| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| D A West, N H James, S C Cosulich, P R Holden, R Brindle, M Rolfe, R A Robert. Role for tumor necrosis factor alpha receptor 1 and interleukin-1 receptor in the suppression of mouse hepatocyte apoptosis by the peroxisome proliferator nafenopin. Hepatology (Baltimore, Md.). vol 30. issue 6. 1999-12-16. PMID:10573520. |
role for tumor necrosis factor alpha receptor 1 and interleukin-1 receptor in the suppression of mouse hepatocyte apoptosis by the peroxisome proliferator nafenopin. |
1999-12-16 |
2023-08-12 |
mouse |
| D A West, N H James, S C Cosulich, P R Holden, R Brindle, M Rolfe, R A Robert. Role for tumor necrosis factor alpha receptor 1 and interleukin-1 receptor in the suppression of mouse hepatocyte apoptosis by the peroxisome proliferator nafenopin. Hepatology (Baltimore, Md.). vol 30. issue 6. 1999-12-16. PMID:10573520. |
in vitro, pps induce rat and mouse hepatocyte dna synthesis and suppress apoptosis, a response mimicked by exogenous tumor necrosis factor alpha (tnfalpha). |
1999-12-16 |
2023-08-12 |
mouse |
| I Matsumoto, S Okada, K Kuroda, I Iwamoto, Y Saito, T Tokuhisa, K Nishioka, T Sumid. Single cell analysis of T cells infiltrating labial salivary glands from patients with Sjögren's syndrome. International journal of molecular medicine. vol 4. issue 5. 1999-12-15. PMID:10534575. |
to analyze the function of t cells infiltrating the labial salivary glands, we analyzed t cell receptor (tcr) beta and alpha chains, the expression of various cytokine mrnas, and apoptosis associated genes in predominant tcr bv2+ t cells in the labial salivary glands of patients with ss at the single cell level. |
1999-12-15 |
2023-08-12 |
Not clear |
| J Y Matroule, A C Hellin, P Morlière, A S Fabiano, R Santus, M P Merville, J Piett. Role of nuclear factor-kappa B in colon cancer cell apoptosis mediated by aminopyropheophorbide photosensitization. Photochemistry and photobiology. vol 70. issue 4. 1999-12-06. PMID:10546550. |
the nf-kappa b turned out to be involved in an antiapoptotic response to app-mediated photosensitization because the hct-116 cell line expressing the dominant negative mutant of inhibitor-kappa b alpha was more sensitive to apoptosis as measured by dna fragmentation and caspase activation. |
1999-12-06 |
2023-08-12 |
Not clear |
| A Pugliese, C Cantamessa, A Saini, A Piragino, L Gennero, C Martini, D Torr. Effects of the exogenous Nef protein on HIV-1 target cells. Cell biochemistry and function. vol 17. issue 3. 1999-12-02. PMID:10451539. |
moreover, we demonstrated a dose-dependent activity of nef on inducing apoptosis in h9 uninfected cells and no effects of this protein on modulation of inf alpha and gamma production in peripheral blood mononucleated cells of health donors. |
1999-12-02 |
2023-08-12 |
Not clear |
| G Z Feuerstei. Apoptosis in cardiac diseases--new opportunities for novel therapeutics for heart diseases. Cardiovascular drugs and therapy. vol 13. issue 4. 1999-11-30. PMID:10516863. |
among the most potent stimuli that elicit cardiomyocyte apoptosis are: oxygen radicals (including no), cytokines, (fas/tnf alpha family of cytokines) and growth factors/energy deprivation. |
1999-11-30 |
2023-08-12 |
human |
| G Z Feuerstei. Apoptosis in cardiac diseases--new opportunities for novel therapeutics for heart diseases. Cardiovascular drugs and therapy. vol 13. issue 4. 1999-11-30. PMID:10516863. |
