| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| R K Schwarting, J P Husto. Unilateral 6-hydroxydopamine lesions of meso-striatal dopamine neurons and their physiological sequelae. Progress in neurobiology. vol 49. issue 3. 1997-01-16. PMID:8878304. |
with respect to the study of dopamine neurons in the brain, especially within the basal ganglia, the neurotoxin 6-hydroxydopamine (6-ohda) provides an important tool. |
1997-01-16 |
2023-08-12 |
human |
| R K Schwarting, J P Husto. Unilateral 6-hydroxydopamine lesions of meso-striatal dopamine neurons and their physiological sequelae. Progress in neurobiology. vol 49. issue 3. 1997-01-16. PMID:8878304. |
this approach has contributed to neuroscientific knowledge at the basic and clinical levels, since it has been used to clarify the neuroanatomy, neurochemistry, and electrophysiology of mesencephalic dopamine neurons and their relationships with the basal ganglia. |
1997-01-16 |
2023-08-12 |
human |
| M Gerlach, M B Youdim, P Riedere. Pharmacology of selegiline. Neurology. vol 47. issue 6 Suppl 3. 1997-01-15. PMID:8959982. |
although not all features of its anti-pd action are known, studies using brain obtained at autopsy from patients who had been treated with 10 mg of selegiline showed that selective inhibition of mao-b, with the concomitant increase of phenylethylamine and dopamine (da) but not of serotonin or noradrenaline, in the basal ganglia may be regarded as its mode of action. |
1997-01-15 |
2023-08-12 |
Not clear |
| W Timmerman, E D Abercrombi. Amphetamine-induced release of dendritic dopamine in substantia nigra pars reticulata: D1-mediated behavioral and electrophysiological effects. Synapse (New York, N.Y.). vol 23. issue 4. 1997-01-07. PMID:8855513. |
dopamine (da) released from dendrites of substantia nigra dopaminergic neurons potentially is in a position to modulate basal ganglia outputs from the substantia nigra pars reticulata (snr) via stimulation of d1 receptors on the terminals of striatonigral afferents. |
1997-01-07 |
2023-08-12 |
rat |
| D Ebert, H Feistel, T Loew, A Pirne. Dopamine and depression--striatal dopamine D2 receptor SPECT before and after antidepressant therapy. Psychopharmacology. vol 126. issue 1. 1996-12-23. PMID:8853222. |
the changes observed might be the result of increased tonic dopamine release in the basal ganglia, but several other explanations exist. |
1996-12-23 |
2023-08-12 |
Not clear |
| K Kuwabara, S Hitoshi, N Nukina, K Ishii, T Momose, S Kubota, Y Seyama, I Kanazaw. PET analysis of a case of cerebrotendinous xanthomatosis presenting hemiparkinsonism. Journal of the neurological sciences. vol 138. issue 1-2. 1996-12-03. PMID:8791252. |
although mri studies could not detect any abnormality in the basal ganglia or midbrain, pet analysis using [18f]-6-fluoro-l-dopa revealed reduced uptake of dopamine into the putamen, suggesting the impairment of presynaptic dopaminergic neurons. |
1996-12-03 |
2023-08-12 |
Not clear |
| J M Vargo, J F Marshal. Frontal cortex ablation reversibly decreases striatal zif/268 and junB expression: temporal correspondence with sensory neglect and its spontaneous recovery. Synapse (New York, N.Y.). vol 22. issue 4. 1996-12-03. PMID:8867024. |
previous studies indicate that compensatory neural alterations involving dopamine (da) occur following this cortical injury and that recovery from neglect produced by frontal injury is accompanied by normalization of glucose utilization within subcortical structures including the basal ganglia. |
1996-12-03 |
2023-08-12 |
rat |
| W H Mec. Neuropharmacology of timing and time perception. Brain research. Cognitive brain research. vol 3. issue 3-4. 1996-11-27. PMID:8806025. |
for example, the internal clock used to time durations in the seconds-to-minutes range appears linked to dopamine (da) function in the basal ganglia, while temporal memory and attentional mechanisms appear linked to acetylcholine (ach) function in the frontal cortex. |
1996-11-27 |
2023-08-12 |
human |
| E J Karle, K D Anderson, L Medina, A Reine. Light and electron microscopic immunohistochemical study of dopaminergic terminals in the striatal portion of the pigeon basal ganglia using antisera against tyrosine hydroxylase and dopamine. The Journal of comparative neurology. vol 369. issue 1. 1996-11-07. PMID:8723706. |
light and electron microscopic immunohistochemical study of dopaminergic terminals in the striatal portion of the pigeon basal ganglia using antisera against tyrosine hydroxylase and dopamine. |
1996-11-07 |
2023-08-12 |
pigeon |
| E J Karle, K D Anderson, L Medina, A Reine. Light and electron microscopic immunohistochemical study of dopaminergic terminals in the striatal portion of the pigeon basal ganglia using antisera against tyrosine hydroxylase and dopamine. The Journal of comparative neurology. vol 369. issue 1. 1996-11-07. PMID:8723706. |
