All Relations between feeding and rhombencephalon

Publication Sentence Publish Date Extraction Date Species
Ai-Jun Li, Qing Wang, Hana Davis, Rong Wang, Sue Ritte. Orexin-A enhances feeding in male rats by activating hindbrain catecholamine neurons. American journal of physiology. Regulatory, integrative and comparative physiology. vol 309. issue 4. 2015-11-10. PMID:26062632. orexin-a enhances feeding in male rats by activating hindbrain catecholamine neurons. 2015-11-10 2023-08-13 rat
Ai-Jun Li, Qing Wang, Hana Davis, Rong Wang, Sue Ritte. Orexin-A enhances feeding in male rats by activating hindbrain catecholamine neurons. American journal of physiology. Regulatory, integrative and comparative physiology. vol 309. issue 4. 2015-11-10. PMID:26062632. both lateral hypothalamic orexinergic neurons and hindbrain catecholaminergic neurons contribute to control of feeding behavior. 2015-11-10 2023-08-13 rat
Ai-Jun Li, Qing Wang, Hana Davis, Rong Wang, Sue Ritte. Orexin-A enhances feeding in male rats by activating hindbrain catecholamine neurons. American journal of physiology. Regulatory, integrative and comparative physiology. vol 309. issue 4. 2015-11-10. PMID:26062632. orexin fibers and terminals are present in close proximity to hindbrain catecholaminergic neurons, and fourth ventricular (4v) orexin injections that increase food intake also increase c-fos expression in hindbrain catecholamine neurons, suggesting that orexin neurons may stimulate feeding by activating catecholamine neurons. 2015-11-10 2023-08-13 rat
Ai-Jun Li, Qing Wang, Megan M Elsarelli, R Lane Brown, Sue Ritte. Hindbrain Catecholamine Neurons Activate Orexin Neurons During Systemic Glucoprivation in Male Rats. Endocrinology. vol 156. issue 8. 2015-10-06. PMID:25978516. hindbrain catecholamine neurons are required for elicitation of feeding responses to glucose deficit, but the forebrain circuitry required for these responses is incompletely understood. 2015-10-06 2023-08-13 rat
Ai-Jun Li, Qing Wang, Megan M Elsarelli, R Lane Brown, Sue Ritte. Hindbrain Catecholamine Neurons Activate Orexin Neurons During Systemic Glucoprivation in Male Rats. Endocrinology. vol 156. issue 8. 2015-10-06. PMID:25978516. orexin neurons, located in the perifornical lateral hypothalamus (peflh), are heavily innervated by hindbrain catecholamine neurons, stimulate food intake, and increase arousal and behavioral activation. 2015-10-06 2023-08-13 rat
Maria Fernanda Condes Areias, Patricia Oliveira Prad. Mechanisms of insulin resistance in the amygdala: influences on food intake. Behavioural brain research. vol 282. 2015-09-21. PMID:25601576. food intake is regulated by a complex mechanism involving the hypothalamus and hindbrain circuitries. 2015-09-21 2023-08-13 human
Hélène Volkof. In vitro assessment of interactions between appetite-regulating peptides in brain of goldfish (Carassius auratus). Peptides. vol 61. 2015-07-29. PMID:25219945. in order to better understand the interactions between appetite-related peptides, the effects of in vitro treatments of hindbrain, forebrain and hypothalamus--a major feeding regulating area--fragments with mch, apelin and orexin on the expression of mch, apelin, orexin, cart (forms 1 and 2) and npy were assessed. 2015-07-29 2023-08-13 Not clear
Zhi Yi Ong, Amber L Alhadeff, Harvey J Gril. Medial nucleus tractus solitarius oxytocin receptor signaling and food intake control: the role of gastrointestinal satiation signal processing. American journal of physiology. Regulatory, integrative and comparative physiology. vol 308. issue 9. 2015-07-06. PMID:25740340. central oxytocin (ot) administration reduces food intake and its effects are mediated, in part, by hindbrain oxytocin receptor (ot-r) signaling. 2015-07-06 2023-08-13 rat
Zhi Yi Ong, Amber L Alhadeff, Harvey J Gril. Medial nucleus tractus solitarius oxytocin receptor signaling and food intake control: the role of gastrointestinal satiation signal processing. American journal of physiology. Regulatory, integrative and comparative physiology. vol 308. issue 9. 2015-07-06. PMID:25740340. we examined the hypothesis that hindbrain ot-r-mediated feeding inhibition results from an interaction between medial nucleus tractus solitarius (mnts) ot-r signaling and the processing of gastrointestinal (gi) satiation signals by neurons of the mnts. 2015-07-06 2023-08-13 rat
Zhi Yi Ong, Amber L Alhadeff, Harvey J Gril. Medial nucleus tractus solitarius oxytocin receptor signaling and food intake control: the role of gastrointestinal satiation signal processing. American journal of physiology. Regulatory, integrative and comparative physiology. vol 308. issue 9. 2015-07-06. PMID:25740340. taken together, these findings provide evidence that mnts neurons are a site of action for hindbrain ot-r signaling in food intake control and that the intake inhibitory effects of hindbrain mnts ot-r signaling are mediated by interactions with gi satiation signal processing by mnts neurons. 2015-07-06 2023-08-13 rat
