| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| Karolina P Skibicka, Amber L Alhadeff, Harvey J Gril. Hindbrain cocaine- and amphetamine-regulated transcript induces hypothermia mediated by GLP-1 receptors. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 29. issue 21. 2009-06-23. PMID:19474324. |
hindbrain cart application reduced food intake and body weight and increased blood glucose levels; no change in hr or activity was observed. |
2009-06-23 |
2023-08-12 |
rat |
| Matthew R Hayes, Lauren Bradley, Harvey J Gril. Endogenous hindbrain glucagon-like peptide-1 receptor activation contributes to the control of food intake by mediating gastric satiation signaling. Endocrinology. vol 150. issue 6. 2009-06-18. PMID:19264875. |
endogenous hindbrain glucagon-like peptide-1 receptor activation contributes to the control of food intake by mediating gastric satiation signaling. |
2009-06-18 |
2023-08-12 |
rat |
| Matthew R Hayes, Karolina P Skibicka, Kendra K Bence, Harvey J Gril. Dorsal hindbrain 5'-adenosine monophosphate-activated protein kinase as an intracellular mediator of energy balance. Endocrinology. vol 150. issue 5. 2009-06-08. PMID:19116341. |
given the role of leptin signaling in food intake inhibition within the medial nts, we also examined whether stimulation of hindbrain ampk by 4th i.c.v. |
2009-06-08 |
2023-08-12 |
rat |
| Matthew R Hayes, Karolina P Skibicka, Kendra K Bence, Harvey J Gril. Dorsal hindbrain 5'-adenosine monophosphate-activated protein kinase as an intracellular mediator of energy balance. Endocrinology. vol 150. issue 5. 2009-06-08. PMID:19116341. |
we conclude that 1) hindbrain ampk activity contributes to energy balance control through regulation of food intake and energy expenditure, 2) leptin's intake-reducing effects in the nts are mediated by ampk, and 3) central nervous system ampk controls whole-body homeostasis at anatomically distributed sites across the neuraxis. |
2009-06-08 |
2023-08-12 |
rat |
| Douglas B Guard, Timothy D Swartz, Robert C Ritter, Gilbert A Burns, Mihai Covas. Blockade of hindbrain NMDA receptors containing NR2 subunits increases sucrose intake. American journal of physiology. Regulatory, integrative and comparative physiology. vol 296. issue 4. 2009-06-05. PMID:19193935. |
these results show that hindbrain injection of competitive nmda antagonists with selectivity or preference for the nmda receptor nr2b or nr2c subunits increases food intake. |
2009-06-05 |
2023-08-12 |
Not clear |
| D B Guard, T D Swartz, R C Ritter, G A Burns, M Covas. NMDA NR2 receptors participate in CCK-induced reduction of food intake and hindbrain neuronal activation. Brain research. vol 1266. 2009-05-26. PMID:19232331. |
nmda nr2 receptors participate in cck-induced reduction of food intake and hindbrain neuronal activation. |
2009-05-26 |
2023-08-12 |
Not clear |
| D B Guard, T D Swartz, R C Ritter, G A Burns, M Covas. NMDA NR2 receptors participate in CCK-induced reduction of food intake and hindbrain neuronal activation. Brain research. vol 1266. 2009-05-26. PMID:19232331. |
previous work has shown that blockade of nmdar by non-competitive (mk-801) and competitive (ap5) antagonists increase food intake by acting in the dorsal hindbrain. |
2009-05-26 |
2023-08-12 |
Not clear |
| D B Guard, T D Swartz, R C Ritter, G A Burns, M Covas. NMDA NR2 receptors participate in CCK-induced reduction of food intake and hindbrain neuronal activation. Brain research. vol 1266. 2009-05-26. PMID:19232331. |
competitive nr2b antagonists potently increase feeding when injected into the hindbrain. |
2009-05-26 |
2023-08-12 |
Not clear |
| D B Guard, T D Swartz, R C Ritter, G A Burns, M Covas. NMDA NR2 receptors participate in CCK-induced reduction of food intake and hindbrain neuronal activation. Brain research. vol 1266. 2009-05-26. PMID:19232331. |
these results confirm that antagonism of hindbrain nmda receptors increases food intake. |
2009-05-26 |
2023-08-12 |
Not clear |
| D B Guard, T D Swartz, R C Ritter, G A Burns, M Covas. NMDA NR2 receptors participate in CCK-induced reduction of food intake and hindbrain neuronal activation. Brain research. vol 1266. 2009-05-26. PMID:19232331. |
in addition our results suggest that nmda receptors outside the hindbrain, perhaps in the periphery, participate in vagally mediated, cck-induced reduction of food intake and nts neuron activation. |
2009-05-26 |
2023-08-12 |
Not clear |
| Karolina P Skibicka, Harvey J Gril. Hindbrain leptin stimulation induces anorexia and hyperthermia mediated by hindbrain melanocortin receptors. Endocrinology. vol 150. issue 4. 2009-04-14. PMID:19056818. |
