| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| David E Cumming. Ghrelin and the short- and long-term regulation of appetite and body weight. Physiology & behavior. vol 89. issue 1. 2006-09-29. PMID:16859720. |
ghrelin crosses the blood-brain barrier and stimulates food intake by acting on several classical body-weight regulatory centers, including the hypothalamus, hindbrain, and mesolimbic reward system. |
2006-09-29 |
2023-08-12 |
Not clear |
| Kimberly P Kinzig, Karen A Scott, Jayson Hyun, Sheng Bi, Timothy H Mora. Lateral ventricular ghrelin and fourth ventricular ghrelin induce similar increases in food intake and patterns of hypothalamic gene expression. American journal of physiology. Regulatory, integrative and comparative physiology. vol 290. issue 6. 2006-07-18. PMID:16424082. |
ghrelin receptors are widely distributed in the brain, and hindbrain ghrelin administration has been shown to potently stimulate feeding, suggesting that there may be other sites for ghrelin action. |
2006-07-18 |
2023-08-12 |
Not clear |
| Chun-Yi Hung, M Covasa, R C Ritter, G A Burn. Hindbrain administration of NMDA receptor antagonist AP-5 increases food intake in the rat. American journal of physiology. Regulatory, integrative and comparative physiology. vol 290. issue 3. 2006-04-03. PMID:16269572. |
hindbrain administration of nmda receptor antagonist ap-5 increases food intake in the rat. |
2006-04-03 |
2023-08-12 |
rat |
| Chun-Yi Hung, M Covasa, R C Ritter, G A Burn. Hindbrain administration of NMDA receptor antagonist AP-5 increases food intake in the rat. American journal of physiology. Regulatory, integrative and comparative physiology. vol 290. issue 3. 2006-04-03. PMID:16269572. |
hindbrain administration of mk-801, a noncompetitive n-methyl-d-aspartate (nmda) channel blocker, increases meal size, suggesting nmda receptors in this location participate in control of food intake. |
2006-04-03 |
2023-08-12 |
rat |
| Chun-Yi Hung, M Covasa, R C Ritter, G A Burn. Hindbrain administration of NMDA receptor antagonist AP-5 increases food intake in the rat. American journal of physiology. Regulatory, integrative and comparative physiology. vol 290. issue 3. 2006-04-03. PMID:16269572. |
therefore, to further assess hindbrain nmda receptor participation in food intake control, we measured deprivation-induced intakes of 15% sucrose solution or rat chow after intraperitoneal injection of either saline vehicle or d(-)-2-amino-5-phosphonopentanoic acid (ap5), a competitive nmda receptor antagonist, to the fourth ventricular, or nucleus of the solitary tract (nts). |
2006-04-03 |
2023-08-12 |
rat |
| Chun-Yi Hung, M Covasa, R C Ritter, G A Burn. Hindbrain administration of NMDA receptor antagonist AP-5 increases food intake in the rat. American journal of physiology. Regulatory, integrative and comparative physiology. vol 290. issue 3. 2006-04-03. PMID:16269572. |
these results support the hypothesis that hindbrain nmda receptors participate in control of food intake and suggest that this participation also may contribute to control of body weight over a 24-h period. |
2006-04-03 |
2023-08-12 |
rat |
| Bruno Bariohay, Bruno Lebrun, Emmanuel Moyse, André Jea. Brain-derived neurotrophic factor plays a role as an anorexigenic factor in the dorsal vagal complex. Endocrinology. vol 146. issue 12. 2006-01-05. PMID:16166223. |
in this study, we addressed whether bdnf can modulate food intake in the hindbrain autonomic integrator of food intake regulation, i.e. |
2006-01-05 |
2023-08-12 |
rat |
| E H E M van de Wall, P Duffy, R C Ritte. CCK enhances response to gastric distension by acting on capsaicin-insensitive vagal afferents. American journal of physiology. Regulatory, integrative and comparative physiology. vol 289. issue 3. 2005-09-20. PMID:15905220. |
capsaicin treatment destroys vagal afferent c fibers and markedly attenuates reduction of food intake and induction of hindbrain fos expression by cck. |
2005-09-20 |
2023-08-12 |
rat |
| Catherine M Kotz, Mary A Mullett, ChuanFeng Wan. Diminished feeding responsiveness to orexin A (hypocretin 1) in aged rats is accompanied by decreased neuronal activation. American journal of physiology. Regulatory, integrative and comparative physiology. vol 289. issue 2. 2005-09-01. PMID:15879054. |
food intake was measured at 1, 2, and 4 h. c-fos-ir in hypothalamic, limbic, and hindbrain regions was measured in two additional sets of rlha-orexin a injected rats. |
2005-09-01 |
2023-08-12 |
rat |
| V Macchi, A Porzionato, D Guidolin, A Parenti, R De Car. Morphogenesis of the posterior inferior cerebellar artery with three-dimensional reconstruction of the late embryonic vertebrobasilar system. Surgical and radiologic anatomy : SRA. vol 27. issue 1. 2005-06-09. PMID:15645157. |
