All Relations between hypertrophic and matrix compartment

Publication Sentence Publish Date Extraction Date Species
V Pelouch, I M Dixon, L Golfman, R E Beamish, N S Dhall. Role of extracellular matrix proteins in heart function. Molecular and cellular biochemistry. vol 129. issue 2. 1994-06-07. PMID:8177233. recent work provides evidence for the role of the renin-angiotensin-aldosterone system in the pathogenesis of abnormal deposition of extracellular matrix in the cardiac interstitium during the development of inappropriate cardiac hypertrophy and failure. 1994-06-07 2023-08-12 Not clear
R J Wardale, V C Duanc. Characterisation of articular and growth plate cartilage collagens in porcine osteochondrosis. Journal of cell science. vol 107 ( Pt 1). 1994-06-06. PMID:8175922. type x collagen was produced in the matrix of the calcifying region of osteochondrotic articular cartilage by small groups of hypertrophic chondrocytes, but was not detected in normal articular cartilage. 1994-06-06 2023-08-12 Not clear
S Moskalewski, A Hyc, T Grzela, J Malejczy. Differences in cartilage formed intramuscularly or in joint surface defects by syngeneic rat chondrocytes isolated from the articular-epiphyseal cartilage complex. Cell transplantation. vol 2. issue 6. 1994-05-27. PMID:8167932. in cartilage nodules produced intramuscularly, chondrocyte hypertrophy and matrix calcification were observed after 2 wk. 1994-05-27 2023-08-12 rat
S Moskalewski, A Hyc, T Grzela, J Malejczy. Differences in cartilage formed intramuscularly or in joint surface defects by syngeneic rat chondrocytes isolated from the articular-epiphyseal cartilage complex. Cell transplantation. vol 2. issue 6. 1994-05-27. PMID:8167932. thus, either intraarticular environment exerted an inhibitory effect on chondrocyte hypertrophy and matrix calcification or articular chondrocytes present among transplanted cells accumulated close to the joint lumen and reconstructed normal articular cartilage. 1994-05-27 2023-08-12 rat
S Katayama, M Abe, M Inaba, A Itabashi, J Ishi. Effects of captopril or nitrendipine on left ventricular collagen or laminin B2 gene expression. Japanese heart journal. vol 34. issue 6. 1994-05-20. PMID:8164345. these results suggest that angiotensin converting enzyme inhibitors prevent and/or cause regression of cardiac hypertrophy by inhibiting the gene expression of extracellular matrix, and that angiotensin ii may have a pivotal role in cardiac hypertrophy. 1994-05-20 2023-08-12 rat
M E Safar, B I Lév. Mechanical function and histological structure of the arterial wall. The response to antihypertensive treatment. Drugs. vol 46 Suppl 2. 1994-05-19. PMID:7512485. firstly, during arterial hypertension, increases in arterial stiffness, associated with medial hypertrophy and/or hyperplasia and alterations in extracellular matrix, are always observed. 1994-05-19 2023-08-12 rat
T Kirsch, R E Wuthie. Stimulation of calcification of growth plate cartilage matrix vesicles by binding to type II and X collagens. The Journal of biological chemistry. vol 269. issue 15. 1994-05-19. PMID:8157677. matrix vesicles (mv), microstructures which rapidly accumulate ca2+ and induce mineral formation in vitro, are linked to type ii and x collagens and proteoglycans in the hypertrophic cartilage. 1994-05-19 2023-08-12 chicken
C W Kische. Contributions of electron microscopy to the study of the hypertrophic scar and related lesions. Scanning microscopy. vol 7. issue 3. 1994-04-29. PMID:8146619. the first sem studies demonstrated a homogeneous, dense dermal matrix which supported the conclusion that the hypertrophic scar and keloid reflected excess collagen. 1994-04-29 2023-08-12 Not clear
E B Hunziker, J Wagner, J Zap. Differential effects of insulin-like growth factor I and growth hormone on developmental stages of rat growth plate chondrocytes in vivo. The Journal of clinical investigation. vol 93. issue 3. 1994-04-21. PMID:8132746. proliferating cell cycle time decreased from 11 to 4.5 and 3 d, and duration of the hypertrophic phase decreased from 6 to 4 and 2.8 d. average matrix volume per cell at each differentiation stage was similar for normal, hormone-treated, and untreated hypophysectomized groups. 1994-04-21 2023-08-12 rat
B Zimmerman. Occurrence of osteoblast necroses during ossification of long bone cortices in mouse fetuses. Cell and tissue research. vol 275. issue 2. 1994-03-28. PMID:8111841. initially, cell necroses in the perichondrium occurred in the region surrounding the hypertrophic cartilage and the matrix of which showed spots of endochondral mineralization. 1994-03-28 2023-08-12 mouse
