| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| T Takizawa, C Tatematsu, K Ohashi, Y Nakanish. Recruitment of apoptotic cysteine proteases (caspases) in influenza virus-induced cell death. Microbiology and immunology. vol 43. issue 3. 1999-09-03. PMID:10338194. |
caspases, a family of cysteine proteases structurally related to interleukin-1-beta-converting enzyme (ice), play crucial roles in apoptosis induced by various stimuli, including fas. |
1999-09-03 |
2023-08-12 |
Not clear |
| S Nandi, P P Banerjee, B R Zirki. Germ cell apoptosis in the testes of Sprague Dawley rats following testosterone withdrawal by ethane 1,2-dimethanesulfonate administration: relationship to Fas? Biology of reproduction. vol 61. issue 1. 1999-09-03. PMID:10377033. |
germ cell apoptosis in the testes of sprague dawley rats following testosterone withdrawal by ethane 1,2-dimethanesulfonate administration: relationship to fas? |
1999-09-03 |
2023-08-12 |
rat |
| S Nandi, P P Banerjee, B R Zirki. Germ cell apoptosis in the testes of Sprague Dawley rats following testosterone withdrawal by ethane 1,2-dimethanesulfonate administration: relationship to Fas? Biology of reproduction. vol 61. issue 1. 1999-09-03. PMID:10377033. |
the fas system has been implicated as a possible key regulator of apoptosis in various cells: binding of fas ligand (fasl), a type ii transmembrane protein, to fas, a type i transmembrane receptor protein, triggers apoptosis in cells expressing fas. |
1999-09-03 |
2023-08-12 |
rat |
| S Nandi, P P Banerjee, B R Zirki. Germ cell apoptosis in the testes of Sprague Dawley rats following testosterone withdrawal by ethane 1,2-dimethanesulfonate administration: relationship to Fas? Biology of reproduction. vol 61. issue 1. 1999-09-03. PMID:10377033. |
we hypothesized that fas protein content would rise in response to reduced levels of testosterone as part of a suicide pathway that would result in germ cell apoptosis. |
1999-09-03 |
2023-08-12 |
rat |
| S Nandi, P P Banerjee, B R Zirki. Germ cell apoptosis in the testes of Sprague Dawley rats following testosterone withdrawal by ethane 1,2-dimethanesulfonate administration: relationship to Fas? Biology of reproduction. vol 61. issue 1. 1999-09-03. PMID:10377033. |
apoptosis was examined in situ and biochemically, and fas protein content in the testis was monitored by western blot analysis. |
1999-09-03 |
2023-08-12 |
rat |
| S Nandi, P P Banerjee, B R Zirki. Germ cell apoptosis in the testes of Sprague Dawley rats following testosterone withdrawal by ethane 1,2-dimethanesulfonate administration: relationship to Fas? Biology of reproduction. vol 61. issue 1. 1999-09-03. PMID:10377033. |
we show that eds injection results in the following sequence of events: apoptotic death of leydig cells by a mechanism that does not involve fas; reduced testosterone; increased testicular fas content; and germ cell apoptosis. |
1999-09-03 |
2023-08-12 |
rat |
| K A Rezai, R T Semnani, L Farrokh-Siar, K J Hamann, S C Patel, J T Ernest, G A van Sevente. Human fetal retinal pigment epithelial cells induce apoptosis in allogenic T-cells in a Fas ligand and PGE2 independent pathway. Current eye research. vol 18. issue 6. 1999-09-02. PMID:10435830. |
human fetal retinal pigment epithelial cells induce apoptosis in allogenic t-cells in a fas ligand and pge2 independent pathway. |
1999-09-02 |
2023-08-12 |
human |
| W S Wold, K Doronin, K Toth, M Kuppuswamy, D L Lichtenstein, A E Tollefso. Immune responses to adenoviruses: viral evasion mechanisms and their implications for the clinic. Current opinion in immunology. vol 11. issue 4. 1999-09-02. PMID:10448144. |
adenoviruses encode proteins that block responses to interferons, intrinsic cellular apoptosis, killing by cd8(+) cytotoxic t lymphocytes and killing by the death ligands tnf, fas ligand and trail. |
1999-09-02 |
2023-08-12 |
Not clear |
| H Liu, K Mihara, A Kimura, K Tanaka, N Kamad. Induction of apoptosis in CD34+ cells by sera from patients with aplastic anemia. Hiroshima journal of medical sciences. vol 48. issue 2. 1999-08-31. PMID:10434475. |
to further explore the mechanism of increased apoptosis induced by the serum of aa patients, the expression of the fas receptor was measured after incubation with serum from the normal controls and aa patients using flow cytometry. |
1999-08-31 |
2023-08-12 |
Not clear |
| H Liu, K Mihara, A Kimura, K Tanaka, N Kamad. Induction of apoptosis in CD34+ cells by sera from patients with aplastic anemia. Hiroshima journal of medical sciences. vol 48. issue 2. 1999-08-31. PMID:10434475. |
after incubation with serum from the aa patients, the fas receptor was overexpressed, correlating with the increased apoptosis induced by the same serum from aa patients. |
1999-08-31 |
2023-08-12 |
