| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| J Poirier, U Beffert, D Dea, R Alonso, D O'Donnell, P Boks. Increased levels of statin, a marker of cell cycle arrest, in response to hippocampal neuronal injury. Brain research. Molecular brain research. vol 34. issue 1. 1997-01-15. PMID:8750861. |
in the present study, neuronal statin was examined following lethal or sublethal neuronal injuries in the hippocampus of alzheimer's disease patients, in rats receiving kainate lesions to the dorsal hippocampus and in entorhinal cortex lesioned rats. |
1997-01-15 |
2023-08-12 |
rat |
| J Poirier, U Beffert, D Dea, R Alonso, D O'Donnell, P Boks. Increased levels of statin, a marker of cell cycle arrest, in response to hippocampal neuronal injury. Brain research. Molecular brain research. vol 34. issue 1. 1997-01-15. PMID:8750861. |
immunolabelling of nuclear statin showed that statin immunoreactivity increased preferentially in ca1 pyramidal neurons of the hippocampus in alzheimer's disease. |
1997-01-15 |
2023-08-12 |
rat |
| P Saftig, C Peters, K von Figura, K Craessaerts, F Van Leuven, B De Stroope. Amyloidogenic processing of human amyloid precursor protein in hippocampal neurons devoid of cathepsin D. The Journal of biological chemistry. vol 271. issue 44. 1996-12-26. PMID:8910296. |
amyloidogenic processing of human amyloid precursor protein in hippocampal neurons devoid of cathepsin d. betaa4-amyloid peptide, the main component of the amyloid plaques in the brain of alzheimer's disease patients is produced from amyloid precursor protein (app) by proteolytical processing. |
1996-12-26 |
2023-08-12 |
mouse |
| D S Woodruff-Pak, M Papka, S Romano, Y T L. Eyeblink classical conditioning in Alzheimer's disease and cerebrovascular dementia. Neurobiology of aging. vol 17. issue 4. 1996-12-20. PMID:8832623. |
eyeblink classical conditioning (ebcc) is severely and consistently impaired in probable alzheimer's disease (ad), presumably due to normal age related changes in the cerebellum and ad-related hippocampal cholinergic disruption. |
1996-12-20 |
2023-08-12 |
human |
| Y Harigaya, M Shoji, T Shirao, S Hira. Disappearance of actin-binding protein, drebrin, from hippocampal synapses in Alzheimer's disease. Journal of neuroscience research. vol 43. issue 1. 1996-12-16. PMID:8838578. |
disappearance of actin-binding protein, drebrin, from hippocampal synapses in alzheimer's disease. |
1996-12-16 |
2023-08-12 |
human |
| Y Harigaya, M Shoji, T Shirao, S Hira. Disappearance of actin-binding protein, drebrin, from hippocampal synapses in Alzheimer's disease. Journal of neuroscience research. vol 43. issue 1. 1996-12-16. PMID:8838578. |
immunocytochemical study demonstrated that widespread drebrin immunoreactivity was observed in hippocampal formations of control human brains, while alzheimer's disease (ad) brains showed remarkable reductions in this immunoreactivity. |
1996-12-16 |
2023-08-12 |
human |
| R J Holden, P A Moone. Interleukin-1 beta: a common cause of Alzheimer's disease and diabetes mellitus. Medical hypotheses. vol 45. issue 6. 1996-12-10. PMID:8771051. |
alzheimer disease is characterized by the presence of beta-amyloid protein deposits, neurofibrillary tangles and cholinergic dysfunction throughout the hippocampal region. |
1996-12-10 |
2023-08-12 |
human |
| R Gray, A S Rajan, K A Radcliffe, M Yakehiro, J A Dan. Hippocampal synaptic transmission enhanced by low concentrations of nicotine. Nature. vol 383. issue 6602. 1996-12-10. PMID:8878480. |
during alzheimer's dementia there are fewer nachrs and the cholinergic inputs to the hippocampus degenerate. |
1996-12-10 |
2023-08-12 |
chicken |
| S E Blac. Focal cortical atrophy syndromes. Brain and cognition. vol 31. issue 2. 1996-12-04. PMID:8811996. |
the other is alzheimer's disease, in which amyloid plaques and neurofibrillary tangles initially develop in the hippocampal region, and spread to the lateral temporal and parietal neocortex and then to the frontal cortex. |
1996-12-04 |
2023-08-12 |
Not clear |
| D S Woodruff-Pak, M Papk. Alzheimer's disease and eyeblink conditioning: 750 ms trace vs. 400 ms delay paradigm. Neurobiology of aging. vol 17. issue 3. 1996-12-03. PMID:8725901. |
patients with probable alzheimer's disease (ad) and hippocampal disruption are severely impaired in eyeblink classical conditioning (ebcc) in the 400 ms delay paradigm. |
1996-12-03 |
2023-08-12 |
Not clear |
| R A Armstron. The spatial pattern of discrete beta-amyloid deposits in Alzheimer's disease reflects synaptic disconnection. Dementia (Basel, Switzerland). vol 7. issue 2. 1996-12-03. PMID:8866681. |
the spatial pattern of discrete beta-amyloid (a beta) deposits was studied in the superficial laminae of cortical fields of different types and in the hippocampus in 6 cases of alzheimer's disease (ad). |
