| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| G Lallement, M Denoyer, A Collet, I Pernot-Marino, D Baubichon, P Monmaur, G Blanche. Changes in hippocampal acetylcholine and glutamate extracellular levels during soman-induced seizures: influence of septal cholinoceptive cells. Neuroscience letters. vol 139. issue 1. 1992-11-06. PMID:1357600. |
these data strongly suggest that, during soman intoxication, septal cholinoceptive cells play a key role in controlling the release of acetylcholine and glutamate in hippocampus. |
1992-11-06 |
2023-08-11 |
rat |
| J Ułas, L C Brunner, J W Geddes, W Choe, C W Cotma. N-methyl-D-aspartate receptor complex in the hippocampus of elderly, normal individuals and those with Alzheimer's disease. Neuroscience. vol 49. issue 1. 1992-11-06. PMID:1407551. |
the hippocampus and parahippocampal gyrus of the normal elderly brain exhibited virtually identical distributions of l-[3h]glutamate, [3h]5-methyl-10,11-dihydro-5h- dibenzo[a,d]cyclohepten-5,10-iminemaleate ([3h]mk-801), [3h][(+/-)2-carboxypiperazine-4-yl]propyl-1-phosphonic acid ([3h]cpp) and strychnine-insensitive [3h]glycine binding sites (r greater than 0.87) suggesting that binding occurred to different domains of the same receptor macromolecule. |
1992-11-06 |
2023-08-11 |
Not clear |
| D Jerusalinsky, M B Ferreira, R Walz, R C Da Silva, M Bianchin, A C Ruschel, M S Zanatta, J H Medina, I Izquierd. Amnesia by post-training infusion of glutamate receptor antagonists into the amygdala, hippocampus, and entorhinal cortex. Behavioral and neural biology. vol 58. issue 1. 1992-11-04. PMID:1417675. |
amnesia by post-training infusion of glutamate receptor antagonists into the amygdala, hippocampus, and entorhinal cortex. |
1992-11-04 |
2023-08-11 |
rat |
| D Jerusalinsky, M B Ferreira, R Walz, R C Da Silva, M Bianchin, A C Ruschel, M S Zanatta, J H Medina, I Izquierd. Amnesia by post-training infusion of glutamate receptor antagonists into the amygdala, hippocampus, and entorhinal cortex. Behavioral and neural biology. vol 58. issue 1. 1992-11-04. PMID:1417675. |
the blockers of glutamate receptors, aminophosphonovaleric acid (ap5) (5.0 micrograms) and cyano-nitroquinoxaline-dione (cnqx) (0.5 microgram), were infused bilaterally into the amygdala, dorsal hippocampus, or entorhinal cortex of rats through indwelling cannulae 0, 90, 180, or 360 min after step-down inhibitory avoidance training. |
1992-11-04 |
2023-08-11 |
rat |
| M Matsumoto, M H Zornow, M S Scheller, M A Strna. AMPA and NMDA receptor antagonists do not decrease hippocampal glutamate concentrations during transient global ischemia. Anesthesiology. vol 77. issue 4. 1992-11-02. PMID:1358005. |
ampa and nmda receptor antagonists do not decrease hippocampal glutamate concentrations during transient global ischemia. |
1992-11-02 |
2023-08-11 |
Not clear |
| M Matsumoto, M H Zornow, M S Scheller, M A Strna. AMPA and NMDA receptor antagonists do not decrease hippocampal glutamate concentrations during transient global ischemia. Anesthesiology. vol 77. issue 4. 1992-11-02. PMID:1358005. |
hippocampal extracellular concentrations of glutamate, aspartate, and glycine were monitored using in vivo microdialysis. |
1992-11-02 |
2023-08-11 |
Not clear |
| M Matsumoto, M H Zornow, M S Scheller, M A Strna. AMPA and NMDA receptor antagonists do not decrease hippocampal glutamate concentrations during transient global ischemia. Anesthesiology. vol 77. issue 4. 1992-11-02. PMID:1358005. |
