| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| M Aoki, R Morishita, H Matsushita, S Hayashi, H Nakagami, K Yamamoto, A Moriguchi, Y Kaneda, J Higaki, T Ogihar. Inhibition of the p53 tumor suppressor gene results in growth of human aortic vascular smooth muscle cells. Potential role of p53 in regulation of vascular smooth muscle cell growth. Hypertension (Dallas, Tex. : 1979). vol 34. issue 2. 1999-09-15. PMID:10454440. |
in vsmc transfected with antisense rb odn, bax, a promoter of apoptosis, was significantly increased in vsmc transfected with antisense rb odn (p<0.01), whereas bcl-2 and fas did not play a pivotal role in the induction of apoptosis. |
1999-09-15 |
2023-08-12 |
human |
| H Yasuda, K Kataoka, H Ichimura, M Mitsuyoshi, T Iida, M Kita, J Imanish. Cytokine expression and induction of acinar cell apoptosis after pancreatic duct ligation in mice. Journal of interferon & cytokine research : the official journal of the International Society for Interferon and Cytokine Research. vol 19. issue 6. 1999-09-14. PMID:10433365. |
significant increases in intrapancreatic mrna levels of tnf-alpha, fas ligand (fasl), and il-1beta-converting enzyme (ice) were observed from day 3 after pdl with the appearance of acinar cell apoptosis. |
1999-09-14 |
2023-08-12 |
mouse |
| X C Li, Y Li, I Dodge, A D Wells, X X Zheng, L A Turka, T B Stro. Induction of allograft tolerance in the absence of Fas-mediated apoptosis. Journal of immunology (Baltimore, Md. : 1950). vol 163. issue 5. 1999-09-14. PMID:10452986. |
our study suggests that: 1) fas is not necessarily essential for allograft tolerance, and 2) fas-mediated apoptosis is not central to the il-2-dependent mechanism governing the acquisition of allograft tolerance. |
1999-09-14 |
2023-08-12 |
mouse |
| A Ortiz, C Lorz, J Egid. New kids in the block: the role of FasL and Fas in kidney damage. Journal of nephrology. vol 12. issue 3. 1999-09-13. PMID:10440512. |
fas ligand (fasl) is a lethal cytokine that promotes apoptosis through cross-linking of the fas receptor, although it also has other, less well understood functions. |
1999-09-13 |
2023-08-12 |
mouse |
| A Ortiz, C Lorz, J Egid. New kids in the block: the role of FasL and Fas in kidney damage. Journal of nephrology. vol 12. issue 3. 1999-09-13. PMID:10440512. |
evidence that fasl and fas participate in kidney damage can be summarized as follows: 1) fasl is expressed by renal cells and its expression increases during kidney damage; 2) activation of the fas receptor promotes apoptosis of non-stimulated or cytokine-primed renal cells in culture; 3) fas agonists kill mesangial cells and induce glomerular injury in vivo, but can also reduce kidney damage by limiting injurious immunological responses; 4) mice with disrupted fasl/fas systems are protected from acute tubular cell injury, although they develop autoimmune glomerulonephritis if other genetic predisposing factors are present. |
1999-09-13 |
2023-08-12 |
mouse |
| T H Holmström, S E Tran, V L Johnson, N G Ahn, S C Chow, J E Eriksso. Inhibition of mitogen-activated kinase signaling sensitizes HeLa cells to Fas receptor-mediated apoptosis. Molecular and cellular biology. vol 19. issue 9. 1999-09-10. PMID:10454546. |
inhibition of mitogen-activated kinase signaling sensitizes hela cells to fas receptor-mediated apoptosis. |
1999-09-10 |
2023-08-12 |
Not clear |
| T H Holmström, S E Tran, V L Johnson, N G Ahn, S C Chow, J E Eriksso. Inhibition of mitogen-activated kinase signaling sensitizes HeLa cells to Fas receptor-mediated apoptosis. Molecular and cellular biology. vol 19. issue 9. 1999-09-10. PMID:10454546. |
the fas receptor (fasr) is an important physiological mediator of apoptosis in various tissues and cells. |
1999-09-10 |
2023-08-12 |
Not clear |
| A D Badley, K Parato, D W Cameron, S Kravcik, B N Phenix, D Ashby, A Kumar, D H Lynch, J Tschopp, J B Ange. Dynamic correlation of apoptosis and immune activation during treatment of HIV infection. Cell death and differentiation. vol 6. issue 5. 1999-09-09. PMID:10381636. |
t cells from hiv infected patients undergo spontaneous apoptosis at a faster rate than those from uninfected patients, are abnormally susceptible to activation induced cell death (aicd), and undergo increased apoptosis in response to fas receptor ligation. |
1999-09-09 |
2023-08-12 |
Not clear |
| A D Badley, K Parato, D W Cameron, S Kravcik, B N Phenix, D Ashby, A Kumar, D H Lynch, J Tschopp, J B Ange. Dynamic correlation of apoptosis and immune activation during treatment of HIV infection. Cell death and differentiation. vol 6. issue 5. 1999-09-09. PMID:10381636. |