Not clear |
| H Liu, K Mihara, A Kimura, K Tanaka, N Kamad. Induction of apoptosis in CD34+ cells by sera from patients with aplastic anemia. Hiroshima journal of medical sciences. vol 48. issue 2. 1999-08-31. PMID:10434475. |
in addition, the induction of apoptosis and fas expression on cd34+ cells by serum from the saa patients was blocked partly by preincubation of the serum with anti-gamma-ifn neutralizing moab. |
1999-08-31 |
2023-08-12 |
Not clear |
| H Liu, K Mihara, A Kimura, K Tanaka, N Kamad. Induction of apoptosis in CD34+ cells by sera from patients with aplastic anemia. Hiroshima journal of medical sciences. vol 48. issue 2. 1999-08-31. PMID:10434475. |
these findings suggested that some aberrant components of the serum in the aa patients, which was confirmed partly to be gamma-ifn, can induce cd34+ progenitors apoptosis through the fas signaling pathway. |
1999-08-31 |
2023-08-12 |
Not clear |
| J Hughes, R J Johnso. Role of Fas (CD95) in tubulointerstitial disease induced by unilateral ureteric obstruction. The American journal of physiology. vol 277. issue 1. 1999-08-30. PMID:10409294. |
we therefore hypothesized that an absence of renal fas may protect resident cells from undergoing apoptosis. |
1999-08-30 |
2023-08-12 |
mouse |
| J Hughes, R J Johnso. Role of Fas (CD95) in tubulointerstitial disease induced by unilateral ureteric obstruction. The American journal of physiology. vol 277. issue 1. 1999-08-30. PMID:10409294. |
in addition, there was no difference in interstitial cell apoptosis or proliferation, suggesting that fas does not play a significant role in interstitial cell death. |
1999-08-30 |
2023-08-12 |
mouse |
| J Hughes, R J Johnso. Role of Fas (CD95) in tubulointerstitial disease induced by unilateral ureteric obstruction. The American journal of physiology. vol 277. issue 1. 1999-08-30. PMID:10409294. |
in conclusion, the absence of functional cell surface fas in uuo provides distal tubular cells with partial protection from apoptosis but does not affect interstitial cell fate in this model of tubulointerstitial injury. |
1999-08-30 |
2023-08-12 |
mouse |
| G Condorelli, G Vigliotta, A Cafieri, A Trencia, P Andalò, F Oriente, C Miele, M Caruso, P Formisano, F Beguino. PED/PEA-15: an anti-apoptotic molecule that regulates FAS/TNFR1-induced apoptosis. Oncogene. vol 18. issue 31. 1999-08-30. PMID:10442631. |
thus, ped/pea-15 is an endogenous protein inhibiting fas and tnfr1-mediated apoptosis. |
1999-08-30 |
2023-08-12 |
Not clear |
| S Guller, L LaChapell. The role of placental Fas ligand in maintaining immune privilege at maternal-fetal interfaces. Seminars in reproductive endocrinology. vol 17. issue 1. 1999-08-26. PMID:10406074. |
over the last several years our group and others have formulated a hypothesis that trophoblast fas ligand (fasl) plays an important role in maintaining fetal immune privilege in human pregnancy by actively promoting apoptosis (programmed cell death) of activated maternal lymphocytes bearing fas (i.e., the fasl receptor). |
1999-08-26 |
2023-08-12 |
human |
| C Cabaner, C Gajate, A Macho, E Muñoz, M Modolell, F Mollined. Induction of apoptosis in human mitogen-activated peripheral blood T-lymphocytes by the ether phospholipid ET-18-OCH3: involvement of the Fas receptor/ligand system. British journal of pharmacology. vol 127. issue 4. 1999-08-26. PMID:10433487. |
induction of apoptosis in human mitogen-activated peripheral blood t-lymphocytes by the ether phospholipid et-18-och3: involvement of the fas receptor/ligand system. |
1999-08-26 |
2023-08-12 |
human |
| C Cabaner, C Gajate, A Macho, E Muñoz, M Modolell, F Mollined. Induction of apoptosis in human mitogen-activated peripheral blood T-lymphocytes by the ether phospholipid ET-18-OCH3: involvement of the Fas receptor/ligand system. British journal of pharmacology. vol 127. issue 4. 1999-08-26. PMID:10433487. |
et-18-och3 induced an increase in fas (apo-1/cd95) ligand mrna expression in activated t-cells, and incubation with a blocking anti-fas (apo-1/cd95) antibody partially inhibited the et-18-och3-induced apoptosis of activated t-lymphocytes. |
1999-08-26 |
2023-08-12 |
human |
| C Cabaner, C Gajate, A Macho, E Muñoz, M Modolell, F Mollined. Induction of apoptosis in human mitogen-activated peripheral blood T-lymphocytes by the ether phospholipid ET-18-OCH3: involvement of the Fas receptor/ligand system. British journal of pharmacology. vol 127. issue 4. 1999-08-26. PMID:10433487. |
these results demonstrate that mitogen-activated t-cells, unlike resting lymphocytes, are able to take up significant amounts of et-18-och3, and are susceptible to undergo apoptosis by the ether lipid via, in part, the fas (apo-1/cd95) receptor/ligand system. |
1999-08-26 |
2023-08-12 |
human |