All Relations between feeding and rhombencephalon

Publication Sentence Publish Date Extraction Date Species
Sergio Polakof, Jesús M Míguez, José L Soenga. Changes in food intake and glucosensing function of hypothalamus and hindbrain in rainbow trout subjected to hyperglycemic or hypoglycemic conditions. Journal of comparative physiology. A, Neuroethology, sensory, neural, and behavioral physiology. vol 194. issue 9. 2008-12-01. PMID:18663455. changes in food intake and glucosensing function of hypothalamus and hindbrain in rainbow trout subjected to hyperglycemic or hypoglycemic conditions. 2008-12-01 2023-08-12 cattle
Shannon J Raboin, Joseph R Reeve, Marvis S Cooper, Gary M Green, Ayman I Sayeg. Activation of submucosal but not myenteric plexus of the gastrointestinal tract accompanies reduction of food intake by camostat. Regulatory peptides. vol 150. issue 1-3. 2008-11-14. PMID:18620003. it has been shown in the rat that endogenous cholecystokinin (cck), released in response to the non-nutrient trypsin inhibitor camostat, reduces food intake at meals and increases fos-like immunoreactivity (fos-li; a marker for neuronal activation) in the dorsal vagal complex (dvc) of the hindbrain but not the myenteric plexus of the duodenum and jejunum. 2008-11-14 2023-08-12 rat
Lucy F Faulconbridge, Harvey J Grill, Joel M Kaplan, Derek Daniel. Caudal brainstem delivery of ghrelin induces fos expression in the nucleus of the solitary tract, but not in the arcuate or paraventricular nuclei of the hypothalamus. Brain research. vol 1218. 2008-10-08. PMID:18514175. ghrelin increases food intake when injected into either the forebrain or hindbrain ventricles. 2008-10-08 2023-08-12 rat
Sergio Polakof, Jesús M Míguez, José L Soenga. Dietary carbohydrates induce changes in glucosensing capacity and food intake of rainbow trout. American journal of physiology. Regulatory, integrative and comparative physiology. vol 295. issue 2. 2008-09-09. PMID:18525014. we hypothesize that variations in dietary carbohydrate levels produce changes in glucosensor parameters in previously characterized glucosensing areas (hypothalamus and hindbrain) related with the regulation of food intake of a carnivorous fish species like rainbow trout. 2008-09-09 2023-08-12 Not clear
Sergio Polakof, Jesús M Míguez, José L Soenga. Dietary carbohydrates induce changes in glucosensing capacity and food intake of rainbow trout. American journal of physiology. Regulatory, integrative and comparative physiology. vol 295. issue 2. 2008-09-09. PMID:18525014. changes in food intake due to dietary carbohydrates were accompanied in hypothalamus and hindbrain of fish fed with hc diet by changes in parameters involved in glucosensing, such as increased glucose, glucose 6-phosphate, and glycogen levels and increased glucokinase (gk), glycogen synthase, and pyruvate kinase activities as well as increased gk and glut2 expression. 2008-09-09 2023-08-12 Not clear
Joanna R Miragaya, Ruth B S Harri. Antagonism of corticotrophin-releasing factor receptors in the fourth ventricle modifies responses to mild but not restraint stress. American journal of physiology. Regulatory, integrative and comparative physiology. vol 295. issue 2. 2008-09-09. PMID:18550868. these data suggest that inhibition of food intake and weight loss induced by rrs or by ms involve different pathways, with hindbrain crfr mediating the effect of ms on body weight and food intake. 2008-09-09 2023-08-12 rat
Matthew R Hayes, Karolina P Skibicka, Harvey J Gril. Caudal brainstem processing is sufficient for behavioral, sympathetic, and parasympathetic responses driven by peripheral and hindbrain glucagon-like-peptide-1 receptor stimulation. Endocrinology. vol 149. issue 8. 2008-08-26. PMID:18420740. hindbrain ventricular ex-4 (0.3 microg) also suppressed food intake in cd rats (4.7+/-0.6 ml ingested) and controls (11.0+/-2.9 ml ingested), compared with vehicle intakes (9.3+/-2.1 and 19.3+/-4.3 ml ingested, respectively). 2008-08-26 2023-08-12 rat
