| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| N Yasuda, K Gotoh, S Minatoguchi, K Asano, K Nishigaki, M Nomura, A Ohno, M Watanabe, H Sano, H Kumada, T Sawa, H Fujiwar. An increase of soluble Fas, an inhibitor of apoptosis, associated with progression of COPD. Respiratory medicine. vol 92. issue 8. 1999-01-29. PMID:9893764. |
an increase of soluble fas, an inhibitor of apoptosis, associated with progression of copd. |
1999-01-29 |
2023-08-12 |
human |
| N Yasuda, K Gotoh, S Minatoguchi, K Asano, K Nishigaki, M Nomura, A Ohno, M Watanabe, H Sano, H Kumada, T Sawa, H Fujiwar. An increase of soluble Fas, an inhibitor of apoptosis, associated with progression of COPD. Respiratory medicine. vol 92. issue 8. 1999-01-29. PMID:9893764. |
fas/apo-1 receptor (fas), fas ligand (fas-l) and soluble fas ligand (sfas-l) are inducers, while soluble fas (sfas) is an inhibitor of apoptosis. |
1999-01-29 |
2023-08-12 |
human |
| S Hodge, F J Novembre, L Whetter, H A Gelbard, S Dewhurs. Induction of fas ligand expression by an acutely lethal simian immunodeficiency virus, SIVsmmPBj14. Virology. vol 252. issue 2. 1999-01-28. PMID:9878614. |
in the present study, we examined whether the ability of this virus to cause widespread apoptosis might be linked to the up-regulation of fas ligand (cd95l) expression in virally infected cells. |
1999-01-28 |
2023-08-12 |
Not clear |
| J Gora-Tybor, M W Deininger, J M Goldman, J V Mel. The susceptibility of Philadelphia chromosome positive cells to FAS-mediated apoptosis is not linked to the tyrosine kinase activity of BCR-ABL. British journal of haematology. vol 103. issue 3. 1999-01-27. PMID:9858222. |
ar230 cells were resistant and sd1 cells were partially resistant to fas-mediated apoptosis induced by ligation of the fas receptor to an anti-fas igm antibody. |
1999-01-27 |
2023-08-12 |
Not clear |
| T Matsuda, H Saito, T Inoue, K Fukatsu, I Han, S Furukawa, S Ikeda, T Mut. Growth hormone inhibits apoptosis and up-regulates reactive oxygen intermediates production by human polymorphonuclear neutrophils. JPEN. Journal of parenteral and enteral nutrition. vol 22. issue 6. 1999-01-26. PMID:9829610. |
therefore, this study was designed to investigate the effects of gh on pmn apoptosis, reactive oxygen intermediates (roi) production, cd16, and fas expression. |
1999-01-26 |
2023-08-12 |
human |
| M Bijl, T van Lopik, P C Limburg, P E Spronk, S M Jaegers, L A Aarden, R J Smeenk, G G Kallenber. Do elevated levels of serum-soluble fas contribute to the persistence of activated lymphocytes in systemic lupus erythematosus? Journal of autoimmunity. vol 11. issue 5. 1999-01-25. PMID:9802929. |
part of this might be explained by a decreased rate of apoptosis, possibly related to elevated levels of soluble fas (sfas) which can inhibit fas mediated apoptosis of lymphocytes. |
1999-01-25 |
2023-08-12 |
human |
| G Pan, T Zhou, W Radding, M S Saag, J D Mountz, J M McDonal. Calmodulin antagonists inhibit apoptosis of CD4+ T-cells from patients with AIDS. Immunopharmacology. vol 40. issue 2. 1999-01-22. PMID:9826024. |
recent studies indicate that fas and fas ligand are involved in apoptosis of t-cells in hiv-infected patients. |
1999-01-22 |
2023-08-12 |
human |
| Y Iwakura, K Itagaki, C Ishitsuka, Y Yamasaki, A Matsuzawa, S Yonehara, S Karasawa, S Ueda, S Saij. The development of autoimmune inflammatory arthropathy in mice transgenic for the human T cell leukemia virus type-1 env-pX region is not dependent on H-2 haplotypes and modified by the expression levels of Fas antigen. Journal of immunology (Baltimore, Md. : 1950). vol 161. issue 12. 1999-01-22. PMID:9862686. |
then, since previous study indicated a defect in fas-mediated apoptosis of transgenic t cells, the effects of fas gene modification on the disease were examined. |
1999-01-22 |
2023-08-12 |
mouse |
| L Gu, A Weinreb, X P Wang, D J Zack, J H Qiao, R Weisbart, A J Lusi. Genetic determinants of autoimmune disease and coronary vasculitis in the MRL-lpr/lpr mouse model of systemic lupus erythematosus. Journal of immunology (Baltimore, Md. : 1950). vol 161. issue 12. 1999-01-22. PMID:9862736. |
although the autoimmune disorder is mediated primarily by mutation of the fas gene (lpr), which interferes with lymphocyte apoptosis, mrl/lpr mice also have other predisposing genetic factors. |
1999-01-22 |
2023-08-12 |
mouse |
| G Stassi, M Todaro, F Bucchieri, A Stoppacciaro, F Farina, G Zummo, R Testi, R De Mari. Fas/Fas ligand-driven T cell apoptosis as a consequence of ineffective thyroid immunoprivilege in Hashimoto's thyroiditis. Journal of immunology (Baltimore, Md. : 1950). vol 162. issue 1. 1999-01-21. PMID:9886394. |
the analysis of itl purified from ht thyroids showed that itl were massively killed by fas crosslinking and that a considerable number (24-36%) underwent spontaneous apoptosis within 36 h of culture. |