based on the evolution of the primitive hindbrain plexus, we suggest that in synchrony with the progressive descent of the cerebellum the branches of the myelencephalic plexus succeed with a rostrocaudal progression in feeding the cerebellum and the morphogenesis of the pica results from the selection of portions of this plexus. |
2005-06-09 |
2023-08-12 |
human |
| Toshiro Kishi, Carl J Aschkenasi, Brian J Choi, Marisol E Lopez, Charlotte E Lee, Hongyan Liu, Anthony N Hollenberg, Jeffrey M Friedman, Joel K Elmquis. Neuropeptide Y Y1 receptor mRNA in rodent brain: distribution and colocalization with melanocortin-4 receptor. The Journal of comparative neurology. vol 482. issue 3. 2005-05-12. PMID:15690487. |
in addition, y1-r hybridization was evident in several extrahypothalamic forebrain and hindbrain sites involved in feeding and/or autonomic regulation in the rat. |
2005-05-12 |
2023-08-12 |
mouse |
| Gregory J Morton, James E Blevins, Diana L Williams, Kevin D Niswender, Richard W Gelling, Christopher J Rhodes, Denis G Baskin, Michael W Schwart. Leptin action in the forebrain regulates the hindbrain response to satiety signals. The Journal of clinical investigation. vol 115. issue 3. 2005-05-09. PMID:15711637. |
these findings demonstrate that forebrain signaling by leptin, a long-term regulator of body adiposity, limits food intake on a meal-to-meal basis by regulating the hindbrain response to short-acting satiety signals. |
2005-05-09 |
2023-08-12 |
rat |
| Ester Fride, Tatyana Bregman, Tim C Kirkha. Endocannabinoids and food intake: newborn suckling and appetite regulation in adulthood. Experimental biology and medicine (Maywood, N.J.). vol 230. issue 4. 2005-04-28. PMID:15792943. |
the endocannabinoids appear to regulate energy balance and food intake at four functional levels within the brain and periphery: (i) limbic system (for hedonic evaluation of foods), (ii) hypothalamus and hindbrain (integrative functions), (iii) intestinal system, and (iv) adipose tissue. |
2005-04-28 |
2023-08-12 |
Not clear |
| M Covasa, Chun-Yi Hung, R C Ritter, G A Burn. Intracerebroventricular administration of MK-801 increases food intake through mechanisms independent of gastric emptying. American journal of physiology. Regulatory, integrative and comparative physiology. vol 287. issue 6. 2005-04-12. PMID:15358605. |
these data confirm previous results that systemic as well as hindbrain injection of mk-801 increases food intake. |
2005-04-12 |
2023-08-12 |
Not clear |
| Gary J Schwart. Biology of eating behavior in obesity. Obesity research. vol 12 Suppl 2. 2005-03-18. PMID:15601957. |
in the central nervous system, adiposity hormones and their downstream mediators have been shown to work at both hindbrain and forebrain sites to affect food intake and metabolism. |
2005-03-18 |
2023-08-12 |
Not clear |
| Mika Kinoshita, Helen I'Anson, Hiroko Tsukamura, Kei-ichiro Maed. Fourth ventricular alloxan injection suppresses pulsatile luteinizing hormone release in female rats. The Journal of reproduction and development. vol 50. issue 3. 2005-01-06. PMID:15226592. |
previous studies have suggested the presence of a glucose-sensing mechanism in the hindbrain that appears to regulate reproductive function as well as feeding behavior. |
2005-01-06 |
2023-08-12 |
rat |
| Cheryl C Miller, Thomas F Murray, Kimberly G Freeman, Gaylen L Edward. Cannabinoid agonist, CP 55,940, facilitates intake of palatable foods when injected into the hindbrain. Physiology & behavior. vol 80. issue 5. 2004-07-13. PMID:14984793. |
it has been previously shown that intracerebroventricular injection of delta-9-tetrahydrocannabinol (delta(9)-thc) causes increased consumption of palatable foods in rats, and we postulated the involvement of the hindbrain in this cannabinoid-induced food intake. |
2004-07-13 |
2023-08-12 |
rat |
| Cheryl C Miller, Thomas F Murray, Kimberly G Freeman, Gaylen L Edward. Cannabinoid agonist, CP 55,940, facilitates intake of palatable foods when injected into the hindbrain. Physiology & behavior. vol 80. issue 5. 2004-07-13. PMID:14984793. |
these results indicate that cp 55,940 may act in the hindbrain to influence feeding behavior in rats. |
2004-07-13 |
2023-08-12 |
rat |
| Nicole M Sanders, Ambrose A Dunn-Meynell, Barry E Levi. Third ventricular alloxan reversibly impairs glucose counterregulatory responses. Diabetes. vol 53. issue 5. 2004-06-24. PMID:15111491. |
in addition, their hyperglycemic response to hindbrain glucoprivation induced with 5-thio-glucose was impaired, whereas fasting blood glucose levels and food intake after an overnight fast were elevated. |
2004-06-24 |
2023-08-12 |
rat |
| Ai-Jun Li, Sue Ritte. Glucoprivation increases expression of neuropeptide Y mRNA in hindbrain neurons that innervate the hypothalamus. The European journal of neuroscience. vol 19. issue 8. 2004-06-18. PMID:15090041. |
the responsiveness of hindbrain npy neurons to glucose deficit suggests that these neurons participate in glucoprivic feeding or other glucoregulatory responses. |
2004-06-18 |
2023-08-12 |
rat |