C Gentili, R Doliana, P Bet, G Campanile, A Colombatti, F D Cancedda, R Cancedd. Ovotransferrin and ovotransferrin receptor expression during chondrogenesis and endochondral bone formation in developing chick embryo. The Journal of cell biology. vol 124. issue 4. 1994-03-24. PMID:8106555. ovotransferrin was expressed by in vitro differentiating chondrocytes in the early phase of the culture and, at least when culture conditions allowed extracellular matrix assembly, also by hypertrophic chondrocytes and derived osteoblast-like cells. 1994-03-24 2023-08-12 chicken
F J Pardo Mindán, A Paniz. Alterations in the extracellular matrix of the myocardium in essential hypertension. European heart journal. vol 14 Suppl J. 1994-02-14. PMID:8281955. hypertension produces myocyte hypertrophy and increases the extracellular matrix. 1994-02-14 2023-08-12 Not clear
J Cosín Aguilar, A Hernandiz Martinez, F Andres Conejo. Mechanisms of ventricular arrhythmias in the presence of pathological hypertrophy. European heart journal. vol 14 Suppl J. 1994-02-14. PMID:8281966. hypertension, electrical changes in the hypertrophic myocyte, the growth of the collagen matrix and ischaemia (even in the absence of macro-angiographic injuries) are factors present in this group of patients that contribute to the appearance of arrhythmias. 1994-02-14 2023-08-12 Not clear
M E Choi, E G Kim, Q Huang, B J Ballerman. Rat mesangial cell hypertrophy in response to transforming growth factor-beta 1. Kidney international. vol 44. issue 5. 1994-01-26. PMID:8264154. central features of progressive glomerular sclerosis are initial glomerular hypertrophy and subsequent accumulation of extracellular matrix proteins. 1994-01-26 2023-08-12 human
M E Choi, E G Kim, Q Huang, B J Ballerman. Rat mesangial cell hypertrophy in response to transforming growth factor-beta 1. Kidney international. vol 44. issue 5. 1994-01-26. PMID:8264154. these findings demonstrate that rat mesangial cells express key components of the tgf-beta system and raise the intriguing possibility that in the glomerular mesangium, tgf-beta 1 may not only induce extracellular matrix synthesis, but may also participate in the process of glomerular hypertrophy in response to injury. 1994-01-26 2023-08-12 human
F J Villarreal, N N Kim, G D Ungab, M P Printz, W H Dillman. Identification of functional angiotensin II receptors on rat cardiac fibroblasts. Circulation. vol 88. issue 6. 1994-01-13. PMID:8252698. cardiac hypertrophy results in an increased deposition of the extracellular matrix (ecm) proteins fibronectin and collagen. 1994-01-13 2023-08-12 rat
W L Garner, S Karmiol, J L Rodriguez, D J Smith, S H Pha. Phenotypic differences in cytokine responsiveness of hypertrophic scar versus normal dermal fibroblasts. The Journal of investigative dermatology. vol 101. issue 6. 1994-01-06. PMID:8245516. the increased rate of collagen synthesis and decreased responsiveness to mitogens are consistent with the increased extracellular matrix content and decreased cellularity of hypertrophic scars. 1994-01-06 2023-08-12 cattle
R J Wardale, V C Duanc. Quantification and immunolocalisation of porcine articular and growth plate cartilage collagens. Journal of cell science. vol 105 ( Pt 4). 1993-12-22. PMID:8227218. types ii and xi are located in the matrix of both cartilages whereas type ix is predominantly pericellular in the calcifying region of articular cartilage and the hypertrophic region of the growth plate. 1993-12-22 2023-08-12 Not clear
M Rintala, M Metsäranta, S Garofalo, B de Crombrugghe, E Vuorio, O Rönnin. Abnormal craniofacial morphology and cartilage structure in transgenic mice harboring a Gly --> Cys mutation in the cartilage-specific type II collagen gene. Journal of craniofacial genetics and developmental biology. vol 13. issue 3. 1993-12-17. PMID:8227287. with the exception of the zone of hypertrophic chondrocytes the matrix also showed a loss of glycosaminoglycans. 1993-12-17 2023-08-12 mouse
F N Ziyade. The extracellular matrix in diabetic nephropathy. American journal of kidney diseases : the official journal of the National Kidney Foundation. vol 22. issue 5. 1993-12-08. PMID:8238022. diabetic nephropathy is characterized by hypertrophy of both glomerular and tubular elements, progressive accumulation of extracellular matrix components in the glomerular mesangium, and thickening of the glomerular and tubular basement membranes. 1993-12-08 2023-08-12 Not clear