Sumpun Thammacharoen, Thomas A Lutz, Nori Geary, Lori Asaria. Hindbrain administration of estradiol inhibits feeding and activates estrogen receptor-alpha-expressing cells in the nucleus tractus solitarius of ovariectomized rats. Endocrinology. vol 149. issue 4. 2008-05-06. PMID:18096668. to test whether direct administration of e2 to this area of the hindbrain is sufficient to inhibit food intake, we first implanted 0.2 microg estradiol benzoate (eb) in cholesterol or cholesterol alone either sc or onto the surface of the hindbrain over the cnts. 2008-05-06 2023-08-12 rat
Sumpun Thammacharoen, Thomas A Lutz, Nori Geary, Lori Asaria. Hindbrain administration of estradiol inhibits feeding and activates estrogen receptor-alpha-expressing cells in the nucleus tractus solitarius of ovariectomized rats. Endocrinology. vol 149. issue 4. 2008-05-06. PMID:18096668. food intake was significantly reduced after hindbrain eb implants but not after sc eb implants. 2008-05-06 2023-08-12 rat
J E Blevins, P K Chelikani, A C Haver, R D Reidelberge. PYY(3-36) induces Fos in the arcuate nucleus and in both catecholaminergic and non-catecholaminergic neurons in the nucleus tractus solitarius of rats. Peptides. vol 29. issue 1. 2008-04-07. PMID:18082288. here we assessed whether pyy(3-36) inhibits feeding by activating neurons in forebrain and hindbrain sites containing y2 receptors and linked to control of food intake, or in hindbrain sites immediately downstream of vagal afferent neurons. 2008-04-07 2023-08-12 rat
Diana L Williams, Michael W Schwartz, L Scot Bastian, James E Blevins, Denis G Baski. Immunocytochemistry and laser capture microdissection for real-time quantitative PCR identify hindbrain neurons activated by interaction between leptin and cholecystokinin. The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society. vol 56. issue 3. 2008-04-02. PMID:18040081. current evidence suggests that leptin reduces food intake in part by enhancing the hindbrain neuronal response to meal-related gastrointestinal signals, including cholecystokinin (cck), but the phenotypes of the relevant cells are not known. 2008-04-02 2023-08-12 rat
Ruth McPherso. Genetic contributors to obesity. The Canadian journal of cardiology. vol 23 Suppl A. 2008-01-10. PMID:17668084. these genes can be considered in four broad categories: regulation of food intake by molecular signalling in the hypothalamus and hindbrain by signals originating in adipose tissue, gut and other organs; regulation of adipocyte differentiation and fat storage; regulation of spontaneous exercise activity; and effect on basal and postprandial thermogenesis. 2008-01-10 2023-08-12 mouse
Gang Li, Yi Zhang, Enda Rodrigues, Donghang Zheng, Michael Matheny, Kit-Yan Cheng, Philip J Scarpac. Melanocortin activation of nucleus of the solitary tract avoids anorectic tachyphylaxis and induces prolonged weight loss. American journal of physiology. Endocrinology and metabolism. vol 293. issue 1. 2007-09-13. PMID:17389713. recombinant adeno-associated viral vector encoding pomc gene was delivered to the nucleus of solitary tract (nts) in the hindbrain, and food intake, body weight, glucose and fat metabolism, brown adipose tissue thermogenesis, and mrna levels of neuropeptides and melanocortin receptors were assessed. 2007-09-13 2023-08-12 rat
Kelli Taylor, Erin Lester, Bryan Hudson, Sue Ritte. Hypothalamic and hindbrain NPY, AGRP and NE increase consummatory feeding responses. Physiology & behavior. vol 90. issue 5. 2007-06-05. PMID:17289093. hypothalamic and hindbrain npy, agrp and ne increase consummatory feeding responses. 2007-06-05 2023-08-12 rat
