All Relations between glutamatergic and prefrontal cortex

Publication Sentence Publish Date Extraction Date Species
J Bagley, B Moghadda. Temporal dynamics of glutamate efflux in the prefrontal cortex and in the hippocampus following repeated stress: effects of pretreatment with saline or diazepam. Neuroscience. vol 77. issue 1. 1997-05-02. PMID:9044375. the present study suggests that: (i) stress produces a rapid increase in glutamate efflux in the prefrontal cortex and hippocampus, (ii) repeated stress reveals tolerance in the glutamatergic response in the prefrontal cortex, (iii) saline and diazepam pretreatment reduce the stress-induced efflux of glutamate in the prefrontal cortex, and (iv) exposure to diazepam may prevent the prefrontal cortex from adapting its response to the subsequent stressor. 1997-05-02 2023-08-12 rat
B Moghaddam, B Adams, A Verma, D Dal. Activation of glutamatergic neurotransmission by ketamine: a novel step in the pathway from NMDA receptor blockade to dopaminergic and cognitive disruptions associated with the prefrontal cortex. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 17. issue 8. 1997-04-28. PMID:9092613. activation of glutamatergic neurotransmission by ketamine: a novel step in the pathway from nmda receptor blockade to dopaminergic and cognitive disruptions associated with the prefrontal cortex. 1997-04-28 2023-08-12 rat
B Moghaddam, B Adams, A Verma, D Dal. Activation of glutamatergic neurotransmission by ketamine: a novel step in the pathway from NMDA receptor blockade to dopaminergic and cognitive disruptions associated with the prefrontal cortex. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 17. issue 8. 1997-04-28. PMID:9092613. a thorough dose-response study using microdialysis in conscious rats indicated that low doses of ketamine (10, 20, and 30 mg/kg) increase glutamate outflow in the pfc, suggesting that at these doses ketamine may increase glutamatergic neurotransmission in the pfc at non-nmda glutamate receptors. 1997-04-28 2023-08-12 rat
M Garcia-Munoz, P Patino, E Masliah, S J Young, P M Grove. Glutamate-dependent long-term presynaptic changes in corticostriatal excitability. Neuroscience. vol 73. issue 1. 1997-03-11. PMID:8783235. we have previously shown that brief high frequency stimulation of the anteromedial prefrontal cortex induces a long-term decrease in excitability of the glutamatergic corticostriatal terminal field. 1997-03-11 2023-08-12 rat
J L Tchélingérian, F Le Saux, C Jacqu. Identification and topography of neuronal cell populations expressing TNF alpha and IL-1 alpha in response to hippocampal lesion. Journal of neuroscience research. vol 43. issue 1. 1996-12-16. PMID:8838580. although not immunohistochemically characterized, neurons in some glutamatergic structures such as the hippocampus and the prefrontal cortex also express these cytokines. 1996-12-16 2023-08-12 mouse
M Karreman, B Moghadda. The prefrontal cortex regulates the basal release of dopamine in the limbic striatum: an effect mediated by ventral tegmental area. Journal of neurochemistry. vol 66. issue 2. 1996-04-03. PMID:8592128. the present study examined whether the prefrontal cortex (pfc) exerts a tonic control over the basal release of dopamine in the limbic striatum and whether this control is mediated by glutamatergic afferents to the dopamine cell body or terminal regions. 1996-04-03 2023-08-12 rat
K N Dudkin, V K Kruchinin, I V Chuev. [The neurophysiological correlates of improvement in the cognitive characteristics of monkeys with modification of the NMDA-ergic structures of the prefrontal cortex]. Fiziologicheskii zhurnal imeni I.M. Sechenova. vol 81. issue 1. 1996-01-03. PMID:7489012. the data obtained suggest that the glutamatergic structures of prefrontal cortex take part in the processes of visual recognition and the sm in monkeys. 1996-01-03 2023-08-12 monkey
G Tsai, L A Passani, B S Slusher, R Carter, L Baer, J E Kleinman, J T Coyl. Abnormal excitatory neurotransmitter metabolism in schizophrenic brains. Archives of general psychiatry. vol 52. issue 10. 1995-11-06. PMID:7575102. to test the glutamatergic hypothesis of schizophrenia, we studied the naag-related glutamatergic variables in postmortem brains from patients with schizophrenia, neuroleptic-treated controls, and normal individuals, with particular emphasis on the prefrontal cortex and hippocampus. 1995-11-06 2023-08-12 Not clear