1996-12-03 |
2023-08-12 |
Not clear |
| Z Nagy, K A Jobst, M M Esiri, J H Morris, E M King, B MacDonald, S Litchfield, L Barnetson, A D Smit. Hippocampal pathology reflects memory deficit and brain imaging measurements in Alzheimer's disease: clinicopathologic correlations using three sets of pathologic diagnostic criteria. Dementia (Basel, Switzerland). vol 7. issue 2. 1996-12-03. PMID:8866679. |
hippocampal pathology reflects memory deficit and brain imaging measurements in alzheimer's disease: clinicopathologic correlations using three sets of pathologic diagnostic criteria. |
1996-12-03 |
2023-08-12 |
Not clear |
| Z Nagy, K A Jobst, M M Esiri, J H Morris, E M King, B MacDonald, S Litchfield, L Barnetson, A D Smit. Hippocampal pathology reflects memory deficit and brain imaging measurements in Alzheimer's disease: clinicopathologic correlations using three sets of pathologic diagnostic criteria. Dementia (Basel, Switzerland). vol 7. issue 2. 1996-12-03. PMID:8866679. |
neurofibrillary tangles (nft), neuritic plaques and amyloid load were quantified in sections of the hippocampus at the level of the lateral geniculate body in 41 consecutive cases fulfilling pathological criteria for diagnosis of alzheimer's disease (ad) and coming to autopsy after longitudinal study during life. |
1996-12-03 |
2023-08-12 |
Not clear |
| C G Gottfries, B Jungbjer, I Karlsson, L Svennerhol. Reductions in membrane proteins and lipids in basal ganglia of classic Alzheimer disease patients. Alzheimer disease and associated disorders. vol 10. issue 2. 1996-11-27. PMID:8727168. |
gangliosides and major membrane components were determined in caudate nucleus, putamen, and hippocampus of 12 cases with alzheimer disease (ad) type i, also termed presenile or pure ad, and in age-matched controls. |
1996-11-27 |
2023-08-12 |
Not clear |
| Z Nagy, M M Esiri, K A Jobst, J H Morris, E M King, B McDonald, S Litchfield, L Barnetso. Clustering of pathological features in Alzheimer's disease: clinical and neuroanatomical aspects. Dementia (Basel, Switzerland). vol 7. issue 3. 1996-11-07. PMID:8740625. |
we have analyzed the tendency of amyloid load, neuritic plaques and neurofibrillary tangles (nft) in the hippocampus and neocortex to occur in clusters in 49 consecutive cases of alzheimer's disease (ad). |
1996-11-07 |
2023-08-12 |
Not clear |
| G B Frisoni, A Beltramello, C Weiss, C Geroldi, A Bianchetti, M Trabucch. Usefulness of simple measures of temporal lobe atrophy in probable Alzheimer's disease. Dementia (Basel, Switzerland). vol 7. issue 1. 1996-10-31. PMID:8788077. |
these results suggest that selected simple indices of hippocampal atrophy might be useful in the diagnosis of alzheimer's disease. |
1996-10-31 |
2023-08-12 |
Not clear |
| P Bertrand, J Poirier, T Oda, C E Finch, G M Pasinett. Association of apolipoprotein E genotype with brain levels of apolipoprotein E and apolipoprotein J (clusterin) in Alzheimer disease. Brain research. Molecular brain research. vol 33. issue 1. 1996-10-23. PMID:8774959. |
this study examines the relationship between the levels of apolipoprotein e (apoe) and apolipoprotein j (apoj, also designated as clusterin) as a function of apoe genotype in the hippocampus and cortex of alzheimer disease (ad) subjects. |
1996-10-23 |
2023-08-12 |
human |
| R Layfield, J Fergusson, A Aitken, J Lowe, M Landon, R J Maye. Neurofibrillary tangles of Alzheimer's disease brains contain 14-3-3 proteins. Neuroscience letters. vol 209. issue 1. 1996-10-04. PMID:8734909. |
the localisation of 14-3-3 proteins compared to that of tau and ubiquitin-protein conjugates in sections of hippocampus from alzheimer's disease (ad) brains was examined by immunohistochemistry. |
1996-10-04 |
2023-08-12 |
Not clear |
| G A Maresh, D Erezyilmaz, C E Murry, D Nochlin, A D Sno. Detection and quantitation of perlecan mRNA levels in Alzheimer's disease and normal aged hippocampus by competitive reverse transcription-polymerase chain reaction. Journal of neurochemistry. vol 67. issue 3. 1996-10-02. PMID:8752120. |
detection and quantitation of perlecan mrna levels in alzheimer's disease and normal aged hippocampus by competitive reverse transcription-polymerase chain reaction. |
1996-10-02 |
2023-08-12 |
human |
| P W Landfiel. Aging-related increase in hippocampal calcium channels. Life sciences. vol 59. issue 5-6. 1996-10-02. PMID:8761327. |
thus, a gradual increase in the density of ca channels appears to be a consistent property of hippocampal neuronal aging and might well be a factor in the vulnerability of aged neurons to alzheimer's disease and other neurodegenerative/traumatic conditions. |
1996-10-02 |
2023-08-12 |
rat |