several complex signal transduction pathways have been implicated in execution of cardiomyocyte apoptosis, including: fas/tnf alpha receptors signaling, stress or mitogen activated protein kinases (sapk/mapk), sphingolipids metabolites (ceramide), g-protein coupled receptor (gpcr) signaling (g alpha i, g alpha q) and nf kappa b activation. |
1999-11-30 |
2023-08-12 |
human |
| D Li, B Yang, J L Meht. Tumor necrosis factor-alpha enhances hypoxia-reoxygenation-mediated apoptosis in cultured human coronary artery endothelial cells: critical role of protein kinase C. Cardiovascular research. vol 42. issue 3. 1999-11-24. PMID:10533621. |
tumor necrosis factor-alpha enhances hypoxia-reoxygenation-mediated apoptosis in cultured human coronary artery endothelial cells: critical role of protein kinase c. ischemia and tumor necrosis factor-alpha (tnf alpha) released during ischemia both cause apoptosis and necrosis of myocardial tissues. |
1999-11-24 |
2023-08-12 |
human |
| D Li, B Yang, J L Meht. Tumor necrosis factor-alpha enhances hypoxia-reoxygenation-mediated apoptosis in cultured human coronary artery endothelial cells: critical role of protein kinase C. Cardiovascular research. vol 42. issue 3. 1999-11-24. PMID:10533621. |
since endothelium may be critically important in determination of cardiac function, we examined the interaction between tnf alpha and hypoxia-reoxygenation with regard to induction of apoptosis and underlying signaling pathway in cultured human coronary artery endothelial cells (hcaecs). |
1999-11-24 |
2023-08-12 |
human |
| H McGrat. Ultraviolet A1 (340-400 nm) irradiation and systemic lupus erythematosus. The journal of investigative dermatology. Symposium proceedings. vol 4. issue 1. 1999-11-17. PMID:10537014. |
the fourth human study showed that the therapeutic action of low-dose uva1 action persisted or progressed long term, a period averaging 3.4 y. uva1 effects on dna repair, cell-mediated immunosuppression, tumor necrosis factor alpha release, and apoptosis contrast markedly with those of ultraviolet b (uvb, 280-320 nm) radiation and afford a possible basis for the salutary action of this modality of treatment. |
1999-11-17 |
2023-08-12 |
mouse |
| H Mühl, M Nold, J H Chang, S Frank, W Eberhardt, J Pfeilschifte. Expression and release of chemokines associated with apoptotic cell death in human promonocytic U937 cells and peripheral blood mononuclear cells. European journal of immunology. vol 29. issue 10. 1999-11-16. PMID:10540334. |
exposure of u937 cells to etoposide (vp-16) or the nitric oxide (no) donor deta-no, both inducers of apoptosis in these cells, was associated with increased expression of the chemokines il-8 and macrophage inflammatory protein-1 alpha. |
1999-11-16 |
2023-08-12 |
human |
| V A Soldatenkov, A Dritschilo, Z Ronai, S Y Fuch. Inhibition of homologue of Slimb (HOS) function sensitizes human melanoma cells for apoptosis. Cancer research. vol 59. issue 20. 1999-11-10. PMID:10537278. |
forced expression of the dominant negative hosdeltaf construct inhibited ikappab degradation and led to sensitization of melanoma cells to apoptosis induced by tumor necrosis factor alpha with cycloheximide, as well as by cisplatin and ionizing and uv irradiation. |
1999-11-10 |
2023-08-12 |
human |
| D T Humphreys, M R Wilso. Modes of L929 cell death induced by TNF-alpha and other cytotoxic agents. Cytokine. vol 11. issue 10. 1999-11-05. PMID:10525316. |
recent studies have variably reported that tumour necrosis factor alpha (tnf-alpha) induces either necrosis or apoptosis in l929 cells. |
1999-11-05 |
2023-08-12 |
Not clear |
