| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| M Didrikse. Effects of antipsychotics on cognitive behaviour in rats using the delayed non-match to position paradigm. European journal of pharmacology. vol 281. issue 3. 1996-01-25. PMID:8521906. |
the acute effects of the dopamine d1 receptor antagonist sch 23390 [(r)-(+)-8-chloro-2,3,4,5-tetrahydro-3-methyl-5-phenyl-1h-3-benzazepi n-7-ol d hemimaleat, the dopamine d2 receptor antagonists raclopride and haloperidol, the compounds with mixed receptor profiles clozapine, risperidone and sertindole, the alpha 1-adrenoceptor antagonist prazosin and scopolamine were investigated in a delay-response task, a test for working memory, for rats. |
1996-01-25 |
2023-08-12 |
rat |
| N S Magoski, L G Bauce, N I Syed, A G Bulloc. Dopaminergic transmission between identified neurons from the mollusk, Lymnaea stagnalis. Journal of neurophysiology. vol 74. issue 3. 1996-01-18. PMID:7500151. |
several dopamine antagonists were used in an attempt to block rped1's synapses: chlorpromazine, ergonovine, fluphenazine, haloperidol, 6-hydroxydopamine, sch 23390, (+/-) sulpiride, and tubocurarine. |
1996-01-18 |
2023-08-12 |
Not clear |
| C R Ashby, H Pan, Y Minabe, A Toor, L Fishkin, R Y Wan. Comparison of the action of the stereoisomers of the psychostimulant 4-methylaminorex (4-MAX) on midbrain dopamine cells in the rat: an extracellular single unit study. Synapse (New York, N.Y.). vol 20. issue 4. 1995-12-14. PMID:7482294. |
administration of haloperidol and the d2/d3 receptor antagonists (-)-sulpiride and (-)-eticlopride but not by the d1 receptor antagonists sch 23390 and sch 39166. |
1995-12-14 |
2023-08-12 |
rat |
| R Corbett, F Camacho, A T Woods, L L Kerman, R J Fishkin, K Brooks, R W Dun. Antipsychotic agents antagonize non-competitive N-methyl-D-aspartate antagonist-induced behaviors. Psychopharmacology. vol 120. issue 1. 1995-12-07. PMID:7480537. |
sch 23390, raclopride, haloperidol, chlorpromazine and risperidone failed to reverse the social withdrawal induced by pcp up to doses which produced significant motor impairment. |
1995-12-07 |
2023-08-12 |
mouse |
| Y Fischer, J Thomas, J Kamp, E Jüngling, H Rose, Carpéné, H Kammermeie. 5-hydroxytryptamine stimulates glucose transport in cardiomyocytes via a monoamine oxidase-dependent reaction. The Biochemical journal. vol 311 ( Pt 2). 1995-11-28. PMID:7487898. |
the effect of 5-ht was not affected by (i) the 5-ht receptor antagonists methysergide (1 microm), ketanserin (1 microm), cyproheptadine (1 microm), mdl 72222 (1 microm) or ics 205-930 (3 microm), nor by (ii) the adrenergic receptor antagonists prazosin (1 microm), yohimbine (1 microm) or propranolol (5 microm), nor by (iii) the dopaminergic antagonists sch 23390 (1 microm) or haloperidol (1 microm). |
1995-11-28 |
2023-08-12 |
rat |
| P M Callahan, K A Cunningha. Modulation of the discriminative stimulus properties of cocaine by 5-HT1B and 5-HT2C receptors. The Journal of pharmacology and experimental therapeutics. vol 274. issue 3. 1995-10-25. PMID:7562516. |
the da receptor antagonists sch 23390 (0.025 or 0.05 mg/kg) and haloperidol (0.125 or 0.25 mg/kg) failed to block the partial substitution of ru 24969 (1 mg/kg) for cocaine. |
1995-10-25 |
2023-08-12 |
rat |
| H Kuribar. Inhibition of methamphetamine sensitization by post-methamphetamine treatment with SCH 23390 or haloperidol. Psychopharmacology. vol 119. issue 1. 1995-10-19. PMID:7675947. |
inhibition of methamphetamine sensitization by post-methamphetamine treatment with sch 23390 or haloperidol. |
1995-10-19 |
2023-08-12 |
mouse |
| H Kuribar. Inhibition of methamphetamine sensitization by post-methamphetamine treatment with SCH 23390 or haloperidol. Psychopharmacology. vol 119. issue 1. 1995-10-19. PMID:7675947. |
both sch 23390 (0.03 mg/kg sc) and haloperidol (0.4 mg/kg sc), dopamine d1 and d2 receptor antagonists, respectively, completely inhibited not only the acute stimulant effect of methamphetamine but also its sensitization when repeated methamphetamine was repeatedly combined with either of these drugs. |
1995-10-19 |
2023-08-12 |
mouse |
| H Kuribar. Inhibition of methamphetamine sensitization by post-methamphetamine treatment with SCH 23390 or haloperidol. Psychopharmacology. vol 119. issue 1. 1995-10-19. PMID:7675947. |
moreover, treatment with sch 23390 2-5 h or haloperidol 1-5 h after each methamphetamine administration significantly antagonized methamphetamine sensitization. |
1995-10-19 |
2023-08-12 |
mouse |
| H Kuribar. Inhibition of methamphetamine sensitization by post-methamphetamine treatment with SCH 23390 or haloperidol. Psychopharmacology. vol 119. issue 1. 1995-10-19. PMID:7675947. |
however, treatments with sch 23390 or haloperidol at 0.5 h, 6 h and 24 h after methamphetamine had no such inhibitory effect. |