Carlos A Campos, Robert C Ritte. NMDA-type glutamate receptors participate in reduction of food intake following hindbrain melanocortin receptor activation. American journal of physiology. Regulatory, integrative and comparative physiology. vol 308. issue 1. 2015-03-04. PMID:25394828. nmda-type glutamate receptors participate in reduction of food intake following hindbrain melanocortin receptor activation. 2015-03-04 2023-08-13 rat
Carlos A Campos, Robert C Ritte. NMDA-type glutamate receptors participate in reduction of food intake following hindbrain melanocortin receptor activation. American journal of physiology. Regulatory, integrative and comparative physiology. vol 308. issue 1. 2015-03-04. PMID:25394828. hindbrain injection of a melanocortin-3/4 receptor agonist, mtii, reduces food intake primarily by reducing meal size. 2015-03-04 2023-08-13 rat
Carlos A Campos, Robert C Ritte. NMDA-type glutamate receptors participate in reduction of food intake following hindbrain melanocortin receptor activation. American journal of physiology. Regulatory, integrative and comparative physiology. vol 308. issue 1. 2015-03-04. PMID:25394828. hindbrain nmdar antagonism also attenuated mtii-induced erk1/2 phosphorylation in nts neurons and prevented synapsin i phosphorylation in central vagal afferent endings, both of which are cellular mechanisms previously shown to participate in hindbrain melanocortinergic reduction of food intake. 2015-03-04 2023-08-13 rat
Carlos A Campos, Robert C Ritte. NMDA-type glutamate receptors participate in reduction of food intake following hindbrain melanocortin receptor activation. American journal of physiology. Regulatory, integrative and comparative physiology. vol 308. issue 1. 2015-03-04. PMID:25394828. together, our results indicate that nmdar activation significantly contributes to reduction of food intake following hindbrain melanocortin receptor activation, and it participates in melanocortinergic signaling in nts neural circuits that mediate reduction of food intake. 2015-03-04 2023-08-13 rat
Tamara N Dunn, Sean H Adam. Relations between metabolic homeostasis, diet, and peripheral afferent neuron biology. Advances in nutrition (Bethesda, Md.). vol 5. issue 4. 2015-02-13. PMID:25022988. it is well established that food intake behavior and energy balance are regulated by crosstalk between peripheral organ systems and the central nervous system (cns), for instance, through the actions of peripherally derived leptin on hindbrain and hypothalamic loci. 2015-02-13 2023-08-13 Not clear
Amy K Sutton, Hongjuan Pei, Korri H Burnett, Martin G Myers, Christopher J Rhodes, David P Olso. Control of food intake and energy expenditure by Nos1 neurons of the paraventricular hypothalamus. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 34. issue 46. 2015-02-09. PMID:25392498. here we show that nos1(pvh) neurons project to hindbrain and spinal cord regions important for food intake and energy expenditure control. 2015-02-09 2023-08-13 mouse
Kristen Kay, Eric M Parise, Nicole Lilly, Diana L William. Hindbrain orexin 1 receptors influence palatable food intake, operant responding for food, and food-conditioned place preference in rats. Psychopharmacology. vol 231. issue 2. 2015-01-12. PMID:23978908. hindbrain orexin 1 receptors influence palatable food intake, operant responding for food, and food-conditioned place preference in rats. 2015-01-12 2023-08-12 rat
Kristen Kay, Eric M Parise, Nicole Lilly, Diana L William. Hindbrain orexin 1 receptors influence palatable food intake, operant responding for food, and food-conditioned place preference in rats. Psychopharmacology. vol 231. issue 2. 2015-01-12. PMID:23978908. most research has focused on forebrain ox1r populations, but hindbrain ox1rs affect feeding. 2015-01-12 2023-08-12 rat
Jennifer E Richard, Imre Farkas, Fredrik Anesten, Rozita H Anderberg, Suzanne L Dickson, Fiona M Gribble, Frank Reimann, John-Olov Jansson, Zsolt Liposits, Karolina P Skibick. GLP-1 receptor stimulation of the lateral parabrachial nucleus reduces food intake: neuroanatomical, electrophysiological, and behavioral evidence. Endocrinology. vol 155. issue 11. 2014-12-31. PMID:25116706. here, we hypothesize that hindbrain glucagon-like peptide 1 (glp-1) neurons provide excitatory inputs to the pbn, activation of which may lead to a reduction in feeding behavior. 2014-12-31 2023-08-13 mouse
Carlos A Campos, Hiroko Shiina, Robert C Ritte. Central vagal afferent endings mediate reduction of food intake by melanocortin-3/4 receptor agonist. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 34. issue 38. 2014-11-11. PMID:25232103. hindbrain injection of a pka inhibitor, kt5720, significantly attenuated mtii-induced reduction of food intake and the increase in synapsin i phosphorylation. 2014-11-11 2023-08-13 rat