Bing Li, Kichoon Lee, Roy J Marti. Overexpression of glucose transporter 2 in GT1-7 cells inhibits AMP-activated protein kinase and agouti-related peptide expression. Brain research. vol 1118. issue 1. 2007-01-23. PMID:16978589. glut2 has been found widely expressed in the brain and glut2 in the hypothalamus and hindbrain has been suggested to be involved in the central glucose sensing and regulation of glucose homeostasis and food intake. 2007-01-23 2023-08-12 Not clear
Sue Ritter, Thu T Dinh, Ai-Jun L. Hindbrain catecholamine neurons control multiple glucoregulatory responses. Physiology & behavior. vol 89. issue 4. 2007-01-04. PMID:16887153. selective destruction of hindbrain catecholamine neurons using the retrogradely transported immunotoxin, anti-dopamine beta-hydroxylase conjugated to saporin (dsap), revealed that spinally-projecting epinephrine (e) or norepinephrine (ne) neurons are required for the adrenal medullary response to glucoprivation, while e/ne neurons with hypothalamic projections are required for feeding, corticosterone and reproductive responses. 2007-01-04 2023-08-12 Not clear
Yukari Date, Takuya Shimbara, Shuichi Koda, Koji Toshinai, Takanori Ida, Noboru Murakami, Mikiya Miyazato, Koichi Kokame, Yuta Ishizuka, Yasushi Ishida, Haruaki Kageyama, Seiji Shioda, Kenji Kangawa, Masamitsu Nakazat. Peripheral ghrelin transmits orexigenic signals through the noradrenergic pathway from the hindbrain to the hypothalamus. Cell metabolism. vol 4. issue 4. 2006-12-21. PMID:17011505. in addition, bilateral midbrain transections rostral to the nts, or toxin-induced loss of neurons in the hindbrain that express dopamine beta hydroxylase (an na synthetic enzyme), abolished ghrelin-induced feeding. 2006-12-21 2023-08-12 Not clear
Harvey J Gril. Distributed neural control of energy balance: contributions from hindbrain and hypothalamus. Obesity (Silver Spring, Md.). vol 14 Suppl 5. 2006-12-20. PMID:17021370. experimental evidence is discussed that supports the following specific conclusions: hindbrain neurons integrate oral and gastrointestinal signals and issue commands to local motor circuits that control meal size; leptin's effect on food intake may be mediated, in part, by a direct action on the hindbrain neurons that respond to gastric distention; deprivation signals, such as the fall in leptin level, affect gene expression outside of the hypothalamus with reductions in proglucagon and proopiomelanocortin message seen in nucleus of the solitary tract-rich tissue; and that hindbrain neurons contribute to the control of energy expenditure seen with food deprivation and increases in expenditure after cold exposure or starvation. 2006-12-20 2023-08-12 Not clear
Aaron W Pardini, Hong T Nguyen, Dianne P Figlewicz, Denis G Baskin, Diana L Williams, Francis Kim, Michael W Schwart. Distribution of insulin receptor substrate-2 in brain areas involved in energy homeostasis. Brain research. vol 1112. issue 1. 2006-11-28. PMID:16925984. in summary, neurons containing irs-2 immunoreactivity were identified in forebrain, midbrain and hindbrain areas and in cell types that are crucial for the control of food intake and autonomic function. 2006-11-28 2023-08-12 rat
Mercedes Gil-Campos, Concepción María Aguilera, Ramón Cañete, Angel Gi. Ghrelin: a hormone regulating food intake and energy homeostasis. The British journal of nutrition. vol 96. issue 2. 2006-10-16. PMID:16923214. ghrelin stimulates the synthesis of neuropeptide y (npy) and agouti-related protein (agrp) in the arcuate nucleus neurons of the hypothalamus and hindbrain, which in turn enhance food intake. 2006-10-16 2023-08-12 Not clear