| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| Chunmin C Lo, Wolfgang Langhans, Maria Georgievsky, Myrtha Arnold, Jody L Caldwell, Stacy Cheng, Min Liu, Stephen C Woods, Patrick Ts. Apolipoprotein AIV requires cholecystokinin and vagal nerves to suppress food intake. Endocrinology. vol 153. issue 12. 2013-01-31. PMID:23027805. |
these observations imply that peripheral apo aiv requires an intact cck system and vagal afferents to activate neurons in the hindbrain to reduce food intake. |
2013-01-31 |
2023-08-12 |
mouse |
| James W Maniscalco, Alison D Kreisler, Linda Rinama. Satiation and stress-induced hypophagia: examining the role of hindbrain neurons expressing prolactin-releasing Peptide or glucagon-like Peptide 1. Frontiers in neuroscience. vol 6. 2013-01-25. PMID:23346044. |
evidence reviewed in this article supports the view that hindbrain prrp and glp-1 neurons contribute importantly to satiation and stress-induced hypophagia by modulating the activity of caudal brainstem circuits that control food intake. |
2013-01-25 |
2023-08-12 |
mouse |
| James W Maniscalco, Alison D Kreisler, Linda Rinama. Satiation and stress-induced hypophagia: examining the role of hindbrain neurons expressing prolactin-releasing Peptide or glucagon-like Peptide 1. Frontiers in neuroscience. vol 6. 2013-01-25. PMID:23346044. |
hindbrain prrp and glp-1 neurons also engage hypothalamic and limbic forebrain networks that drive parallel behavioral and endocrine functions related to food intake and homeostatic challenge, and modulate conditioned and motivational aspects of food intake. |
2013-01-25 |
2023-08-12 |
mouse |
| Adeline Orts-Del'immagine, Nicolas Wanaverbecq, Catherine Tardivel, Vanessa Tillement, Michel Dallaporta, Jérôme Trouslar. Properties of subependymal cerebrospinal fluid contacting neurones in the dorsal vagal complex of the mouse brainstem. The Journal of physiology. vol 590. issue 16. 2013-01-08. PMID:22570378. |
using immunohistological techniques and whole-cell electrophysiological recordings in brain slices obtained from pkd2l1:egfp transgenic adult mice, we looked for and determined the functional properties of s-csf-cns in the dorsal vagal complex (dvc), a hindbrain structure controlling autonomic functions such as blood pressure, energy balance and food intake. |
2013-01-08 |
2023-08-12 |
mouse |
| Cara L Ooi, James L Kennedy, Robert D Levita. A putative model of overeating and obesity based on brain-derived neurotrophic factor: direct and indirect effects. Behavioral neuroscience. vol 126. issue 4. 2012-12-20. PMID:22687148. |
the first "direct" pathway focuses on the specific role of bdnf as a mediator of food intake control at brain areas rich in bdnf receptors, including the hypothalamus and hindbrain. |
2012-12-20 |
2023-08-12 |
Not clear |
| Marta Librán-Pérez, Sergio Polakof, Marcos A López-Patiño, Jesús M Míguez, José L Soenga. Evidence of a metabolic fatty acid-sensing system in the hypothalamus and Brockmann bodies of rainbow trout: implications in food intake regulation. American journal of physiology. Regulatory, integrative and comparative physiology. vol 302. issue 11. 2012-11-29. PMID:22496361. |
this response seems to be exclusive for the hypothalamus, since the other center controlling food intake (hindbrain) was unaffected by treatments. |
2012-11-29 |
2023-08-12 |
Not clear |
| Scott E Kanoski, Shiru Zhao, Douglas J Guarnieri, Ralph J DiLeone, Jianqun Yan, Bart C De Jonghe, Kendra K Bence, Matthew R Hayes, Harvey J Gril. Endogenous leptin receptor signaling in the medial nucleus tractus solitarius affects meal size and potentiates intestinal satiation signals. American journal of physiology. Endocrinology and metabolism. vol 303. issue 4. 2012-11-19. PMID:22693203. |