results show that hindbrain leptin receptor stimulation produced long-lasting (>6 h) increases in core temperature and heart rate and also decreased food intake and body weight. |
2009-04-14 |
2023-08-12 |
rat |
| Ai-Jun Li, Qing Wang, Thu T Dinh, Sue Ritte. Simultaneous silencing of Npy and Dbh expression in hindbrain A1/C1 catecholamine cells suppresses glucoprivic feeding. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 29. issue 1. 2009-04-08. PMID:19129404. |
simultaneous silencing of npy and dbh expression in hindbrain a1/c1 catecholamine cells suppresses glucoprivic feeding. |
2009-04-08 |
2023-08-12 |
Not clear |
| Ai-Jun Li, Qing Wang, Thu T Dinh, Sue Ritte. Simultaneous silencing of Npy and Dbh expression in hindbrain A1/C1 catecholamine cells suppresses glucoprivic feeding. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 29. issue 1. 2009-04-08. PMID:19129404. |
previous data have strongly implicated hindbrain catecholamine/neuropeptide y (npy) coexpressing neurons as key mediators of the glucoprivic feeding response. |
2009-04-08 |
2023-08-12 |
Not clear |
| John-Paul Baird, Angela Choe, Jasmine L Loveland, Janine Beck, Carrie E Mahoney, Julia S Lord, Lindsay A Grig. Orexin-A hyperphagia: hindbrain participation in consummatory feeding responses. Endocrinology. vol 150. issue 3. 2009-03-24. PMID:19008313. |
orexin-a hyperphagia: hindbrain participation in consummatory feeding responses. |
2009-03-24 |
2023-08-12 |
rat |
| J E Blevins, J Overduin, J M Fuller, D E Cummings, K Matsumoto, D H Moralej. Normal feeding and body weight in Fischer 344 rats lacking the cholecystokinin-1 receptor gene. Brain research. vol 1255. 2009-03-18. PMID:19111529. |
a large body of evidence has demonstrated that one mechanism by which cholecystokinin (cck) inhibits food intake through activation of cck1 receptors (cck1r) on vagal afferent neurons that innervate the gastrointestinal tract and project to the hindbrain. |
2009-03-18 |
2023-08-12 |
rat |
| J E Blevins, J Overduin, J M Fuller, D E Cummings, K Matsumoto, D H Moralej. Normal feeding and body weight in Fischer 344 rats lacking the cholecystokinin-1 receptor gene. Brain research. vol 1255. 2009-03-18. PMID:19111529. |
these results confirm that in rats with a f344 background, cck-1r mediates cck-8-induced inhibition of food intake and fos activation in the hindbrain and demonstrate that selective genetic ablation of cck1r is not associated with altered meal patterns, hyperphagia, or excessive weight gain on a palatable diet. |
2009-03-18 |
2023-08-12 |
rat |
| Shampa De, Jack E Turma. Krox-20 gene expression: influencing hindbrain-craniofacial developmental interactions. Archives of histology and cytology. vol 68. issue 4. 2009-01-29. PMID:16477142. |
the krox-20 null mutation results in hindbrain anomalies that result in neonatal death due to respiratory and feeding deficits. |
2009-01-29 |
2023-08-12 |
Not clear |
| Dawna Salter-Venzon, Alan G Watt. The role of hypothalamic ingestive behavior controllers in generating dehydration anorexia: a Fos mapping study. American journal of physiology. Regulatory, integrative and comparative physiology. vol 295. issue 4. 2008-12-04. PMID:18667712. |
we hypothesize that this inhibition is directed at those neurons in the paraventricular nucleus and lateral hypothalamic area that constitute the hypothalamic "behavior controller" for feeding rather than their afferent inputs from the arcuate nucleus or hindbrain that convey critical feeding-related sensory information. |
2008-12-04 |
2023-08-12 |
rat |
| Dawna Salter-Venzon, Alan G Watt. The role of hypothalamic ingestive behavior controllers in generating dehydration anorexia: a Fos mapping study. American journal of physiology. Regulatory, integrative and comparative physiology. vol 295. issue 4. 2008-12-04. PMID:18667712. |
in the arcuate nucleus and those regions of the hindbrain that provide afferent inputs critical for the feeding response to 2dg, the fos response to 2dg was unaffected by dehydration. |
2008-12-04 |
2023-08-12 |
rat |
| Ai-Jun Li, Thu T Dinh, Sue Ritte. Hyperphagia and obesity produced by arcuate injection of NPY-saporin do not require upregulation of lateral hypothalamic orexigenic peptide genes. Peptides. vol 29. issue 10. 2008-12-03. PMID:18577407. |
the source of the seemingly unregulated stimulatory drive for feeding in these animals has not been identified, but may be associated with hindbrain or endocrine mechanisms. |
2008-12-03 |
2023-08-12 |
rat |