in the present study, we used immunohistochemical labeling with antibodies against tyrosine hydroxylase (th) or dopamine (da) to study the dopaminergic input to the striatal portion of the basal ganglia in pigeons (i.e., lobus parolfactorius and paleostriatum augmentatum). |
1996-11-07 |
2023-08-12 |
pigeon |
| L I Nefedov, E M Doroshenko, V Iu Smirnov, G N Zav'ialova, I I Klimovic. [Taurine induces an imbalance in the pool of neuroactive amino acids and biogenic amines in regions of the brain]. Ukrainskii biokhimicheskii zhurnal (1978). vol 68. issue 1. 1996-11-06. PMID:8755096. |
the tau load was followed by an elevation of the concentrations of glutamate, aspartate and their amides, gaba and tyrosine concentrations in cerebellum whereas in basal ganglia that of tyrosine and dopamine. |
1996-11-06 |
2023-08-12 |
rat |
| R Sandy. Freezing of gait in Parkinson's disease is improved by treatment with weak electromagnetic fields. The International journal of neuroscience. vol 85. issue 1-2. 1996-10-24. PMID:8727687. |
the pathophysiology of parkinsonian freezing, which is considered a distinct clinical feature independent of akinesia, is poorly understood and is believed to involve abnormalities in dopamine and norepinephrine neurotransmission in critical motor control areas including the frontal lobe, basal ganglia, locus coeruleus and spinal cord. |
1996-10-24 |
2023-08-12 |
Not clear |
| D M Gash, Z Zhang, W A Cass, A Ovadia, L Simmerman, D Martin, D Russell, F Collins, B J Hoffer, G A Gerhard. Morphological and functional effects of intranigrally administered GDNF in normal rhesus monkeys. The Journal of comparative neurology. vol 363. issue 3. 1996-10-24. PMID:8847404. |
the studies included evaluating whole animal behavior, electrochemical recordings of striatal dopamine release, neurochemical determinations of basal ganglia and nigral monoamine levels, and immunohistochemical staining of the nigrostriatal dopamine system. |
1996-10-24 |
2023-08-12 |
human |
| I Bodis-Wollner, A Anta. On the functional significance of primate retinal dopamine receptors. Journal of neural transmission. Supplementum. vol 45. 1996-10-23. PMID:8748611. |
an understanding of the logic performed by retinal d1 and d2 receptors may be useful to discern properties of the role of diverse dopamine receptors in basal ganglia da circuits. |
1996-10-23 |
2023-08-12 |
human |
| b' T D Sotnikova, R R Ga\\xc4\\xadnetdinov, T V Grekhova, K S Raevski\\xc4\\xa. [Dopamine autoreceptors subtype D3 predominantly regulate the liberation of dopamine in rat brain basal ganglia]. Biulleten\' eksperimental\'noi biologii i meditsiny. vol 121. issue 4. 1996-10-16. PMID:8829459.' |
[dopamine autoreceptors subtype d3 predominantly regulate the liberation of dopamine in rat brain basal ganglia]. |
1996-10-16 |
2023-08-12 |
rat |
| F Blandini, R H Porter, J T Greenamyr. Glutamate and Parkinson's disease. Molecular neurobiology. vol 12. issue 1. 1996-10-04. PMID:8732541. |
degeneration of dopamine nigral neurons is followed by striatal dopaminergic denervation, which causes a cascade of functional modifications in the activity of basal ganglia nuclei. |
1996-10-04 |
2023-08-12 |
Not clear |
| A Moser, J Scholz, H Bamberg, V Böhm. The effect of N-methyl-norsalsolinol on monoamine oxidase of the rat caudate nucleus in vitro. Neurochemistry international. vol 28. issue 1. 1996-10-03. PMID:8746770. |
these results suggest that 2-mdtiq, the naturally occurring compound found in parkinsonian brain and cerebrospinal fluid, perturbs dopamine metabolism in basal ganglia. |
1996-10-03 |
2023-08-12 |
rat |
| A J Cooper, I J Mitchel. Fos immuno-positive neurons in the subthalamic nucleus following reversal of parkinsonian symptoms by antagonism of excitatory amino acid transmission in the entopeduncular nucleus of the monoamine depleted rat. Neuroscience letters. vol 201. issue 3. 1996-09-26. PMID:8786852. |
a series of experiments was conducted in order to determine whether this manipulation affected the pattern of fos immunoreactivity in the basal ganglia elicited by dopamine depletion. |
1996-09-26 |
2023-08-12 |
rat |
| R Dayne Mayfield, G Larson, R A Orona, N R Zahnise. Opposing actions of adenosine A2a and dopamine D2 receptor activation on GABA release in the basal ganglia: evidence for an A2a/D2 receptor interaction in globus pallidus. Synapse (New York, N.Y.). vol 22. issue 2. 1996-09-26. PMID:8787129. |
opposing actions of adenosine a2a and dopamine d2 receptor activation on gaba release in the basal ganglia: evidence for an a2a/d2 receptor interaction in globus pallidus. |
1996-09-26 |
2023-08-12 |
rat |
| R Dayne Mayfield, G Larson, R A Orona, N R Zahnise. Opposing actions of adenosine A2a and dopamine D2 receptor activation on GABA release in the basal ganglia: evidence for an A2a/D2 receptor interaction in globus pallidus. Synapse (New York, N.Y.). vol 22. issue 2. 1996-09-26. PMID:8787129. |
there is increasing evidence that adenosine (ado) and dopamine (da) interact directly in the basal ganglia via actions at ado a2a and da d2 receptors, respectively. |
1996-09-26 |
2023-08-12 |
rat |