1999-01-21 |
2023-08-12 |
Not clear |
| S B Brown, J Savil. Phagocytosis triggers macrophage release of Fas ligand and induces apoptosis of bystander leukocytes. Journal of immunology (Baltimore, Md. : 1950). vol 162. issue 1. 1999-01-21. PMID:9886423. |
phagocytosis triggers macrophage release of fas ligand and induces apoptosis of bystander leukocytes. |
1999-01-21 |
2023-08-12 |
human |
| F Novelli, A Allione, V Wells, G Forni, L Mallucc. Negative cell cycle control of human T cells by beta-galactoside binding protein (beta GBP): induction of programmed cell death in leukaemic cells. Journal of cellular physiology. vol 178. issue 1. 1999-01-21. PMID:9886496. |
analysis of possible mechanisms of induction of apoptosis does not support fas and fas l as having a main role but points instead to bcl-2 and bax. |
1999-01-21 |
2023-08-12 |
human |
| A J Seely, D E Swartz, B Giannias, N V Christo. Reduction in neutrophil cell surface expression of tumor necrosis factor receptors but not Fas after transmigration: implications for the regulation of neutrophil apoptosis. Archives of surgery (Chicago, Ill. : 1960). vol 133. issue 12. 1999-01-20. PMID:9865647. |
reduction in neutrophil cell surface expression of tumor necrosis factor receptors but not fas after transmigration: implications for the regulation of neutrophil apoptosis. |
1999-01-20 |
2023-08-12 |
Not clear |
| A J Seely, D E Swartz, B Giannias, N V Christo. Reduction in neutrophil cell surface expression of tumor necrosis factor receptors but not Fas after transmigration: implications for the regulation of neutrophil apoptosis. Archives of surgery (Chicago, Ill. : 1960). vol 133. issue 12. 1999-01-20. PMID:9865647. |
to test the hypothesis that loss of polymorphonuclear neutrophil tumor necrosis factor alpha (tnf-alpha) receptors during transmigration renders the exudate neutrophil refractory to tnf-alpha-mediated stimulation of apoptosis; and to investigate the surface expression of fas on both circulating and exudate neutrophils. |
1999-01-20 |
2023-08-12 |
Not clear |
| S H Lee, J J Jang, J Y Lee, S Y Kim, W S Park, M S Shin, S M Dong, E Y Na, K M Kim, C S Kim, S H Kim, N J Yo. Fas ligand is expressed in normal skin and in some cutaneous malignancies. The British journal of dermatology. vol 139. issue 2. 1999-01-19. PMID:9767230. |
fas, a cell surface receptor and member of the tumour necrosis factor receptor superfamily, induces apoptosis upon oligomerization by its ligand (fas ligand: fasl). |
1999-01-19 |
2023-08-12 |
human |
| D G Wingett, R E Vestal, K Forcier, N Hadjokas, C P Nielso. CD40 is functionally expressed on human breast carcinomas: variable inducibility by cytokines and enhancement of Fas-mediated apoptosis. Breast cancer research and treatment. vol 50. issue 1. 1999-01-19. PMID:9802617. |
enhanced apoptosis appears to be attributable, at least in part, to an up-regulation of fas expression caused by cd40 ligation. |
1999-01-19 |
2023-08-12 |
human |
| R M Pitti, S A Marsters, D A Lawrence, M Roy, F C Kischkel, P Dowd, A Huang, C J Donahue, S W Sherwood, D T Baldwin, P J Godowski, W I Wood, A L Gurney, K J Hillan, R L Cohen, A D Goddard, D Botstein, A Ashkenaz. Genomic amplification of a decoy receptor for Fas ligand in lung and colon cancer. Nature. vol 396. issue 6712. 1999-01-19. PMID:9872321. |
fas ligand (fasl) is produced by activated t cells and natural killer cells and it induces apoptosis (programmed cell death) in target cells through the death receptor fas/apol/cd95. |
1999-01-19 |
2023-08-12 |
Not clear |
| M Sata, K Wals. Endothelial cell apoptosis induced by oxidized LDL is associated with the down-regulation of the cellular caspase inhibitor FLIP. The Journal of biological chemistry. vol 273. issue 50. 1999-01-14. PMID:9837872. |
normal vascular endothelial cells are resistant to fas-mediated apoptosis though they express both fas and fasl. |
1999-01-14 |
2023-08-12 |
Not clear |
| D A Ladin, Z Hou, D Patel, M McPhail, J C Olson, G M Saed, D P Fivenso. p53 and apoptosis alterations in keloids and keloid fibroblasts. Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair Society. vol 6. issue 1. 1999-01-12. PMID:9776848. |
this phenotype is reversed in the older areas of the keloid and may prevent malignant degeneration, thus favoring normal apoptosis as evidenced by prominent fas expression. |
1999-01-12 |
2023-08-12 |
Not clear |
| M Muzi. Signalling by proteolysis: death receptors induce apoptosis. International journal of clinical & laboratory research. vol 28. issue 3. 1999-01-08. PMID:9801924. |
death receptors (fas, tnfr-2, dr3, and trail receptors) induce apoptosis upon ligation to cognate ligands or ectopic expression. |
1999-01-08 |
2023-08-12 |
Not clear |