All Relations between glutamatergic and prefrontal cortex

Publication Sentence Publish Date Extraction Date Species
Shannon L Gourley, Alexia T Kedves, Peter Olausson, Jane R Taylo. A history of corticosterone exposure regulates fear extinction and cortical NR2B, GluR2/3, and BDNF. Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology. vol 34. issue 3. 2009-03-30. PMID:18719621. extinction of conditioned fear is an active learning process thought to involve glutamatergic targets--including specific nmda and ampa receptor subunits--in the ventromedial prefrontal cortex (vmpfc), which includes the prelimbic, infralimbic, and medial orbitofrontal cortices. 2009-03-30 2023-08-12 rat
Julián de Almeida, Guadalupe Mengo. Serotonin 1A receptors in human and monkey prefrontal cortex are mainly expressed in pyramidal neurons and in a GABAergic interneuron subpopulation: implications for schizophrenia and its treatment. Journal of neurochemistry. vol 107. issue 2. 2008-12-24. PMID:18761712. we used double in situ hybridization histochemistry to quantify the percentage of glutamatergic and gabaergic neurons expressing 5-ht(1a) receptors in human and monkey prefrontal cortex. 2008-12-24 2023-08-12 human
K Fuxe, D Marcellino, A Rivera, Z Diaz-Cabiale, M Filip, B Gago, D C S Roberts, U Langel, S Genedani, L Ferraro, A de la Calle, J Narvaez, S Tanganelli, A Woods, L F Agnat. Receptor-receptor interactions within receptor mosaics. Impact on neuropsychopharmacology. Brain research reviews. vol 58. issue 2. 2008-12-18. PMID:18222544. such treatment may ensure the proper glutamatergic drive from the mediodorsal thalamic nucleus to the prefrontal cortex, one which is believed to be reduced in schizophrenia due to a dominance of d(2)-like signaling in the ventral striatum. 2008-12-18 2023-08-12 Not clear
A Dervaux, X Laqueill. [Smoking and schizophrenia: epidemiological and clinical features]. L'Encephale. vol 34. issue 3. 2008-10-23. PMID:18558153. smoking may transiently alleviate negative symptoms in schizophrenic patients by increasing dopaminergic and glutamatergic neurotransmission in the prefrontal cortex. 2008-10-23 2023-08-12 human
Tsung-Ung W Woo, Amy M Kim, Emma Viscid. Disease-specific alterations in glutamatergic neurotransmission on inhibitory interneurons in the prefrontal cortex in schizophrenia. Brain research. vol 1218. 2008-10-08. PMID:18534564. disease-specific alterations in glutamatergic neurotransmission on inhibitory interneurons in the prefrontal cortex in schizophrenia. 2008-10-08 2023-08-12 human
Tsung-Ung W Woo, Amy M Kim, Emma Viscid. Disease-specific alterations in glutamatergic neurotransmission on inhibitory interneurons in the prefrontal cortex in schizophrenia. Brain research. vol 1218. 2008-10-08. PMID:18534564. these findings raise the possibility that glutamatergic innervation of inhibitory interneurons via the nmda receptor in the prefrontal cortex may be selectively altered in schizophrenia. 2008-10-08 2023-08-12 human
Mary M Torregrossa, X-C Tang, Peter W Kaliva. The glutamatergic projection from the prefrontal cortex to the nucleus accumbens core is required for cocaine-induced decreases in ventral pallidal GABA. Neuroscience letters. vol 438. issue 2. 2008-09-19. PMID:18455875. the glutamatergic projection from the prefrontal cortex to the nucleus accumbens core is required for cocaine-induced decreases in ventral pallidal gaba. 2008-09-19 2023-08-12 Not clear
Mary M Torregrossa, X-C Tang, Peter W Kaliva. The glutamatergic projection from the prefrontal cortex to the nucleus accumbens core is required for cocaine-induced decreases in ventral pallidal GABA. Neuroscience letters. vol 438. issue 2. 2008-09-19. PMID:18455875. the present study investigated the neural mechanism of this cocaine-induced decrease in vp gaba by determining if activity of the glutamatergic projection from the medial prefrontal cortex (pfc) to the nucleus accumbens is required for the effect. 2008-09-19 2023-08-12 Not clear
Christophe Rouillon, Jacques H Abraini, Hélène N Davi. Prefrontal cortex and basolateral amygdala modulation of dopamine-mediated locomotion in the nucleus accumbens core. Experimental neurology. vol 212. issue 1. 2008-07-17. PMID:18501353. though this may be of critical importance to better understand the neural mechanisms of motivated behaviors and certain psychiatric diseases, the specific role of the glutamatergic afferents arising from the pfc and the bla in the modulation of locomotion produced by activation in the nucleus accumbens (nacc) of d1-like receptors or d2-like postsynaptic receptors yet has not been examined. 2008-07-17 2023-08-12 Not clear
Christophe Rouillon, Jacques H Abraini, Hélène N Davi. Prefrontal cortex and basolateral amygdala modulation of dopamine-mediated locomotion in the nucleus accumbens core. Experimental neurology. vol 212. issue 1. 2008-07-17. PMID:18501353. these data demonstrate that the glutamatergic inputs arising from the pfc and the bla specifically control d2-like- and d1-like-mediated locomotor responses, respectively. 2008-07-17 2023-08-12 Not clear