studies here employ genetic [adeno-associated virally mediated rna interference (aav-rnai)] and pharmacological methodologies to specify the neurons and the mechanisms through which hindbrain leprb signaling contributes to the control of food intake. |
2012-11-19 |
2023-08-12 |
Not clear |
| Bart C De Jonghe, Matthew R Hayes, Derek J Zimmer, Scott E Kanoski, Harvey J Grill, Kendra K Benc. Food intake reductions and increases in energetic responses by hindbrain leptin and melanotan II are enhanced in mice with POMC-specific PTP1B deficiency. American journal of physiology. Endocrinology and metabolism. vol 303. issue 5. 2012-11-02. PMID:22761160. |
food intake reductions and increases in energetic responses by hindbrain leptin and melanotan ii are enhanced in mice with pomc-specific ptp1b deficiency. |
2012-11-02 |
2023-08-12 |
mouse |
| Bart C De Jonghe, Matthew R Hayes, Derek J Zimmer, Scott E Kanoski, Harvey J Grill, Kendra K Benc. Food intake reductions and increases in energetic responses by hindbrain leptin and melanotan II are enhanced in mice with POMC-specific PTP1B deficiency. American journal of physiology. Endocrinology and metabolism. vol 303. issue 5. 2012-11-02. PMID:22761160. |
therefore, we examined the effects of hindbrain administration (4th ventricle) of leptin (1.5, 3, and 6 μg) or the melanocortin 3/4r agonist melanotan ii (0.1 and 0.2 nmol) in pomc-ptp1b(-/-) (ko) and control ptp1b(fl/fl) (wt) mice on food intake, body weight, spontaneous physical activity (spa), and core temperature (t(c)). |
2012-11-02 |
2023-08-12 |
mouse |
| Bart C De Jonghe, Matthew R Hayes, Derek J Zimmer, Scott E Kanoski, Harvey J Grill, Kendra K Benc. Food intake reductions and increases in energetic responses by hindbrain leptin and melanotan II are enhanced in mice with POMC-specific PTP1B deficiency. American journal of physiology. Endocrinology and metabolism. vol 303. issue 5. 2012-11-02. PMID:22761160. |
the results show that ko mice were hypersensitive to hindbrain leptin- and mtii-induced food intake and body weight suppression and spa compared with wt mice. |
2012-11-02 |
2023-08-12 |
mouse |
| Susan A Wright, Martha C Washington, Carlos Garcia, Ayman I Sayeg. Gastrin releasing peptide-29 requires vagal and splanchnic neurons to evoke satiation and satiety. Peptides. vol 33. issue 1. 2012-08-24. PMID:22210008. |
second, we examined if the duodenum communicates the feeding signals (ms and imi) of grp-29 (0, 0.3, 1.0, 2.1, 4.1, 10.3 and 17.2 nmol/kg) with the feeding control areas of the hindbrain by performing duodenal myotomy (myo), a procedure that severs some layers of the duodenal wall including the vagal, splanchnic and enteric neurons. |
2012-08-24 |
2023-08-12 |
rat |
| Carlos A Campos, Jason S Wright, Krzysztof Czaja, Robert C Ritte. CCK-induced reduction of food intake and hindbrain MAPK signaling are mediated by NMDA receptor activation. Endocrinology. vol 153. issue 6. 2012-08-14. PMID:22508518. |
cck-induced reduction of food intake and hindbrain mapk signaling are mediated by nmda receptor activation. |
2012-08-14 |
2023-08-12 |
rat |
| Carlos A Campos, Jason S Wright, Krzysztof Czaja, Robert C Ritte. CCK-induced reduction of food intake and hindbrain MAPK signaling are mediated by NMDA receptor activation. Endocrinology. vol 153. issue 6. 2012-08-14. PMID:22508518. |
previously, we demonstrated that hindbrain administration of n-methyl-d-aspartate (nmda)-type glutamate receptor antagonists attenuate reduction of food intake after ip cck-8 injection, indicating that these receptors play a necessary role in control of food intake by cck. |
