposition, typically contributes to hypertrophic scar(hs) formation. 2023-04-09 2023-08-14 Not clear
Minghao Qu, Mingjue Chen, Weiyuan Gong, Shaochuan Huo, Qinnan Yan, Qing Yao, Yumei Lai, Di Chen, Xiaohao Wu, Guozhi Xia. Pip5k1c Loss in Chondrocytes Causes Spontaneous Osteoarthritic Lesions in Aged Mice. Aging and disease. vol 14. issue 2. 2023-04-03. PMID:37008048. pip5k1c loss promotes extracellular matrix (ecm) degradation, chondrocyte hypertrophy and apoptosis, and inhibits chondrocyte proliferation in the articular cartilage of aged mice. 2023-04-03 2023-08-14 mouse
Qian Yu, Qiang Dai, Zonglin Huang, Chen Li, Li Yan, Xin Fu, Qian Wang, Yi Zhang, Lei Cai, Zhigang Yang, Ran Xia. Microfat exerts an anti-fibrotic effect on human hypertrophic scar via fetuin-A/ETV4 axis. Journal of translational medicine. vol 21. issue 1. 2023-04-02. PMID:37004048. hypertrophic scar is a fibrotic disease following wound healing and is characterized by excessive extracellular matrix deposition. 2023-04-02 2023-08-14 human
Manjula P Mony, Kelly A Harmon, Ryan Hess, Amir H Dorafshar, Sasha H Shafikhan. An Updated Review of Hypertrophic Scarring. Cells. vol 12. issue 5. 2023-03-11. PMID:36899815. hypertrophic scarring (hts) is an aberrant form of wound healing that is associated with excessive deposition of extracellular matrix and connective tissue at the site of injury. 2023-03-11 2023-08-14 Not clear
Shenyi Jiang, Shuhao Qian, Lingxi Zhou, Jia Meng, Rushan Jiang, Chuncheng Wang, Xinguo Fang, Chen Yang, Zhihua Ding, Shuangmu Zhuo, Zhiyi Li. Mapping the 3D remodeling of the extracellular matrix in human hypertrophic scar by multi-parametric multiphoton imaging using endogenous contrast. Heliyon. vol 9. issue 2. 2023-03-06. PMID:36873151. mapping the 3d remodeling of the extracellular matrix in human hypertrophic scar by multi-parametric multiphoton imaging using endogenous contrast. 2023-03-06 2023-08-14 human
Irina V Kologrivova, Natalia V Naryzhnaya, Olga A Koshelskaya, Tatiana E Suslova, Elena S Kravchenko, Olga A Kharitonova, Vladimir V Evtushenko, Alla A Boshchenk. Association of Epicardial Adipose Tissue Adipocytes Hypertrophy with Biomarkers of Low-Grade Inflammation and Extracellular Matrix Remodeling in Patients with Coronary Artery Disease. Biomedicines. vol 11. issue 2. 2023-02-25. PMID:36830779. association of epicardial adipose tissue adipocytes hypertrophy with biomarkers of low-grade inflammation and extracellular matrix remodeling in patients with coronary artery disease. 2023-02-25 2023-08-14 Not clear
Lei Huang, Meng Jin, Ruiying Gu, Kunlin Xiao, Mengnan Lu, Xinyu Huo, Mengyao Sun, Zhi Yang, Zhiyuan Wang, Weijie Zhang, Liqiang Zhi, Ziang Meng, Jie Ma, Jianbing Ma, Rui Zhan. miR-199a-5p Reduces Chondrocyte Hypertrophy and Attenuates Osteoarthritis Progression via the Indian Hedgehog Signal Pathway. Journal of clinical medicine. vol 12. issue 4. 2023-02-25. PMID:36835852. further investigations demonstrated that mir-199a-5p could directly regulate ihh expression and reduce chondrocyte hypertrophy and matrix degradation via the ihh signal pathway in the primary human chondrocytes. 2023-02-25 2023-08-14 human
Chin-Chan Lee, Chia-Chun Chen, Cheng-Kai Hsu, Yih-Ting Chen, Chun-Yu Chen, Kai-Jie Yang, Ming-Jui Hung, I-Wen W. Urinary microRNA in Diabetic Kidney Disease: A Literature Review. Medicina (Kaunas, Lithuania). vol 59. issue 2. 2023-02-25. PMID:36837555. previous literatures have reported its potential role in the pathophysiology of diabetic kidney disease, including regulation of transforming growth factor-β1-mediated fibrosis, extracellular matrix and cell adhesion proteins, cellular hypertrophy, growth factor, cytokine production, and redox system activation. 2023-02-25 2023-08-14 Not clear
Joshua S Godwin, Casey L Sexton, Nicholas J Kontos, Bradley A Ruple, Darryn S Willoughby, Kaelin C Young, C Brooks Mobley, Michael D Robert. Extracellular matrix content and remodeling markers do not differ in college-aged men classified as higher- and lower-responders to resistance training. Journal of applied physiology (Bethesda, Md. : 1985). 2023-02-09. PMID:36759158. we determined if skeletal muscle extracellular matrix (ecm) content and remodeling markers adapted with resistance training or were associated with hypertrophic outcomes. 2023-02-09 2023-08-14 human
Charly Robert, Frédéric Kerff, Fabrice Bouillenne, Maxime Gavage, Marylène Vandevenne, Patrice Filée, André Matagn. Structural analysis of the interaction between human cytokine BMP-2 and the antagonist Noggin reveals molecular details of cell chondrogenesis inhibition. The Journal of biological chemistry. 2023-01-15. PMID:36642181. additionally, several in vitro cell-based assays, including enzymatic measurements, rt-qpcr and matrix staining, demonstrated their biological activity during cell chondrogenic and hypertrophic differentiation. 2023-01-15 2023-08-14 human