| V Goll, E Alexandre, C Viollon-Abadie, L Nicod, D Jaeck, L Richer. Comparison of the effects of various peroxisome proliferators on peroxisomal enzyme activities, DNA synthesis, and apoptosis in rat and human hepatocyte cultures. Toxicology and applied pharmacology. vol 160. issue 1. 1999-11-04. PMID:10502499. |
furthermore, pps inhibited transforming growth factor beta (tgfbeta)-induced apoptosis but not tumor necrosis factor alpha (tnfalpha)/alpha amanitine (alphaama)-induced apoptosis in rat hepatocyte cultures. |
1999-11-04 |
2023-08-12 |
human |
| Y Takada, M Hachiya, Y Osawa, Y Hasegawa, K Ando, Y Kobayashi, M Akash. 12-O-tetradecanoylphorbol-13-acetate-induced apoptosis is mediated by tumor necrosis factor alpha in human monocytic U937 cells. The Journal of biological chemistry. vol 274. issue 40. 1999-11-02. PMID:10497185. |
12-o-tetradecanoylphorbol-13-acetate-induced apoptosis is mediated by tumor necrosis factor alpha in human monocytic u937 cells. |
1999-11-02 |
2023-08-12 |
human |
| J L Cook, B A Routes, C Y Leu, T A Walker, K L Colvi. E1A oncogene-induced cellular sensitization to immune-mediated apoptosis is independent of p53 and resistant to blockade by E1B 19 kDa protein. Experimental cell research. vol 252. issue 1. 1999-11-01. PMID:10502412. |
in this study, the roles of p53 and e1b 19 kda were tested for e1a sensitization to cl-induced apoptosis in contrast with apoptosis triggered by tnf alpha or chemical injuries. |
1999-11-01 |
2023-08-12 |
mouse |
| N Ohta, J Y Tsai, E F Secchi, P F Kador, S Sat. Neutrophils in galactose-fed dogs: suppressed apoptosis and increased adhesion to retinal capillary endothelial cells. Journal of diabetes and its complications. vol 13. issue 3. 1999-11-01. PMID:10509875. |
apoptosis of neutrophils was microscopically examined after incubation at 37 degrees c for 3 hours with either 100 u/ml tumor necrosis factor alpha (tnf-alpha), 2 microg/ml cycloheximide or 50 ng/ml phorbol 12-myristate 13-acetate (pma). |
1999-11-01 |
2023-08-12 |
dog |
| R K Srivastava, A R Srivastava, P Seth, S Agrawal, Y S Cho-Chun. Growth arrest and induction of apoptosis in breast cancer cells by antisense depletion of protein kinase A-RI alpha subunit: p53-independent mechanism of action. Molecular and cellular biochemistry. vol 195. issue 1-2. 1999-10-28. PMID:10395066. |
growth arrest and induction of apoptosis in breast cancer cells by antisense depletion of protein kinase a-ri alpha subunit: p53-independent mechanism of action. |
1999-10-28 |
2023-08-12 |
human |
| R K Srivastava, A R Srivastava, P Seth, S Agrawal, Y S Cho-Chun. Growth arrest and induction of apoptosis in breast cancer cells by antisense depletion of protein kinase A-RI alpha subunit: p53-independent mechanism of action. Molecular and cellular biochemistry. vol 195. issue 1-2. 1999-10-28. PMID:10395066. |
interestingly, ri alpha antisense inhibited cell viability and induced apoptosis in the absence of p53, suggesting that these actions of ri alpha antisense are exerted independent of p53. |
1999-10-28 |
2023-08-12 |
human |
| R K Srivastava, A R Srivastava, P Seth, S Agrawal, Y S Cho-Chun. Growth arrest and induction of apoptosis in breast cancer cells by antisense depletion of protein kinase A-RI alpha subunit: p53-independent mechanism of action. Molecular and cellular biochemistry. vol 195. issue 1-2. 1999-10-28. PMID:10395066. |
these results demonstrate that the ri alpha antisense, which efficiently depletes the growth stimulatory molecule ri alpha, induces cell differentiation and apoptosis, providing a new approach to combat breast cancer cell growth. |
1999-10-28 |
2023-08-12 |
human |