2012-08-14 |
2023-08-12 |
rat |
| Carlos A Campos, Jason S Wright, Krzysztof Czaja, Robert C Ritte. CCK-induced reduction of food intake and hindbrain MAPK signaling are mediated by NMDA receptor activation. Endocrinology. vol 153. issue 6. 2012-08-14. PMID:22508518. |
however, the signaling pathways through which hindbrain nmda receptors contribute to cck-induced reduction of food intake have not been investigated. |
2012-08-14 |
2023-08-12 |
rat |
| Carlos A Campos, Jason S Wright, Krzysztof Czaja, Robert C Ritte. CCK-induced reduction of food intake and hindbrain MAPK signaling are mediated by NMDA receptor activation. Endocrinology. vol 153. issue 6. 2012-08-14. PMID:22508518. |
we conclude that activation of nmda receptors in the hindbrain is necessary for cck-induced erk1/2 phosphorylation in the nts and consequent reduction of food intake. |
2012-08-14 |
2023-08-12 |
rat |
| Andrea M Spaeth, Scott E Kanoski, Matthew R Hayes, Harvey J Gril. TrkB receptor signaling in the nucleus tractus solitarius mediates the food intake-suppressive effects of hindbrain BDNF and leptin. American journal of physiology. Endocrinology and metabolism. vol 302. issue 10. 2012-07-17. PMID:22374757. |
hindbrain ventricular bdnf suppressed body weight through reductions in overall food intake and meal size and by increasing core temperature. |
2012-07-17 |
2023-08-12 |
Not clear |
| Andrea M Spaeth, Scott E Kanoski, Matthew R Hayes, Harvey J Gril. TrkB receptor signaling in the nucleus tractus solitarius mediates the food intake-suppressive effects of hindbrain BDNF and leptin. American journal of physiology. Endocrinology and metabolism. vol 302. issue 10. 2012-07-17. PMID:22374757. |
mnts bdnf administration reduced food intake significantly, and this effect was blocked by preadministration of a highly selective trkb receptor antagonist {[n2-2-2-oxoazepan-3-yl amino]carbonyl phenyl benzo (b)thiophene-2-carboxamide (ana-12)}, suggesting that trkb receptor activation mediates hindbrain bdnf's effect on food intake. |
2012-07-17 |
2023-08-12 |
Not clear |
| Andrea M Spaeth, Scott E Kanoski, Matthew R Hayes, Harvey J Gril. TrkB receptor signaling in the nucleus tractus solitarius mediates the food intake-suppressive effects of hindbrain BDNF and leptin. American journal of physiology. Endocrinology and metabolism. vol 302. issue 10. 2012-07-17. PMID:22374757. |
because both bdnf and leptin interact with melanocortin signaling to reduce food intake, we also examined whether the intake inhibitory effects of hindbrain leptin involve hindbrain-specific bdnf/trkb activation. |
2012-07-17 |
2023-08-12 |
Not clear |
| Benjamin J Lamont, Yazhou Li, Edwin Kwan, Theodore J Brown, Herbert Gaisano, Daniel J Drucke. Pancreatic GLP-1 receptor activation is sufficient for incretin control of glucose metabolism in mice. The Journal of clinical investigation. vol 122. issue 1. 2012-03-06. PMID:22182839. |
systemic glp-1r activation with the glp-1r agonist exendin-4 had no effect on food intake, hindbrain c-fos expression, or gastric emptying but improved glucose tolerance and stimulated insulin secretion in pdx1-hglp1r:glp1r-/- mice. |
2012-03-06 |
2023-08-12 |
mouse |
| Alison V Roland, Suzanne M Moente. Regulation of gonadotropin-releasing hormone neurons by glucose. Trends in endocrinology and metabolism: TEM. vol 22. issue 11. 2012-02-23. PMID:21855365. |
existing evidence suggests that neural pathways originating in the hindbrain, as well as in the hypothalamic feeding nuclei, transmit information concerning glucose availability to gnrh neurons. |
2012-02-23 |
2023-08-12 |
Not clear |