these results do not support the existence of a positive feedback loop for glutamate mediated via ampa or nmda autoreceptors in the hippocampus during transient global ischemia or reperfusion. |
1992-11-02 |
2023-08-11 |
Not clear |
| N Nakata, H Kato, Y Liu, K Kogur. Effects of pretreatment with sublethal ischemia on the extracellular glutamate concentrations during secondary ischemia in the gerbil hippocampus evaluated with intracerebral microdialysis. Neuroscience letters. vol 138. issue 1. 1992-10-26. PMID:1357599. |
effects of pretreatment with sublethal ischemia on the extracellular glutamate concentrations during secondary ischemia in the gerbil hippocampus evaluated with intracerebral microdialysis. |
1992-10-26 |
2023-08-11 |
Not clear |
| N Nakata, H Kato, Y Liu, K Kogur. Effects of pretreatment with sublethal ischemia on the extracellular glutamate concentrations during secondary ischemia in the gerbil hippocampus evaluated with intracerebral microdialysis. Neuroscience letters. vol 138. issue 1. 1992-10-26. PMID:1357599. |
to elucidate the role of glutamate in the pathogenesis of altered neuronal vulnerability following sublethal ischemia, we measured the extracellular concentrations of glutamate in the gerbil hippocampus using a microdialysis technique. |
1992-10-26 |
2023-08-11 |
Not clear |
| K Akiyama, Y Yoneda, K Ogita, T Itoh, A Daigen, I Sora, I Kohira, H Ujike, S Otsuk. Ionotropic excitatory amino acid receptors in discrete brain regions of kindled rats. Brain research. vol 587. issue 1. 1992-10-22. PMID:1326375. |
nor was there a significant change in nmda-sensitive [3h]glutamate, strychnine-insensitive [3h]glycine, [3h]spermidine, [3h]kainate or [3h]alpha-amino-3-hydroxy-5-methylisoxazole-4-propionic acid ([3h]ampa) binding in any brain region 7 days after the last amygdala kindled seizure, or in the hippocampus 28 days after the last amygdala kindled seizure. |
1992-10-22 |
2023-08-11 |
rat |
| T S Whittingham, H Assaf, W R Selman, R A Ratcheson, W D Lus. Glutamate-induced energetic stress in hippocampal slices: evidence against NMDA and glutamate uptake as mediators. Metabolic brain disease. vol 7. issue 2. 1992-10-22. PMID:1356225. |
glutamate-induced energetic stress in hippocampal slices: evidence against nmda and glutamate uptake as mediators. |
1992-10-22 |
2023-08-11 |
Not clear |
| T S Whittingham, H Assaf, W R Selman, R A Ratcheson, W D Lus. Glutamate-induced energetic stress in hippocampal slices: evidence against NMDA and glutamate uptake as mediators. Metabolic brain disease. vol 7. issue 2. 1992-10-22. PMID:1356225. |
the introduction of exogenous glutamate to normally respiring hippocampal slices produced substantial reductions in atp, phosphocreatine (pcr) and intracellular ph (phi) when the concentration exceeded 1 mm. |
1992-10-22 |
2023-08-11 |
Not clear |
| M Sega. Acetylcholine enhances NMDA-evoked calcium rise in hippocampal neurons. Brain research. vol 587. issue 1. 1992-10-22. PMID:1525651. |
the effects of acetylcholine (ach) on changes in [ca]i produced by the glutamate agonist n-methyl-d-aspartate (nmda) were measured in cultured rat hippocampal neurons loaded with the fluorescent calcium indicator fluo-3 in a confocal laser scanning microscope. |
1992-10-22 |
2023-08-11 |
rat |
| M Virgili, P Migani, A Contestabile, O Barnabe. Protection from kainic acid neuropathological syndrome by NMDA receptor antagonists: effect of MK-801 and CGP 39551 on neurotransmitter and glial markers. Neuropharmacology. vol 31. issue 5. 1992-10-20. PMID:1356249. |