furthermore, spontaneous apoptosis decreases in both cd4 and cd8 t cells (cd4 apoptosis 17.4 vs 2.6%, p=0.005; cd8 apoptosis 15.0 vs 1.0%, p<0.001), as does both fas mediated apoptosis (cd4 apoptosis 19.0 vs 3.5%, p=0.03; cd8 apoptosis 13.7 vs 1.5%, p=0.002) and cd3 induced aicd (cd4 apoptosis 13.7 vs 3.2%, p=0.001; cd8 apoptosis 29 vs 2.2%, p=0.08). |
1999-09-09 |
2023-08-12 |
Not clear |
| A D Badley, K Parato, D W Cameron, S Kravcik, B N Phenix, D Ashby, A Kumar, D H Lynch, J Tschopp, J B Ange. Dynamic correlation of apoptosis and immune activation during treatment of HIV infection. Cell death and differentiation. vol 6. issue 5. 1999-09-09. PMID:10381636. |
changes in apoptosis are not associated with changes in fas receptor expression, but are significantly correlated with changes in activation marker profiles. |
1999-09-09 |
2023-08-12 |
Not clear |
| C K Sen, R Sashwati, L Packe. Fas mediated apoptosis of human Jurkat T-cells: intracellular events and potentiation by redox-active alpha-lipoic acid. Cell death and differentiation. vol 6. issue 5. 1999-09-09. PMID:10381641. |
fas mediated apoptosis of human jurkat t-cells: intracellular events and potentiation by redox-active alpha-lipoic acid. |
1999-09-09 |
2023-08-12 |
human |
| C K Sen, R Sashwati, L Packe. Fas mediated apoptosis of human Jurkat T-cells: intracellular events and potentiation by redox-active alpha-lipoic acid. Cell death and differentiation. vol 6. issue 5. 1999-09-09. PMID:10381641. |
activation of caspases is required in fas receptor mediated apoptosis. |
1999-09-09 |
2023-08-12 |
human |
| W Barthlen, J Schäfer, R Girgert, M Schuster, P Schweize. Apoptosis of cultured neuroblastoma cells is induced by ceramide and not by ligation of the Fas/Apo-1/CD95 receptor. Chemotherapy. vol 45. issue 4. 1999-09-09. PMID:10394009. |
the induction of apoptosis was attempted by incubation with fas antibody igm and igg, fas ligand and with ceramide. |
1999-09-09 |
2023-08-12 |
Not clear |
| W Barthlen, J Schäfer, R Girgert, M Schuster, P Schweize. Apoptosis of cultured neuroblastoma cells is induced by ceramide and not by ligation of the Fas/Apo-1/CD95 receptor. Chemotherapy. vol 45. issue 4. 1999-09-09. PMID:10394009. |
however, propidium iodide staining in facs showed that neither incubation with the fas antibody nor with the fas ligand resulted in convincing apoptosis of the neuroblastoma cells. |
1999-09-09 |
2023-08-12 |
Not clear |
| I Bechmann, G Mor, J Nilsen, M Eliza, R Nitsch, F Naftoli. FasL (CD95L, Apo1L) is expressed in the normal rat and human brain: evidence for the existence of an immunological brain barrier. Glia. vol 27. issue 1. 1999-09-09. PMID:10401633. |
fasl-positive astrocytes were often situated in close vicinity to cerebral blood vessels in vivo and induced apoptosis of fas expressing jurkat cells in vitro. |
1999-09-09 |
2023-08-12 |
human |
| M Lotz, S Hashimoto, K Küh. Mechanisms of chondrocyte apoptosis. Osteoarthritis and cartilage. vol 7. issue 4. 1999-09-09. PMID:10419776. |
activation of the fas receptor triggers apoptosis in these cells. |
1999-09-09 |
2023-08-12 |
human |
| G Matute-Bello, W C Liles, K P Steinberg, P A Kiener, S Mongovin, E Y Chi, M Jonas, T R Marti. Soluble Fas ligand induces epithelial cell apoptosis in humans with acute lung injury (ARDS). Journal of immunology (Baltimore, Md. : 1950). vol 163. issue 4. 1999-09-09. PMID:10438964. |
soluble fas ligand induces epithelial cell apoptosis in humans with acute lung injury (ards). |
1999-09-09 |
2023-08-12 |
Not clear |
| G Matute-Bello, W C Liles, K P Steinberg, P A Kiener, S Mongovin, E Y Chi, M Jonas, T R Marti. Soluble Fas ligand induces epithelial cell apoptosis in humans with acute lung injury (ARDS). Journal of immunology (Baltimore, Md. : 1950). vol 163. issue 4. 1999-09-09. PMID:10438964. |
bal from patients with ards induced apoptosis of distal lung epithelial cells, which express fas, and this effect was inhibited by blocking the fas/fasl system using three different strategies: anti-fasl mab, anti-fas mab, and a fas-ig fusion protein. |
1999-09-09 |
2023-08-12 |
Not clear |
| B Lin, C Williams-Skipp, Y Tao, M S Schleicher, L L Cano, R C Duke, R I Scheinma. NF-kappaB functions as both a proapoptotic and antiapoptotic regulatory factor within a single cell type. Cell death and differentiation. vol 6. issue 6. 1999-09-08. PMID:10381655. |
treatment with pma plus ionomycin (p/i) results in the upregulation of fas ligand (fasl) and induction of apoptosis. |
1999-09-08 |
2023-08-12 |
Not clear |
| T Takizawa, C Tatematsu, K Ohashi, Y Nakanish. Recruitment of apoptotic cysteine proteases (caspases) in influenza virus-induced cell death. Microbiology and immunology. vol 43. issue 3. 1999-09-03. PMID:10338194. |
influenza virus infection induces apoptosis in cultured cells with an augmented expression of fas (apo-1/cd95). |
1999-09-03 |
2023-08-12 |
Not clear |