| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| Carlos A Campos, Hiroko Shiina, Robert C Ritte. Central vagal afferent endings mediate reduction of food intake by melanocortin-3/4 receptor agonist. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 34. issue 38. 2014-11-11. PMID:25232103. |
altogether, our results suggest that reduction of food intake following hindbrain mc4r activation is mediated by central vagal afferent endings. |
2014-11-11 |
2023-08-13 |
rat |
| Amber L Alhadeff, Harvey J Gril. Hindbrain nucleus tractus solitarius glucagon-like peptide-1 receptor signaling reduces appetitive and motivational aspects of feeding. American journal of physiology. Regulatory, integrative and comparative physiology. vol 307. issue 4. 2014-10-09. PMID:24944243. |
hindbrain nucleus tractus solitarius glucagon-like peptide-1 receptor signaling reduces appetitive and motivational aspects of feeding. |
2014-10-09 |
2023-08-13 |
Not clear |
| Jacqueline M Ho, Vishwanath T Anekonda, Benjamin W Thompson, Mingyan Zhu, Robert W Curry, Bang H Hwang, Gregory J Morton, Michael W Schwartz, Denis G Baskin, Suzanne M Appleyard, James E Blevin. Hindbrain oxytocin receptors contribute to the effects of circulating oxytocin on food intake in male rats. Endocrinology. vol 155. issue 8. 2014-09-19. PMID:24877632. |
hindbrain oxytocin receptors contribute to the effects of circulating oxytocin on food intake in male rats. |
2014-09-19 |
2023-08-13 |
mouse |
| Jacqueline M Ho, Vishwanath T Anekonda, Benjamin W Thompson, Mingyan Zhu, Robert W Curry, Bang H Hwang, Gregory J Morton, Michael W Schwartz, Denis G Baskin, Suzanne M Appleyard, James E Blevin. Hindbrain oxytocin receptors contribute to the effects of circulating oxytocin on food intake in male rats. Endocrinology. vol 155. issue 8. 2014-09-19. PMID:24877632. |
because plasma ot levels increase after a meal, we hypothesized that circulating ot acts at both peripheral and hindbrain ot receptors (otrs) to limit food intake. |
2014-09-19 |
2023-08-13 |
mouse |
| Jacqueline M Ho, Vishwanath T Anekonda, Benjamin W Thompson, Mingyan Zhu, Robert W Curry, Bang H Hwang, Gregory J Morton, Michael W Schwartz, Denis G Baskin, Suzanne M Appleyard, James E Blevin. Hindbrain oxytocin receptors contribute to the effects of circulating oxytocin on food intake in male rats. Endocrinology. vol 155. issue 8. 2014-09-19. PMID:24877632. |
to initially determine whether circulating ot inhibits food intake by acting at hindbrain otrs, we pretreated rats with an otr antagonist administered into the fourth ventricle (4v) followed by either central or systemic ot administration. |
2014-09-19 |
2023-08-13 |
mouse |
| Sayaka Akieda-Asai, Paul-Emile Poleni, Yukari Dat. Coinjection of CCK and leptin reduces food intake via increased CART/TRH and reduced AMPK phosphorylation in the hypothalamus. American journal of physiology. Endocrinology and metabolism. vol 306. issue 11. 2014-07-22. PMID:24735891. |
furthermore, by using midbrain-transected rats, we investigated the role of the neural pathway from the hindbrain to the hypothalamus in the interaction of cck and leptin to reduce food intake. |
2014-07-22 |
2023-08-13 |
rat |
| Liangyou Ru. Brain regulation of energy balance and body weight. Reviews in endocrine & metabolic disorders. vol 14. issue 4. 2014-07-01. PMID:23990408. |
the hypothalamic circuits regulate food intake in part by modulating the sensitivity of the hindbrain to short-term satiety hormones. |
2014-07-01 |
2023-08-12 |
Not clear |
| Ayman I Sayeg. The role of bombesin and bombesin-related peptides in the short-term control of food intake. Progress in molecular biology and translational science. vol 114. 2014-06-05. PMID:23317790. |
studies have shown that the vagus and the splanchnic nerves in the upper gastrointestinal tract, which communicate with the feeding areas of the hindbrain, are necessary for reduction of ms and prolongation of the imi by bn, grp, and nmb. |
2014-06-05 |
2023-08-12 |
Not clear |
| Ai-Jun Li, Qing Wang, Thu T Dinh, Bethany R Powers, Sue Ritte. Stimulation of feeding by three different glucose-sensing mechanisms requires hindbrain catecholamine neurons. American journal of physiology. Regulatory, integrative and comparative physiology. vol 306. issue 4. 2014-04-10. PMID:24381177. |
stimulation of feeding by three different glucose-sensing mechanisms requires hindbrain catecholamine neurons. |
2014-04-10 |
2023-08-12 |
rat |
| Sayaka Akieda-Asai, Paul-Emile Poleni, Kazuya Hasegawa, Yukari Dat. Role of the neural pathway from hindbrain to hypothalamus in interaction of GLP1 and leptin in rats. The Journal of endocrinology. vol 220. issue 2. 2014-03-11. PMID:24222040. |