M T Taber, S Das, H C Fibige. Cortical regulation of subcortical dopamine release: mediation via the ventral tegmental area. Journal of neurochemistry. vol 65. issue 3. 1995-09-18. PMID:7643120. these results indicate that ionotropic glutamate receptors in the vta are critically involved in basal and evoked dopamine release in the nucleus accumbens and suggest that a glutamatergic projection from the prefrontal cortex regulates the activity of dopaminergic neurons in the vta. 1995-09-18 2023-08-12 Not clear
D Law-Tho, J C Hirsch, F Crepe. Dopamine modulation of synaptic transmission in rat prefrontal cortex: an in vitro electrophysiological study. Neuroscience research. vol 21. issue 2. 1995-05-23. PMID:7724066. these findings indicate that da decreases both glutamatergic and gabaergic synaptic transmission in neurons located in layer v of rat prefrontal cortex. 1995-05-23 2023-08-12 rat
B Moghadda. Recent basic findings in support of excitatory amino acid hypotheses of schizophrenia. Progress in neuro-psychopharmacology & biological psychiatry. vol 18. issue 5. 1994-12-28. PMID:7972857. these include studies on the functional links between glutamatergic and dopaminergic systems, effect of acute and chronic antipsychotic drug treatment on excitatory amino acid function, and stress-induced activation of excitatory amino acid release, in particular in the prefrontal cortex. 1994-12-28 2023-08-12 human
J Gigg, A M Tan, D M Finc. Glutamatergic hippocampal formation projections to prefrontal cortex in the rat are regulated by GABAergic inhibition and show convergence with glutamatergic projections from the limbic thalamus. Hippocampus. vol 4. issue 2. 1994-12-20. PMID:7951693. glutamatergic hippocampal formation projections to prefrontal cortex in the rat are regulated by gabaergic inhibition and show convergence with glutamatergic projections from the limbic thalamus. 1994-12-20 2023-08-12 rat
J Gigg, A M Tan, D M Finc. Glutamatergic hippocampal formation projections to prefrontal cortex in the rat are regulated by GABAergic inhibition and show convergence with glutamatergic projections from the limbic thalamus. Hippocampus. vol 4. issue 2. 1994-12-20. PMID:7951693. these data confirm that the hf projection to prefrontal cortex is, at least in part, glutamatergic; suggest that the responses of mpfc neurons to activity in this hf pathway are regulated by gabaergic inhibition; and indicate that projections from hf and md converge onto single mpfc neurons. 1994-12-20 2023-08-12 rat
F Trovero, P Marin, J P Tassin, J Premont, J Glowinsk. Accelerated resensitization of the D1 dopamine receptor-mediated response in cultured cortical and striatal neurons from the rat: respective role of alpha 1-adrenergic and N-methyl-D-aspartate receptors. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 14. issue 10. 1994-11-14. PMID:7931580. as previously shown in vivo, noradrenergic and glutamatergic neurons can regulate the denervation supersensitivity of d1 dopaminergic (da) receptors in the rat prefrontal cortex and striatum respectively. 1994-11-14 2023-08-12 rat
H P Jedema, B Moghadda. Glutamatergic control of dopamine release during stress in the rat prefrontal cortex. Journal of neurochemistry. vol 63. issue 2. 1994-08-12. PMID:7518503. glutamatergic control of dopamine release during stress in the rat prefrontal cortex. 1994-08-12 2023-08-12 rat
M T Taber, H C Fibige. Electrical stimulation of the medial prefrontal cortex increases dopamine release in the striatum. Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology. vol 9. issue 4. 1994-03-17. PMID:8305127. we hypothesized that stimulation of the prefrontal cortex (pfc) would phasically enhance striatal da release via the glutamatergic corticostriatal pathway. 1994-03-17 2023-08-12 rat
G B Schacter, C R Yang, N K Innis, G J Mogenso. The role of the hippocampal-nucleus accumbens pathway in radial-arm maze performance. Brain research. vol 494. issue 2. 1989-10-26. PMID:2776019. similar injections were made in rats with ibotenic acid lesions of the prefrontal cortex in order to eliminate the glutamatergic prefrontal cortex-nucleus accumbens pathway so as to investigate the glutamatergic hippocampal-nucleus accumbens pathway. 1989-10-26 2023-08-11 rat
W Hauber, W J Schmid. Effects of intrastriatal blockade of glutamatergic transmission on the acquisition of T-maze and radial maze tasks. Journal of neural transmission. General section. vol 78. issue 1. 1989-09-07. PMID:2569029. prefrontal cortex and neostriatum constituting the prefrontal system are connected by glutamatergic neurones. 1989-09-07 2023-08-11 rat