1995-10-19 |
2023-08-12 |
mouse |
| H Kuribar. Inhibition of methamphetamine sensitization by post-methamphetamine treatment with SCH 23390 or haloperidol. Psychopharmacology. vol 119. issue 1. 1995-10-19. PMID:7675947. |
the mice treated with sch 23390 or haloperidol after each saline administration (the control administration for methamphetamine) did not show significant change in the sensitivity to methamphetamine. |
1995-10-19 |
2023-08-12 |
mouse |
| S Honma, K Honm. Phase-dependent phase shift of methamphetamine-induced circadian rhythm by haloperidol in SCN-lesioned rats. Brain research. vol 674. issue 2. 1995-08-03. PMID:7796108. |
haloperidol, a non-selective dopamine receptor antagonist, was injected intraperitoneally in to suprachiasmatic nucleus (scn)-lesioned rats at various phases of the locomotor activity rhythm induced by methamphetamine (map) treatment. |
1995-08-03 |
2023-08-12 |
rat |
| J Koistinaho, S M Saga. Light-induced c-fos expression in amacrine cells in the rabbit retina. Brain research. Molecular brain research. vol 29. issue 1. 1995-07-06. PMID:7770001. |
nicotine, kainic acid, nmda and sch 38393, a dopamine d1 receptor agonist, induced fos immunostaining in the inner nuclear and ganglion cell layers, but administration of the corresponding receptor blockers mecamylamine, kynuretic acid, mk-801, haloperidol and sch 23990 did not prevent light-induced fos expression. |
1995-07-06 |
2023-08-12 |
rabbit |
| B Ferger, K Kuschinsk. Effects of morphine on EEG in rats and their possible relations to hypo- and hyperkinesia. Psychopharmacology. vol 117. issue 2. 1995-06-22. PMID:7753968. |
this effect was antagonized by naloxone (0.5 mg/kg ip) but only in part by the blocker of d1 receptors sch 23390 (0.2 mg/kg ip) and not by haloperidol in a dose which mainly blocks d2 receptors (0.1 mg/kg ip). |
1995-06-22 |
2023-08-12 |
rat |
| N E Lezcano, S R De Barioglio, M E Celi. alpha-MSH changes cyclic AMP levels in rat brain slices by an interaction with the D1 dopamine receptor. Peptides. vol 16. issue 1. 1995-05-18. PMID:7716065. |
therefore, we measured camp in tissues and medium in response to alpha-msh in the presence of haloperidol, the selective d1 (sch 23390) or d2 (sulpiride) antagonists, or the selective d1 (skf 38393) or d2 (bromocriptine) agonists. |
1995-05-18 |
2023-08-12 |
rat |
| E M Byrnes, B J Johnson, J P Brun. D1- and D2-receptor mediation of motoric behavior in rats depleted of DA as neonates: effects of age and size of depletion. Neuroscience letters. vol 181. issue 1-2. 1995-04-27. PMID:7898774. |
moreover, since important changes in da receptor ontogeny occur during the first postnatal week, we compared the ability of the d1-like antagonist, sch 23390 (0.2 mg/kg), or the d2-like antagonists, clebopride (10.0 mg/kg) and haloperidol (1.0 mg/kg), to induce akinesia or catalepsy in adults depleted of da on either day 1 or on day 3. |
1995-04-27 |
2023-08-12 |
rat |
| L W Fitzgerald, A Y Deutch, G Gasic, S F Heinemann, E J Nestle. Regulation of cortical and subcortical glutamate receptor subunit expression by antipsychotic drugs. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 15. issue 3 Pt 2. 1995-04-17. PMID:7891180. |
by immunoblotting procedures using antibodies specific for the nmdar1, glur1, and glur2 subunits, we found that haloperidol (predominantly a d2-like antagonist) increased nmdar1 subunit immunoreactivity (and mrna levels) in the striatum, while the d1-like antagonist sch 23390 had the opposite effect. |
1995-04-17 |
2023-08-12 |
Not clear |
| L W Fitzgerald, A Y Deutch, G Gasic, S F Heinemann, E J Nestle. Regulation of cortical and subcortical glutamate receptor subunit expression by antipsychotic drugs. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 15. issue 3 Pt 2. 1995-04-17. PMID:7891180. |
the second major finding of the present study was the ability of haloperidol and clozapine to increase glur1 levels in the medial prefrontal cortex (pfc), whereas chronic sch 23390 treatment decreased glur1 levels. |
1995-04-17 |
2023-08-12 |
Not clear |
| A Verma, S K Kulkarn. D1/D2 dopamine and N-methyl-D-aspartate (NMDA) receptor participation in experimental catalepsy in rats. Psychopharmacology. vol 109. issue 4. 1995-04-04. PMID:1365866. |
b-ht 920, a d2 agonist, reversed the cataleptogenic effects of perphenazine, haloperidol and sch 23390. |
1995-04-04 |
2023-08-11 |
rat |
| A Verma, S K Kulkarn. D1/D2 dopamine and N-methyl-D-aspartate (NMDA) receptor participation in experimental catalepsy in rats. Psychopharmacology. vol 109. issue 4. 1995-04-04. PMID:1365866. |
combined administration of b-ht 920 (0.1 mg/kg) and skf 38393 (5 mg/kg) enhanced the protective effect of b-ht 920 in sch 23390-treated animals but not in animals treated with haloperidol or perphenazine. |
1995-04-04 |
2023-08-11 |
rat |