in this study, by using midbrain-transected rats, we investigated the role of the neural pathway from the hindbrain to the hypothalamus in the interaction of glp1 and leptin in reducing food intake. |
2014-03-11 |
2023-08-12 |
rat |
| Karolina P Skibick. The central GLP-1: implications for food and drug reward. Frontiers in neuroscience. vol 7. 2014-02-18. PMID:24133407. |
in accordance with the classical model of food intake control glp-1 action on feeding has been primarily ascribed to receptor populations in the hypothalamus and the hindbrain. |
2014-02-18 |
2023-08-12 |
Not clear |
| Laura E Rupprecht, Elizabeth G Mietlicki-Baase, Derek J Zimmer, Lauren E McGrath, Diana R Olivos, Matthew R Haye. Hindbrain GLP-1 receptor-mediated suppression of food intake requires a PI3K-dependent decrease in phosphorylation of membrane-bound Akt. American journal of physiology. Endocrinology and metabolism. vol 305. issue 6. 2013-11-15. PMID:23900416. |
hindbrain glp-1 receptor-mediated suppression of food intake requires a pi3k-dependent decrease in phosphorylation of membrane-bound akt. |
2013-11-15 |
2023-08-12 |
rat |
| Laura E Rupprecht, Elizabeth G Mietlicki-Baase, Derek J Zimmer, Lauren E McGrath, Diana R Olivos, Matthew R Haye. Hindbrain GLP-1 receptor-mediated suppression of food intake requires a PI3K-dependent decrease in phosphorylation of membrane-bound Akt. American journal of physiology. Endocrinology and metabolism. vol 305. issue 6. 2013-11-15. PMID:23900416. |
because camp/pka activity can promote pi3k/pip3-dependent translocation of akt to the plasma membrane, we hypothesize that hindbrain glp-1r-mediated control of feeding involves a pi3k-akt-dependent pathway. |
2013-11-15 |
2023-08-12 |
rat |
| Carlos A Campos, Hiroko Shiina, Michael Silvas, Stephen Page, Robert C Ritte. Vagal afferent NMDA receptors modulate CCK-induced reduction of food intake through synapsin I phosphorylation in adult male rats. Endocrinology. vol 154. issue 8. 2013-10-29. PMID:23715865. |
finally, hindbrain inhibition of calcineurin enhanced and prolonged synapsin i phosphorylation and potentiated reduction of food intake by cck. |
2013-10-29 |
2023-08-12 |
rat |
| Hyun-Ju Kim, Xian-Hua Zhang, Eun-Young Park, Kyung-Ho Shin, Sang-Hyun Choi, Boe-Gwun Chun, Dong-Hoon Ki. Metformin decreases meal size and number and increases c-Fos expression in the nucleus tractus solitarius of obese mice. Physiology & behavior. vol 110-111. 2013-09-24. PMID:23391573. |
however, it has not been investigated how oral metformin affects meal patterns, or whether the reduced food intake is associated with neuronal activation in the hindbrain. |
2013-09-24 |
2023-08-12 |
mouse |
| S Zhao, S E Kanoski, J Yan, H J Grill, M R Haye. Hindbrain leptin and glucagon-like-peptide-1 receptor signaling interact to suppress food intake in an additive manner. International journal of obesity (2005). vol 36. issue 12. 2013-05-15. PMID:22249232. |
hindbrain leptin and glucagon-like-peptide-1 receptor signaling interact to suppress food intake in an additive manner. |
2013-05-15 |
2023-08-12 |
Not clear |
| S Zhao, S E Kanoski, J Yan, H J Grill, M R Haye. Hindbrain leptin and glucagon-like-peptide-1 receptor signaling interact to suppress food intake in an additive manner. International journal of obesity (2005). vol 36. issue 12. 2013-05-15. PMID:22249232. |
the physiological control of feeding behavior involves modulation of the intake inhibitory effects of gastrointestinal satiation signaling via endogenous hindbrain leptin receptor (lepr) and glucagon-like-peptide-1 receptor (glp-1r) activation. |
2013-05-15 |
2023-08-12 |
Not clear |
| Jacqueline M Ho, James E Blevin. Coming full circle: contributions of central and peripheral oxytocin actions to energy balance. Endocrinology. vol 154. issue 2. 2013-03-19. PMID:23270805. |
its effects on food intake have largely been attributed to limiting meal size through interactions in key regulatory brain regions such as the hypothalamus and hindbrain. |
2013-03-19 |
2023-08-12 |
Not clear |
| Zhaofei Wu, Yuanzhong Xu, Yaming Zhu, Amy K Sutton, Rongjie Zhao, Bradford B Lowell, David P Olson, Qingchun Ton. An obligate role of oxytocin neurons in diet induced energy expenditure. PloS one. vol 7. issue 9. 2013-02-28. PMID:23028821. |
pharmacologic doses of oxytocin suppress feeding through a proposed melanocortin responsive projection from the pvh to the hindbrain. |
2013-02-28 |
2023-08-12 |
mouse |
| Michael M Scott, Mario Perello, Jen-Chieh Chuang, Ichiro Sakata, Laurent Gautron, Charlotte E Lee, Danielle Lauzon, Joel K Elmquist, Jeffrey M Zigma. Hindbrain ghrelin receptor signaling is sufficient to maintain fasting glucose. PloS one. vol 7. issue 8. 2013-02-07. PMID:22952883. |
notably, selective hindbrain ghrelin receptor expression was not sufficient to restore ghrelin-stimulated food intake. |
2013-02-07 |
2023-08-12 |
mouse |