M W Feltenstein, R E Se. The neurocircuitry of addiction: an overview. British journal of pharmacology. vol 154. issue 2. 2008-06-19. PMID:18311189. although drugs of abuse possess diverse neuropharmacological profiles, activation of the mesocorticolimbic system, particularly the ventral tegmental area, nucleus accumbens, amygdala and prefrontal cortex via dopaminergic and glutamatergic pathways, constitutes a common pathway by which various drugs of abuse mediate their acute reinforcing effects. 2008-06-19 2023-08-12 Not clear
G Baiardi, A M Ruiz, A Beling, J Borgonovo, G Martínez, A I Landa, M A Sosa, P A Gargiul. Glutamatergic ionotropic blockade within accumbens disrupts working memory and might alter the endocytic machinery in rat accumbens and prefrontal cortex. Journal of neural transmission (Vienna, Austria : 1996). vol 114. issue 12. 2008-05-28. PMID:17616844. glutamatergic ionotropic blockade within accumbens disrupts working memory and might alter the endocytic machinery in rat accumbens and prefrontal cortex. 2008-05-28 2023-08-12 rat
Vinay Parikh, Kingson Man, Michael W Decker, Martin Sarte. Glutamatergic contributions to nicotinic acetylcholine receptor agonist-evoked cholinergic transients in the prefrontal cortex. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 28. issue 14. 2008-05-19. PMID:18385335. glutamatergic contributions to nicotinic acetylcholine receptor agonist-evoked cholinergic transients in the prefrontal cortex. 2008-05-19 2023-08-12 Not clear
Vinay Parikh, Kingson Man, Michael W Decker, Martin Sarte. Glutamatergic contributions to nicotinic acetylcholine receptor agonist-evoked cholinergic transients in the prefrontal cortex. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 28. issue 14. 2008-05-19. PMID:18385335. glutamatergic mechanisms necessarily mediate the cholinergic effects of nachr agonists in the prefrontal cortex. 2008-05-19 2023-08-12 Not clear
Fabio Fumagalli, Angelisa Frasca, Giorgio Racagni, Marco Andrea Riv. Dynamic regulation of glutamatergic postsynaptic activity in rat prefrontal cortex by repeated administration of antipsychotic drugs. Molecular pharmacology. vol 73. issue 5. 2008-05-06. PMID:18250147. dynamic regulation of glutamatergic postsynaptic activity in rat prefrontal cortex by repeated administration of antipsychotic drugs. 2008-05-06 2023-08-12 rat
Fabio Fumagalli, Angelisa Frasca, Giorgio Racagni, Marco Andrea Riv. Dynamic regulation of glutamatergic postsynaptic activity in rat prefrontal cortex by repeated administration of antipsychotic drugs. Molecular pharmacology. vol 73. issue 5. 2008-05-06. PMID:18250147. by using western blot and immunoprecipitation techniques, we investigated the expression, trafficking, and interaction of essential components of glutamatergic synapse in rat prefrontal cortex. 2008-05-06 2023-08-12 rat
Fabio Fumagalli, Angelisa Frasca, Giorgio Racagni, Marco Andrea Riv. Dynamic regulation of glutamatergic postsynaptic activity in rat prefrontal cortex by repeated administration of antipsychotic drugs. Molecular pharmacology. vol 73. issue 5. 2008-05-06. PMID:18250147. the global attenuation of glutamatergic function in prefrontal cortex might explain, at least in part, the cognitive deterioration observed in patients treated with haloperidol. 2008-05-06 2023-08-12 rat
Pablo Henny, Barbara E Jone. Projections from basal forebrain to prefrontal cortex comprise cholinergic, GABAergic and glutamatergic inputs to pyramidal cells or interneurons. The European journal of neuroscience. vol 27. issue 3. 2008-04-22. PMID:18279318. projections from basal forebrain to prefrontal cortex comprise cholinergic, gabaergic and glutamatergic inputs to pyramidal cells or interneurons. 2008-04-22 2023-08-12 rat
Pablo Henny, Barbara E Jone. Projections from basal forebrain to prefrontal cortex comprise cholinergic, GABAergic and glutamatergic inputs to pyramidal cells or interneurons. The European journal of neuroscience. vol 27. issue 3. 2008-04-22. PMID:18279318. the present study was undertaken to characterize the pre- and postsynaptic constituents of the basal forebrain (bf) projection to the prefrontal cortex in the rat, and determine whether it includes glutamatergic in addition to established gamma-aminobutyric acid (gaba)ergic and cholinergic elements. 2008-04-22 2023-08-12 rat
Peter W Kaliva. Cocaine and amphetamine-like psychostimulants: neurocircuitry and glutamate neuroplasticity. Dialogues in clinical neuroscience. vol 9. issue 4. 2008-04-11. PMID:18286799. this review outlines the affect of psychostimulants on mesocorticolimbic dopamine projections that mediate their reinforcing effect, and how this action ultimately leads to enduring pathological neuroplasticity in glutamatergic projections from the prefrontal cortex to the nucleus accumbens. 2008-04-11 2023-08-12 Not clear