appropriate doses of both compounds (1 mg/kg mk-801 and 25 mg/kg cgp 39551) completely reversed the decrease of high affinity uptake of glutamate and activity of glutamate decarboxylase in the olfactory cortex, amygdala, hippocampus and lateral septum. |
1992-10-20 |
2023-08-11 |
Not clear |
| Z A Bortolotto, G L Collingridg. Activation of glutamate metabotropic receptors induces long-term potentiation. European journal of pharmacology. vol 214. issue 2-3. 1992-10-08. PMID:1325361. |
the specific glutamate metabotropic receptor agonist 1s,3r-aminocyclopentane dicarboxylate (acpd), but not its inactive enantiomer 1r,3s-acpd, induced a slowly developing potentiation of synaptic transmission in rat hippocampal slices. |
1992-10-08 |
2023-08-11 |
rat |
| G J Lee. Effects of anaesthetics, anticonvulsants and glutamate antagonists on kainic acid-induced local and distal neuronal loss. Journal of the neurological sciences. vol 108. issue 2. 1992-10-08. PMID:1517756. |
a semi-quantitative estimation has been made of the effect of anaesthetics, anticonvulsants and glutamate antagonists on the extent of neuronal loss in the hippocampus caused by the local injection of the excitotoxin kainic acid, and on the vulnerability of neurons in various extrahippocampal regions due to the resulting seizure activity. |
1992-10-08 |
2023-08-11 |
Not clear |
| G J Lee. Effects of anaesthetics, anticonvulsants and glutamate antagonists on kainic acid-induced local and distal neuronal loss. Journal of the neurological sciences. vol 108. issue 2. 1992-10-08. PMID:1517756. |
following the intrahippocampal injection of 0.47 nmol kainic acid (a submaximal dose), the amount of neuronal loss in the dorsal hippocampus was greater when given under the short-acting anaesthetics halothane and ketamine (a non-competitive glutamate antagonist), than when given under pentobarbital anaesthesia (with or without co-administration of ketamine (30 mg/kg)). |
1992-10-08 |
2023-08-11 |
Not clear |
| D H Penning, J Patrick, S Jimmo, J F Brie. Release of glutamate and gamma-aminobutyric acid in the ovine fetal hippocampus: ontogeny and effect of hypoxia. Journal of developmental physiology. vol 16. issue 5. 1992-10-06. PMID:1688088. |
release of glutamate and gamma-aminobutyric acid in the ovine fetal hippocampus: ontogeny and effect of hypoxia. |
1992-10-06 |
2023-08-11 |
Not clear |
| D H Penning, J Patrick, S Jimmo, J F Brie. Release of glutamate and gamma-aminobutyric acid in the ovine fetal hippocampus: ontogeny and effect of hypoxia. Journal of developmental physiology. vol 16. issue 5. 1992-10-06. PMID:1688088. |
the effects of increased potassium ion concentration (50 mm) and hypoxia on the efflux of glutamate and gamma-aminobutyric acid (gaba) were studied in ovine fetal hippocampal slices using the static-pool-interface superfusion method at three selected gestational ages (85 days, 105 days, 135 days; term, about 147 days). |
1992-10-06 |
2023-08-11 |
Not clear |
| D H Penning, J Patrick, S Jimmo, J F Brie. Release of glutamate and gamma-aminobutyric acid in the ovine fetal hippocampus: ontogeny and effect of hypoxia. Journal of developmental physiology. vol 16. issue 5. 1992-10-06. PMID:1688088. |
potassium ion stimulated the efflux of glutamate in the hippocampus of the 85-days-old fetus only, and this efflux of glutamate was not calcium-ion dependent. |
1992-10-06 |
2023-08-11 |
Not clear |